theory Flashcards

1
Q

what is treating the whole person. taking into account the social/mental and physical rather than just the physical symptoms

A

holism

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2
Q

the theory that relates scientific method to philosophy by affirming that all beings and events in the universe are natural. consequently, all things fall under the pale of scientific investigation

A

naturalism

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3
Q

the practice of analyzing and interpreting a complex phenomenon that are held to represent a simpler or more fundamental level. ie the idea that a person is a collection of atoms

A

humanism

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4
Q

the philosophy or theory that the main objective of the human experience is to pursue personal happiness and respect other humans. ie a person who works hard on a farm his entire life to be completely self-sustaining

A

objectivism

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5
Q

a belief or theory that opinions and actions should be based on reason and knowledge rather than on religious belief or emotional response

A

rationalism

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6
Q

an approach that assesses the truth of meaning of theories or beliefs in terms of the success of their practical application.

A

pragmatism

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7
Q

school of scientific thought- starts with aristotle- that attempts to explain the nature of life as resulting from a vital force peculiar to living organisms and different from all other forces found outside living things

A

vitalism

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8
Q

the predominant form of materialism, which holds that natural phenomena can and should be explained by reference to matter and motion and their laws. a mechanism results in the interaction between human agents, intervention, and structures

A

mechanism

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9
Q

philosophical position that states that the universe and its various parts ought to be considered alive and naturally ordered, much like a living organism

A

organicism

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10
Q

devotion and belief in things- greed

A

materialism

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11
Q

ability to perform activities of daily living includes ROM, stability, strength and coordination

A

function

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12
Q

innate is part of the all wise, part of the creator

A

innate intelligence

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13
Q

god creator

A

universal intelligence

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14
Q

our brain power and the things that we learn via both formal and informal education

A

educated intelligence

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15
Q

what is cavitation?

A

is the formation of vapor and gas bubbles primarily carbon dioxide within the synovial fluid in a joint capsule through a local rapid reduction of pressure

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16
Q

the actual mechanical process of cavitation is called?

A

tribonucleation- decrease in pressure to viscous adhesion that also occurs between the two articular surfaces,

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17
Q

what does cavitation represent?

A

cavitation represents a physical event that includes- joint separation, stretching of periarticular tissues, and stimulation of joint mechanoreceptors.

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18
Q

what are some physiological changes when a cavitation is done?

A
  • associated with transitory increase in PROM
  • temporary increase in joint space
  • increased joint separation
  • 20 minute refractory period before cavitation can occur again.
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19
Q

joint fixation hypotheses offers an explanation for what?

A

why a joint is not moving as it should

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20
Q
  • interarticular adhesions are due to?
  • what reactions does spinal manipulation offer?
A

-development of adhesions between the articular surface of facet joints- joint injury, inflammation or immobilization-
- SM induces gapping of the facet joints, and if adhesions are the problem the gaping would lead to adhesions being broken up.

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21
Q

explain the interarticular block theory-

A
  • restrictions are due to the product of some derangement within the synovial joint- arises from poorly coordinated spinal movements or sustained stressful postures that lead to entrapment or entrapment of the fibrous cap of the meniscoid.
  • SM gap the joint allowing for the meniscoid to become free of the entrapment-
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22
Q

these people are health care professionals for the dx, care and prevention of disorders of the spine, as well as other parts of the musculoskeletal system

A

chiropractor

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23
Q

what is the focus of chiropractic?

A

maintaining proper spinal function and its relationship with the neurological system

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24
Q

what is chiropractic based on?

A

based on the premise that the nervous system functions to control and coordinate all organs and structures of the body, and relates the individual to ones environment

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25
Q

explain chiropractic foundation

A

abnormal or dysfunctional spinal movement can adversely impact nerve function- affecting the communication between the brain and the body

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26
Q

what is a subluxation

A
  • spinal joint dysfunction- vertebral subluxation or VSC (complex) which is a condition of disturbed joint function
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27
Q

what does the chiropractic exam identify?

A
  • identifies dysfunctional joints whether they are painful or not- joints can have disturbed biomechanics without pain or symptoms
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28
Q

what type of disturbed biomechanics can arise due to a dysfunctional joint

A
  • reduced performance-
  • alter load distrubution
  • increase risk of injury
  • accelerate joint degeneration without pain or other symptoms
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29
Q

what does the adjustment produce?

