Physical and cognitive development in adulthood Flashcards Preview

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Flashcards in Physical and cognitive development in adulthood Deck (25)
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1
Q

biological - 3

A

genetically predetermined path way - birth to death, mature, digress, multidirectional
asunchronous - never the same, each system move through diff patterns of aging and development
all the nurturing that were exposed to has an effect - food, PA, address

2
Q

senescence -2

A

late adulthood

harmful events in terms of appearance and function like wrinkles and age spots

3
Q

senility - 2

A

accumulation of senescence events to death - attached to disease processes
late adulthood - or earlier, some sort of disease

4
Q

How to calculate life expectancy

A

go back to the year you were born

5
Q

1900 life expectancy

A

50

6
Q

2012 life expectancy M vs F

A

80 vs 84

7
Q

life expectancy trend

A

went up and now slowing down

8
Q

Do women outlive men?

A

gap thinning, originally thought to be x chromosome, industrialized (environmental

9
Q

max life expectancy

A

under ideal conditions 120, oldest 122, but how long do you wanna live

10
Q

2 theories of biological aging

A

levels of DNA and body cells

levels of organs and tissues

11
Q

aging at the level of DNA and body cells - 2

A

programmed effects of genes - DNA make up
- aging genes called telomeres - caps that protect us from biological processes every time they replicate they shorten (supplementation?)
- disorders that support aging genes - closely linked to telomeres
cumulative effects of random events - phases where they want to then dont want to, body transitioning of what we do - to prevent aging

12
Q

3 disorders supporting the aging genes

A

down’s syndrome - avg lifespan of 45, usually by CVD, accelerated aging process (decline of telomeres) like sensory deficits - spectrum disease
progeria - chr1, avg lifespan 13, CVD, 5-10 times their actual age, extremely small telomere and fast deterioation 0 most aggressive aging order thats rare and no way to predict - no genetic link - harmful senescense events but physiologically still the age they are
werners syndrome - chr8, recessive, onset 18-20, infertile, may reproduce before hand, accelerated process, 3-4 times regular, late diagnosis - 40-50

13
Q

3 cumulative effects of random events

A

damage to DNA in cells - mutations
accumulations - less efficiency in repaire and replacement - aging
probable cause - free radicals - antioxidants to slow them down (good food gps)

14
Q

levels of organs and tissues

A

cross linkage theory - fibres bend and crosslink to other fibres to make an x pattern which deterioate and speed up aging process quicker - predicable from biological parents

15
Q

motor performance changes

A

decrease heart and lung function with gradual muscle loss - change in motor performance -reduced capacity/change sports/ maintain

16
Q

other reasons for aging and bad at PA

A

motivation and practice

17
Q

at what age does athletic skill peak?

A

20-30 then declines

18
Q

in practice continues, what does decline look like?

A

2%/decade - we have the capacity

19
Q

what sport peaks later

A

endurance - long distance running and golf

20
Q

cognitive development

A

3 career changes in a life time

21
Q

post formal thought

A

cognitive turning point - body and mind senescence, may not be wisdom, lose simplicity of thinking

22
Q

schaie’s theory

A

no age

- goals shift from acquiring knowledge to using it - occurs in stages

23
Q

4 stages of schaie’s theory

A

acquisitive - sensory info, schemas, motor skills, collecting info - what we need to know
achieving - info vs things we want to do - desire to achieve
responsibility - career, roles
reintegrative - late adulthood, prioritize, reevaluate surroundings - why do we need to know

24
Q

achieving and responsibility

A

how do we use the knowledge

25
Q

adulthood

A

transitional period between adolescense and older adulthood - long