msk Flashcards

1
Q

sprains and strains

A

Sprains (ligaments) and strains (muscles)
nursing interventions: warm up exercises
Most common symptoms: Edema, decreased function, bruising, pain
Immediate care: RICE
Movement of joint while supported

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2
Q

dislocation and subluxation

A

(thumb, elbow, shoulder, hip and patella)
Most common symptoms: deformity, pain, tenderness, loss of function and swelling
Nursing interventions: dislocation is an orthopaedic emergency: priority is to realign the joint
Nursing management: pain relief, support of joint; extensive rehab program
Patient will be at increased risk of repeat dislocation

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3
Q

carpal tunnel syndrome

A

Nursing and collaborative care: prevention is best; wrist splints, special keyboards. Corticosteriod injections; surgery (open release or endoscopic carpal tunnel release); outpatient procedure
Months of recovery needed; neurovascular assessment of hand important

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4
Q

rotator cuff injury and meniscus issues

A

Nursing and collaborative care: treated with ice and heat, rest, NSAIDS, corticosteroids and physio.
Surgery may be required. Avoid ‘frozen’ shoulder. Recovery may take 6 months

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5
Q

anterior cruciate ligament

A

most common sports injury
Nursing and collaborative care: a positive Lachman test; MRI-shows co-existing conditions. Treatment of intact ACL includes: ice, rest, NSAIDS, elevation
Reconstructive surgery: for complete tear in active patients. Allograft or autograft (hamstring or patellar ) ligament used. Rehab is crucial for functional return of knee—6-8 month recovery

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6
Q

nursing interventions and collaborative care for closed reduction msk fractures

A

nonsurgical, manual realignment of bone fragments. Traction and countertraction utilized while pt under general anesthesia. Casting immobilizes the injured part to allow healing

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7
Q

nursing interventions and collaborative care for open reduction msk fractures

A

correction of bone alignment through surgical incision. Internal fixation requires wire, screws, plates, pins. Infection a risk. Continuous passive ROM machines may aid healing.

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8
Q

msk fractures supports

A

Traction: application of a pulling force to an injured body part. Skin traction (Buck) or skeletal traction.
Risk for skeletal traction: infection and prolonged immobility
Casts: typical with closed reduction; immobilizes joint above or below the fx.
Upper extremities: sugar-tong splint, short arm, long arm, posterior splint
Vertebral injury: body jacket
Lower extremity: long cast, short cast, cylinder cast, Jones dressing or splint. Hip spica cast used in pediatric setting

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9
Q

msk fracture nursing care

A

Pain medication, muscle relaxants (Robaxin), tetanus immunization and antibiotics (cefazolin) may all be required
Nutritional therapy: adequate protein (1g/kg of body weight); vitamins, calcium, phosphorus, and magnesium necessary for healing
Cast care:
neuro-vascular assessments, observe for edema, compartment syndrome, NO scratching or putting objects inside the cast, keep cast dry.
Cast removal in outpatient setting
Ambulation important: non-weight bearing, touch down weight bearing, toe-touch weight-bearing, partial weight bearing , weight bearing as tolerated, full weight bearing

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10
Q

goals of fracture treatment

A

Realignment of bone fragments
Immobilization to maintain realignment
Restoration of normal function of injured parts

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11
Q

complications of fractures

A
Bone infection
Bone malunion or nonunion
Avascular necrosis 
Compartment syndrome
Deep vein thrombosis
Fat embolism
Traumatic or hypovolemic shock
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12
Q

fracture infections

A

High incidence in open fractures and soft tissue injuries
Massive or blunt soft tissue injury often has more serious consequences than fracture.
Devitalized and contaminated tissue is an ideal medium for pathogens.

