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Congenital Defects & Pediatrics COPY > Congenital Heart Defects: ASD/VSD- Topic 9 > Flashcards

Congenital Heart Defects: ASD/VSD- Topic 9 Flashcards

1
Q

CPB Normorthermic Blood Flow

A

0-3 kg = 200 cc/kg/min 3-10 kg= 150 cc/kg/min 10-15 kg= 125 cc/kg/min 15-30 kg= 100 cc/kg/min >30 kg= 75 cc/kg/min >55 kg= 65 cc/kg/min

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2
Q

Atrial Septal Defects most commonly occur as defects in what?

A

Septum primum within the fossa ovalis (secundum ASD)

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3
Q

Atrial Septal defects CAN involve the secundum near what?

A

SVC (sinus venosus defects are less common)

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4
Q

Atrial septal defects will do what to physiology?

A

Will cause pressure mediated shunting L to R R to L

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5
Q

Which shunt is of most concern to you? L to R or R to L?

A

R to L; skips the lungs; cyanotic shunts *BUT both are of concern; one can become the other because it is pressure mediated

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6
Q

How is blood flow quantitated? (In shunts)

A

Pulmonary Blood Flow (Qp) Systemic Blood Flow (Qs)

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7
Q

What would expect the Qp/Qs ratio to be in a normal individual?

A

1 (1:1 ratio)

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8
Q

When do shunts require treatment?

A

When they are symptomatic

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9
Q

When do shunts require treatment?

A

When they are symptomatic Pulmonary over circulation Shunts causing increase in right heart size

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10
Q

When do shunts require treatment?

A

When they are symptomatic Pulmonary over circulation Shunts causing increase in right heart size Qp:Qs > 1.5

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11
Q

When do shunts require treatment?

A

When they are symptomatic Pulmonary over circulation Shunts causing increase in right heart size Qp:Qs > 1.5

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12
Q

What is the most common type of atrial septal defect?

A

Ostium Secundum

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13
Q

What are some common types of atrial septal defects?

A

Ostium Secundum Patent Foramen Ovale Ostium Primum Sinus Venosus

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14
Q

Ostium Secundum

A

Most common ASD type Formed by failed growth of the septum secundum OR rapid reabsorption of the septum primum Mid atrial

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15
Q

What is the only type of ASD suitable for percutaneous closure?

A

Ostium Secundum ASD; defect in the middle of the atrial septum

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16
Q

Patent foramen Ovale (PFO)

A

Small channel that has little hemodynamic consequence; it is a remnant of the fetal foramen ovale *In some cases PFO can be larger and require treatment

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17
Q

Patent foramen Ovale (PFO)

A

-Small channel that has little hemodynamic consequence; it is a remnant of the fetal foramen ovale -Channel from birth; “Flap valve” *In some cases PFO can be larger and require treatment

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18
Q

Why would a PFO normally close?

A

Closes due to pressure changes very early in life

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19
Q

Why would a PFO normally close?

A

Closes due to pressure changes very early in life

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20
Q

Closure of Fetal Shunts

A

*The initial inflation of the lungs causes changes: *Decreases PVR results in increased BF from PA *Increased BF from RA to RV and into the PA’s and less BF through the foramen ovale to the left atrium *More blood returns from lungs increases pressure inLA *Increased LA pressure and decreased RA pressure (due to pulmonary resistance) forces blood against septum primum causing hte formane ovale to close

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21
Q

What action functionally completes the separation of the heart into two pumps?

A

Closure of the PFO in a normal fetal heart after birth (changes in pulmonary pressures, resistance, BF)

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22
Q

Ostium Primum ASD Location

A

Low in the septum and can be considered a type of AV septal defect (AVSD)

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23
Q

Ostium Primum Sat Changes

A

Could have RA saturations lower than RV without a VSD

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24
Q

Ostium Secundum

A

Most common ASD type (90%) Formed by failed growth of the septum secundum OR rapid reabsorption of the septum primum Mid atrial

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25
Q

Ostium Primum Sat Changes

A

Could have RA saturations lower than RV without a VSD

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26
Q

Sinus Venosus ASD Location

A

high in the septum where the vena cava intersects with the right atrium; may be inferior or superior

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27
Q

What is sinus venosus ASD frequently associated with?

A

Partial anomalous venous return (PAPVR)

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28
Q

What is sinus venosus ASD frequently associated with?

A

Partial anomalous venous return (PAPVR)

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29
Q

What forms separate chambers of the heart?

A

Embryonic septation

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30
Q

What are key aspects in heart development?

A
  1. Embryonic/fetal circulation is different to the neonatal circulation 2. Embryonic septation forms separate chambers 3. Several septal defects of heart septation may only become apparent on this transition
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31
Q

Which septal defect occurs in us all?

