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Flashcards in Self Study 6 Deck (40):
1

what does allgx and H dermatoses involve

inflm respoonse to multi exogenous/endogenous agents

2

what is allgx and H deramtoses charcterized by

epidermal edieam with seperation of epidermal cells

3

what dermatitis' does allgx and H deramtoses involve

irritant contact dermatitis, allgx contact dermatits, atopic and nummular eczema, drug induced skin interuptions

4

contact dematitis

common inflm of skin

5

2 types of contact dermatitis

allergic and irritant CD

6

what does allgx CD result from

cell mediated, type4 H rxn

7

what type of reaction occurs in ACD

t cell only. no abs.

8

2 kinds of ACD reaction

direct cytotoxic T cell destr of macrophage
deylaed t cell prod of lymphokines that destroy ag bearing cell days later

9

primary lesions ACD

mild erythema with edema to vesicles or lrage bullae

10

secondary lesions ACD

dt bacterial infc

11

where do lesions in ACD occur

anywhere

12

ICD cause

caused by chemicals that irritate skin, environmental triggers

13

risk for ICD

hx of atopic dermatitis

14

4 types of ICD

subjective, acute, chonic, chemical burns

15

subjective ICD

no clinical mnfts
burning or stinging when exposed to irritant

16

acute ICD

due to single exposures to irritant
dryness, erythema, edmea, inflm, vesicles

17

chronic ICD

results from contd exposure to irritant , even when irritant removed.
dryness, erythema, sclaes, fissures, vesicles

18

chemical burn ICD

deep necrosis from contact with cement prod

19

Tx ICD

remove source
change lifestyle
minor: wash area, apply anti itch cream, topical corticosteroids?
mod - extreme: tx with wet dressing, oral antihistamine, systemic corticosteroids

20

eczema

inflm of skin

21

2 types of eczema

atopic dermatits
nummular eczema

22

atopic derm

ithcy, inflm skin disorder

23

what is atopic derm characterized by

poorly defind erythema with edema, vesicles, weeping (acute)
skin thickening (chronic)

24

how is atopic derm often described

IgE mediated H. Similar to Allgx patho.

25

how do mnfts present in atopic derm

differently depending on age and race

26

when do 70% of atopic derm cases start

peds

27

what is atopic derm characterized with (mnfts)

vesicles formation, oozing, cursting

28

where does atopic derm begin (peds)

cheeks, progress to scalp, arms, trunk, legs

29

atopic derm mnfts adolsecent (acute)

dry, red patches on face, neck, upper trunk-no thickening

30

atopic derm mfnts adolescent (chronic)

dry, leathery, lichenfied spin

31

is there a gen predisopostion in atopic derm

yes

32

tx for atopic derm

avoid temp changes/stress
skin care
avoid triggers
dec contact with awater
meds
wet derssing
probiotics

33

why should a person with atopic derm avoid temp changes

minimize vasscular and sweat response

34

what triggers does a person with atopic derm have to avoid

evironmental
food

35

what meds are used to tx atopic derm

topical corticosteroids
topical immune modulators

36

nummular eczema lesions

coin shaped lesions
papulovesicular patches on arms and legs

37

what is normal for NE leision patterns

initial leisions heal, followed by secondary outbreak of mirror image leisons on opposite side of body

38

pattern of exacerbation for NE

chornic, with weeks and years between exacerbations. happens mostly in winter

39

tx for NE (lifestyle

avoid iodides and bromides
avoid frequent bathing and stress
inc environmental humidty
t

40

tx for NE (pharm)

topical emollients, corticosteroids, coal tar preperations, uv light