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13
Q

compartment syndrome

A

Two basic types of compartment syndrome
↓ compartment size
Resulting from restrictive dressing, splints, casts, excessive traction, or premature closure of fascia
↑ compartment size
Related to fracture, bleeding, edema, chemical response to snakebite, or IV filtration

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14
Q

compartment syndrome occurrence

A

Early recognition and treatment essential
Ischemia may occur within 4 to 8 hours after onset.
Regular neurovascular assessments
May occur initially or may be delayed for several days

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15
Q

6 P’s of compartment syndrome

A

Pain: distal to injury that is not relieved by opioid analgesics and pain on passive stretch of muscle travelling through compartment
Paresthesia: numbness and tingling
Pressure: ↑ in compartment
Pallor: coolness and loss of normal colour of extremity
Paralysis: loss of function
Pulselessness: diminished/absent peripheral pulses

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16
Q

compartment syndrome assessments and signs

A

Urine output must be assessed because there is a possibility of muscle damage.
Myoglobin released from damaged muscle cells precipitates as a gel-like substance.
Large amounts of myoglobin may result in acute tubular necrosis.
Acute tubular necrosis causes acute renal failure.
Common signs of myoglobinuria
Dark reddish brown urine
Clinical manifestations associated with acute renal failure

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17
Q

fat embolism syndrome

A

Presence of systemic fat globules from fracture that are distributed into tissues and organs after a traumatic skeletal injury

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18
Q

fat embolism treatment

A

Early recognition crucial in preventing potentially lethal course
Most patients manifest symptoms 24 to 48 hours after injury.
Clinical course of fat embolus may be rapid and acute.
Patient frequently expresses a feeling of impending disaster.
In a short time skin colour changes from pallor to cyanosis.
Treat symptoms
Use of corticosteroids is controversial but often administered

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19
Q

preoperative care

A

Day surgery: procedures lasting 2 hrs or less; requires 3-4 hr stay in post anesthesia care unit (PACU)
Pre-Op clinic: decreases surgical delays, reduces pt anxiety through education and answering questions
Instructions for: routine medications on day of surgery, which medications or herbal remedies to stop (ie. Anticoagulants), NPO instructions, pain management options, infection prevention, post-op discharge and care
Day of surgery: OR checklist and consents signed; pre-op meds and IV insertion
Hair clipped, NOT shaved

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20
Q

malignant hypothermia

A

genetic condition, rare, potential fatal metabolic disease of hyperthermia with rigidity of skeletal muscles. Screened during pre-op for familial hx

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21
Q

hip fracture

A

70-90% caused by osteoporosis; 95% result from a fall
10% die within the 1st month; 20% at 4 months; 30% at 1 yr
‘fracture of the hip’ defined as proximal 1/3 of femur
Buck traction may be applied pre-surgery
Surgery options: internal fixation devices; partial hip replacement (replacement of femur with prosthesis); total hip replacement (involves both femur and acetabulum)

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22
Q

typical post-op assessments for hip fracture

A

Typical post-op assessments: vitals, in/out fluids, resp status (deep breathing and coughing, monitoring pain and incision (bleeding or infection). Goal: pt out of bed within 24 hrs
Neuro-vascular assessment: color, T, capillary refill, distal pulses, edema, sensation, motor function, pain

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23
Q

posterior approach for hip fractures

A

Posterior approach: dislocation prevention needed (first 6 wks)
Precautions: never >900 of hip flexion
Adduction across the midline
Internal rotation
Taking a bath or driving is not allowed for 6 wks
Abductor pillow between pt’s legs prevents adduction

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24
Q

anterior or anterolateral approach for hip fractures

A

hip muscles left intact; fewer dislocation and infection issues

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25
Q

weight bearing and hip fractures

A

variable depending on how fragile the fx—full weight bearing may be delayed until 6-12 wks based on x-ray

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26
Q

arthroplasty vs hemiarthroplasty

A
arthroplasty = reconstruction or replacement of a joint 
hemiarthroplasty = replacement of a part of the joint
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27
Q

indications for joint replacement

A

Relieve chronic pain

Improve joint mobility

Correct malalignment

Remove intra-articular causes of erosion

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28
Q

amputation

A

Older age group: related to diabetes mellitus: atherosclerosis, vascular changes, PVD

Two types: residual limb and disarticulation
Residual limb: closed amputation: anterior skin flap with soft tissue padding over the bony prominence. Skin flap is situated posteriorly.
Disarticulation: through a joint
Body image: psychological and social implications
Phantom limb sensation/pain (neurogenic pain)