A
  • applied to the joints can restore motion when bodys own muscles cannot
  • helps to relieve pain, restore and maintain normal movement, biomechanics and function
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30
Q

what does function mean? what does it include?

A
  • the ability to perform activities of daily living
  • includes range of motion, flexibility, stability, strength and coordination
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31
Q

what percentage of patient complaint pertains to chiropractic care?

A
  • back pain 60%
  • other extremeties etc. 20%
  • headache 10%
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32
Q

what is ACC

A

the association of chiropractic colleges

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33
Q

what consensus did ACC come to?

A

statement to promote a unified voice to the profession and the public in an attempt to further clarify the unique role the chiropractic profession serves.

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34
Q

what is the statement that ACC came up with

A

chiropractic is a health care discipline which emphasizes the inherent recuperative powers of the body to heal itself without the use of drugs or surgery

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35
Q

what do chiros do to heal the body without drugs or surgery? the ACC says?

A
  • the practice of chiropractic focuses on the relationship between structure (spine) and function (ns) and how that relationship affects the preservation and restoration of health
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36
Q
  • doctors of chiropractic recognize the value and resp. of ________ with other health care practitioners when in the best interest of the patient
A
  • working in cooperation
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37
Q

what does the ACC advocate

A
  • a profession that generates, develops and utilizes the highest level of evidence possible in the provision of effective prudent and cost conscious patient evaluation and care
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38
Q

what else does ACC say about chiropractic?

A
  • the purpose of chiropractic is to optimize health- the bodys innate recuperative power is affected by and integrated through the nervous system
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39
Q

according to ACC what is a subluxation

A

is a complex of functional and or structural and or pathological articular changes that compromise neural integrity and may influence organ system function and general health

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40
Q

what are the subluxation models/

A
  • faye 5 component model
  • 9 component lantz model
  • 3 component kent model,
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41
Q

-what is the faye model of subluxation?
- what is its primary emphasis ?
- what is a key to an adjustment?

A
  • 5 component model of VSC
  • primary emphasis on joint motion, fixation or loss of segmental mobility
  • mobilize the fixation
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42
Q

what does the faye 5 component model of VSC consists of?

A
  1. neuropathophysiology
  2. kinesiopathology
  3. myopathology
  4. histopathology
  5. biochemical changes.
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43
Q

what is neuropathophysiology in faye 5 model
- what is it related to?
- how can this exist?

A

-facilitation, degeneration, decreased axoplasmic flow-
- related to pathological changes in physiology related to the nervous system.
- can exist in form of irritation, pressure or decreased axoplasmic flow- can also exist indep. or in combo.

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44
Q

what is kinesiopathology in faye 5 model

A

hypomobility, hypermobility, loss of joint play

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45
Q

what is myopathology in faye 5 model

A

spasm , atonia, includes hiltons law

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46
Q

what is histopathology in faye 5 model?

A

inflammation

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47
Q

what is biochemical changes in faye 5 model

A

hormonal and chemical effects

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48
Q
  • Neuropathophysiology a part of faye 5- what is facilitation?
  • what term relates to facilitation?
  • what can the internal and external be described as?
A
  • the term irritation relates to facilitation of afferent nerve cells, which lowers the threshold for firing- this may lead to the CNS receiving an inappropriate picture of what is really going on with the environment.
  • internal and external can be broadly described as dysafferentation.
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49
Q

regarding faye 5 NPP- explain dysafferentation
- what parts of the spine could be effected and how.

A
  • CNS received inappropriate picture of what is really going on in environment
    this is dysafferentation- it may evoke activity of efferent neurons with nerve cell bodies located in the spinal cord.
  • the anterior horn of the spinal cord (motor function) may lead to hypertonicity of muscles or spasm. AND the lateral horn (visceral function) may lead to vasomotor changes like vasoconstriction. AND the posterior horn (sensory function) may lead to changes in sensation.
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50
Q

regarding NPP Faye 5
- what is the cause of compression
- what are the nerve fibers affected and what are the outcomes?
- what is the result of all types of compression

A
  • increased pressure within a tissue, stretching, angulation, (abnormal bends around structures) or distortion to neural elements in or around the IVF (intervertebral foramen)- fascial layers, muscles with increased tone (hypertonicity)
  • motor fx changes can include muscular atrophy, sensory changes can include decreased sensation (hypoesthesia) or altered sensation ( paresthesia)
  • autonomic (visceral) fx changes include vasodilation and resultant vascular stasis
  • degeneration over time
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51
Q

regarding NPP faye 5
- what is decreased axoplasmic flow- what is it tied to?
- this idea is included in what hypothesis?
- is there evidence for immune function?

