DKA / HHS Flashcards
Fast Facts on DKA:
- ____ insulin present –> gluconeogisis and lipolysis –> _______ —> and ________
- More common in Type ___ DM
- Onset is ____ ( _____
- Mortality rate
- NO insulin present –> gluconeogisis and lipolysis –> ketone production –> ketoacidosis and hyperglycemia!
- More Common in Type 1 DM
- Onset is 13.9
- Mortality rate
What can cause DKA / HHS to develop? (7)
- infection
- MI
- Tx errors with insulin
- Dirarrhea / vomiting
- Stroke
- Trauma
- Pancreatitis
The stresses that lead to DKA / HHS developing causes what 3 things to be released?
- glucagon
- cortisol
- catecholamines
What are the S/S of DKA?
polyuria, polydipsia, polyphagia, blurred vision, weakness, headache, orthostatic hypotension, anorexia, n/v, abdmn pain, acetone (fruity) breath, hyperventiliation, altered LOC
Why would a pt. with DKA be hyperventilating?
In an attempt to breath off the acidosis
Dx for DKA:
- Elevated (5)
- Low (1)
- Accumulation of what? (1)
- Changes in what electrolytes? (4)
- Elevated: BG, Creatinine, BUN, Hgb, Hct
- Low bicarb
- Accumulation of ketones in blood and urine
- Na+, K+, PO4, Cl-
Treatment involves what 3 key concepts for DKA
- Rehydration
- Restoring electrolytes
- Reversing acidosis
Nursing care Includes the nurse:
- Admin IV fluids (NS or .45% NS, 6-10 L at 200-300 ml/hr for 2-3 hours)
- Admin IV insulin
- Admin K+
- Assessing I/O (renal function)
- ECG
- VS
- ABGs
- BG
- Monitor electrolytes
- Admin medications for what may have been the stressor to cause DKA
- Chest X ray, blood samples, urinlaysis (attempting to find infection)
boom
Why might K+ levels decrease once the pt. is receiving insulin? (3)
- Rehydration dilutes the concentration of K+
- Renal excretion of K+
- Insulin then causes K+ to move back into the cells, which can then lead to low levels of it in the bood stream
HHS:
- Insulin ____ –> gluconeogesis & lipolysis mainly ______ –> ketones mild/ ____ –>pH normal/mild
- Mostly type __ DM
- Onset _____
- BG > ____
- Mortality rate _____
- Insulin present –> gluconeogesis & lipolysis mainly inhibitied –> ketones mild / absent –> pH normal / mild
- Mostly Type 2 DM
- Onset days –> weeks (develops more slowly)
- BG >33.3
- Mortality rate 10-40%
S/S of HHS include (4)
- hypotension
- profound dehydration (dry mucous membranes, poor skin turogour)
- tachycardia
- varying neurological signs
Treatment of HHS is similar to DKA. What are the considerations that are different?
Consider the neuro symptoms may take 3-5 days to clear
Consider this is often in older adults so consider age/factors /education