Neuro Flashcards

(76 cards)

1
Q

Kernigs Sign is?

And is a postive sign when found in ________ or _________

A

The Inability to extend a patients legs past 135o with out pain. This is done with a patient supine

Positive sign Of meningittis, or subarachnoid Hemorrhage.

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2
Q

Brudizinski reflex is ?

A

When you bend a patients head at the neck toward the chest and this causes the same flexation at the hips.

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3
Q

What are the three major catagories of Meningitis

A

Viral

Fungal

Bacterial

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4
Q

What are the S/S ofViral meningitis

A

Generally follows another major viral illness, such as measles, mumps, herpes simplex, herpes zoster

Increased Temps, Head Ache, Neucol Ridgitity(Stiff Neck), Nausea, Vomiting

Essentially presents as flu like symptoms

Flu = Virus

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5
Q

What is the Most commonly encountered Meningitis and how is it treated

A

Viral meningitis, is the most common, it is treated by monitoring for Seziures and treating Symptoms, the general course takes 1-2 weeks

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6
Q

What type of patient is fungal meningitis generally found in.

What are the general s/s

how is it treated

A

immunosupressive patients.

S/S were dependent on the type of immunosupression

treatment is generally IV antifungals, and symptomatic

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7
Q

what is the worst type of meningitis?

A

Bacterial Meningitis is the worst type of meningitis

It is a medical emergency

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8
Q

What are the S/S of Bacterial Meningitis

A

Very Rapid Progression and action

High Fever Nuchal rigidity

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9
Q

What nursing interventions are needed for Bacterial Meningitis

A

Rapid thourogh assessment

Blood Draw immediately

Prep for L/P

Adminster Broad Spectrum antibiotics

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10
Q

What is the Name of the Vaccine for Meningitis

A

Menomune

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11
Q

What are two test you can do during assessment that would lead you to suspect meningitis

A

Brudizinski reflex and Kernigs Sign

Brudinski’s- neck bends feet body draws to core

kernigs- Painful extension of the legs

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12
Q

what are some lab/ Diagnostics for Meningitis

A

Culture and sensitivity

Cbc with Differential

L/P

CT/ MRI

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13
Q

What are the primary nursing care items for a person with Meningitis

A

Monitor Neruo with special attention cranial nerves

observe for s/s of ICP

seziure Precations

Monitor for septic shock

Assessment will show Fever, hard to arouse, decreased appetite, decreased mental status

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14
Q

Who are at the greatest risk for Meningitis?

Why?

A

Antivaxers, Imnunocompromised)

people who live in grouped settings (dorm, prisons, etc)

This is spread by respiratory so close quarters breeds illness

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15
Q

Important Patient care considerations for People with Meningitis

A

Vital signs at least q2-4 hours

Frequent Neuro Checks

Pain management-balance with ability to arouse

I+0

Decrease environmental stimuli

Bed Rest HOB always 300

Isolation precautions

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16
Q

What are some Prevetions complications for a nurse treating a patient with Meningitis

A

Intercrainial presssure ICP

Vascualar Dysfunction

F+E imbalances

Seziures

Shock

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17
Q

What is a CVA Stroke

A

Cerbreal vascular attack, is an interuption of blood flow to the brain similar to a heart attack, that is caused either by a blockage or bleed

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18
Q

What are the types of CVA

A
  • Ischeimic- Occlusive
  1. Thrombolytic
  2. embolitic
  • Hemorrhagic
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19
Q

What is an Ischemic Stroke

A

It is a interuption of blood flow to the brain by some form of thrombus or embolus

Thrombus come from larger venus stasis

Embolus tend to come from a cardiac event such as A-fib

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20
Q

Diagnosis of ischemic stroke is done by?

A

CT MRI

EKG ECG

ABG

CMP CKP

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21
Q

Treatment for an ischemic stroke is

A

TPA(clot busters)