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29
Q

amputation aftermath

A

Prosthesis: pt may weight bear 3 months post-surgery
Proper residual limb bandaging fosters shaping and moulding. Compression bandage : supports soft tissue, reduces edema, minimizes pain, hastens healing, promotes limb shrinkage
Physio essential
Prosthetist: fits limb fit for prosthetic
Walking : below the knee prosthesis requires 40% more energy; above the knee amputation requires 60% more energy

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30
Q

intraoperative care

A

Circulating nurse role: interprofessional time-out
Not scrubbed, gowned or wearing sterile gloves; documents
Scrub nurse role: scrubbed, gowned, with sterile gloves.
Assists with surgery, instruments, surgical count of equipment
Surgeon and assistant
Anesthesiologist: medications for pain and sedation; intubation

Positioning the pt (Lewis Figure 21-6)
Electrocautery: common equipment used for incisions and cauterizing blood vessels. Requires grounding—pad often placed on thigh of pt

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31
Q

intraoperative: anesthesia

A

General anesthesia: loss of consciousness, skeletal muscle relaxation, amnesia and analgesia
Local anesthesia: loss of sensation in a specific area, no loss of consciousness; may be achieved through topical, SC, or intracutaneous administration route.
Regional anesthesia: spinal, epidural, peripheral nerve blocks
Procedural (conscious ) sedation: mild depression of consciousness. Patient maintains their airway

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32
Q

postoperative care

A
PACU (post anesthesia care unit) : Located in close proximity to OR
Monitor: ABC’s
Serial Vital signs
Resp: oxygen status
Pain assessment
Post-op bleeding
GU: urine output
Cardiac: ECG
delayed ambulation
Short stay (a few hours)

OR—to PACU—pt transferred back to day surgery or surgical unit
SBAR handoff report important at each stage

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33
Q

msk assessments

A
color 
warmth 
sensation 
movement
the 6 P's
34
Q

post op day of surgery

A
Monitor: ABC’s
Vital signs (including pain assessment)
Resp: oxygen status
Surgical wounds (Post-op bleeding, drains)
GU: urine output
GI: N/V, diet
Limited Ambulation
Labs
35
Q

post op day 1

A
Monitor: 
Vital signs (including pain assessment)
Ambulation
Resp: oxygen status
Surgical wounds (Post-op bleeding, drains, infection)
GU: urine output
GI: N/V, diet
Labs
36
Q

post op discharge

A
Monitor: 
Vital signs (including pain assessment)
Ambulation
Surgical wounds (infection)
Discharge: restricted activities: ambulating, lifting, driving, work, sex; follow-up with surgeon
Labs
Resp: oxygen status
GU: urine output
GI: N/V, diet
37
Q

post op teaching for hip fractures

A
No flexion of affected limb past 90 degrees
No crossing of legs
No bending over to reach for objects
No driving for 6 wks
No heavy lifting
38
Q

osteomyelitis

A

(infection of the bone)
Collaborative care and nursing interventions
IV antibiotics
Pain control (NSAIDS, opioid analgesics, muscle relaxants)
Surgical debridement and decompression
Possible: septic shock
Debridement: Surgical removal of chronically infected tissue that is not healing
Long-term complications: muscle flaps, skin grafting, amputation

39
Q

facial fractures

A

Ensure patent airway
Treat pt also for c-spine injury (often concurrent) until proven otherwise
Nutrition a priority
Mandible fx may also be therapeutic for malocclusion
Post-op care: ensure oral hygiene, communication, pain management
Wire cutters required in close proximity

40
Q

pelvic fractures

A

High mortality rate
May be missed on initial triage
Possible damage to abdominal, GI/ GU organs
high pain levels
Pt at high risk for DVT, sepsis or fat embolism syndrome (FES)

41
Q

acute back pain

A

NSIDs or muscle relaxants
Stay active
Bed rest should be limited; may be harmful

42
Q

chronic back pain

A
NSAIDs					
Weight reduction
Sufficient rest periods
Heat or cold applications
Exercise
Tricyclic antidepressants
43
Q

osteoarthritis collaborative care

A

Focuses on:
Managing pain and inflammation
Preventing disability
Maintaining and improving joint function
Foundation for OA management is nonpharmacological interventions
Drug therapy serves as an adjunct