A
  • tied to the idea that large molecules (macromolecules) are not transported as efficiently - neurotrophic substances or substances that help provide necessarily materials for target cells.
  • neuroimmune hypothesis
  • there is no evidence to support spinal manipulation affects immune function
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52
Q

regarding faye 5
- what is kinesiopathology?
- what part of PART is this?

A
  • lack of mobility associated with changes in nociceptive and mechanoreceptive reflex function that include proprioception.- shows up in the clinical manifestation of subluxation
  • it is the R in PART
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53
Q

regarding faye 5
- what is myopathology?
- what law is associated with this?
- what part of PART is this?

A
  • this includes spasm, hypertonicity from compensation or facilitation.
  • Hiltons law- states that a nerve supplying a joint also supplies the muscles which move the joint and the skin covering the articular insertion of those muscles.
  • T and possibly A of PART ( tight muscles)
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54
Q

regarding faye 5
- what is histopathology?
- what are the laws tied to this? define the laws.

A
  • relates to inflammation and can result from trauma, irritation ties to hypermobility or as part of a tissue repair process.
  • weigerts law: loss of destruction of a part of a joint is likely to result in compensatory replacement and and overproduction of tissue during the process of regeneration and repair.
  • wolffs law: every change in the form and function of a bone is followed by definite changes in its internal architecture, and secondary alterations in its external conformation- internal changes will lead to external changes.
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55
Q

regarding faye 5
- what is biochemical changes?
- what does this result in?
- what is this often tied to?

A
  • related to hormonal and chemical effects or imbalance tied to the pre-inflammatory stress syndrome.
  • as result of trauma or lack of mobility in spinal joints, histamine, prostaglandin and bradykinin are produced and released- this results in altered aberrant- somatic input in the segmental levels of the spinal cord. (dysafferentation)
  • often tied to Selye’s general adaptation syndrome and local adaptation syndrome
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56
Q
  • the faye model proposes the adjustment affects _____ component directly and the other components indirectly.
A
  • kinesiopathology component
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57
Q
  • define selye’s general adaptation syndrome
  • what are the 3 stages
A
  • general adaptation syndrome or GAS- developed by hans Selye- describes the pattern of responses that the body goes through after being prompted by a stressor.
  • the stages are alarm, resistance, and exhaustion - ARE!
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58
Q
  • what is local adaptation syndrome?
  • what are some characteristics?
  • what are the two localized responses?
A
  • LAS is response of a body tissue or organ or part to the stress of trauma, illness or other physiological change
  • characteristics include- response localized- does not involve entire body system/ response is adaptive, stressor is necessary to stimulate/ response is short term- does not persist indefinitely/ response is restorative- restoring homeostasis to the body region or part.
  • the two localized responses are REFLEX PAIN RESPONSE AND INFLAMMATORY RESPONSE.
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59
Q

regarding local adaptation syndrome:
- what is reflex pain response?

A
  • localized response of the CNS to pain. adaptive response and protects tissue from further damage. response involves a sensory receptor, a sensory nerve from the spinal cord, and an effector muscle.
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60
Q

regarding local adaptation syndrome:
- what is the inflammatory response?

A
  • stimulated by trauma or infection. localizes the inflammation containing spread and promotes healing. inflammatory response may produce localized pain, swelling, heat, redness and changes in functioning.
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61
Q

regarding local adaptation syndrome:
- what are the 3 phases of inflammatory response?

A
  • first phase- narrowing of blood vessels occurs at the injury to control bleeding. Histamine is released at the injury, increasing the number of white blood cells to combat infection.
  • second phase- characterized by release of exudates from the wound
  • third phase- the last phase is repair of tissue by regeneration or scar formation. regeneration replaces damaged cells with identical or similar cells.
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62
Q

regarding GAS/ Stress syndrome:
- define the 3 stages and sub stages.

