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Neuro 1 > test 2 > Flashcards

test 2 Flashcards

1
Q

How does RAS work

A

Brain processes the space between the beats not the beat itself - anticipatory cue

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2
Q

principle of entrainment

A

Temporal locking process in which one system’s motion or signal frequency entrains the frequency of another system

Stronger oscillator locks the weaker into its frequency, or if equal in strength the systems move towards each other

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3
Q

how does entrainment relate to RAS

A

creates fast, precise, and stable synchronization between sensory and motor

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4
Q

anatomy in neural entrainment
time keepers
and tract

A

Intermotor timekeepers  sensorimotor, cerebellum, BG, posterior inferior frontal (BROCAS)
Reticulospinal  has auditory fibers linked with it

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5
Q

patterned sensory enhancement

A

Using musical patterns to create functional movement patterns and sequencing to enhance direction and accuracy of movement

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6
Q

Dx that have evidence to support the use of RAS

A

CVA
PD
disorders of consciousness
epilepsy

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7
Q

inclusion criterial for RAS

A

Able to walk >20-100ft
Hearing intact
MMSE mini mental >24
No major CV dysfunction

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8
Q

RAS golden lesions

A

cerebellum, brainstem, thalamus

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9
Q

3 phases of RAS gait training

A

phase 1 - warm up 1-2 mins with RAS

phase 2 gait training w/ RAS at 10% increase in cadence

phase 3 intermittent fading of RAS for carryover

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10
Q

most life threatening TBI

A

SAH - subarachnoid hemorrhage

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11
Q

post TBI moa

A

causes metabolic cascade –> apoptosis

electrolyte imbalance and release of dmginng neurotransmitters

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12
Q

ICP ranges

A

norm - 5-10
abnormal ICP > 20
caution >= 15

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13
Q

S/s of increase ICP

A

increased BP decreased HR

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14
Q

post traumatic seizures

when can you mob

A

increased risk of seizure after TBI

must be seizure free for 24 hrs before mob

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15
Q

PAID

s/s

A

sympathetic storming - don’t work with them while they are stomping

tachypnea
tachycardia
HTN
diaphoresis
posturing
agitation
GCS 3-8
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16
Q

heterotropic ossification

A

formation of abnormal bone growth - common in large joints

onset 4-12 wks after injury

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17
Q

most common cog deficit TBI

A

attention

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18
Q

dysexecutive syndrome
orbitofrontal

frontal convexity

A

orbitofrontal - poor judgment, innapp jocular, impulsive behavior

frontal convexity - indifference, motor preservation

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19
Q

decorticate

decerebrate

A

corti - LE ex UE fl

cere - LE and UE EX

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20
Q 
mild v mod v severe TBI
GCS
LOC
alteration of consciousness
amnesia
imaging
A 
mild - GCS: 13-15
LOC: 0-30 minutes
Brief (<24 hours) alteration of consciousness
Post-traumatic amnesia <1 day
Imaging: normal
mod - GCS: 9-12
LOC: >30min but    <24 hours
Alteration of consciousness >24 hours
Post traumatic amnesia >1 but <7 days
Imaging: normal or abnormal
severe - GCS: 3-8
LOC: >24 hours
Alteration of consciousness >24 hours
Post-traumatic  amnesia >7 days
Imaging: normal or abnormal
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21
Q

neg prog indi

A

older
female
edu level
very low GOS/GCS

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22
Q

what type of injury is concussion

A

metabolic

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23
Q 
gait speed 
household ambulator
limited community ambulator
community ambulator
cross street  ambulator
A

i) 0.0-0.4 m/s: Household ambulator
ii) 0.4-0.8 m/s: Limited community ambulator
iii) 0.8-1.2 m/s: Community ambulator
iv) 1.2-1.4 m/s: Cross street; normal walking speed

24
Q

hemi gait abnormalities
hemi leg
overall

A

hemi leg - decreased step/stride length, variable step width
decreased step height

temporal asymmetries
decreased SLS
increased DLS
increased swing time
decreased cadence
25
Q

stirrup/double upright ortho

A

i) Only one for fluctuation of edema

26
Q

solid AFO

A

(1) Significant LE weakness or hypotonia requiring maximum stability
(1) Good for M/L stability