44
Q

osteoarthritis nursing interventions

A

Early on: rest relieves symptoms
Heat and cold applications
Nutritional therapy and exercise
Complimentary therapies: yoga, massage, acupuncture
Medications
Long-term: surgery arthroscopic surgery removes debris

45
Q

osteoarthritis medications

A

Acetaminophen: max 4 g/day
Topical agents: capsaicin or diclofenac diethylamine (Voltaren Emulgel)
NSAIDs as the disease progresses: Celecoib (Celebrex), diclofenac(Voltaren), ibuprofen, indomethacin, ketoprofen, ketorolac, meloxicam, nabumetone, paroxen, piroxicam, sulindac
Antibiotics: doxycycline or minocycline
Corticosteriods: methylprednisolone acetate, triamcinolone, dexamethasone, hydrocortisone sodium succinate, methylprednisolone sodium succinate, prednisone, triamcinolone
Disease-Modifying Antirheumatic Drugs (DMARDs): methotrexate, sulphasalazine, leflunomide, D-penicillamine
Gold compounds: oral–auranofin, parenteral—gold sodium aurothiomalate
Antimalarials: hydroxychloroquine
Immuno-suppressants: azathioprine, cyclophosphamide
JAK (Janus Kinase) inhibitors: tofacitinib
Biological and targeted therapies: tumour necrosis factor inhibitors, interleukin-1 receptor antagonist, interleukin-6 receptor antagonist

46
Q

fibromyalgia

A

collaborative interventions: symptom management
Medications: pregabalin and duloxetine; antidepressants
Ongoing support of healthcare team support important for pt and family
Many alternate therapies are helpful: massage, yoga, tai chi, low-impact exercise, biofeedback, mindfulness, CBT therapy.

47
Q

systemic exertion intolerance disease

A

Diagnosis is by exclusion of other diseases

Supportive management is essential as loss of livelihood is common due to fatigue, cognitive impairment

48
Q

osteoporosis

A

1 in 3 Cdn women and 1 in 5 Cdn men will experience a fx due to osteoporosis.
Definition: low bone density and deterioration of bone tissue
Supplemental Calcium and Vit D helps prevent osteoporosis
Calcium: women over the age of 50yrs: 1200 mg of calcium/ day with diet or supplementation
Vitamin D: 800-2000 units/ day for adults over 50 yrs or high risk young adults
Exercise: Weight-bearing activities build and maintain bone mass

49
Q

osteoporosis medications

A
Bisphosphonates
SERMS
Calcitonin and teriparatide
Denosumab
Selective Estrogen Receptor Modulators (SERMS):
Raloxifene
Tamoxifen

Teriparatide: stimulates bone formation
Denosumab: prevents bone resorption

50
Q

bisphosphonates

A

Alendronate
Etindronate disodium with calcium carbonate (Didrocal and Etidroncal
Risondronate
sodium demipentahydrate (Actonel, Actonel DR)
Zoledronic Acid

51
Q

bisphosphonates mechanism of action

A

Inhibit osteoclast mediated bone resorption

52
Q

SERMS mechanism of action

A

stimulates estrogen receptors in bone

53
Q

teriparatide mechanism of action

A

only drug available that stimulates bone formation. Modulates the body’s metabolism of calcium and phosphorus

54
Q

denosumab mechanism of action

A

monoclonal antibody that blocks osteoclast activation (prevents bone resorption)

55
Q

osteoporosis drugs indications

A

Ralixofene: prevention
Bisphosphonates and calcitonin: for prevention and treatment
Teriparatide: high risk osteoporosis

56
Q

osteoporosis drugs contraindications

A

Bisphosphonates: drug allergy, hypocalcemia, esophageal dysfunction, inability to sit upright for 30 min after taking the med
SERMs: known allergy, who are or may become pregnant, women with a history of venous thromboembolic disorder
Teriparatide: known drug allergy
Denosumab: hypocalcemia, kidney impairment and infection