A
  • GAS or Stress syndrome- characterized by chain of physiologic events.
  • alarm reaction- initial rx of the body which alerts the body defenses- divided into 2 parts: shock phase and countershock phase
  • stage of resistance- occurs when bodys adaptation takes place, the body attempst to adjust with the stressor and to limit the stressor to the smallest area of the body that can deal with it.
  • stage of exhaustion- adaptation that the body made during the second stage cannot be maintained- the ways used to cope with the stressors have been exhausted.
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63
Q

what is the bodys reaction to stress

A
  • stressors stimulate the sympathetic nervous system which stimulates the hypothalamus-
  • the hypothalamus releases coricotrophin releasing hormone- during times of stress the adrenal medulla secretes epinephrine and norepinephrine in response.
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64
Q

regarding body reaction to stress:
- what is body responses to epinephrine?

A
  • increase myocardial contractility- increasing cardiac output and blood flow to active muscles
  • bronchial dilation which allows increased oxygen intake
  • increased blood clotting
  • increased cellular metabolism
  • increased fat mobilization to make energy available and to synthesize other compound needed by the body
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65
Q

what are some physiological indicators of stress?

A
  • pupils dilate to increase visual perception
  • seat production
  • increase heart rate and cardiac output
  • skin is pallid due to constriction of vessels
  • sodiaum and water retention increase
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66
Q

what is the rationale for adjustments according to faye?

A
  • find the hypomobility
  • use adjustments to mobilize the fixation
  • recheck to confirm that movement has improved.
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67
Q

define doctors of chiropractic.
what is the focus of chiropractic?

A
  • DC of chiropractic are health care professionals for the dx care and prevention of disorders of the spine as well as other parts of the musculoskeletal system
  • the focus is on maintaining proper spinal function and its relationship with neuro system
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68
Q

what is chiropractic based on?

A
  • the premise that the nervous system fx to control and coordinates all organs and structures of the body and relates the individual to ones environment
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69
Q

what is a subluxation

A

a technical term for spinal joint dysfunction - disturbed joint fx

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70
Q

explain the adjustment

A
  • specifically applied to joints can restore motion when the body’s own muscles cannot.
  • adjustments help relieve pain, as well as restore and maintain normal movement, biomechanics and fx.
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71
Q

what does function mean?
what does it include?

A
  • the ability to perform activities of daily living
  • includes range of motion, flexibility, stability, strength and coordination
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72
Q

what is dd palmer paradigm?

A
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73
Q

what did dd palmer start as ?
what was the publication he started?
what became a central defining aspect of chiropractic paradigm>

A

magnetic healer
- the magnetic cure
- dis-ease is a condition of not-diseas, lack of ease.

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74
Q

in 1902 what core tenets of chiro were in place?

A
  • subluxation caused impinged nerves, which caused dysfunction - the adjustment of subluxation led to normal fx, improved tone and health. chiropractic was the cure for many diseases because it went directly to the cause.
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75
Q

in 1903 what did dd palmer develop?
- what governed what?
- what was central?

A
  • theory of innate intelligence and educated intelligence
  • innate governed the vital systems- interior processes and educated was in charge of the motor systems and looked out for exterior threats.
  • central is adaptation
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76
Q

Who is AP Davis?

A
  • med. doctor- naturopathic doctor, osteopath
  • AT stills first class at kirkville
  • published osteopathy illustrated in 1889 same year he became dd palmer 2nd student
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77
Q

what is davis’ theory?

A
  • circulation of the body being effected by heat and cold.
  • if blood flow is impeded the tissue fails to get what they need and emaciation ensues.
  • goal is parasympathetics
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78
Q

who is OG (oakley) smith?

A
  • dd palmer 10th student
  • he started as a patient when he was in highschool
  • he became protege and friends with bj
  • he used SUBLUXATION
  • eventually joined dd’s rival sol langworthy
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79
Q

who is sol langworthy?

A
  • graduated from palmer in 1902- he is pioneer for TRACTION in chiropractic.
  • he started the American School- with partners Smith and Paxson which was direct competitor with Palmer
  • ## he wanted to include other health practices in school.
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80
Q

who is willard carver

A
  • lawyer- helped dd and bj in separation- he also became a chiropractor and formed his own school
  • taught first generation chiro’s- influence on chiro education.
  • psychological theories into chiropractic- known for developing the structural school of chiropractic
  • THE PRINCIPLE OF BASIC AND COMPENSATORY DISTORTION
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81
Q

what is the definition of principle of basic and compensatory distortion?