27
Q

pre hinged AFO

A

progress to articulated AFO

28
Q

hinged AFO

A

need pf strength
provides ankle control
M/L stability

29
Q

ground reactive AFO

A

helps with KNEE

drop foot

30
Q

posterior leaf spring

A

drop foot with min to no M/L stability

swing phase only

31
Q

BWSTT

A

has to be <40% support to remain functional

improve gait speed and endurance

32
Q

Bioness L300

A

foot drop

33
Q

bioness L300+

A

for knee instability
hamstring during swing
and DF during swing

34
Q

jargon aphasia replaces words with

A

random sounds

35
Q

tulia is used for

A

apraxia

36
Q

dysconjugate gaze means they are going to struggle with

A

maintaining focus

37
Q

KF-NAP used for

A

used for neglect

38
Q

medial inferior pontine s/s

A

a. Nystagmus
b. Ipsi ataxia
c. N/V
d. Paralysis of deconjugate gaze
e. UE/LE/face Hemisensory – dorsal
f. UE/LE/face Weakness
g. Diplopia

39
Q

medial medullary s/s

A

a. Ipsi tongue weak

b. UE/LE hemiplegia and sensory - dorsal

40
Q

inferior lateral pontine s/s

A

a. Nystagmus
b. Ipsi ataxia
c. Vertigo/N/V
d. Facial weakness
e. Paralysis of deconjugate gaze
f. CN 5 – sensory
g. Hearing loss
h. UE/LE/facial sensory – ant lat

41
Q

lateral medullary s/s

A

a. UE/LE hemisensory- dorsal and anterolat
b. Face pain/temp loss
c. Vertigo/N/V
d. Nystagmus
e. Ipsi Ataxia
f. Dysphagia, dysphonia, impaired gag
g. Ipsi Horner’s

42
Q

superior lateral pontine

A

a. Nystagmus
b. Ipsi ataxia
c. Face pain/temp loss
d. Lateral pulsion
e. D/N/V
f. Paralysis of deconjugate gaze
g. LE>UE sensory – dorsal
h. Ispi Horner’s

43
Q

BDNF

A

increases neuro plasticity

44
Q

prediction calc for independent ambulation post stroke

A

Berg

MMT of LE

45
Q

CPG for locomotion post stoke

A 
should perform
walk training at mod to high aerobic intensities
walk training VR
may consider
strength training at 70%
circuit training, cycling at 75-85 HR
balance training
46
Q

ambulation prediction rule

A

age
motor
sensory

47
Q

HIIT training neuroplasticity principles

A

specificity
intensity
repetition
variablity

48
Q

HIIT protocol for

intensity

A

> 85 % HR max

49
Q

BB for HR max adjustment

A

drop 10 bpm in their usual percentage

50
Q

VS parameter
active
Rest
for HR BP SaO2

A 
active
HR - >85 HR max
BP - 250/115, >10 mmHg in systolic
SaO2 - 92
Rest
HR - > 120 <60
BP - > 180/110
SaO2 - < 92
51
Q

intervention and management for rancho 6

A

phase away from errorless
less cues
incorporate more complex mem, reasoning, and sequencing
may be able to do formal OMs
increase overall mobility and independence - incorporate mod busy environment

52
Q

intervention and management of rancho 5

A 
structure w/ variety
slowly increase stim
behavior mod plan
begin visual cues for mem aid
small increase in PA
53
Q

intervention and management for rancho 4

A 
re orient
prevent over stim
identify and manage triggers
structure
family edu
physical restraints last resort
54
Q

intervention and management for rancho 7

A 
more self directed therapy
increase stim
increase difficulty w/ prb solving
more group participation
increased sequencing steps
55
Q

intervention and management for rancho 8-10

A 
enviro can be more typical
pt driven goals
promote indi
practice ADLs
large focus on divided attention and multi tasking
improved speed and intensity
56
Q

pseudobulbar affect

A

state of inappropriate emotional (inconsistent with mood) outbursts due to neuro insult

57
Q

aneurysm gets what surgical intervention

A

surgical clipping

Neuro 1 (4 decks)

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