57
Q

osteoporosis drugs adverse effects

A

SERMs: hot flashes, and leg cramps; increased risk of venous thrmobembolism and are teratogenic; puts pt at risk fo leukopenia

Bisphosphonates: headache, GI upset, joint pain. A chance for esophageal burn: take with a full glass of water and remain sitting for 30 minutes.
Teriparatide: chest pain, dizziness, hypercalcemia, nausea and arthralgia
Denosumab: infections

58
Q

osteoporosis drugs interactions

A

Raloxifene: decreased absorption with cholestyramine and ampicillin’ decreases the absorption of warfarin sodium

Bisphosphonates: absorption interfered by calcium supplements and antacids; aspirin and NSAIDs may irritate GI symptoms

59
Q

kyphosis

A

Abnormal convex angulation in the curvature of the thoracic spine
May occur secondary to TB, chronic arthritis, osteodystrophy, compression fractures
Most common is ‘postural’: often when bone growth outpaces muscle growth
Postural kyphosis accompanied by compensatory lordosis

60
Q

kyphosis nursing interventions

A

Range from simple exercises to surgery

Empower the adolescent that exercise to strengthen shoulder and abdominal muscles has cosmetic appearance value
Regular therapy sessions
Brace may be required until muscle maturity occurs
Surgery is a last resort for spinal fusion in severe, and painful deformities

61
Q

lordosis

A

Accentuation of the cervical or lumbar curvature
May be secondary to a disease process; may be from trauma; or idiopathic
Associated with flexion contractures of the hip, scoliosis, obesity, developmental dysplasia of the hip, slipped capital femoral epiphysis
Often painful

Treatment: treat predisposing cause; support garments

62
Q

scoliosis

A

Complex spinal deformity in 3 planes:
Lateral curvature, spinal rotation, thoracic hypokyphosis

Most common spinal deformity

Classified according to age of onset: congenital, infantile, childhood, adolescent

Sometimes secondary to another disease; most often idiopathic

63
Q

scoliosis diagnosis

A
Adams forward test
Scoliometer: measures truncal rotation
 x-rays are definitive 
100 of curvature needed for diagnosis
250 of curvature= mild and requires ongoing observation
Risser scale
64
Q

scoliosis treatment

A

Observation
Based on magnitude, location and type of curve
Bracing: often the treatment of choice for mild curves
Boston and Wilmington braces
TLSO—thoracolumbosacral orthotic
Surgery: anterior or posterior approach may be used
Luque-rod
Cotrel-Dubouset
Dwyer or Zielke

65
Q

scoliosis nursing interventions

A

Difficult time as adolescence is a time of identify formation
Surgical pre-op:
X-ray; pulmonary function tests, lab tests
Significant blood loss expected
Post-op:
PCA—significant pain, foley catheter, log-roll
Possible brace
Assessment: wound, neuro, circulation, VS
Mobilization (depending on surgical approach)
Personal items for comfort and distraction

66
Q

cerebral palsy

A

A group of permanent disorders of movement and posture. This results in limitation of activity attributed to nonprogressive disturbances in the developing fetal or infant brain
Musculoskeletal problems and epilepsy (15-60%) results in: disturbances of sensation, perception, communication, cognition and behavior
Characterized by abnormal muscle tone and coordination
Most common permanent motor disability in childhood
Low birth weight infants at high risk (100x)
Cause: believed to be from existing prenatal brain abnormalities

67
Q

cerebral palsy diagnosis

A

Diagnostic: neuro assessment and history main source
Also: MRI or CT
Metabolic and genetic testing is recommended if no structural abnormality is found
Often diagnosis cannot be confirmed until age 2
Primitive reflexes remaining after 4 months
Asymmetrical Tonic neck reflexes or persistent Moro reflex
Crossed extensor reflex
Permanent disability; therapy is preventive and symptom control

68
Q

cerebral palsy therapy goals

A

To establish locomotion, communication and self-help skills
To gain optimal appearance and integration of motor functions
To correct associated defects
To provide educational opportunities adapted for the child
To promote socialization experiences with other children
Accomplished with multi-disciplinary team