A
  • when basic structure moves in relation to the body gravity line, relative and superimposed structures must alter their relation to that line or equilibrium is lost.
  • when a weight bearing structure is distorted, nature automatically attempts to restore body balance by developing compensatory counter distortions. SFMA
  • law of equilibrium- if the bottom floor is not level the floors above must compensate.
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82
Q

who is andrew still?

A
  • founder of osteopathy 1874
  • manipulation of joints and bones- to dx and treat illness.
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83
Q

who is sam weed?

A
  • reverend weed coined the term chiropractic-
  • chiro= hand and practic= to practice
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84
Q

who is mabel palmer?

A
  • known as the first lady of chiropractic- author of first anatomy text for chiropractic (she was not the first female chiro)
  • wife of BJ- professor of anatomy for 40 years
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85
Q

who is minor paxson

A
  • the first femal chiropractic graduate
  • co author of the first chiro text book
  • first female to get a state license in Illinois
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86
Q

who is shegataro morikubo DC

A
  • impacted chiropractic with landmark case ruling that chiro was separate and distinct with philosophy and science and art
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87
Q

who is john howard DC

A
  • graduate of palmer opened his own college
  • suggested that linguistic maneuvering for legal reasons
  • wanted to reshape the profession along the lines of biomedical paradigm
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88
Q

who is leroy baker

A
  • the first chiro student of DD palmer- did not graduate.
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89
Q

who were the first 2 graduates of palmer?

A
  • william seeley was first then AP Davis was second
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90
Q

who was harvy lillard

A

he was the janitor who was first treated with the technique known as chiropractic by DD

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91
Q

where did the term subluxation come from?

A
  • medical term adapted by DD to describe the phenomenon that he experienced in his newly discovered practice.
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92
Q

bj and subluxation

A

subluxation could occur only at the atlas because of unique position in relation to whole body function. “foot on the hose”

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93
Q

innate intelligence

A
  • part of the all wise- innate is a part of the creator- innate spirit is a part of universal intelligence- individualized and personified.
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94
Q

what is universal intelligence

A

god- the universal intelligence- the life force of creation

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95
Q

what is educated intelligence

A

brain power and the things that we learn via both formal and informal education

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96
Q

what is vitalism
who was considered a vitalist/

A
  • a theory that an organic molecule cannot be produced from INORGANIC molecules, but instead can only be produced from a living organism or some part of a living organism.
  • vitalism suggested that an organic molecule such as urea cannot by synthesized
    solely from inorganic sources.
  • DD considered himself a vitalist
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97
Q

the first state to license chiros was?

A

kansas 1913

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98
Q

the last state to license chiros is

A

louisiana 1974

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99
Q

who wrote the 33 principles of chiropractic?

A

RW stephenson and endorsed by BJ palmer

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100
Q

what are the core elements of cleveland chiropractic?

A
  • the body is a self regulating and self healing organism with an innate ability to adapt to a changing environment
  • the NS is the master system that regulates and controls all other organs and structures and relates the indiv. to his or her environment
  • spinal biomechanical dysfunction in the form of vertebral subluxation complex may adversely affect the nervous system ability to regulate function
  • the central focus of the doctor of chiropractic is to optimize patient health by correcting, managing or minimizing vertebral subluxation through the chiropractic spinal adjustment.
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101
Q

what is the Lantz’s 9 model

A
  • describes the VSC vertebral subluxation complex by describing a hierarchy of orgnization and a pattern of inter relatedness of components.
  • the common denominator is the focus on restricted motion and its affects on nervous system function
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102
Q

what are these?

A

A- kinesiologic
B- vascular
C- myologic
D- neurologic
E- connective tissue
F- inflammatory response
G- anatomic
H- physiologic
I- biochemic

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103
Q

regarding lantz 9 model
- what does kinesiologic rep?
- what does myologic rep?
- what does neurologic rep?
- what does vascular rep?
- what does connective tissue rep?

