Final Exam Flashcards

Know it all.

1
Q

Aural Atresia reconstruction time periods

A

Microtia repair: 6-7 y/o

Atresia repair: 1-2 years later

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2
Q

Exostoses side effects:

A

Traps debris
impinges on TM
reduces visibility of TM
can cause CHL

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3
Q

What are exostoses

A

benign bony growths in medial ear canal secondary to cold water exposure (scuba)

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4
Q

Why can otitis externa become concerning?

A

Infection of pinna or ear canal can turn from brief problems to life-threatening situation in immunocompromised individuals

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5
Q

What is otitis externa?

A

Infection of pinna or ear canal that presents with mucopurulent matter/otorrhea, redness, edema, and pain.

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6
Q

What is a furuncle?

A

Abscess from hair follicle. Swelling

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7
Q

Otomycosis:

A

Fungal infection of the ear canal. Most seen in hearing aid users.

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8
Q

Otomycosis presentation:

A

Pruritis, otorrhea, reduced hearing, otalgia (otoscopy: fungal hyphae)

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9
Q

Dermatitis (Alt names and etiology)

A

Chronic external otitis or seborrheic external otitis
Etiology: irritants (q-tips, hairspray) and secondary to bacterial infection.
Chronic drainage causing pruritis and edema.

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10
Q

What is myringitis

A

inflammation and infection, presence of painful vessicles on TM.

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11
Q

Tm Perforation

A

Usually heal by themselves or may require tympanoplast if not healing. Concerns: CHL & Cholesteatoma

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12
Q

Myringoplasty Vs Tympanoplasty

A

M: TM reconstructive surgery
T: Middle ear reconstructive surgery.

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13
Q

Tympanosclerosis

A

Calcified plaques or connective tissue. Associated with chronic otitis media & other inflamm disorders, PE tubes.

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14
Q

Tympanosclerosis presentation:

A

Tymps: A or A(s)
AR: Depends on severity, CHL pattern (probe ear)
Audio: CHL possible if more than TM is involved

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15
Q

TM Perforation etiology and presentation:

A
Etiology: Trauma/blast/penetration/head trauma
Presentation: 
Otoscopy-perf
Tymp: Type B, HIGH volume
Audio: CHL up to 50 dB (depends on size)
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16
Q

Retracted TM Etiology & presentation

A

etiology: Hx of secretory or ROM, ETD
presentation:
otoscopy: small retraction pockets to more global involvement
tymp: varies (usually not A)
Reflexes: varies
Audio: possible CHL

17
Q

Cholesteatoma (The 3 types)

A

Congenital- Ant to malleus
-Dx’d between 6mo to 5y
-May obstruct ET, CHL, chronic middle ear effusion
Primary - TM Retraction, ETD, or infection (acquired after impingement on ossicles = CHL)
Secondary- Injury to TM (perf, PE tube)
-Squamous cells migrate through TM to ME. (foul smell due to bacterial colonization)

18
Q

Hallmark Sign of Cholesteatoma:

A

Otorrhea w/o otalgia

19
Q

Cholesteatoma Presentation:

A

Otorrhea w/o otalgia (possibly dizziness)
Tymp: Variable
Reflexes: Variable
Audio: CHL (obstruction of ME space), mixed (ossicular chain damage), or SNHL (Erosion into cochlea)

20
Q

Glomus Jugulare

A

Develop from tissue in/near jugular bulb

May extend through skull base, into and through temporal bone

21
Q

Glomus Tumors

A

Slow glowing paragangliomas.
Develop from neuroendocrine (PNS cells, highly vascularized)
Most common tumor of middle ear.

22
Q

Glomus tumor MC signs and symptoms:

A
CHL
Pulsatile tinnitus (hallmark)
Aural fullness
Otalgia
Otorrhea
Bruit (audible vascular flow)
Expansion into mastoid: CN VII paralysis, labyrinthine erosion (vertigo, SNHL)
23
Q

Glomus tumor treatment

A

Surgical removal (mastoidectomy)
Radiation
Therapy (for CN involvement)
Audiologic management for residual HL

24
Q

Eustachian Tube Functions

A

Ventilate ME
Protect ME from nasopharynx
Clear secretions from ME
(Should be closed at rest)

25
Q

Patulous Eustachian Tube Symptoms:

A

Autophony
breathing sounds in ear
Head or aural pressure
Tinnitus