Exam 3: Stomach Flashcards

1
Q

What are the (2) types of bloat in ruminants?

A

Primary = frothy bloat
Secondary = gaseous bloat (obstruction)

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2
Q

What is the pathogenesis of frothy bloat?

A

Change in diet to some types of legumes such as alfalfa or grain concentrate –> lower rumen pH –> stable foam –> foam mixed with rumen contents blocks the cardia –> failure to eructate and bloat

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3
Q

What is the pathogenesis of grain overload and ruminitis-liver abscess complex?

A

Rapidly fermentable grain
Acidosis = low rumen pH
Damage to mucosa = F. necrophorum overgrowth
Rumen wall abscess, inflammation
Bacterial access and travel to liver via portal vein
Liver abscesses

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4
Q

What is the sequela of grain overload and rumitis-liver abscess complex?

A

Multifocal hepatic abscesses
Caudal vena caval pyremia (fatal septic embolism)
Pulmonary embolic pneumonia and hemorrhage (rupture of pulmonary artery)
Pulmonary hemorrhage = hemoptysis, nasal hemorrhage; sudden death due to blood loss

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5
Q

Where do pulmonary emboli arise from with the ruminitis-liver abscess complex?

A
  1. rupture of a hepatic abscess into the caudal vena cava
  2. vegetative valvular endocarditis (tricuspid valve)
  3. jugular thrombosis
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6
Q

What is the gross appearance of mycotic ruminitis?

A

Circular, well delineated lesions
Fungi can be angioinvasive resulting in infarction of vessels

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7
Q

What dog breeds are associated with gastric dilation and volvulus?

A

Large “barrel” chested dogs
Rarely pigs, guinea pigs

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8
Q

What is the pathogenesis of gastric dilation and volvulus?

A

Dilated stomach and displacement –> compression of diaphragm, vena cava, and portal vein –> decreased venous return –> decreased CO –> shock

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9
Q

How do you distinguish antemortem from postmortem rupture?

A

Antemortem = evidence of hemorrhage

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10
Q

What is the pathogenesis of displaced abomasum?

A

High grain diet and hypocalcemia –> fermentation –> atony –> production of gas –> recent parturition –> displacement of the abomasum to the cranial left abdomen –> obstruction –> HCl secretion –> chloride sequestration –> hypochloremia and metbolic alkalosis

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11
Q

How are left and right abomasal displacement different?

A

RDA can be fatal
20% can have volvulus
Can lead to vagal indigestion

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12
Q

What are the causes of gastric ulcers?

A

Mechanical - FB
Chemical
Drugs - steroids, NSAIDs
Stress
Hepatic disease
Vasculitis
Gastritis
Gastrinomas
Mast cell tumors
Addison’s
Equine gastric ulceration syndrome
Pigs (finely ground feed)
Idiopathic

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13
Q

What are the clinical signs of gastric ulcers?

A

Pale liver
Melena
Pale Body
Fibrinous/fibrous peritonitis

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14
Q

What causes braxy?

A

Clostridium septicum

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15
Q

What are the common gastric parasites of horses?

A

Gastric bots (gasterophilus intesinalis, nasalis)
Draschia megastoma (tumor like nodules, ulcers, glandular mucosa)
Trichostrongylus

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16
Q

What are the common gastric parasites of ruminants?

A

Haemonchus contortus, placei (blood loss, PLE, bottle jaw)
Ostertagia (mucosal metaplasia, hyperplasia)
Trichostrongylus

17
Q

What are the common gastric parasites of swine?

A

Hyostrongylosis

18
Q

What are the benign gastric neoplasms?

A

Adenoma (epithelial)
Leiomyoma (sm muscle)

19
Q

What are the malignant gastric neoplasms?

A

Squamous cell carcinoma (horses)
Adenocarcinoma (dogs)
Leiomyosarcoma
Lymphoma (abomasum)

20
Q

What are the gross characteristics of gastric lymphoma?

A

Smooth surface
White, bulges on cut surface
Soft