A
  • kinesiologic= importance of motion
  • myologic= tissue level comp of VSC/ muscles initiate and control movement
  • neurologic- ties the nervous system to the system that ultimately controls movement
  • vascular= nutrition to and removal waste products from all tissues
  • connective tissue= limiting and stabilizing movement.
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104
Q

regarding lantz 9 model
- - what may the neurologic component affect?

A
  • affect nerve roots, dorsal root, ganglia, spinal nerves, recurrent meningeal nerve, articular neurology and spinal reflex pathways.
  • this can lead to perception of spinal joint pain.
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105
Q

regarding lantz 9 model
- how does connective tissue comp. make impact?

A
  • impacts joint immobilization and associated connective tissue changes
  • changes include synovial fluid- articular cart. might shrink. adhesions might form. cascade changes contribute to further loss of motion
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106
Q

regarding lantz 9
- what is the vascular component

A
  • related to the anatomical arrangement of segmental artery and veins passing back through the IVF supplying a motion seg. including the dorsal and ventral nerve roots.
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107
Q

regarding lantz 9 model
- what is the inflammatory comp.

A
  • related to chemical radiculits or inflammation surrounding tissues altering nerve function.
  • inflamed dorsal root ganlion can cause an action potential discharge to continue after the mechanical stimulus has ceased.
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108
Q

what is the 3 component kent model

A
  • the 3 DYS’s
    dyskinesia, dysponesis, dysautonomia
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109
Q

regarding kents 3 model
what is dyskinesia?

A

the distortion of difficulty with or impairment of voluntary movement- changes may be segmental or regional- VSC models all tie nicely to motion

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110
Q

regarding kents 3 model
what is dysponesis

A
  • reversible pathophysiologic state consisting of unnoticed, misdirected neurophysiologic reactions through out the organism.
  • emotions and thought included
  • errors in energy expenditure, errors in action potential output from the motor and premotor areas of the cerebral cortex that may result in longer appropriately responding and adapting to the environment due to subluxation.
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111
Q

regarding kent 3
- what is dysautomonia

A
  • functional abnormalities of the autonomic nervous system. sympathetic and parasympathetic systems primarily tied to visceral function.
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112
Q

what is afferentation

A

the input of sensory information from a body part to the brain

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113
Q

what is dysafferentation

A
  • a dysfunction of afferentation or input ot the CNS
  • spinal biomechanical dysfunction may result in alteration of normal nociception, mechanoreception or both, increase in nociceptor input and a reduction in mechanoreceptor input
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114
Q

regarding nociceptive axons
- what is hooshmand description?

A
  • describes the mechanism as restricted joint mobility resulting in decreased firing of large diamter mechanoreceptors axons type II and increased firing of nociceptive axons type III and c-fibers - type IV
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115
Q

does pain have receptors?

A
  • no pain is an interpretation of the CNS that occurs in the brain as a result of information that it receives
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116
Q

what does henderson propose regarding how an adjustment affects dysafferentation ?

A

-altered somatic afferent input theory- proposes that spinal adjustments may normalize articular afferent input and reestablish normal nociceptive and kinesthetic reflex thresholds

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117
Q

what does kent propose?

A
  • aberrated afferent input to the CNS may lead to DYSPONESIS
  • dysponisis is a reversible physiopathologic state consisting of unnoticed, misdirected neurophysiologic reactions to various agents and the reercussions of these reactions throughout the organism.
  • agents involved in the condition of dysponesis can include environmental events, body sensation, emotion and thoughts
  • correction or reduction of vertebral subluxtion FACILITATES the restoration of proper tone throughout the nervous system
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118
Q

what is salutogenesis?

A
  • the origins of health and focuses on factors that support human health and well being rather than on factors that cause disease-
  • salutogenic model concerned with the relationship between health, stress, and coping through a study of holocaust survivors.
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119
Q

what is Seamans proposal?

A
  • symptom relief and health improvement following chiropractic adjustments is the restoration of normal afferent input.
  • joint restriction reduces large diameter afferent nerve fiber input from MECHANORECEPTORS IN THE ARTICULAR CAPSULE AND FROM INTRINSIC MUSCLES OF THE SPINE (HILTONS LAW) leads to excitation of type III and C-fibers producing autonomic symptoms, pain, sympathetic vasoconstriction and reflex muscle spasm.
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120
Q

what other connection does Seaman make in the mechanism of the adjustment?

