Lecture 12 - Head, Neck and Back Flashcards

1
Q

TMJ

A

temporal mandibular joint
is the synovial joint that connects the jaw to the skull
these two joints are located just in front of each ear
each joint is composed of the condyle of the mandible, an articulating disk, and the articular tubercle or the temporal bone
the movements allowed are side to side, up and down, as well as protrusion and retrusion
this complicated joint along with its attached muscles, allows movements needed for speaking, chewing, and making facial expressions

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2
Q

Which type of arthritis is at increased risk of joint destruction of TMJ?

A

pts with RA

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3
Q

MPD

A

myofasial pain dysfunction syndrome

pain at the TMJ due to various causes of increased muscle tension and spasm
it is believed that MPD syndrome is a physical manifestation of psychological stress
no primary disorder of the joint itself is present
pain is secondary to events such as nocturnal jaw clenching and teeth grinding
treatment is focused on behavioral modification as opposed to joint repair

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4
Q

Who gets TMJ?

A

F > M
hx of jaw trauma
MC in 3rd and 5th decade of life
associated with other chronic pain syndromes and depression

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5
Q

Bruxism

A

teeth clenching

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6
Q

What PE is done for TMJ?

A

abnormal mandibular movements:
observe opening and closing of the mouth, with attention to any jaw or dental protrusion. Check to see if the upper teeth and lower teeth are aligned. Look for jaw deviation on opening and closing

Decreased ROM of TMJ: measure functional ROM of jaw opening. normal functional opening is 35 - 55mm

Muscles of mastication tenderness: palpate the TMJ and muscles of mastication assessing for muscle tenderness, joint crepitus, or jaw click during opening and closing

Pain with dynamic loading: to elicit pain with dynamic loading, have pt bite on a cotton toll or grasp the mandible on both sides, pressing down and back to compress the TMJ bilaterally

Bruxism: examine the oral cavity and dentition, looking for signs of tooth wear. This exam may be done by the pts dentist

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7
Q

What is the DDx for TMJ?

A
AOM
mastoiditis 
AOE
Gout
Temporal arteritis 
mandibular fracture or dislocation 
RA
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8
Q

What is the treatment for TMJ?

A

NSAIDs, muscle relaxers, lifestyle modification
avoid gum

refer to oral surgery for possible:
bite block/splint or other means of reducing bruxism
joint injection
surgery

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9
Q

Cervical spine nerve roots are above or below cervical vertebrae?

A

above

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10
Q

Radiculopathies

A

pain in arm or leg which radiates in a dermatomal distribution of a peripheral nerve
pain may be unilateral or bilateral
typically in cervical or lumbar spines, but occasionally in thoracic spine

several causes: 
trauma 
herniated disc 
osteophytes (arthritis) 
nerve root swelling 
invasion of cancer into bone or spinal canal
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11
Q

How do pts with radiculopathies present?

A

pain in localized dermatome
pain often described as “electrical shock” or “tingling” or “pins and needles”
pain often worse with certain positions
often initiated by trauma, heavy lifting, repetitive motions or prolonged sitting

have you lost control of bowel or bladder?

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12
Q

What are early PE findings of radiculopathies?

A

Early PE:

pain reproducible with stretching of nerve over bone (straight leg raise test for lumbar radiculopathy)

pain reproduced by narrowing the foramen (Spurling’s test - cervical radiculopathy)

pt moves slowly and very carefully

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13
Q

What are later PE findings of radiculopathies?

A

muscle weakness in affected dermatome

significant sensory loss detectable by pin prick or light touch

diminished reflexes in affected area

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14
Q

What are concerning PE findings of radiculopathies?

A

MOST CONCERNING PE FINDINGS:

laxity of rectal tone
fecal incontinence or urinary retention

CALL SPINE SURGEON NOW

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15
Q

What imaging is useful in radiculopathies?

A

lumbar is POINTLESS

cervical spine films with obliques can show foraminal narrowing

MRI is BEST test (CT will do in a pinch)

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16
Q

Disc Herniation

A

tough connective tissue of intervertebral disc, the annulus fibrosis, ruptures and spills out the gel like nucleus pulposis onto the spinal nerves
significant inflammatory reaction occurs

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17
Q

Disc Bulge

A

bulging of the annulus fibrosis can also cause compression of the spinal nerves

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18
Q

What is the treatment for radiculopathies?

