Arrhythmia 1 Flashcards

1
Q

What does supraventricular mean and include?

A

Non-specific term as it means origin is above the ventricle

ie. AV node, SA node, atrial muscle. Slide 4

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2
Q

What are the possible supraventricular arrhythmias?

A
Atrial Fibrillation
Atrial Flutter
Ectopic Atrial Tachycardia
Sinus bradycardia
Sinus pauses. 
Slide 5
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3
Q

What are the possible ventricular arrhythmias?

A
Ventricular ectopics or premature ventricular complexes (PVC)
Ventricular tachycardia
Ventricular Fibrillation
Asystole.
Slide 6
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4
Q

What are possible arrhythmias that could arise in the AV node?

A

AVN re-entry tachycardia
AV reciprocating tachycardia
AV block from 1st to 3rd degree. Slide 7

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5
Q

What are the anatomical and autonomic nervous system causes for arrhythmias?

A

Anatomy:
LV hypertrophy
Accessory pathways
Congenital HD

Nervous:
Sympathetic stimulation e.g. stress
Increased vagal tone. Slide 9

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6
Q

Apart from anatomical and nervous causes, what else could cause arrhythmias?

A
Metabolic e.g. hypoxia
Inflammation e.g. myocarditis
Drugs
Genetics e.g long QT syndrome
Slide 9
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7
Q

What are the electrophysiological mechanisms of an arrhythmia?

A

Ectopic beats:
Altered automaticity
Triggered Activity

Re-entry:
Accessory pathway tachycardia.
Slide 10

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8
Q

What are the 4 ways to alter automaticity?

A

Change the slope to threshold
Change the threshold itself
Change resting memebrane potential
PNS can afftect phase 4 slope. Slide 13

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9
Q

Why does hypokalaemia cause an increase in heart rate?

A

Due to the increase in phase 4 slope and prolongs repolarisation. Slide 14

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10
Q

What is triggered activity and what are the potential causes?

A

In phase 3 a small depolarisation may occur whcih if sufficient then may triger a sustained train of depolarisations which is triggered activity.
Digoxin toxicity
Long QT syndrome and hypokalaemia
Slide 15

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11
Q

How does Re-entry of a potential cause an arrhythmia and what are some structural causes of re-entry?

A

There is a second conduction pathway and causes another potential to split off from the main potential and become a recurrent potential circling back to the atrium.
Structural: Accessory pathways, scars from previous MI and congenital HD. Slide 17+21

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12
Q

What are the symptoms of an arrhythmia?

A
Palpitations
SOB
Dizziness
Syncope
Faintness
Sudden cardiac death
Angina
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13
Q

What are the investigations of arrhythmias?

A
12 lead ECG
CXR
Echocardiogram
Stress ECG for exercise related arrhythmias
24hr ECG
Slide 26
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14
Q

On an ECG what indicates pre-excitation?

A

The slope up to the QRS called a delta wave and a short PR interval. Slide 28+44

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15
Q

Sinus arrhythmia is bad. True or False?

A

False, sinus arrhythmia is the only normal arrythmia where during inspiration and expiration the heart can speed up or slow down. Slide 35

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16
Q

What is the treatment for sinus bradycardia?

A

Atropine

Slide 36

17
Q

What does orthodromic and antidromic AV reciprocating tachycardia present as on a ECG?

A

Orthodomic AVRT:
Inverted P wave after QRS
Antidromic AVRT:
Wide QRS complex with delta wave. Slide 44

18
Q

What is the acute and chronic management of supraventricular tachycardia management?

A
Acute:
Slow AV node conduction
-IV adenosine/verapamil
Increase vagal tone:
-Carotid massage
Chronic:
Avoid stimulants
Radiofrequency ablation in young patients
B blockers
Anti-arrhythmic drugs. Slide 46
19
Q

What is radiofrequency catheter ablation?

A

Selective cautery of cardiac tissue to prevent tachycaria. Slide 47

20
Q

What are possible causes of heart block?

A
Ageing
Acute MI
Myocarditis
Amyloid
Drugs e.g. B blockers, Ca antagonists
Aortic valve disease
Genetic e.g. Lenegre's disease. Slide 51
21
Q

What is 1st degree AV block and what is the treatment?

A

When the conduction from P wave to QRS is longer.

There is no treatment just monitor incase it gets worse. Slide 52

22
Q

What are the two types of 2nd degree AV block?

A

Mobitz I: progressive lengthening of the PR interval and results in a dropped beat.
Mobitz II: Pathological and is usually in a 2:1 P wave to QRS.
Slide 53

23
Q

What is 3rd degree heart block?

A

When there is no action potential from the SA node.

Instead there is only rhythm coming spontaneously from the ventricular myocytes. Slide 57

24
Q

If the QRS complex is narrow it is ____, if the QRS complex is broad it is ____.

A

Atrial - narrow
Ventricular - broad
Slide 57

25
What are the types of pacemakers?
Single chamber or dual chamber. Slide 59
26
What are the possible causes for ventricular ectopic beats?
Structural: LVH, myocarditis Metabolic: Ischaemic heart disease Slide 61
27
If there is a man who is acute SOB at rest and chest pain 70 male, hypertension, diabetes, Smoker. Had a remote MI six months ago. What is the most common diagnosis?
Ventricular tachycardia until proven otherwise. Slide 63
28
What is ventricular tachycardia?
When patients have significant heart disease and is life threatening but is harmodynamically stable. Slide 64
29
Why do some patients feel light headed or syncope due to ventricular tachycardia?
As there is a large sustained reduction of arterial pressure. Slide 65
30
What are the differences between monomorphic VT and polymorphic and what does polymorphic VT have monomorphic doesn't?
Monomorphic is haemodynamically stable as the VT rhythm is regular Polymorphic is haemodynamically unstable and has an irregular VT rhythm. Polymorphic has signs of torsades de pointes. Slide 66+67
31
What is Ventricular fibrillation?
Chaotic ventricular electrical activity which causes the heart ot lose the ability to function as a pump. Slide 68
32
What is the acute and long term treatment of VT?
Acute Unstable: Direct current cardioversion Stable: Pharmacologic cardioversion. ``` Chronic Correct ischaemia Anti-arryhtmic drugs Implantable cardiovertor defibs. VT catheter ablation. Slide 69+70 ```
33
What does an ICD do?
Termination the occurance of a VT or VF by defibirillation. Slide 72