A
  • emotion, descending inhibitory pathways can influence and may be affected by emotions.
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121
Q

what is aberrant spinal reflex hypothesis?

A
  • VSC affects the NS (body reflexes- such as muscular contraction and glandular secretion- heart beat, respiration, digestive and postural adjustments.
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122
Q

what is the refex arc?

A
  • receptor stimulus (skin) - in through dorsal horn- is an actual horn
  • sensory neuron- integration center (lateral horn)
  • motor neuron from anterior horn (near the crack)
  • to the effector (muscle)
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123
Q

what is somatosomatic reflex?

A
  • receptor and effector are part of somatic division of PNS
  • somatic supplies and recieves info from skin, skeletal muscles, joints and tendons
  • visceral division of PNS supplies and receives info from smooth muscle, cardiac muscles and glands.
  • visceral motor fibers make up the ANS
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124
Q

what are the 4 types of spinal reflexes?

A
  • somatosomatic- afferents and efferents are somatic nerve fibers
  • viscerovisceral- afferents and efferents are visceral sensory fibers and autonomic nerve fibers
  • somatovisceral- afferents are somatic sensory fibers and efferents are autonomic efferent fibers
  • viscerosomatic- afferents are visceral sensory fibers and efferents are somatic motor nerve fibers.
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125
Q

aberrant spinal reflex hypothesis-

A
  • joint subluxation can alter spinal reflexes adversely
  • irvin korr
  • demonstrated that a number of nervous system changes occur associated with movement restriction in the afferent receptors and heightened reflexive activity at spinal cord levels assoc. with palpable lesions.
  • korr says this is FACILITATION- FACILITATED SEGMENT- FACILITATED LESION
  • korrs theory of facilitation is included in most models of vertebral subluxation complex
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126
Q

what are korrs thoughts on facilitation?

A
  • the spinal adjustment influences facilitation both directly and indirectly
  • directly- the joint and muscle receptors in the are produce a reflex response that modifies or inhibits the current facilitated reflex activity. - this causes relaxation of the paraspinal muscles.
  • indirectly- the adjustment normalizes joint mechanics, removing the dysfunction that produces abnormal levels of spinal reflex activity that resulted in a facilitated segment in the first place.
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127
Q

what is a somatosomatic reflex- example?
who/what is related to this thought?

A
  • occurs when a stimulus at one area of the musculoskeletal system produces reflex activity in the nervous systemm which is then exhibited elsewhere in the musculskeletal system
  • deep tendon reflexes are the most well known example
  • related to this is role of interneurons of the dorsal horn in pain inhibition.
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128
Q

explain Walls gate control theory - somatosomatic reflexes of interneurons of the dorsal horn

A
  • afferent inhibition occurs due to mechanically activating the large, fast conducting alpha fibers type Ia and Ib fibers which in turn inhibit the synaptic transmission of signals interpreted as pain by blocking the synaptic gates normally used by the smaller c fibers.
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129
Q

what is the wyke theory

A
  • wyke suggests spinal manipulation stretches mechanoreceptors in the joint capsule type II leading to an inhibitory effect, mediated through spinal cord interneurons on nociceptive activity
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130
Q

what is proprioceptive insult hypothesis

A
  • subcomponenet of somatosomatic reflex model - receptors in the highly innervated soft tissue in and around joints become irritated leading to reflex modification in postural tone and neural integration of postural activities.
  • JANSE PROPOSES AN AFFERENT BARRAGE OF IMPULSES INTO THE NS DISTURBS EQUILIBRIUM, CREATING SOMATOSOMATIC REFLEXES AND CAUSING ABERRANT SOMATOVISCERAL AND SOMATOPSYCHIC REFLEXES.
  • Koors facilitation would be muscle spasm or hypertonicity of skeletal muscle
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131
Q

what is somatovisceral reflex theory

A
  • aka somatosympathetic, or somatoautonomic-
  • stimulus to nerves or receptors related to spinal structures produces reflexive responses influencing function in visceral organs.
  • indirect effect from spinal nerves and visceral organs through interaction with the autonomic ganglia or through vasomotor responses to vasculature supplying the visceral organ.
  • this theory explains mechanism of tachycardia- hypertension etc.
    ( there are no direct connections between spinal nerves and visceral organs, this theory proposes an indirect effect )
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132
Q

what is viscersomatic reflex

A
  • occurs when a stimulus to nerves or receptors related ot visceral organs produces reflexive response influencing function in the musculoskeletal system - examples are asthma- causing muscle tension and joint function/ colic / ab pain/ functional GI pain.
  • patient with muscular skeletal symptoms cause by visceral condition may not respond to manual therapies- many visceral conditions refer pain to other areas of the body. - this is why history taking is important
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133
Q

