W3 - Resistance Training Flashcards

1
Q

why does resistance training result in strength increase?

A
  • Muscle hypertrophy
    • Muscle hyperplasia
    • Neural adaptations
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2
Q

How does training impact muscles?

A

muscle hypertrophy (fibre size ↑)

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3
Q

How does the increase in muscle size effect performance?

A

Increases performance

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4
Q

How does sex difference effect muscles?

A

Different muscle size, hormones (testosterone, males more muscle mass, less fat mass)

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5
Q

How does resistance training impact gains in muscular fitness?

A
  • Resistance training yields strength gains via neuro-muscular changes
    • After 3 to 6 months of resistance training:
      ~25 to 100% strength gain
      ○ More effective force production (physiological adaptations) - bigger fibres & neural system is better at accessing the working muscles
      ○ Optimisation of movements (technique) - coordination & technique between muscles (only accessing muscles that facilitate the movement)
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6
Q

Describe the mechanisms of muscle strength gain

A
  • Strength gain can occur without hypertrophy
    ○ Normally motor units are recruited asynchronously
  • Strength training makes the muscle recruitment from asynchronous to synchronous - increase in force over time is steeper
  • Nerves send signals in a more synchronous fashion
    ○ Resistance training → more synchronous recruitment → strength ↑
  • Improved rate of force development
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7
Q

Describe how motor unit recruitment can generate strength gains

A
  • Strength gains may also result from greater motor unit recruitment
    • Neural drive during maximal contraction ↑
    • Frequency of neural discharge ↑ (higher neural output makes us contract more forcefully)
    • Inhibitory impulses ↓ (e.g.: these become lost as we fall asleep, sudden kicking) Improved capability to exert steady forces
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8
Q

Explain autogenic inhibition

A
  • Normal intrinsic inhibitory mechanisms
    – Golgi tendon organs (inhibit muscle contraction if tendon tension too high, monitor stretch of the tendon)
    → Prevent damage to bones and tendons via inhibitory neuron
  • Decreasing inhibitory impulses
    – Training → Force ↑
    – Superhuman feats of strength(e.g.: under extreme stress, mother child reaction can prevent these inhibitory impulses)
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9
Q

Describe the muscle strength gain in antagonists vs agonists

A
  • Co-activation of agonists and antagonists
    • Normally antagonists oppose agonist force (training reduces the antagonist force to as little as possible)
    • Reduced co-activation may result in (slight) strength gain
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10
Q

What are the short/long term muscle strength gains?

A
  • Short-term increase in muscle strength
    – Substantial increase in maximum force
    – Increase in voluntary neural activation
    – Neural factors critical in first ~8 to 10 weeks
  • Long-term increase in muscle strength
    – Associated with fibre hypertrophy
    – Net increase in protein synthesis takes time to occur
    – Hypertrophy major factor after first ~10 weeks that drives strength gain
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11
Q

Explain the chronic and transient hypertrophy that takes place to increase muscle size

A
  • Hypertrophy: increase in muscle size
    • Transient muscle hypertrophy (after exercise bout)
      ○ Due to edema formation from plasma fluid
      ○ Disappears within hours
    • Chronic muscle hypertrophy (long term)
      ○ Reflects actual structural change in muscle
      ○ Fibre hypertrophy (bigger fibres), fibre hyperplasia (more fibres), or both: animal experiments show before training there is 1 big fibre, after training there are 2 smaller fibres
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12
Q

What are some benefits of fibre hypertrophy?

A
  • more myofibrils,
  • more actin and myosin filaments,
  • more sarcoplasm,
  • more connective tissue, or
  • any combination of these.
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13
Q

What are the 3 main mechanisms of muscle strength gain through fibre hypertrophy?

A
  • Resistance training → protein synthesis ↑
    – During exercise: synthesis ↓, degradation ↑
    – After exercise: synthesis ↑, degradation ↓
  • Hormones
    – Testosterone facilitates fibre hypertrophy (natural anabolic steroid hormone)
    – Synthetic anabolic steroids → large increases in muscle mass
  • Genetics: e.g., mutation in myostatin gene (Belgian Blue cows)
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14
Q

How does eccentric training impact muscle strength gain ?

A
  • Maximized by eccentric training → Force increase
  • Maximized by eccentric training
    – Disruption of sarcomere Z-lines
    • After a training session → protein remodelling → overreaching of capabilities
    • Long term trajectory increases
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15
Q

How does concentric training impact muscle strength gain?

A
  • Concentric training may limit muscle hypertrophy and strength gains
    • Left side of diagram is where muscle lengthens because the weight is too heavy for muscle shortening
    • Why not only eccentric training is done:
      ○ More risk of injury
      ○ Training the specific movements for competition
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16
Q

Explain fibre hyperplasia

A
  • Most hypertrophy due to fibre hypertrophy
  • Fibre hyperplasia: fibre splitting, each half grows to size of parent fibre
    – may also contribute (some evidence in animal studies, very little evidence in humans)
17
Q

How does fibre type determine the type of exercises for strength gain?

A
  • Fibre type conversion is possible under certain conditions
    – High-intensity treadmill or resistance training:
    Type I → Type IIa
    – Heavy resistance training programme:
    Percent Type IIx ↓, percent Type IIa ↑
18
Q

What is the role of the sarcomere Z-disks?

A
  • Sarcomere Z-disks: anchoring points of contact for contractile proteins
    – Transmit force when muscle fibres contract
    – Z-disk, actin/myosin damage after eccentric work
    e.g.: After a marathon there is a lot of damage to muscle cells
19
Q

What does DOMS stand for?

Explain what DOMS is & what it tells us

A
  • 1-2 days after exercise
  • Muscle strain (range: stiffness → severe, restrictive pain)
  • Major cause: eccentric contractions
    – Level run pain < downhill run pain
    – Cycling: mostly concentric → mostly no DOMS
    – Not caused by increases in blood lactate concentrations
  • Indicated by muscle enzymes in blood (e.g., creatine kinase)
    – Suggests structural damage to muscle membrane (leak)
    – Index of degree of muscle breakdown
20
Q

What effect does DOMS have on a persons strength?

How does this effect performance?

A
  • DOMS → muscle force generation ↓
    • Loss of strength:
      – Physical disruption of muscle / Loss of contractile protein
      – Failure in excitation-contraction coupling (appears to be most important) - neuron signal doesn’t travel as efficiently as it did before
21
Q

What are some strategies that athletes can use to reduce DOMS?

A
  • Athletes must reduce excessive DOMS for effective training!
  • Strategies to reduce DOMS:
    – Minimize eccentric work early in training
    – Start with low intensity training and gradually increase throughout session
22
Q

How does DOMS effect white blood cells?

Explain inflammation of the muscle:
Muscle damage → inflammation → pain

A
  • White blood cells (neutrophils, macrophages) defend body against foreign materials and pathogens, clean up altered/destroyed cells– Damaged muscle cells attract neutrophils
    – Neutrophils release attractant chemicals, radicals - stimulates pain receptors
    – Released substances and swelling stimulate pain nerves
    – Macrophages remove cell debris