CVP Unit 3 Flashcards

1
Q

The amount of blood pumped into the aorta each minute by the heart is called?

A

Cardiac output

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2
Q

CO=VR also CO=SVxHR Stands for?

A

CO- cardiac output= venous return VR CO- cardiac output= stroke volume SVx heart rate HR

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3
Q

The quantity of blood flowing from the veins in the right atrium is called

A

Venous return

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4
Q

CO= equals?

A

VR venous return

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5
Q

What are the factors that influence the VR?

A

Body metabolism Age Body size Gender

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6
Q

How does body metabolism influence VR and CO?

A

Local flow and autoregulation

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7
Q

VR is the summation of all local _____?

A

Blood flows

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8
Q

How does gender influence VR?

A

Men- resting heart rate is 5.6l/m Women 4.9l/m Average 5l/m

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9
Q

Factors affecting HR include-

A

Autonomic innervation Hormones Fitness level Age

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10
Q

Factors affecting SV include?

A

Heart size- hypertrophy Fitness Gender Contractility Duration of contractions Preload and afterload

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11
Q

The cardiac index rises rapidly at what age?

A

10

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12
Q

What does the declining of the cardiac index indicate?

A

That cardiac output is regulated by metabolic activity- decline is indicative of the declining activity- muscle mass and age.

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13
Q

The CO will match the VR through the use of which mechanisms?

A

Frank Starlings mechanism Bainbridge reflex SA node stretch

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14
Q

When increased quantities of blood flow into the heart, the increase blood stretches the walls of the heart chambers- as a result of the stretch the cardiac muscle contract with increased force- this action empties the extra blood that has entered the systemic circulation- this describes the?

A

Frank starlings mechanism

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15
Q

The stretch of the heart also causes the heart to pump faster causing increased heart rate/ contraction- this has an affect of the rhythmicity of the node to increase heart rate. This is called?

A

SA node stretch

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16
Q

The stretch on the right atrium initiates a nervous reflex called _________- this passes to the vasomoter of the brain, then back to the heart by __________nerves and vagis to increase the heart rate.

A

BainBridge reflex Sympathetic nerves

17
Q

In bainbridge reflex what happens to myosin and actin?

A

The are brought to a more nearly optimal degree of overlap for force generation.

18
Q

How are the limits of the heart expressed?

A

Cardiac output curves

19
Q

Define cardiac output curves- how does it effect VR?

A

They demonstrate the effectiveness of cardiac function at different levels of right atrial pressure- which reflects venous return.

20
Q

What is considered a normal maximum of cardiac output?

A

13 L/m 2.5 more than the norm of 5L/m

21
Q

Cardiac output curve above 13L/m is considered?

A

Hypereffective

22
Q

Cardiac output curve below 13L/m is considered?

A

Hypoeffective Add photo

23
Q

2 factors that can cause hypereffective cardio output curve.

A
  1. Nervous stimulation 2. Hypertrophy of the heart muscle
24
Q

What is the cause of the nervous heart stimulation?

A

Combination of sympathetic stimulation and parasympathetic inhibition- which does 2 things

25
Q

Regarding nervous stimulation of the heart- the Combination of sympathetic stimulation and parasympathetic inhibition- does 2 things to increase the pumping effectiveness of the heart- what are they?

A
  1. Greatly increases the heart rate 2. It increases the strength of heart contraction called CONTRACTILITY
26
Q

any factor that decreases the hearts ability to pump blood causes_________.

A

Hypoeffective heart

27
Q
  1. Hypertension 2. Sympathetic inhibition of nervous excitation 3. Heart pathology 4. Arterial blockage 5. Valve or heart disease 6. Myocarditis 7. Cardia hypoxia Are all factors that decrease the hearts ability to pump blood which is called?
A

Hypoeffective heart

28
Q

Pathologically high cardiac output is due to factors that decrease the PERIPHERAL RESISTANCE- while increasing the cardiac output- these include?

A
  1. Berberi 2. Arteriovenous fistula 3. Hyperthyroidism 4. Anemia
29
Q

Not enough thiamine B1- lack of this vitamin causes diminished ability of the tissues to use cellular nutrients- and the local blood flow is compromised cause compensatory peripheral vasodilation, so blood is moving quickly causing higher cardiac output sometimes 2x normal.

A

Beriberi

30
Q

Large amount of blood move from the large artery to the large vein- decreasing the total peripheral resistance and increasing the venous return and cardiac output- Two tubes- an artery and vein connected- could be a defect or fusion or tumor.

A

Arteriovenous fistula

31
Q

Metabolism of the tissues become greatly increased. O2 usage increases, and vasodilator products are released from the tissues. Total peripheral resistance decreases due to local tissue blood flow control. Therefore the venous return and cardiac output increase to 40 to 80 percent above normal

A

Hyperthyroidism

32
Q

Two peripheral effects greatly decrease total peripheral resistance. 1. Reduced viscosity results from RBC concentration. 2. Diminished delivery of O2 to the tissue- causing vasodilation and increase CO

A

Anemia

33
Q
A