mental health1 Flashcards

1
Q

obsessions are defines by

A

recurrent and persistent thoughts, urges, or images that are experiences, at some time during the disturbance, as intrusive and unwanted, and that in most individuals cause marked anxiety or distress
the individual attempts to ignore or suppress such thoughts, urges, or images, or to neutralize them with some other thought or action

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2
Q

compulsions are defined by

A

repetitive behaviours or mental acts that the individual feels driven to perform in response to an obsession or according to rules that must be applied rigidly
the behaviours or mental acts are aimed at preventing or reducing anxiety or distress, or preventing some dreaded event or situation, however, these behaviours or mental acts are not connected in a realistic way with what they are designed to neutralize or prevent, or are clearly excessive

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3
Q

DSM V criteria for ocd

A

presence of obsessions, compulsions, or both
the obsessions or compulsions are time consuming or cause clinically significant distress or impairment in social, occupational, or other important areas of functioning
the obsessive compulsive symptoms are not attributable to the physiological effects of a substance
the disturbance is not better explained by the symptoms of another mental disorder

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4
Q

prevalence of ocd

A

About 1.2 % will develop OCD over a lifetime

Early onset predicts poorer treatment outcomes

Females are diagnosed slightly more than males

Higher rates among individuals who are
Young, divorced or separated, and unemployed

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5
Q

prevalence of ocd across the lifespan

A

Children & Adolescence
1-3%
More males than females
Young children are more difficult to diagnose

Older Adult
Late onset is more likely to occur in females
Increased occurrence of PTSD

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6
Q

comorbidities of ocd

A

Mood disorders
Particularly depression and bipolar disorder

Anxiety and panic disorders

Impulse control disorders
Eating disorders

Tourette syndrome
Frequently occurs with OCD

Personality disorders
Occurs in about 80% of individuals diagnosed with OCD

Somatic Disorder

Substance disorders
Occurs in about 1/3 of individuals diagnosed with OCD

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7
Q

genetics and ocd

A

Genetic
Strong Heritability

First degree relatives

Increased prevalence if relatives also have Tourette’s syndrome, an anxiety disorder, or a mood disorder

Polygenic disease

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8
Q

neuropathological and ocd

A

Neuropathological

Hyperactivity of the

orbitofrontal cortex
Has the most dopamine receptors
anterior cingulated cortex
Part of the limbic system
caudate nucleus 
Part of the basal ganglia where GABA mediates dopamine

Increased cerebral glucose metabolism

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9
Q

biochemical and ocd

A

Biochemical

The most studied neurotransmitter in conjunction with OCD is Serotonin

This is based on the effectiveness of Serotonin specific reuptake inhibitors (SSRI)

Serotonin is one of the transmitters that
Initiates the fight or flight response
Influences how emotions are prioritized in the amygdala
Influences how meaning is connected to memories in the pre-frontal cortex

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10
Q

psychodynamic and ocd

A

Psychodynamic
Arise form unconscious defense mechanisms
Isolation – separation of affect from thoughts and impulse
Undoing – performing a behavior to avoid the consequences of another behavior
Reaction formation – a behavior or attitude that opposes another behavior or attitude

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11
Q

behavioral and ocd

A

Behavioral
Conditioned stimuli
Behaviors that would typically be considered neutral, provoke anxiety
To manage anxiety individuals begin to perform other behaviors
The more a behavior decreases anxiety the more frequently an individual will engage in the behavior

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12
Q

assessments for ocd

A

MSE

Risk Assessment

Physical Assessment

Family

SEDoHs

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13
Q

mental status exam for ocd

A
General Appearance
Affect & Mood
Speech & Language
Thought Process
Thought Content
Perceptual Functioning
Cognitive Functioning
Insight
Judgment
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14
Q

risk assessment for ocd

A

Assess both the obsession and compulsion for
Type
Severity

Inquire about
Access to means
Protective factors

It is importance to:
Allow enough time for the assessment
Gather collateral information

Consider insight and judgment
Is there increased impulsivity? Does the patient feel the need to punish themselves?

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15
Q

physical assessment for ocd

A
Dermatological
Includes assessments of skin and hair
Behaviors include:
Repetitive hand washing
Excessive cleaning (skin breakdown also due to cleaning agents)
Skin picking
Pulling out hair

Dental Care
Behaviors include:
Excessive teeth brushing (leads to decreased tooth enamel)

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16
Q

possible outcomes of physical assessment for ocd

A
Possible Outcomes
Osteoarthritis
Trichotillomania
Body dysmorphic disorder
Infection 
Electrolyte imbalances
Particularly if there have been changes in nutrition due to decreased enamel.