A

initial treatment is conservative unless bowel/bladder control is lost:
NSAIDs
PT
brief rest +/- brief rx with opiates
remove the exacerbating factors (lifting, sitting, etc.)
avoid bed rest
avoid systemic steroids

if conservative measures don’t work, can consider:
-epidural steroid injection (lumbar mainly)
-surgery
1 year after initial sxs, those who had surgery and those who did not had the same outcomes

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19
Q

Cervical spinal stenosis

A

cervical myelopathy

narrowing of the cervical canal causing pressure on the spinal cord typically causing upper motor neuron signs and sxs

MC causes:
disk herniation
osteophyte formation (arthritis)
ligamentum flavum hypertrophy and ossification

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20
Q

What is the clinical presentation of cervical myelopathy?

A

motor:
earliest finding in triceps and hand intrinsics weakness
clumsiness is common - problems with shirt buttoning and fine motor control
spasticity in LE

bilateral sensory changes:
+/- radicular. often “stocking-glove”
posterior columns (vibration and proprioception) function impaired. + electrical shock pains in the arms
spastic or scissors gait
bladder dysfunciton - urgency and/or retention
hyperreflexia, including upgoing toes and clonus
+Hoffman’s signs

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21
Q

Which imaging test is best for cervical myelopathy?

A

MRI

note: you can’t look for cervical stenosis if you dont think of it! often missed dx

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22
Q

What will you see on MRI for cervical myelopathy?

A

hyperintensity within the spinal cord at the area of compression on T2
myelomalacia = cord edema
spinal cord atrophy

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23
Q

myelomalacia

A

cord edema

24
Q

What is the treatment for cervical myelopathy?

A
conservative management (not ideal) 
surgical decompression (anterior and posterior)
25
Q

Lumbar spinal stenosis

A

narrowing of the lumbar canal/foramen causing pressure on the spinal nerves

26
Q

Why is there no pressure on the spinal cord in lumbar stenosis?

A

NOT DONE HERE

27
Q

What is the most common cause of lumbar stenosis?

A

disc herniation
osteophyte formation (arthritis)
neuroforaminal narrowing
invasive tumor

28
Q

How do pts with lumbar spinal stenosis present?

A

neuroclaudication (aka psudoclaudication) = pathognomonic
pain, sensory loss and weakness which occur mainly when the pt is moving

sxs are typically bilateral, but also asymmetrical
sxs are almost always in more than one dermatome
weakness or sensory loss in more than one dermatome and on more than one side
wide-based gain and + Romberg sign

29
Q

What is the treatment of lumbar spinal stenosis?

A

can try conservative therapy if neuro deficits are not substantial
PT
NSIADs

surgical decompression/ laminectomy/ fusion
these are typically long, complicated surgeries. Ideally surgery performed by a fellowship-trained spine surgeron

30
Q

Cauda equina syndrome

A

SURGICAL EMERGENCY

a radiculopathy in which the cauda equina is compressed
may have “saddle anesthesia” - area of the pts perineum
often associated with loss of bowel or bladder control

31
Q

What is the treatment for cauda equina syndrome?

A

call a surgeon
possibly steroids
once a pt loses bowel/bladder control, may not get it back unless quickly decompressed
admission to the hospital for neuro checks both pre and post op

32
Q

____ of adults will experience low back pain during their lives

A

85%

33
Q

Treatment of back pain +/- sciatica

A
exercise - not bedrest 
NSAIDs
PT
massage or acupuncture 
consider tricyclic antidepreesants if NSAIDs are ineffective 

AVOID lumbar spine films - high radiation burden with little clinical utility

AVOID opiates for anything but the very acute phase - increasing evidence of addiction risk

AVOID systemic steroids - not effective

34
Q

Scoliosis

A

abnormal curvature of the spine in any direction
most commonly, the abnormal curve is in the coronal plane, but can also be rotational or in the sagittal plane

most common in adolescents right just before or at onset of puberty (80-85% of cases start at this point in the lifespan)

35
Q

Cobb angle

A

the cobb angle is formed by the intersection of a line parallel to the superior end plate of the most cephalad vertebra in a particular curve, with the line parallel to the inferior end plate of the most caudad vertebra of the curve
the intersection of these lines may occur outside the border of the actual film

therefore by convection, perpendiculars to the parallels are drawn, and the angle between their intersection is measured

36
Q

What cobb angle defines scoliosis?

