IV - Hemodynamic Disorders, Thrombosis and Shock Flashcards Preview

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Flashcards in IV - Hemodynamic Disorders, Thrombosis and Shock Deck (153)
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61

Fate of a thrombus wherein it may induce inflammation and fibrosis and establish some degree of blood flow.

Organization and recanalization(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.97

62

True or false:Therapeutic administration of fibrinolytic agents is generally effective only within a few hours of thrombus formation.

True(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.97

63

Most common site of venous thrombosis.

Superficial or deep veins of the leg(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.97

64

Most common sequelae of deep venous thrombosis.

Pulmonary embolism(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.97

65

Tumor-associated procoagulant release largely responsible for the increased risk of thromboembolic phenomena seen in disseminated cancers.

Migrating thrombophlebitis or Trousseau's syndrome(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.98

66

Hardening or thickening of the arteries as a result of the accumulation of fatty materials, macrophages, platelets and other inflammatory mediators.

Atherosclerosis(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.98

67

Fates of a thrombus (4)

PropagationResolution/DissolutionOrganization and recanalizationEmbolization(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.98

68

Embolus occluding a bifurcation in the pulmonary tree.

Saddle embolus(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.99

69

True or false:A patient who has had one pulmonary embolus has a decreased risk of developing another embolus.

False.The patient is at risk of developing more pulmonary emboli.(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.99

70

A venous embolus which entered the systemic circulation through an interarterial or interventricular defect.

Paradoxical embolus(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.99

71

Most common symptom of pulmonary embolism.

None/ Asymptomatic (60-80%)(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.99

72

Right Ventricular failure secondary to pulmonary hypertension.

Cor pulmonale(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.99

73

Emboli in the arterial circulation.

Systemic thromboembolism(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.99

74

Most common origin of systemic thrombi.

Intracardiac mural thrombi (80%)(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.99

75

Major site of arteriolar embolization.

Lower extremities (75%)Brain (10%)(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.99

76

Microscopic fat globules found in the circulation after fractures of long bones or after soft-tissue trauma.

Fat embolism(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.99

77

Symptoms of pulmonary insufficiency, neurologic symptoms, anemia, and thrombocytopenia characterize what syndrome?

Fat embolism syndrome(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.99

78

Gas bubbles within the circulation obstructing vascular flow and causes distal ischemic injury.

Air embolism(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.99

79

Amount of air in the circulation which produces clinical effects of air embolism.

>100 mL(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.99

80

This occurs when individuals are exposed to sudden changes in atmospheric pressure (e.g. Deep sea divers, scuba divers).

Decompression sickness(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100

81

The rapid formation of gas bubbles within skeletal muscles and supporting tissues in and around joints causing pain.

Bends(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100

82

Gas bubbles in the lung vasculture causing edema, hemorrhages, focal atelectasis and emphysema.

Chokes(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100

83

More chronic form of decompression sickness where persistence of gas emboli in the bones leads to multiple foci of ischemic necrosis.

Caisson disease(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100

84

Treatment of choice for decompression sickness.

Hyperbaric compression chamber(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100

85

Underlying cause of amniotic fluid embolism.

Entry of amniotic fluid into the maternal circulation through a tear in the placetal membranes and rupture of uterine veins.(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100

86

Presence of marked pulmonary edema, diffuse alveolar damage, and presence of squamous cells in the pulmonary circulation shed from fetal skin, lanugo hair, fat and mucin.

Amniotic fluid embolism(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100

87

White or red infarct?Venous occlusion

Red infarct(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100

88

White or red infarct?Lung infarction

Red infarct(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100

89

White or red infarct?Intestinal infarct

Red infarct(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100

90

White or red infarct?Myocardial infarction

White infarct(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100