drugs Flashcards
RAAS-ACE INHIBITORS
END IN PRIL
SIDE EFFECTS-COUGH, ANGIOEDEMA, HYPERKALEMIA
BECAUSE YOU ARENT MAKING ANGIOTENSIN 2 SO ITS NOT MAKING ALDOSTERONE SO YOU LOSE NA+ AND H20 THEREFORE RETAINING K+
ANGIOTENSIN 2 BLOCKER-SARTANS
DOESNT INTERFERE WITH PRODUCTION OF ANGIOTENSIN, JUST BLOCKS THE RECEPTOR.
USE IF PT CANT TAKE ACEI BECAUSE OF ACE COUGH OR ANGIOEDEMA (STILL RISK)
CAUSES HYPERKALEMIA
ALDOSTERONE ANTAGONIST
SPIRONOLACTONE/ EPPLERANONE
ALDOSTERONE ANTAGONIST
SPIRONOLACTONE/ EPPLERANONE
BLOCKS ALDOSTERONE RECEPTORS
ELIMINATES VOLUME EXPANSION (ESPECIALLY FOR HYPER ALDOSTERONISM)
HORMONAL SIDE EFFECTS LIKE GYNECOMASTIA, DEEPENING OF VOICE
CAUSES HYPERKALEMIA
ALPHA 2 AGONISTS
CLONADINE
ALPHA 2 AGONISTS
CLONADINE
BLOCKS NOR EPI
NOR EPI CONTINUES TO BUILD
CAUSES REBOUND HTN IF ABRUPTLY STOPPED, DROWSINESS, XEROSTOMIA
RESERPINE
DIMINISHES SYNTHESIS OF NOR EPI
S/E IS DEPRESSION
BETA BLOCKER
LOL
ATENALOL, METATROPROLOL
DECREASES HEART RATE AND CONTRACTIITY
ALPHA AND BETA BLOCKER
CARVETALOL AND LABETALOL
ALPHA 1 ANTAGONIST (SIN)
ALPHA 1 RECEPTORS ARE ON ARTERIES AND VEINS
SIN DRUGS DIALATE ARTERIES AND VEINS
SIDE EFFECT- 1ST DOSE RESPONSE
TAMSALOSIN, DOXAZASIN
DIHYDROPYRIDINE CALCIUM CHANNEL BLOCKERS
SUBSTANTIAL AFTERLOAD REDUCERS NIFEDIPINE PROFOUND BLOCKADE OF CALCIUM CHANNELS IN ARTERIES NO EFFECT ON HEART CAN TRIGGER BARORECEPTORS
DIHYDROPYRIDINE CALCIUM CHANNEL BLOCKERS- S/E
DIZZINESS HA **PERIPHERAL EDEMA GINGIVAL HYPERPLASIA REFLEX TACH FAST ACTING NIFEDIPINE LINKED TO INCREASED MORTALITY IF TAKEN IN EXCESS, IT LOSES SELECTIVITY
NONDIHYDROPYRIDINE CHANNEL BLOCKERS
BLOCK CALCIUM CHANNELS ON ARTERIES AND ON THE HEART
NONDIHYDROPYRIDINE CHANNEL BLOCKERS
VERAPAMIL ANTI-ARRHYTHMIA ACTS LIKE A BETA BLOCKER ON HEART DECREASES AFTERLOAD, REDUCES CO THERAPEUTIC USE- ANGINA CAUSES BAD CONSTIPATION! CP450 INHIBITOR
NONDIHYDROPYRIDINE CHANNEL BLOCKERS
DILTIAZEM AND VERAPIMIL