Must be adminstered within 3 hours of onset of stroke to be successful

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22
Q

TPA dosing is based on

A

Dosing is based on weight, with max dose being 90mg

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23
Q

What is the Norm for INR

A

2-3

the higher the number the thiner the blood

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24
Q

What is a TIA

A

Trans Ischemic attack

Generally Preceeds stroke is a warning sign

It allows for Reversable neruo Deficits or RIND

Damage can be seen by MRI or CT

Multiple TIAs ^ risk for Stroke

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25
Number one cause of Stroke IS?
HTN Prevention is key so controlled BP is paramont
26
Blood Thiners and Antiplatets are used to prevent what type of stroke
Ischemic
27
Can you use TPA for all strokes"
NO TPA is only for ISCHEMIC Strokes In hemmoragic you want the blood to clot
28
Hemoragic Stroke is
A bleed in the brain caused by a rupture of a vessle, Aneurysm, or rupture of an AV fistula malformation
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Hemorrhagic stroke risk factors include
Smoking Coccaine use obesity sedintary lifestyle ^stress levels, ^ HTN ^Cholesterol Previous CVA/TIA Sudden discontiuance of HTN meds
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Treatment for Hemmoragic stroke includes
Clipping, or spiraling artery, montioring for ICP Regular Neruo assessments monitor basic Asssesment HOB 300
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Management of Pts with Stroke
Prevention is key: maintain blood pressure pharmacology surgical interventions if needed prevent complications Assess FAST
32
The assessment acryonmm FAST stands for
**F-** Face Drooping, numbness **A**- Arm Weakness, Raising **S**- Slurred Speach **T- Time to ED ASAP**
33
Remember TREATMENT FOR ALL STROKES IS IN WHAT PRIORITY
**A B C** **Oxygenate**
34
What are some Degenerative Neurological Conditions
MS ALS( amytrophic lateral sclerosis) parkinsons myasthenia Gravis Guillain-Barre Syndrome Huntingtons
35
What are some commonalities between all Degenerative Neuro Disorders
* Altered Motor activity * Altered coordination * Altered Sensory Function * altered urinary/Bowel functions * Altered role/Self preception * altered speech/Swallowing * impaired Physical Mobility * risk for Injury * Nutrition Less than body req. * Risk for aspiration
36
MS is characterized by periods of remission and exacerbation but what is it
it is an inflamatory response that is autoimmune where the body attacks its own mylean sheaths on the nerve cells
37
MS is more common in women then men and more prevailant in northern climates, when does the onset generally begin
typically occurs between 20-40 y/o, believed to be related to childberaring and new stressors, and there is thought to be a link to family there is no cure
38
there are two focus types of ms they are
Relapse/Remit vs Chronic R/R is treatable and less debilitating, Chronic is progressive and leads to a rapid decrease in function and need for total care
39
MS Trigger Factors Include
* Virus or infectious agent * living in a cold climate * physical injury * emotional stress * pregnancy * fatigue * overexertion * temperature extremes * hot bath/shower
40
MS is diagnosed how
History, and symptoms, there is no test to confirm
41
What are the primary Symptoms of MS
Fatigue weakness numbness Difficulty with coordination, loss of balance visual disturbances * blurring * dilopia(double vison) * patchy or total blindness * change in peripheral vision * nystagmus(repetative uncontroled movements)
42
More Primary symptoms of MS include
Speech defecits (stutter) Dysarthria Dysphagia spastic weakness, ataxia, tremors, dysmeteria Emontial labilit, depression, euphoria bladder and bowl dysfunction-loss of sensation Tinitus, vertigo, decrease hearing Cognative changes
43
MS Secondary Symptoms - Problems related to primary symptoms
* Repeated utis * loss of muscle tona and disuse weakness * poor posture and torso control * decreased body density * shallow ineffcient breathing * pressure ulcers from immobility *
44
MS drug of Choice is ? What are some other common drugs used
Avonex(inteferon beta 1) given subq once a week also used are prednisone, baclofen(spacitiy) Keppra
45
Parkinsons is a disease that affects?
The neuro transmission of impulses causeing tremors, ridgitity slow movement(Bradykinesia) poor posture, depression/psych, dementia, autonomic symptoms, sleep distrubances
46
Cause of Parkisons is unknown but it affects what
Affects Gross motor Function, Degeneration of the nerves that control voluntary movment Occurs in the brain affects response to dopamines causes death of cells that create dopamine
47
MOST SEEN Symptoms IN PARKINSONS
Temors Rigidity Poor Posture Bradykinesia(Slow Movement)
48
What is the most used and gold standard treatment for Parkinsons meds
Levodopa and Carbidopa- may take up to 6 months to reach theraputic effect
49
what are some adverse affects of Levodopa-Carbidopa
Involuntary movements ataxia(loss of full control of body movements) Increased Tremors anorexia
50
Symmetrel is
An antiviral antiparkonsionian med that is thought to increase dopamine release. it is used to treat Bradykinesia, tremor, and ridgity, but is commonly avoided due to adverse effects: Dizziness, insomnia, confusion, orthostatic hypo-tension, which can all lead to increased risk for falls
51
Major Complications from parkinsons include
Aspiration Pnemonia from aphasia Altered cognition/ Dementia
52
Other than medication what is another theraputic option for parkinsons treatment
Deep brain stimulation- This is like a pacemaker to the heart but is place in the brain to help regulate
53
