HTN: Exam 1 Flashcards

(37 cards)

1
Q

The “idea” BP

A

119/79

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2
Q

CPG NEW BP Guidelines

A

see pics

**CAN BE ONE OR THE OTHER

SBP OR DBP

doesn’t need to be both #s to be in that category!!!

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3
Q

NORMAL BP

A
  • SBP
    • ​<120
  • DBP
    • ​<80
  • Ideal== 119/79
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4
Q

ELEVATED BP

used to be “Pre-HTN”

A

SBP== 120-129

OR

DBP== <80

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5
Q

Hypertension

Stage 1

A

SBP== 130-139

OR

DBP== 80-89

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6
Q

HTN

Stage 2

A

SBP== >/= 140

OR

DBP== >/= 90

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7
Q

NOTE:

** indiv’s w/ SBP and DBP in 2 categories should be designated to _______

A

HIGHER BP category

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8
Q

BP======

controlled by what 2 mechs?

A

BP= CO(HR*SV) * TPR

controlled by:

1. ANS

2. Hormonal system (Renin-angio system)

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9
Q

Types of BP

Primary

essential or idiopathic

A

from Risk factors–90% of people

ex. sedentary, stress, genetics

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10
Q

Types of BP

Secondary

A

caused by SOMETHING ELSE IN SYSTEM

ex. kidney dis.,

hard to treat

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11
Q

Types of BP:

Labile

A

fluctuating BP

hormonal affects

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12
Q

Types of BP

Anxiety induced HTN

“white coat HTN

A

exactly what it sounds like

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13
Q

HTN:

The Sequela

explain

A
  • Initially
    • INC SNS stim from risk factors cause:
      • periph vasoconstrict
      • inc blood plasma vol
      • sustained + inotrope and/or + chronotropic effect
    • High Na+ diet==> inc plasma volume
    • lack of ex, inact, sedent,
      • decondition heart==> non-optimal SV w/ reflexive + chrono effect
    • obesity ==> inc press in abd area/LE/UE
      • inc’s TPR
    • sleep deprivation
      • activates SNS
          • chronotropic effect==INC HR/BP

**ALL of this produces overload in L. VENT, aorta, and periph arteries=====> EARLY HTN

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14
Q

Sustained HTN

DECd compliance/distensibility of aa’s:

elastic tissue of aa’s and arterioles are replaced by fibrous collagen tissue =====

A

no recoil

prolonged or irreversible HTN

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15
Q

sustained HTN

diminished SV is detected by KIDNEYS—->

activates RAS causing vasoconstriction and Na+/H2O retention=====

A

prolonged or irrev. HTN

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16
Q

Sustained HTN

DEC compliance of CA’s leads to DEC O2 supply to myocardium ======

A

prolonged or irrev. HTN

17
Q

sustained HTN

L. vent working against elevated HTN for prolonged pds will become HYPERTROPHIED or rigid or less kinetic

this cannot be reduced =====

A

prolonged or irrev. HTN

18
Q

Sx’s HTN

early Tx the better!!!!

A
  • asymptomatic
    • or just used to sx’s
  • HA
  • vertigo
  • flushed face
  • epistaxis
    • nausea, dry heave
  • blurred vision
  • nocturnal urinary freq
    • Nocturia
  • dyspnea
  • angina
19
Q

Basic med. tx HTN

Goal (3)

A
  • normalize BP BOTH @ rest and BP resp during exertion
  • PREVENT prolonged HTN patho
    • LVH, art stenosis, CVA
  • IMPROVE CA O2 supply

*LIFE-STYLE CHANGES!!!

20
Q

MED TX HTN

meds

A

beta blocks

alpha blocks

ACE inhibitors

Ca+ channel blocks

Diuretics

21
Q

HTN Tx

Category + Tx

A
  • Pre-HTN
    • lifestyle mod
  • stage 1 HTN
    • diuretics
    • maybe ACE inhibitors
  • Stage 2 HTN
    • combo of 2+ meds or cocktail
      • ALWAYS INCLUDES BETA BLOCKER
        • **blunted HR resp***
          • ​use borg!!!
22
Q

Systemic effects HTN

Brain

A

cerebral aneurysm

Hemorrhagic CVA

23
Q

Systemic effects HTN

Eyes

A

Retinopathy—-arterioloar damage

24
Q

Systemic effects HTN

A

Atherosclerosis

25
Systemic effects HTN **Heart**
CHF atherosclerosis angina MI
26
systemic effects HTN ## Footnote **Kidneys**
Nephrosclerosis (hardened tubules) CRF
27
systemic effects HTN BP
persistent elevation
28
Prognosis of HTN
HTN===sig. risk factor for dev. of **CAD, stroke, CHF and renal failure** ## Footnote **precedes heart failure in 90% of all cases and INCing in all other assoc'd cond's**
29
Exercise is like a DRUG for HTN
reduces systolic AND diastolic BP @ rest ## Footnote **DOSE dependent (more==better)** **may be able to reverse early HTN** **may be able to AVOID, DISCONT, DEC dosage of med mgmt in prolonged HTN**
30
High % of pts referred to PT will have \_\_\_\_\_\_
dx or undx'd HTN * **Monitor during tx sessions:** * **​**any pt over 45 * any pt w/ risk factors or **Dx** * **​CVA, DM, CAD, aortic aneurysm, PAD, obesity, CRF, alcoholism, chronic pain syndrome**
31
3 categories to follow when to take vitals
* Healthy indiv's * during eval * pts w/ risk factors * EVERY session pre/post aerobic * Pts w/ Dx HTN * keep cuff on * **monitor constantly!!!**
32
Exercise Capacity is pts w/ **prolonged HTN is REDUCED 15-30%** ## Footnote **why?**
* Sub-opt SV * SV inc's faster w/ ex * HIGHER RHR * LOWER peak HR * diminished CO **Prescribe accordingly!!!!**
33
Indiv's medicated for HTN may have altered ___________ **if taking Beta blocker**
altered resp to EX **Do a warmup\*\*\*** **use RPE\*\*\***
34
ASCM Guidelines: **Ex testing is CONTRAINDICATED IF:**
BP \>200/110
35
ASCM GUIDELINES: ## Footnote **Discontinue Ex testing if:**
BP \>220/110 **in indivs w/ known CV dx** BP \>250/115 in **healthy indivs** **Anytime there is a DROP in SBP** **Anytime there is an ELEVATION or DROP in DBP \>/= 10**
36
Pulm AA HTN== **MAP \> 25** 3 types
1. Idiopathic Pulm HTN 2. Secondary Pulm HTN 3. Chronic thromboembolic pulm HTN (**CTEPH) ---\> dev clots over time==HTN**
37