A

> /= 10 degrees

37
Q

What is the epi of scoliosis?

A

3% of the population meet the criteria but only 1:10 of those people will need treatment

girls are 10x more likely to need treatment

etiology is unknown although it can run in families, most are idiopathic

38
Q

Which scoliosis pts get xrays?

A

those with >7degree or with clinically evident scoliosis

order standing, full length PA and lateral views of the spine (C7 to sacrum and iliac crest)

special PA long cassette film to minimize radiation to breasts and thyroid

39
Q

What is the treatment for scoliosis?

A

cobb angle
>49 surgery
30-49 = brace
20-29 = observe, family may elect bracing
10-20 = repeated observation every 6 months

40
Q

What are the screening recommendations for scoliosis?

A

pediatricians screen peri-pubertal visits

no more school screening

41
Q

Kyphosis

A

vertebral fractures, esp due to osteoporosis
degenerative disc disease
decreased postural flexibility
muscle weakness
ossification of intervertebral ligaments
scheurmann’s disease = congenital short vertebral height

42
Q

Who is more likely to get kyphosis?

A

W > M

>60 yo MC

43
Q

What is the common workup for kyphosis?

A

measure the angle with goniometer

lateral spine xrays

DEXA for osteoporosis

44
Q

What is the sequelae of kyphosis?

A
impaired pulmonary function 
falls 
diminished physical performance (walking speed, muscle strength, get up and go test) 
fx of vertebral spine
chronic pain 
GI issues such as GERD and hiatal hernia
increased all cause mortality for age
45
Q

What is the treatment for kyphosis?

A

no high quality evidence for any of the treatments

PT/exercise may be helpful for restoring muscle strength and preventing falls
bracing
surgery has exceedingly high complication rate and is only recommended for those in severe pain or with significant pulmonary compromise

46
Q

Whiplash

A

traumatic injury to the neck due to abrupt flexion/extension movement of the C spine

multiple structures can be injured including muscles, soft tissues, ligaments, bones and intervertebral discs

very common in MVC
usually self limited

50% of pts have some degree of pain 1 year later

47
Q

What is the workup for whiplash?

A

detailed PE to include good peripheral nerve damage

usually get C spine xrays on day of injury

MRI if focal neuro deficit, pain not improving after af ew weeks of conservative treatment

48
Q

What is the treatment for whiplash?

A

NSAIDs
muscle relaxants
early neck mobilization after serious injury rule out
opiates for 1-3 days max
PT if pain becomes chronic
resolving legal issues is associated with improvement in sxs

49
Q

What are the first steps in scalp lacerations?

A

first step - ABCs

clear C spine

evaluate for potential skull fracture

always consider MOI

50
Q

PE of Scalp Lacerations

A

NOT DONE HERE

51
Q

What is the treatment of scalp lacerations?

A

if the pt has been scalped, will need plastic surgery

otherwise:
-irrigate and clean. may need to clip hair
-local anesthesia - lido with epi
-close if wound within 24 hours
–staples preferable
–suturing if needed for deep wound or hemostasis
–can consider hair apposition
update tetanus immunization
abx almost never needed

52
Q

What are the MC causes of skull fx in adults?

A

falls
assaults
MVC
penetrating trauma/GSW

53
Q

What are the MC causes of skull fx in children?

A

falls
recreational activities
MVC
child abuse

54
Q

What are the steps in evaluating a skull fracture?

A

first step - ABCs

second step - check for neurological dysfunction that may indicate elevated ICP or neurologic injury

clear C spine or assume a sC spine injury

palpate skull for step offs

get a CT

55
Q

What is the treatment for a skull fx?

A

only linear skull fractures without underlying parenchymal damage can be managed on the outpt basis
the rest need to be admitted to a hospital with neurosurgical capacity
treatment may include:
surgery
abx to prevent meningitis
observation for neurologic deterioration or CSF leak