*These outcomes would require additional assessment

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17
Q

family assessment for ocd

A

Individuals are more likely
To remain single
Have higher rates of celibacy

Evaluate the families understanding of OCD
Is education required?
Is the family ready to change their behaviors?
How much is the family able to adapt?
Is the family enabling the patient’s compulsions?
Does the family need assistance caring for the patient?

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18
Q

impact of sedohs and ocd

A
Impact of SEDoHs
Financial 
Can the patient work?
Is the patient safe alone or is constant caregiving required ?
Is funding required and/or available

Outside supports
Does the patient have social supports (friends, volunteer groups, etc.)?

Education
What education has the patient completed?
How does this impact employment?
How does this impact peer groups?

Social Functioning
Is it awkward to participate in social activities?

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19
Q

consequences of ocd on the patient

A

Alienation from family and friends

Lack of intimate relationships

Additional health problems
Co-morbid conditions
Con-current conditions

Legal
Acting on obsessions

Financial
Unable to work

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20
Q

body image definition

A

a mental picture of how one’s own body looks

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21
Q

body image distortion definition

A

an individual perceives his/her own body different than the world perceives it

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22
Q

interoceptive awareness definition

A

describes the sensory response to emotional and visceral cues, such as hunger

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23
Q

purge definition

A

purposeful evacuation of stomach or bowel contents through artificial means such as vomiting or laxatives

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24
Q

DSM V criteria for anorexia nervosa

A

restriction of energy intake relative to requirements, leading to a significantly low body weight in the context of age, sex, developmental trajectory, and physical health. significantly low weight is defined as a weight that is less than minimally normal or, for children and adolescents, less than minimally expected
intense fear of gaining weight or becoming fat, or persistent behaviour that interferes with weight gain, even though at a significantly low weight
disturbance in the way in which one’s own body weight or shape is experienced, undue influence of body weight or shape on self evaluation, or persistent lack of recognition of the seriousness of the current low body weight

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25
Q

prevalence of anorexia

A

About 0.5 – 1%
Onset is in early Adolescence (age 14-16)
Females
10x more likely than males
Early menses (age 10/11) is an important predictor
Usually a chronic condition with relapses
Precipitated by distorted body image
Impacted by
Culture
SEDoHs (Decreased socioeconomic status, Decreased education

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26
Q

anorexia across the lifespan

A

Most often begin in childhood and adolescence

May have serious dieting before the signs of starvations are noticeable

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27
Q

comorbidities of anorexia

A

Anxiety disorders
OCD predates anorexia nervosa by 5 years

Perfectionism

Depression

Co-morbidities seem to resolve when the anorexia is successfully treated

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28
Q

changes in the orbitofrontal cortex and striatum and anorexia

A

The orbitofrontal cortex has the most dopamine receptors in the brain
Decreased dopamine may decrease the pleasure an individual experiences with eating

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29
Q

genetics and anorexia

A

Strong Heritability

First degree relatives account for 50-80% of heritability

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30
Q

neuroendocrine and neurotransmitter and anorexia

A

Increase in endogenous opioids
Patients exercise excessively

Decreased thyroid function
Related to malnutrition and chronic stress this puts on the body systems

Decreased serotonergic functioning
Decreased weight

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31
Q

history and anorexia

A

It has been thought that the underlying psychological factors have been related to unresolved conflict in developing autonomy.
Dieting and weight control were a means to defend against feelings of inadequacy

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32
Q

internalization of peer pressure and anorexia

A

learned from peers

Reflect: How is a therapeutic environment created on an in-patient child/adolescent psychiatric unit?

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33
Q

body dissatisfaction and anorexia

A

Comparison with others (especially in the media) results in a struggle to develop his/her own identity
Implications when the individual falls short of the ideal
Reflect: Why is it important for nurses to educate adolescents and families about normal growth and development patterns, in particular the increased weight gain that supports changes that occur in puberty?

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34
Q

societal theories and anorexia

A

Societal messages
Appearance
Roles
What roles are women most likely to pursue?
If a women is in a significant leadership role, are there expectations for how they will look and behave?
Achievements
Do we have expectations for what successful people looks like?

Confusion between character and appearance
“It doesn’t matter what is on the outside. It’s what’s on the inside that matters?”
Does this align with values and beliefs about equity?
Do we live out these values and beliefs?

