Cerebral Palsy Flashcards Preview

Conditions neuro > Cerebral Palsy > Flashcards

Flashcards in Cerebral Palsy Deck (18):
1

What is CP?

static lesion in the immature brain that leaves children with a permanent motor impairment

2

How does a lesion occur to cause CP?

developmental defect, MCA occlusion causing an infarction, trauma during or after delivery

3

What is the etiology breakdown for CP?

Prenatal (genetic syndroms, teratogenic (alcohol), viral infection, twin pregnancies= 40%

Perinatal (pre-exclampsia, birth asphyxia, cerebral artery infarction)= 55%

Postnatal (hypoxic ischemic injury, anoxia, cerebral traumatic injury, meningitis)= 5%

4

What are associated disorders with CP?

seizures
cognitive/behavioral challenges
persistence of primitive reflexes
feeding disorders

5

what are the topographical classifications of CP

monoplegic
hemiplegic (all 1 side)
diplegic (both UE and LE)
triplegic (both feet and 1 UE)
quadriplegic

6

What are the two physiologic classifications of CP?

pyramidal or spastic type

7

What is spasticity?

disorder of muscle tone characterized by an initial increased resistance to stretch (75% of CP cases)

8

Extrapyramidal CP is damage to what brain structures and causes what

damage to the basal ganglia or cerebellum which results in movement disorders

9

Athetoid CP effects what part of the brain

extrapyramidal CP
involuntary movements
intelligence normal

10

Atonic CP

extrapyramidal CP
damage to motor area of cerebral cortex
flaccid muscle tone, "floppy"
no DTRs
clumsy/uncoordinated
child will over or under-reach for objects (dysmetria)
gait is ataxic and wide-based

11

spastic hemiplegia

involvement of one side of body
arm more affected than leg
definite hand preference in children less than 12 months
sitting and crawling at normal age, but walking is delayed

12

spastic diplegia

lower limbs more involved than upper limbs
DTRs exaggerated
shuffling gait, w/flexion/adduction of hips and flexion of knees
growth of lower limbs suffers, but upper limbs grow normally.
can use a WALKER

13

spastic quadriplegia

severely disabled
microcephaly
severe mental retardation
epilepsy
visual and hearing deficits
hip subluxation
hypertonicity leads to arching of the back
scissoring of legs
arms internally rotated

14

athetosis

purposeless movements which are uncontrollable. These movements may be slow or fast, writhing, jerky, swiping, tremor, or rotary patterns

fluctuates with emotional state. fatigue decreases athetosis

15

Patterns that show increase in tone

arm adducted
shoulder internal rotation
elbow flexed
forearm pronated
wrist flexed
thumb adducted

16

Medical Management of CP

Baclofen: muscle relaxer and antispastic agent

Botox: reduces spasticity by blocking neurmuscular transmissions by binding to receptors sites and inhibits release of acetycholine.

CITM: forced use of involved extremity by using a splint/cast and doing repetitive adaptive tasks with involved extremity

17

What is the widely used intervention for CP?

Neurodevelopmental Treatment (NDT): key points are the places of physical contact b/w therapist's parts of the body/equipment and client's body

18

Athetoid effects on movement and intelligence

Causes involuntary movements
And usually intelligence is normal