A

The foramen ovale (between the 2 atria) which in general closes in the neonate over time

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32
Q

At what day does cardiac septation occur?

A

Day 27

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33
Q

How long does cardiac septation last?

A

10 days

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34
Q

When do the formation of cardiac septa occur?

A

Simultaneously

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35
Q

During cardiac septation, how does the external appearance change?

A

No major changes in external appearance

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36
Q

Atrial Septation at what days?

A

27-28 (for 10 days)

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37
Q

What happens during atrial septation?

A

*The paired atria fuse together to form a common atrium *Atrial septation occur simultaneously and in cooperation with ventricular septation *Atrial septation also lasts 10 days

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38
Q

In normal individuals, are left or right sided pressures higher? Why?

A

Left heart pressures > Right heart pressures Thick LV has to produce enough pressure to pump blood throughout the entire body, while the thin RV only has to produce enough pressure to pump blood to the lungs

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39
Q

In normal individuals, are left or right sided pressures higher? Why?

A

Left heart pressures > Right heart pressures Thick LV has to produce enough pressure to pump blood throughout the entire body, while the thin RV only has to produce enough pressure to pump blood to the lungs

40
Q

What size ASD would be considered “large”?

A

> 9mm

41
Q

A large ASD may result in what kind of shunt?

A

Clinically remarkable left-to-right shunt; blood shunted LA to RA. This extra blood from LA may cause a volume overload of both the RA and the RV (RA dilatation leading the RA fibrosis)

42
Q

What could happen if a large ASD went untreated?

A

Can result in enlargement of the right side of the heart and ultimately heart failure

43
Q

What could happen if a large ASD went untreated?

A

Can result in enlargement of the right side of the heart and ultimately heart failure

44
Q

How could an ASD result in pulmonary hyptertension?

A

Increased right sided volume Right atrial and ventricular dilatation Tricuspid annular dilatation (TR) Pulmonary congestion Pulmonary hypertension

45
Q

How do Qp and Qs compare in pulmonary hypertension?

A

Qp > Qs

46
Q

What could cause worsening of a left-to-right-shunt?

A

Any process that increases the pressure in the LV can cause worsening of the left-to-right shunt

47
Q

What could cause worsening of a left-to-right-shunt?

A

Any process that increases the pressure in the LV can cause worsening of the left-to-right shunt; also works on the right heart Systemic hypertension, increases pressure that the LV has to generate in order to open the aortic valve

48
Q

What could cause worsening of a left-to-right-shunt?

A

Any process that increases the pressure in the LV can cause worsening of the left-to-right shunt; also works on the right heart Systemic hypertension, increases pressure that the LV has to generate in order to open the aortic valve

49
Q

If there is a L to R shunt, which ventricle has to push our more blood? What does this result in?

A

Right ventricle; constant overload of the right side of the heart will cause an overload of the entire pulmonary vasculature (pulmonary over-circulation)

50
Q

If there is a L to R shunt, which ventricle has to push our more blood? What does this result in?

A

Right ventricle; constant overload of the right side of the heart will cause an overload of the entire pulmonary vasculature (pulmonary over-circulation)

51
Q

Qp/Qs > 1.5/1.0

A

Problem!! Eventually pulmonary hyptertension will develop

52
Q

What does pulmonary hyptension cause for the RV?

A

Increased afterload (PVR) In addition to increased preload that the shunted blood form the LA to RA caused RV will be forced to generate higher tension/pressures to try to overcome the pulmonary HTN Could lead to RV failure (dilatation and decreased systolic function of the RV)

53
Q

If Left to Right shunt is left uncorrected….

A

Pressure in right heart> pressure in left heart RA pressure > LA pressure Pressure gradient reverses across the ASD Shunt reverses (R to L shunt)= Eisenmenger’s syndrome Oxygen-poor blood gets shunted to the left heart Cyanosis

54
Q

Eisenmenger’s Syndrome

A

Reversal of a L to R shunt to become a R to L shunt Oxygen-poor blood gets shunted to the left heart Causes cyanosis

55
Q

Eisenmenger’s Syndrome

A

Reversal of a L to R shunt to become a R to L shunt Oxygen-poor blood gets shunted to the left heart Causes cyanosis

56
Q

Surgical Correction of ASD’s

A

Percutanous Closure (Amplatzer) Surgical Closure (Primary closure; patch closure)

57
Q

What is the only ASD that could undergo Amplatzer closure?