Awareness of obesity
Media awareness of plus sized models
Dedicated clothing stores for particular sizes

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35
Q

risk factors for anorexia

A

Puberty

Low self-esteem

Dieting/Attitudes about healthy eating

Feelings of inadequacy

Athleticism

SEDoHs

36
Q

assessments for anorexia

A

MSE

Risk Assessment

Physical Assessment

Family

SEDoHs

37
Q

general appearance for anorexia

A

Dry skin, sunken facial features,
Wears baggy clothing, long sleeve shirts, long pants, layers
May look older than actual age
May have difficulties with balance

38
Q

affect and mood for anorexia

A

Blunted affect
May be unable to describe mood
Ambivalence (affect incongruent with context)

39
Q

speech and language for anorexia

A
Speech generally at normal rate, volume, and rhythm
Circumstantial speech (talking around the core problem – around the circumstance)
40
Q

thought process for anorexia

A

Obsessional thinking
Irrelevant details
May eventually get to the point

41
Q

thought content for anorexia

A

*Especially important to listen for strengths
Often hopelessness and helplessness are present
Depressive or anxious cognitions (may be about talking about the subject or other concerns or worries)

42
Q

perceptual functioning for anorexia

A

Maybe altered due to physical functioning (significantly altered electrolytes)

43
Q

cognitive functioning for anorexia

A

Maybe altered due to physical functioning and physical changes in the brain
Decreased ability to remember, concentrate

44
Q

insight for anorexia

A

May not acknowledge there is a problem even thought they become physically unwell

45
Q

judgement for anorexia

A

Poor. Continue to want to loose weight

46
Q

risk assessment for anorexia

A

7% - 10 % mortality rate

Suicide is the leading cause of death
Use highly lethal means
Inquire about 
A plan (time/date/place)
Access to means (even if there is no plan as insight and judgment are usually impaired)
Protective factors

Collaborative information

47
Q

physical assessment for anorexia

A

Multiple physical systems are compromised by starvation

48
Q

musculoskeletal and anorexia

A

Loss of muscle mass, fat (leads to osteoporosis)

49
Q

metabolic and anorexia

A

Hypothyroidism
Hypoglycemia
Decreased insulin sensitivity

50
Q

cardiac and anorexia

A

Bradycardia and ventricular tachycardia
Arrhythmias (atrial and ventricular premature contractions)
Hypotension
Loss/diminished cardiac muscle
Prolonged QT interval (consider potentiated effect with antidepressants like Remron)
Sudden death syndrome

51
Q

gastrointestinal and anorexia

A

Delayed gastric emptying
Bloating
Constipation/diarrhea (outcome of continued laxative use and poor nutrition)
Abdominal pain

52
Q

reproductive and anorexia

A

Irregular menses to Amenorrhea
Low levels of luteinizing hormone
Low levels of follicle-stimulating hormone

53
Q

dermatological and anorexia

A

Dry, cracking skin & brittle nails (dehydration)
Lanugo (fine baby –like hair) over the whole body
Edema
Bluish hands and feet (result of poor nutrition and dehydration –decreased blood volume and decreased iron to carry oxygen)
Thinning hair

54
Q

hematological and anorexia

A

Leukopenia
anemia,
Thrombocytopenia
Increased cholesterol

55
Q

neuropsychiatric and anorexia

A
Abnormal taste sensation (related to zinc deficiency)
Depression
Apathy
Sleep disturbances (decreased serotonin)
Fatigue (but will continue to exercise)
56
Q

psychological symptoms and anorexia

A
Decreased interoceptive awareness
Sexuality conflict or fears
Maturity fears
Ritualistic behaviors
Difficulty expressing negative emotions
Low self esteem
Perfectionism
Body dissatisfaction
57
Q

family assessment and anorexia

A

There is no evidence that family interactions are the primary cause of eating disorders.

58
Q

Behaviors which may contribute to an individual’s need to control eating and weight

A

Unrealistic attitudes (weight, shape and size)
Decreased affection, communication and time spent together
Inability to manage conflict
Enmeshment (Erikson’s stages of development – low autonomy, excessive lack of boundaries intrudes privacy)
Overprotectiveness (decreases the development of autonomy)
Rigidity (maintaining the status quo; change and conflict are avoided)

59
Q

consequences of anorexia for patient

A

Alienation from family and friends

Lack of intimate relationships

Additional health problems
Co-morbid conditions
Con-current conditions

Financial
Unable to work

Death

60
Q

DSM V criteria for bulimia

A

recurrent episodes of binge eating (eating in a discrete period of time, an amount of food that is definitely larger than what most individuals would in a similar period of time or circumstance. a sense of lack of control over eating during the episode)
recurrent inappropriate compensatory behaviors in order to prevent weight gain such as self induced vomiting, misuse of laxatives, diuretics, or other medications, fasting, excessive exercise
the binge eating an inappropriate compensatory behaviours occur both at least once a week for 3 months
self evaluation is unduly influenced by body weight and shape
the disturbance does not occur exclusively during episodes of anorexia

61
Q

prevalence of bulimia

A

About 1% – 2.3%

Onset is between 15-24 years old

Females
10x more likely than males

Influenced also by
Culture
SEDoHs

62
Q

comorbidities of bulimia

A

Anxiety disorders

Depression

Substance abuse

Borderline and avoidant personality

History of childhood sexual abuse

63
Q

biological factors of bulimia

A

Genetic
Strong Heritability

Biochemical
Decreased serotonin 
Contributes to vegetative shifts which can contribute to weight gain
Decreased plasma tryptophan
Can lead to depressed mood
64
Q

cognitive perspective and bulimia

A

Cognitive distortions form the basis of binge eating (i.e. all or nothing thinking, discounting positive changes, fortune telling)

