Chap 24: Mgmt. of patients with chronic pulm. disease Flashcards

1
Q

what does a rapid assessment of respiratory status include?

A

Assessment of airway and breathing, O2 sat and O2 delivered, BP, HR, work of breathing, lung and heart sounds, LOC

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2
Q

what is the nursing management for Asthma?

A

Monitor: severity of symptoms, breath sounds, peak flow, pulse ox, and V/S.
Interventions: obtain a hx of allergies, identify meds that patient is taking, admin. fluids if needed.

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3
Q

what is the pharmacologic steps given in their order for asthma?

A
  1. SABA, PRN
  2. Low dose ICS: inhaled cortico steroid
  3. Low dose ICS and LABA OR medium dose ICS
  4. preferred medium-dose: ICS + LABA
  5. preferred: high dose ICS + LABA AND consider omalizumab
  6. preferred: high dose ICS + LABA + oral corticosteroid and consider omalizumab.
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4
Q

What medications are used in Asthma and what are their effects?

A

Bronchodilators: 1. beta agonists: epi, albuterol, isoetharine.

  1. anticholinergics: block Parasymp. nerv. syst. and promote bronchodilation.
  2. xanthines: decrease bronchospasm
  3. antiinflammatory agents: cromolyn sodium: decreases airway inflammation, decrease edema, inhibit release of histamine, prev. mast cells from opening, and increase permeability. (5-15mcg/mL)
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5
Q

what are the treatment steps of acute exacerbation?

A

oxygen, SABA (inhaled or IV), High dose syst. corticosteroids (PO or IV)
Monitor: VS. Pulse ox, ABGs IF o2 sat is <94% and symptomatic OR if sat is < 90% regardless of symptoms

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6
Q

what are the risks of Long acting asthma medications?

A

ICS: cough, dysphonia, oral thrush, H/A. high doses: systematic effects, adrenal suppression, osteoporosis, skin thinning, easy bruising
Systematic CS: Short-term: increase in appetite, fluid retention, wt.gain, mood alteration, HTN, peptic ulcer, aseptic necrosis.
Long term: adrenal suppression, growth suppression, dermal thinning, HTN, diabetes, Cushings, cataracts, musc. weakness, impaired immune function
LABA: decreased prot. from EIA, tachy, musc. tremor, hypokalemia, ECG changes, seizure, fatal/life threatening exacerbation.
Xanthines: tachy, N/V, tachyarrhythmia, CNS stim, HA, seizures, hemataemesis, hyperglycemia, hypokalemia, insomnia, GI upset,
Leukotriene Receptor Agonists: Smooth muscle relaxation

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7
Q

What are the short term meds for asthma and risks?

A

Inhaled SABA: tachy, muscle tremor, hypokalemia, increase lactic acid, ha, hyperglycemia
Anticholinergics: dry mouth, increased wheezing, does not stop EIB.
Corticosteroids: blood glucose anomalies, increase in appetite, wt. gain, HTN, mood alt., fluid retention, peptic ulcer.

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8
Q

how does the WBC differentiate between bacterial and viral PNA? What does the WBC indicate about asthma control?

A

Bacterial PNA: increased neutrophils: bandemia
Viral PNA: increased lymphocytes
Asthma: increased WBC is the A1C of asthma. eosinophils.

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9
Q

what is the asthma action plan?

A

Green Zone: 80%+ peak flow, can do usual activities, no symptoms. NO INTERVENTION
Yellow Zone: asthma is getting worse, symptoms present, waking d/t asthma, cant do some of act. peak flow 50-79% of base peak flow. INTERVENTION: 1st SABA- 2-4puffs q20min up to 1hr. Neb. once
2nd: take SABA 2-4 puffs, neb, add oral CS for _ #of days.
Red Zone: Very SOB, no effect with SABA, cant do usual activities, symptoms same or worse p 24hrs in yellow zone. Peak flow: < 50% of best peak flow. INTERVENTION: SABA: 2-4puffs, or neb, oral CS, CALL DR. NOW. Call ambulance if you are still in Red Zone after 15m or DR. has not been reached.

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10
Q

What are some causes of asthma?

A

constriction of bronchioles, swelling of bronchial membranes, increased mucous production.

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11
Q

What is the pathophysiology of an asthma attack?

A

Bronchiole muscles enlarge, thick mucous is produced, airways are hyperinflated: air trapping. AG>AB response, mast cells release histamine, bradykinin, prostaglandins, and SRSA (slow reacting substance of anaphylaxis)
AG: antigen AB: antibody

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12
Q

stimulation of alpha receptors cause?

stimulation of beta receptors cause?

A

alpha receptors: bronchoconstriction

beta receptors: bronchodilation

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13
Q

what are some non-allergic triggers for asthma?

A

viral illnesses, common cold, cold air, infection, stress, and smoke

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14
Q

how is sinusitis treated in patients who have asthma?

A

monitor closely for S/S of super infection, start with Amoxicillin, if no improvement after 5 days, switch to Augmentin for 10 days. May need CS.

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15
Q

How can asthma exacerbations be prevented?

A

flu vaccines, exercise sleep and optimal nutrition are encouraged.

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16
Q

What role might Magnesium play in asthma?

A

It may act as a bronchodilator. Beta 2 agonists may lead to Mg deficiency.

17
Q

What is included in resuscitation for status asthmaticus?

A

Oxygen therapy, admin of SABA, ICS, LABA, theophylline, hydration, monitor K and Ph, O2.

18
Q

what is the way to teach a patient to take a peak flow measurement?

A

stand up straight, indicator is at bottom of meter, take a deep breath, fill lungs completely, put mouthpiece in mouth, lightly bite, close lips, blast air out as hard and fast as poss. in a single blow. Record number on meter. Repeat three times, Record highest reading. Repeat test over 2-3 week period, to find personal best.

19
Q

what is PaO2/FiO2 ratio? what is normal and how is this number used?

A

amount of O2 in the blood in relation to the amount of inspired oxygen.
Normal: 380-476mmHg. Used to describe the degree of lung injury.

20
Q

What is the order of assessment in the diagnosis of COPD

A

Assess symptoms (mRC 0-1 or CAT <10) no to little symptoms. MrC >2 or CAT >10)
Airflow limitations: FEV1/FVC < 70
Risk of exacerbations: 0-1 exacerbation risk low, 2+ risk is high
Comorbidities: CV disease, osteoporosis, URI, DM, Lung Ca, anxiety/depression

21
Q

What are the Severity of airflow limitations using GOLD standards

A

Gold 1: Mild FEV1, > or = to 80% predicted
Gold 2: Moderate 50-80% predicted
Gold 3: Severe 30-50% predicted
Gold 4: Very severe, FEV1, < 30% predicted

22
Q

What are the treatment strategies for COPD?

A

Manage modifiable risk, Vaccinations: flu and pneumococcal, medications

23
Q

which meds are used for COPD

A

long acting inhaled bronchodilators: reduce exacerbations, hospitalizations, and improve symptoms.
Inhaled corticosteroids: improve symptoms, lung function, and quality of life and reduces exacerbations. CAN increase risk of PNA and withdrawal can cause exacerbations.

24
Q

Common cause and treatment of exacerbation?

A

Viral URI

Rx: SABAs, Systemic steriods (30-40mg/day) for 10days, O2 therapy maintain sat at >89%.