Not every case of parkinsons is believed to be from brain issues, what are some other thoughts of what may contribute to parkinsons
Medications(esp Psych) Heavy metal poisoning, pestasides mid-brain injuries and High levels of CO poisoning
54
True or False Parkinsons is a slowly progressing neurological dissorder that eventually leads to disablity
TRUE
55
Assessment and Care of parkinsons patients
Focus on degree of disabiltiy TEACHING AND SAFETY ARE KEY
56
Goals of treatment for Parkinsons Include
Improved functional ability, maintaining indepence with adls achieveing adequate bowel elimination maintiaining appropiate nutritional status, copiing skills, and communicatoin
57
Ways to focus on improving mobility for a parkinsons patient are
Daily excersise program, ROM Postural excersises PT EVAL and TX Techniques to ensure saftey and balance while walking Frequent rest, proper footwear Use of assistive devices PRN
58
What is ALS Amyotrophic Lateral Sclerosis
Loss of motor neurons in the anterior horn of the spinal cord and the motor nuclei of the brainstem: Leads to progressive weakness of extremities and trunk then, Muscle Atrophy, and weakness of the bulbar muscles which impairs swallowing and speech. Respiratory function is also imparied Due to Difficulty clearing secretions leading to drowning in own fluids
59
How is ALS Diagnosed
There is no diagnostice test but some test are used to attempt to rule it out. Some diagnostic proceedures Include: * CK- will be increased Muscle damage * EMG(electromyography) muscle Fasciculations-muscle twitching * Muscle Biopsy- testing atrophy * Serial muscle Testing- loss of stregnth * Pulmonary Function Testing- VIP to do at DX to establish a baseline for monitoring of progression *
60
ALS is a traditional assessment, with focus on speech and airway function, Primarily AIRWAY. What are some interventions.
* Excercise and mobility are used * Managment of Swallowing * Maitain Respiratory function * includes L/S assessments * prn suctioning * Cough and DB * Meds for Issues Rilusole, Quinine, Baclofen, Dantrolene, Diazepam * Pt Family Teaching
61
Meds used treatment of ALS include Rilusole, Quinine, Levsin, Baclofen, Dantrolene, Diazepam, what is each used for
Rilusole- Used to prolong Survival time of the Trachea Levsin-Dry Secretion Quinine - Muscle Cramps BACLOFEN, dantrolene Diazepam- Antispasmodics
62
When is Rilusole given
Generally toward the end of the disease process, and is used and good for 12 weeks
63
What is Diplopia
Double Vision
64
Myasthenia Gravis is
A progressive autoimmune disease, that leads to severe muscle weakness that affects women 15-35 or men over 40
65
Myastheia Gravis presents as
Extreme muscle weakness, Fatigue diplopia and ptosis are early signs Sleepy mask like expression dysphonia problems with chewing and swallowing leading to aspiration progressive weakness of Diaphram leading to Resp. Distress
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Diagnosed with
* Pt Hist & Phys * Tensiloon Testing- * 10 cc's IV, 2cc's pushed to monitor for reaction then remaining 8cc's Tensilon test reverse the effects of MG within 30 secs * ENSURE ATROPINE IS AT THE BEDSIDE, in case of adverse effects
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Theraputic Treatments of Myasthenia Gravis include
Plasmapheresis- which removes antibodies that are believed to be the cause of the condition
68
Medications given for M Gravis
* Anticholinesterase meds- * pyridostigmine(Mestinon)- give with food due to gi upset, and must be taken sametime each day to ensure theraputic levels * ambenonium (mytelase) * Both may cause Cholinergic Crisis * Chorticosteroids and Immunosupressents to tx inflamation
69
What are two potential complications with medication of MGravis
1. Myasthenic crisis- Under medication 2. cholinergic crisis- over medication
70
What are the S/s Of Myasthenic crisis(undermedication)
* Resp Muscle weakness \> need Mechanical Ventilation * Myasthenic Symptoms \> Weakness incotinece fatigue, hypertension, * temporary relief of symptoms with tensilon * PT's Cant breath from stopping medications
71
12 Cranial nerves Ooo, ooo, ooo, to touch and feel a girls vagina ah heaven
Most typically, humans are considered to have twelve pairs of cranial nerves (I–XII). They are: olfactory nerve (I), optic nerve (II), oculomotor nerve (III), trochlear nerve (IV), trigeminal nerve (V), abducens nerve (VI), facial nerve (VII), vestibulocochlear nerve (VIII), glossopharyngeal nerve (IX) vagus nerve (X), accessory nerve (XI), and hypoglossal nerve (XII)
72
Cholinergic Crisis Presentations Include
* Muscle twitching that results in muscle weakness and causes need for mechanical ventilation * Leads to hypersecretions, hypermotility * Hypotension * Tensolin has no positive effect on symptoms * Symptoms will improve with Anti cholinergic meds due to med overdose.
73
What is GBS, Guilian Barre Syndrome
It is an acute inflammatroy disease usually preceeded by a viral infection(URI or GI) 1-4 weeks Degeneration of the mylein sheath of the peripheral nerves, which is diagnosis by H&Pyshical and CSF
74
Guillian Barre Syndome Presents
Ascending paralysis that starts at the feet ascending up the body until It decides to stops which can take up to 3 weeks to progress, and may take up to 6 months to a year to go away
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Interventions For GBS
Monitor Cerebral vascular status Monitor Respitory treat accordingly, Vent, HOB ^, Chest PT Trach Nutritional interventions PRN, NG, TPN, Bowel sounds, Ensure communication, Lip reading, pic cards, eye blinks Pyschological support, prevention of deformities as ROM, Skin care prevention of Breakdown, Consider Steroids if needed **Plasmaphoresis- attempt to remove antibobies believed to be a possible cause**
76