Final sweep 1 Flashcards

1
Q

Med history - high risk

A

ASA 3+

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2
Q

Vasoconstrictor intolerance can be due to

A

Significant cardiac disease
Anti-hypertensive medications may increase activity of vasoconstrictors
Antiadrenergics
Nonselective beta-blockers (propranolol)

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3
Q

Failures occur in dentistry as a result of many factors some of which can be controlled by the operator whilst others are unavoidable.
The number of failures can be reduced by adhering to accepted treatment procedures and by avoiding ‘short cuts’.
May be a consequence of failures of the

A

coronal restoration rather than being due to failure of the root canal filling itself.

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4
Q

Disasters are usually related to —– and they may have detrimental effects on the outcome of treatment in the long term, eventually becoming ——. Endodontic disasters will require special techniques to salvage them.

A

operator errors

catastrophes

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5
Q

Catastrophes usually result in

A

loss of the tooth and every effort should be made to prevent such problems from occurring.

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6
Q

Ledged canal more likely

A

IF greater than 30 degree curvature, and especially if you don’t have good access (file is getting deflected out of canal and you don’t have straight line access, ledge is more likely to happen).

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7
Q

Zipped canal - May need to repair with

A

MTA depending on location of root perforation

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8
Q

Strip perforation - Pretty much drilling

A

out a wall. More common with rotary stuff like GG.

Almost always around a curved canal.

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9
Q

Strip perforations are more likely to occur
at the —— aspect of maxillary premolars
and distal aspect of —— roots of
mandibular molars

A

MESIAL

MESIAL

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10
Q

bleach incident - Most likely to occur in:

A

Maxillary posterior

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11
Q

With bleach incident, DO NOT try to flush the tooth out with saline –

A

pushes it further down.

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12
Q

Vital (Irreversible pulpitis or normal pulp):

A

thermal sensitivity should dissipate; no swelling expected because no “infection” (just tender gingiva from rubber dam clamp, or may feel swollen);
expect: tender to touch and biting; “achy” for up to 1 week.
Most of these patients feel better after treatment!

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13
Q

Necrotic:

A

swelling may develop if it hasn’t already
may get worse before it gets better
always warn your patient of a “flare-up.”

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14
Q

Ibuprofen (Advil):

A

600 mg every 6 hours x 24-48 hours postop

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15
Q

Acetaminophen (Tylenol/ES Tylenol)

A

1000 mg every 4-6 hours postop

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16
Q

Combination narcotics/acetaminophen

Requires Rx.

A

Example: Norco 5/325 #20 tabs. Take 1 tab q4-6 hrs prn pain. 0 refills.

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17
Q

Diagnosis: The identification of a disease from its signs and symptoms. Include the tooth

A

, pulpal diagnosis and periodontal diagnosis.

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18
Q

Disposition:

A

List the option for treatment that the patient has chosen, and where the patient is to go to have treatment accomplished. “Refer to ________ for _______ tooth #______”

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19
Q

Healed:

A

functional, asymptomatic; no or minimal radiographic periradicular pathosis

20
Q

Nonhealed:

A

nonfunctional, symptomatic; with or without radiographic periradicular pathosis

21
Q

Healing:

A

periradicular pathosis present, but asymptomatic and functional; or, with or without radiographic pathosis, symptomatic but for which intended function is not altered

22
Q

Functional:

A

treated tooth or root that is serving its intended purpose in the dentition

23
Q

Luxations

A

(tooth moved)

24
Q

Luxation is the most common injury to —–

Crown fractures are more common in ———

A

primary dentition.

permanent dentition.

25
Q

Enamel infraction:

A

crack
in enamel with no loss of
tooth structure

26
Q
Enamel fracture (uncomplicated
crown fracture):
A

fracture with loss

of enamel

27
Q

Enamel-dentin fracture (uncomplicated

crown fracture):

A

fracture with loss of
enamel and dentin, but not involving
the pulp

28
Q

Concussion -

A

Injury to tooth-supporting structures without abnormal loosening or displacement of the tooth, but with marked reaction to percussion

29
Q

Subluxation

A

Injury to tooth and supporting structures with abnormal loosening, but without displacement of the tooth

30
Q

Extrusive luxation

A

Partial displacement of the tooth out of its socket

31
Q

Lateral luxation

A

Displacement of the tooth in a direction other than axially; accompanied by comminution or fracture of the alveolar socket

32
Q

Intrusive luxation

A

Displacement of the tooth into the alveolar bone; accompanied by comminution or fracture of the alveolar socket

33
Q

RCT absolutely necessary when

A

pulp connection to periodontium has been severed (avulsion, extrusion, intrusion, lateral luxation, severe root fracture).

34
Q

Hyaline layer of Hopewell-Smith binds

A

cementum to dentin

35
Q

Lymphatics: in —–
Remove ——-
Drain into ——

A

periphery of the pulp

inflammatory exudates, transudates, cellular debris

submental, submandibular or cervical lymph nodes, then empty into the subclavian and internal jugular veins

36
Q

Sympathetic fibers alter

A

pressure, flow, distribution of blood at precapillary sphincters

37
Q

Sensory fibers release —— via axon reflexes - - this neurogenic inflammation mechanism results in increased blood flow and increased capillary permeability

A

neuropeptides

38
Q

Sympathetic motor innervation:

A

T1, C8, T2 via superior cervical ganglion – maintains vasomotor tone in precapillary sphincters which controls pressure and distribution of blood

39
Q

NO —- innervation of the pulp

A

parasympathetic

40
Q

Symptomatic apical periodontitis

A

Inflammation, usually of the apical periodontium, producing clinical symptoms including a painful response to biting and/or percussion or palpation. It might or might not be associated with an apical radiolucent area.

41
Q

Asymptomatic apical periodontitis

A

Inflammation and destruction of apical periodontium that is of pulpal origin, appears as an apical radiolucent area, and does not produce clinical symptoms.

42
Q

Acute apical abscess

A

An inflammatory reaction to pulpal infection and necrosis characterized by rapid onset, spontaneous pain, tenderness of the tooth to pressure, pus formation, and swelling of associated tissues.

43
Q

Chronic apical abscess

A

An inflammatory reaction to pulpal infection and necrosis characterized by gradual onset, little or no discomfort, and the intermittent discharge of pus through an associated sinus tract.

44
Q

Acute apical abscess (AAA)

A

An inflammatory reaction to pulpal infection and necrosis characterized by rapid onset, spontaneous pain, tenderness of the tooth to pressure, pus formation, and swelling of associated tissues.

45
Q

Symptomatic apical periodontitis (SAP)
Inflammation, usually of the apical periodontium, producing clinical symptoms including a ———–. It might or might not be associated with an apical radiolucent area.

A

painful response to biting and/or percussion or palpation

46
Q

Asymptomatic apical periodontitis (AAP)

Inflammation and destruction of apical periodontium that is of pulpal origin, appears as an

A

apical radiolucent area, and does not produce clinical symptoms.