Psychological triggers can cause physiological responses and therefore are currently seen as an explanation for continuation of the behavior (bulimia nervosa) not as a cause

65
Q

psychological triggers and bulimia

A

Increased stress
Negative emotions
Both physical and emotional trigger the opioid system in the anterior cingulate cortex (ACC) to release endogenous opioids. Our bodies do not make an endless supply of endogenous opioids and these can be depleted over a prolonged experience of pain.
Environmental cues
Environmental stimuli can be linked to memories in the hippocampus
In this way, these cues trigger neurochemicals to respond in a way similar to when the initial event was experienced.
As a result, even a visual or the smell of a desired food can cause a increase in dopamine levels.

66
Q

separation-individuation process and bulimia

A

Moving between developmental stages
Usually occurs between adolescence and adulthood
Individuals are unprepared for emotional separation

Blurred boundaries
Individuals may feel guilt about making their own decisions

67
Q

additional risk factors for bulimia

A

Societal perceptions

Dietary restraint

Low self-esteem

History of sexual abuse

Feelings of inadequacy

68
Q

assessments for bulimia

A

MSE

Risk Assessment

Physical Assessment

Family

SEDoHs

69
Q

general appearance with bulimia

A

Wears baggy clothing, long sleeve shirts, long pants
May look older than actual age
May look overweight, but may also appear an appropriate weight or under weight

70
Q

affect and mood with bulimia

A

May be unable to describe mood

Ambivalence (affect incongruent with context)

71
Q

speech and language with bulimia

A
Speech generally at normal rate, volume, and rhythm
Circumstantial speech (talking around the core problem – around the circumstance)
72
Q

thought process with bulimia

A

Obsessional thinking
Irrelevant details
May eventually get to the point

73
Q

thought content with bulimia

A

*Especially important to listen for strengths
May perseverate on weight, size or shape of their body
Depressive or anxious cognitions (may be about talking about the subject or other concerns or worries)

74
Q

perceptual functioning with bulimia

A

no negative changes

75
Q

cognitive functioning with bulimia

A

Decreased ability to remember, concentrate due to perseveration

76
Q

insight with bulimia

A

May not acknowledge there is a problem even thought they become physically unwell

77
Q

judgement with bulimia

A

Poor. Continue the binge-purge cycle

78
Q

risk assessment for bulimia

A

High suicide risks
Independent of other co-morbid/con-current disorders

High risk for self-mutilation
Increased impulsivity

Legal and Financial difficulty
Related to increased impulsivity

Inquire about
A plan (date/time/location)
Access to means (even if there is no plan)
Protective factors

79
Q

metabolic and bulimia

A
Electrolyte abnormalities (hypokalemia, hypomagnesemia
Increased blood urea nitrogen levels (possible kidney damage)
80
Q

cardiac and bulimia

A

Ipecac-related cardiomyopathy arrhythmias
Ipecac syrup is used to induce vomiting
Chronic vomiting can alter sodium and calcium levels

81
Q

gastrointestinal and bulimia

A

Salivary gland and pancreatic inflammation and enlargement (increased serum amylase)
Esophageal and gastric erosion/rupture
Dysfunctional bowel syndrome

82
Q

dental and bulimia

A

Dental
Erosion of dental enamel (front teeth)
Decay
*Both 1 & 2 occur because of increased acid in the mouth when the individual is vomiting.

83
Q

integument and bulimia

A

Fingers and knuckles may be dry and cracked which is the result of the acidity of the vomit

84
Q

neuropsychiatric and bulimia

A
Seizures 
Due to the fluid shift and electrolyte disturbances
Mild neuropathies
Fatigue 
Weakness
85
Q

psychological assessment and bulimia

A

Decreased interoceptive awareness

Sexuality conflict or fears
Related to their perception of their body

Maturity fears
Difficulties with taking responsibility and being accountable
Fear of failure

Low self-esteem

Body dissatisfaction
Could be related to weight, size, and shape

86
Q

Behaviors which may contribute to an individual’s need to control eating and weight

A

Inability to manage conflict
Enmeshment
Erikson’s stages of development - excessive boundaries intrudes privacy which may lead to sneaking behavior; lack of boundaries may lead to decreased sense of the individual’s own identity
Overprotectiveness
Impedes the development of autonomy
Rigidity
Maintaining the status quo – change and conflict are avoided

87
Q

consequences of bulimia on patient

A

Alienation from family and friends

Lack of healthy relationships

Additional health problems
Co-morbid conditions
Con-current conditions

Financial and legal implications