A

Ostium Secundum

58
Q

Surgical Closure of ASD: Incision

A

Median Sternotomy Right thoracotomy Sub-mammary (under breast tissue on right front of chest/very difficult)

59
Q

Surgical Closure of ASD: Primary

A

Closure by direct vision suture

60
Q

Surgical Closure of ASD: Patch

A

uses pericardial tissue or gore-tex patch for closure

61
Q

SurgicalClosure of ASD: Cannulation

A

Arterial: Aortic VEnous: Bicaval (Total CPB) *Single atrial if infant and small and DHCA is anticipated

62
Q

Surgical Closure of ASD: Venting

A

May use direct venting with a flexible since the heart is open

63
Q

Surgical Closure of VSD: Cardioplegia

A

Antegrade; usually a single dose will suffice

64
Q

Surgical Closure of VSD: Case Notes

A

Very, very quick (5-10 min pump run) Will XC, Stay warm “ drift down temp “ Can be challenging: (on CPB, XC, CPG, warm, correct Ca++, lytes, ABG’s off CPB-MUF)

65
Q

VSD

A

defect in the ventricular septum, the wall dividing the left and right ventricles of the heart

66
Q

What does the ventricular septum consist of?

A

Inferior muscular portion Superior membranous portion

67
Q

Common Regions of VSDs

A

Inlet Outlet (supracristal) Peri-membranous septum Muscular septum

68
Q

What is the #1 type of VSD we will see? (What percent)

A

Per-membranous septum (75%)

69
Q

What percent of VSDs are muscular?

A

20%

70
Q

What percent of VSDs are supracristal (outflow)?

A

5%

71
Q

Muscular VSDs are found in what four locations?

A

Anterior Mid-ventricular Posterior Apical

72
Q

Muscular VSDs are found in what part of the septum?

A

lower part of the septum (they’re surrounded by muscule)

73
Q

The membranous portion of the ventricle is close to what?

A

AV node

74
Q

Membranous VSDs are located near what?

A

Heart valves

75
Q

What is the #1 type of VSD we will see? (What percent)

A

Peri-membranous septum (75%)

76
Q

Membranous VSDs are located near what?

A

Heart valves; can close at any time

77
Q

Supracristal VSD

A

outflow tract VSD sub-valvular in nature

78
Q

Crista Supraventricularis can be considered synonymous with what?

A

Infundibular (or conus) ventricular septum

79
Q

Outflow VSDs are found in what part of the ventricle?

A

Where blood leaves the heart; these are the rarest types of VSD

80
Q

What are the rarest types of VSDs?

A

Outflow VSDs

81
Q

What separates the tricuspid and pulmonary valves and accounts for the more superior placement of the pulmonary valves relative to the aortic valve?

A

Infundibular (or conus) septum

82
Q

What separates the tricuspid and pulmonary valves and accounts for the more superior placement of the pulmonary valves relative to the aortic valve?

A

Infundibular (or conus) septum

83
Q

Infundibular (or conus) septum provides muscular rigid support for what valve?

A

Aortic valve, especially the right coronary cusp (think prolapse)

84
Q

VSD

A

defect in the ventricular septum, the wall dividing the left and right ventricles of the heart

85
Q

Infundibular (or conus) septum provides muscular rigid support for what valve?

A

Aortic valve, especially the right coronary cusp (think prolapse)

86
Q

VSD Blood Flow Pattern During Systole

A

some of the blood from the left ventricle leaks into the right ventricle, passes through the lungs and reenters the left ventricle via the pulmonary veins and left atrium

87
Q

VSD Blood Flow Pattern During Systole

A

some of the blood from the left ventricle leaks into the right ventricle, passes through the lungs and reenters the left ventricle via the pulmonary veins and left atrium Results: Overloads LV Because the left ventricle normally has a must higher systolic pressure (120 mmHg) than the right ventricle (20 mmHG) a left to right shunt persists Elevated RV pressure and volume –> pulmonary HTN

88
Q

VSD Symptoms when there is a larger defect

A

Breathlessness, poor feeding, failure to thrive in infancy (patients with smaller defects may be asymptomatic)

89
Q

Ventricular septum is formed by what?

A

Outgrowth of the muscular ridge at the interventricular foramen; ventricular septum grows upward from the apex of the heart to the base of the heart

90
Q

VSD Cannulation

A

Arterial: Aortic Venous: Bicaval (total Cpb) Single atrial if the infant is small and DHCA is anticipated

91
Q

VSD Repair: Venting

A

May use direct venting with a flexible since the heart is open

92
Q

VSD Repair: Cardioplegia

A

Antegrade, usually with a single dose

93
Q

VSD Repair: Case Notes

A

case is quick depending on VSD location Case may be 32C or DHCA if small infant Can be challenging, but usually you have more time with VSD Ventricular function may be related to the length of time the VSD has been present

94
Q

What could Ventricular function in a pt with a VSD be related to?

A

Length of time the VSD has been present

95
Q

Surgical Correction of VSDs

A

Percutaneous closure *can be tough with VSDs Surgical closure (Primary, patch) *probably not primary

Congenital Defects & Pediatrics COPY (19 decks)

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