IV - Hemodynamic Disorders, Thrombosis and Shock Flashcards Preview

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Flashcards in IV - Hemodynamic Disorders, Thrombosis and Shock Deck (153)
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91

White or red infarction?Splenic infact

White infarct(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100

92

White or red infarction?Wedge infarct

White infarct(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.100

93

The dominant histologic characteristic of infarction.

Ischemic coagulative necrosis(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.101

94

Histologic characteristic of brain infarcts.

Liquefactive necrosis(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.101

95

This occurs when bacterial vegetations from a heart valve embolize or when microbes seed an area of necrotic tissue.

Septic infarct(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.101

96

Most common sequalae of septic infarcts.

Abscess(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.101

97

Major determinants of the eventual outcome of an infarct. (4)

Nature of vascular supplyRate of development of occlusionVulnerability to hypoxiaOxygen content of blood(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.101

98

Neurons undergo irreversible damage when deprived of their blood supply for _______.

3-4 minutes(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.102

99

Myocardial cells undergo irreversible damage after ______ minutes of ischemia.

20-30 minutes(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.102

100

It is the final common pathway for severe hemorrhage, extensive trauma, burns, large MI, pulmonary embolism and sepsis.

Shock(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.102

101

End results of shock (3)

HypotensionImpaired tissue perfusionHypoxia(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.102

102

This type of shock results from failure of the cardic pump which maybe caused by MI, ventricular arrythmias, cardiac tamponade or outflow obstruction.

Cardiogenic shock(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.102

103

This type of shock results from loss blood or plasma volume.

Hypovolemic shock(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.102

104

This type of shock is caused by microbial infection, caused by gram negative and gram positive bacteria and fungi

Septic shock(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.102

105

True or false:Systemic bacteremia must be present to induce septic shock.

FalseHost inflammatory response to local extravascular infections may be sufficient to induce septic shock.(TOPNOTCHRobbins Basic Pathology, 8th ed. p.102

106

Type of shock which occurs in the setting of an anesthetic accident or spinal cord injury as a result of loss of vascular tone and peripheral pooling of blood.

Neurogenic shock(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.102

107

This type of shock represents systemic vasodilation and increased vascular permeability caused by IgE hypersensitivity reaction.

Anaphylactic shock(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.102

108

Septic shock caused by gram negative bacilli.

Endotoxic shock(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.103

109

Criteria for SIRS.

Temp 38 CelciusHR >90 bpmRR >20 or PaCO2 12,000 cells/mm3 or 10% bands(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.103

110

Adrenal changes in shock.

Cortical cell lipid depletion(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.106

111

Kidney changes in shock.

Acute tubular necrosis resulting in oliguria, anuria, and electrolyte disturbances.(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.106

112

Gastrointestinal changes in shock.

Focal mucosal hemorrhage and necrosis(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.106

113

Lung changes in shock.

Diffuse alveolar damage if due to bacterial sepsis and trauma.(TOPNOTCH)Robbins Basic Pathology, 8th ed. p.106

114

The main mechanism of edema in inflammatory disease is:

increase vascular permeability (TOPNOTCH)

115

Sudden death in pulmonary embolism is due to:

Acute right heart failure (TOPNOTCH)

116

Accumulation of fluid within tissues

Edema (TOPNOTCH) Robbins Basic Pathology, 9th ed., p. 113

117

Accumulation of fluid within body cavities

Effusion (TOPNOTCH) Robbins Basic Pathology, 9th ed., p. 113

118

Patient presented with pain, swelling, and tenderness of left leg. What is the mechanism of edema in this condition?

Increased hydrostatic pressure. It is caused by impaired venous return as a result of DVT.(TOPNOTCH)

119

Patient presented with dyspnea, orthopnea, easy fatigability, and bipedal edema. The mechanism of edema in this condition is.

Increased hydrostatic pressure (TOPNOTCH)

120

Patient presented with periorbital edema and ankle edema. Lab result shows high lipid levels, and low serum albumin. What is the mechanism of edema in this condition?

Reduced plasma oncotic pressure secondary to protein/albumin loss (case of Nephrotic syndrome) (TOPNOTCH)