69
Q

typical cerebral palsy treatment

A

Ankle-foot orthoses/ Wheeled go-carts for mobility

Orthopedic surgery: due to contractures and spastic deformities, spinal fusion; dorsal rhizotomy

70
Q

cerebral palsy medications

A

Medications: dantrolene sodium, baclofen, diazepam, Botulinum toxin A
carbamazepine (Tegretol), divalproex (valproate sodium and valproic acid)

71
Q

cerebral palsy effects

A

Dental hygiene
Toilet training
Electronic devices: give biofeedback; may improve, eye-hand coordination and body mechanics
Behavioral issues: ADHD and learning delays
Vision difficulties
Speech pathologist may help with speech and feeding issues
Physiotherapist: assist with mobility and stretching

72
Q

early treatment for cerebral palsy

A

therapeutic hypothermia within 6 hrs of birth may be neuroprotective

73
Q

cerebral palsy interventions and care

A
ADLs (majority of time and effort spent)
Frequent rest periods needed
Diet: gastrostomy feedings may be necessary
Jaw control an issue
Vocational training
Participate as able
Nursing Interventions
Emotional support 
Siblings
Hospitalization
Possible respite
74
Q

neural tube defects

A

Neural tube defects: largest group of congenital anomalies
Decreased rates of NTD due to prenatal supplementation and termination of pregnancies have affected incidence rates (folic acid supplements)
Encephalocele and anencephaly are abnormalities from the anterior end of the neural tube not closing
Encephalocele: herniation of brain and meninges through a skull defect; may be from hydrocephalus; shunting main intervention
Anencephaly: most serious NTD; both hemispheres of brain are absent; incompatible with life

75
Q

spina bifida

A

Myelodysplasia: any malformation of the spinal canal and cord
Spina bifida occulta: a defect not visible externally; often in lumbosacral area
Spinal bifida cystica: visible defect with external saclike protrusion. May be meningocele: encases meninges and spinal fluid but no neural elements or myleomeningocele: containing meninges, spinal fluid and nerves

Children have normal intelligence but many physical hardships

76
Q

spina bifida diagnosis

A
Diagnosis based on clinical manifestations
CT, MRI, ultrasound, neuro evaluation
Spina bifida cystica: 
Bowel and bladder dysfunction
Motor dysfunction of lower extremities
Hydrocephalus
Orthopedic deformities
Spina bifida occulta:
Skin depression, port-wine angiomatous nevi or dark tufts of hair
Gait disturbance with foot weakness
Bowel and bladder sphincter disturbances
77
Q

myelomeningocele

A

Closure within 24 hrs of birth ideal when CSF leaking
Objective is skin coverage
Shunt procedures may be required to deal with hydrocephalus
Chiari II malformation only deal with if child is symptomatic
Prenatal surgery more effective than postnatal surgery
Postnatal surgery: prevent infection with broad spectrum antibiotics, skin closure of myelomeningocele within 12-72 hours

78
Q

spina bifida long term issues

A
Hydrocephalus and shunt malfunctions
Chiari II development
Scoliosis
Bowel and bladder management issues
Latex allergy
Epilepsy
79
Q

spina bifida prevention

A

Rates dropped from 1997-2007: 0.86/1000 to 4.0/10,000 births
Folic acid supplementation
Early abortions

80
Q

muscular dystrophy

A

DMD: Duchenne muscular dystrophy; most common form of muscular dystrophy
Genetic origin: gradual degeneration of muscle fibers
Lifespan affected
Supportive measures of physio and orthopedic procedures
High genetic component: 65% have family history
Dystrophin is absent or reduced (a protein product in skeletal muscle)

81
Q

DMD

A

X-linked recessive trait; rarely a female will have DMD
Normal development; muscle weakness appears between 3-7 yrs
Ambulation impossible by 12 yrs of age
Other symptoms: osteoporosis, fractures, constipation, skin breakdown, atrophy of facial oropharyngeal and respiratory muscles—leading to the requirement for long term respiratory support
Cardiomyopathy seen in 50-80% of DMD pts
Mild to moderate cognitive impairment commonly found

82
Q

DMD with family members

A

Isolation of pt and family requires support
Long term care options
End of life directives needed
Palliative care should be discussed