23. Deep venous thrombosis Flashcards Preview

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Flashcards in 23. Deep venous thrombosis Deck (13):
1

Deep venous thrombosis definition

blood clot within deep vein

2

deep venous thrombosis sx

1.swelling
2.redness
3.warmth
4. pain

simia flegmonis

3

deep venous thrombosis is predisposed by

Virhow triad
1. stasis
2. hypercoagulability
3.endothelial damage

4

Homan sign

DVT
dorsiflexion of foot --> calf pain

5

prophylaxis and acute management of DVT

unfractionated heparin OR low molecular weight heparin

6

treatment (long time prevention ) of DVT

oral anticoagulants (e.g warfarin, rivaroxaban)

7

• Which three factors that promote blood coagulation are known as the Virchow triad?






Stasis, Hypercoagulability, and Endothelial damage (SHE)

8

• A patient diagnosed with factor V Leiden is at risk of developing deep venous thrombosis. Which element of the Virchow triad is affected?

Hypercoagulability

9

• How does endothelial damage lead to the formation of a clot?

Exposed collagen triggers the clotting cascade

10

• A patient presents with dyspnea and tachycardia. Chest CT shows a pulmonary embolus. From where did the embolus most likely originate?

The deep veins of the leg

11

• Describe the Homan sign.

In the Homan sign, dorsiflexion of the foot results in a tender calf muscle because of the presence of a deep venous thrombosis

12

• A patient presents with a red, swollen leg. Venous Doppler confirms a deep venous thrombosis. Acutely, what is your treatment of choice?

Unfractionated heparin or heparins of low molecular weight

13

• A patient with recurrent deep venous thromboses requires long-term anticoagulation. Which medications may be prescribed?

Oral anticoagulants (e.g., warfarin, rivaroxaban)