Dirty Half Dozen Flashcards

1
Q

LBP - What do physicians look for?

A

Reflex loss
Sensory loss
Motor Weakness

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2
Q

The Dirty Half Dozen Study

A

Looking for mechanical reasons and interventions for persistent LBP

183 patients with “Failed low back syndrome” (79 male, 104 female)

Avg. age 40.8

Disabled avg. 30.7 mo

Found 6 somatic dysfunction present in most patients

  • 2.7% had none of the DHD
  • 55% had three or more of the DHD
  • 75% of the patients returned to full employment and ADL’s following treatment plan and home exercise/stretch regimen despite and average of 2.5 years disability!!!
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3
Q

The Dirty Half Dozen

A

Non-neutral dysfunction within the lumbar spine
Pubic symphysis dysfunctions
Posterior extensions or torsions of the sacrum
Innominate shears
Short-leg, pelvic-tilt syndrome
Muscle imbalance of the trunk and LE

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4
Q

Pubic symphysis

A

Pubic rami must be able to move superiorly and inferiorly during the gait cycle
Aids in distribution of forces from the femur into the pelvis and across the SIJ’s

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5
Q

Pubic symphysis somatic dysfunction

A

75% of patients in the DHD study had pubic symphysis unleveling
Somatic dysfunction causes distortion of the pelvic brim
Increases the force load across the SIJ’s

Most commonly right inferior (32%), left superior (25%)

  • Hypertonic adductors
  • Weak lower abdominals
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6
Q

Innominate dysfunctions

A

24% of DHD patients showed the presence of innominate shears

  • 19% superior
  • 4% inferior

Shears, while not common, are most troublesome
- SIJ dysfunction reflexly inhibits gluteus maximus firing destabilizing pelvis and lumbar spine

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7
Q

Short-leg, pelvic-tilt

A

63% of DHD study patient had this present

Short leg on one side creates a constellation of structural dysfunctions
- LE somatic dysfunction
- Innominate rotations
- Pelvic unleveling 
- Spinal asymmetry
---- Coronal plane (sidebending)
----- Chronic Type I dysfunctions
----- Muscular imbalance
Shoulder asymmetry
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8
Q

Innominate rotations

A

NOT part of the DHD, but may be indicative of the short leg, pelvic tilt syndrome
Axis of innominate rotation posterior to the acetabulum, influencing the rotational motion
May also be indicative of muscular imbalance

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9
Q

Checking for short leg/pelvic tilt

A
Standing flexion test
Foot arches
Greater trochanter height
Iliac crest height
Pelvic side shift
Presence of persistent Type I dysfunctions
Shoulder unleveling
CCJ unleveling
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10
Q

Sacral dysfunctions

A

48.8% of DHD study patients had a restriction to anterior nutation of the sacrum

  • Backward torsion
  • — L on R = 17.5%
  • — R on L = 15.8%

Bilaterally extended= 15.3%

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11
Q

Posterior sacral dysfunctions are painful! - form closure

A

Form closure
Flat surfaces of the SIJ are susceptible to shear forces
Wedge shape of the sacrum in the AP as well as the vertical planes protects against this

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12
Q

Force closure

A

Begins in the LE through force coupling
Heel strike: peroneus longus and biceps femoris into the sacrotuberous ligament and across the ipsilateral SIJ
Midstance: downward force of Biceps femoris contraction countered by upward contraction of ipsilateral gluteus maximus and contralateral latissimus dorsi through thoracolumbar fascia

Creates a muscle-tendon-fascial sling that is temporally precise: any delay in firing of these mm decreases the stability of the entire mechanism

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13
Q

Counternutation dysfunctions

A

by definition, decrease the ability of the sacrum to nutate
Leads to loss of form closure
Leads to arthrokinematic inhibition of Gluteus Maximus diminishing force closure and destabilizing the muscle-tendon-fascia sling

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14
Q

Anterior sacral torsions

A

Not part of the Dirty Half Dozen

Recurrent presence may be indicative of short leg/pelvic tilt syndrome

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15
Q

Unilateral sacral dysfunctions

A

Present in chronic recurrent LBP with minimal radiation to the LE below the buttocks
Standing for longer periods of time increases pain; walking sitting decreases pain
Usually one legged standers (creates postural imbalance)
Most common pattern:
- Left superior pubic symphysis
- Left unilateral sacral flexion
- Left posterior innominate
- L5 ERS left
- Hypertonic erector spinae, weak abdominals, weak pelvic stabilizers (Glut med/min)

Tx:

  • Correct somatic dysfunctions present
  • Postural retraining
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16
Q

Lumbar dysfunctions

A

84% of DHD patients had Type II dysfunctions of the lumbar spine
ERS = 34.9%
FRS = 83.6%

Most had multiple dysfunctions and were clustered at L4 & L5

17
Q

Type II dysfunctions

A

inhibit transversospinal function
These work synergistically with transversus abdominus and internal obliques, thoracolumbar fascia, iliopsoas and quadratus lumborum
Inhibition of transversospinal mm leads to decoupling of entire core mechanism

–> Loss of core control

18
Q

Muscle imbalance

A

Greater than 95 % of population had significant muscle imbalance between trunk and lower extremities.
Primarily tight hip flexors, tight piriformis, tight adductors, weak abdominals, weak gluteus maximus and medius.
Poor proprioceptive balance and trunk rotator control.

19
Q

Core control is key

A

Abdominal muscles notoriously weak

  • Transversus abdominus essential for core control
  • TA fires first with all upper and lower extremity motion
  • In patients with persistent or recurrent LBP TA shows delayed firing
  • Leads to decreased core control and increased instability
20
Q

Treatment sequence

A
Pubic symphysis dysfunctions
Innominate shears
Sacral dysfunctions
Lumbar Type II’s
Correct short leg if present
Correct muscle imbalance with home exercise and retraining program