Lecture 34 - Ostertagia Flashcards

(28 cards)

1
Q

what nematode causes disease in calves and young cattle

A

ostertagia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

where are L4s in ostertagia

A

arrested in the lumen of gastric glands

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

describe acquired immunity to ostertagia

A

important for cattle
develops at approx. 2 years
mature cows will not show signs of infection

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

T/F: most ostertagia pathology is due to growth and development of L4s in the lumen of the gastric glands

A

TRUE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

what are clinical signs of ostertagia

A

persistent watery D+
hypoproteinemia (bottle jaw and edema)
rough hair coat
stunted growth, weight loss
weakness

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

negative nitrogen balance results in

A

protein catabolism

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

how does ostertagia initiate clinical signs

A

decreases gastric acid so pH increases

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

what is apparent on necropsy of ostertagia infection

A

“moroccan leather”
inflamed papillae

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

is ostertagiasis seasonal? if so, when?

A

yes during grazing seasons

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Describe Type I Ostertagiasis

L4 role
pathology
morbidity : mortality
treatment

A

L4s: develop directly to adult worms in young calves during 1st grazing season

pathology: summer & fall (cool regions) and winter & spring (arid regions)

high morbidity, low mortality (slow, progressive pathology)

tx: treat & move or treat & repeat

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

how does type II differ from type I ostertagiasis

A

early L4s arrest, and remain arrested for weeks to months whereas in type I L4s directly mature to L5s

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Describe Type II Ostertagiasis

L4 role
pathology
morbidity : mortality
treatment

A

L4: arrest and reactivate later in yearling calves during 2nd grazing season

pathology: spring (cool region) and fall (arid region)

low morbidity, high mortality (acute pathology)

tx: treat older calves and target L4s

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

T/F: southern calves moved to northern feedlots in the fall have potential for type II ostertagiasis

A

TRUE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

how is ostertagia diagnosed

A
  1. FEC
  2. MOO ELISA
  3. increased serum pepsinogen levels
  4. abomasal-centesis
  5. deworm & observe
  6. moroccan leather on necropsy
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

type I should be treated with ____ while type II should be treated with ____

A

adulticide; larvicide

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

T/F: there is reported resistance to dewormers for ostertagia in cattle

17
Q

what are the best pasture management techniques to avoid infective ostertagia L3s

A
  1. rotational grazing (naive calves first)
  2. co-grazing (cow/calf operations)
18
Q

the pathology of trichostrongylus is due to

A

activity of adult worms

19
Q

describe the life cycle of trichostrongylus spp.

A
  1. L3s over winter well on pasture, die off in summer, and recontaminate in fall
  2. L4s migrate below the mucosal surface but do not arrest
20
Q

T. axei exist in the stomach of what animals

A

horse, rabbit, humans

21
Q

what is caused by T. colubriformis

A
  1. damage to intestinal mucosa
  2. villus atrophy
  3. dark, watery D+ (fly strike complications)
  4. anorexia
22
Q

how is T. colubriformis diagnosed

A
  1. clinical signs, history
  2. FEC
  3. larval speciation
23
Q

what is the 5-point check in small ruminants

A
  1. FAMACHA
  2. Body condition score
  3. DAG score
  4. Nasal discharge
  5. Bottle Jaw
24
Q

what are the deworming strategies for trichostrongylosis

A
  1. tactical - salvage deworming at clinical signs
  2. strategic - scheduled deworming
25
T/F: sheep/goats should not be co-grazed with horses to avoid T. axei infection
TRUE
26
what are L3 hatching requirements of nematodirus? what does this cause?
eggs must be chilled over winter before hatching major larval storms
27
describe nematodirus
- pathology is due to adult activity and manifests as enteritis and severe D+ - large eggs
28
describe cooperia spp.
- pathology due to adult activity causing enteritis, D+, anorexia, emaciation - prominent in cow-calf operations - resistance to macrocyclic lactones