Ib

A

sensory only
- golgi tendon organ- afferent
- largest diameter
- fastest
- yes to myelin
PROPREOCEPTION

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134
Q

II

A
  • sensory only- fLOWER SPRAY ENDING
  • secondary afferent of muscle spindles, touch pressure
  • medium
  • medium speed
  • yes to mylin
    PROPREOCEPTION AND MECHANORECEPTORS
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135
Q

Ia

A
  • sensory only
  • muscle spindles afferent
  • largest
  • fastest
  • myelin yes
    PROPREOCEPTION
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136
Q

III

A
  • sensory only
  • touch, pressure, fast pain temp.
  • small diameter
  • med. conductive
  • yes to myelin
    NOCICEPTOR, THERMORECEPTOR, MECHANORECEPTORS
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137
Q

IV

A
  • sensory only
  • pain temp, OLFACTION
  • small diameter
  • slowest
  • NO MYELIN
    NOCICEPTORS, THERMORECEPTORS
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138
Q

C

A
  • sensory and motor
  • slow pain, post ganglia, olfaction ANS
  • smallest
  • slowest
  • NO MYELIN
    ANS
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139
Q

B

A
  • sensory and motor
  • preganglionic, ANS
  • small
  • med conductivity
  • yes to myelin
    ANS
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140
Q

A alpha

A
  • sensory and motor
  • largest
  • fastest
  • myelin
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141
Q

A beta

A
  • sensory and motor
  • touch pressure
  • med. diameter
  • med speed
  • yes to myelin
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142
Q

A gamma

A
  • sensory and motor
  • muscle spindles
  • med. diameter
  • med. speed
  • yes to myelin
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143
Q

A delta

A
  • sensory and motor
  • touch pressure, temp, fast pain
  • small
  • med.
  • yes to myelin
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144
Q

regarding selye:
during the stress response:
- what happens to the to the hormones?
- what happens to the cardiac region?

A
  • there is a stress hormone release
  • increased cardiac output vasoconstriction/ blood pressure increase/ BP increases to move the hormones through the system faster
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145
Q

regarding selye and stress response:
- what type of catabolic activities occur?
- what type of anabolic activities occur?
- what effect does this have on immunity?

A
  • there is uptick in catabolic activities/ this breaks down the body because we need all these energy stores quickly for acute stress situations
  • this will decrease healing, growth and repair (anabolic activities) over a long period of time if the stress becomes chronic
  • this will decrease immunity due to the high degree of metabolic expense. Energy used makes the individual tired especially during long stressful period.
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146
Q

regarding selye and the stress response:
- how does the stress effect the brain and learning?
- what is the brain focused on during these times?
- what happens to the brain cells- in what specific area especially?
- what is the brain then trained to anticipate- and not able to concentrate on long term memory?

A
  • factual learning decreases during stress brain activity
  • brain is focused only on the task or danger at hand
  • the brain cells shrink under long tern stress- esp. in the hippocampus
  • this stress event trains the brain to anticipate a future event- and not long term memory.
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147
Q

regarding selye and stress response:
- what tends to increase under stress response?
- what is an example of this?

A
  • increase in sensitivity to sensory organs to survive - if they are consistently being stimulated the can’t concentrate due to exhaustion-
  • example would be adhd and test taking
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148
Q

regarding selye and stress response:
- what happens to cholesterol
- what happens to blood sugar and fatty acids

A
  • cholesterol increases and blood starts to clott
  • increase in blood sugar and fatty acids for burning and energy to survive
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149
Q

regarding selye and stress response:
- what is a stressor
- what is stress response

A
  • any time you eat, move or think that is not conducive to health or lack of movement.
  • stress response is an intelligent response by the body.
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150
Q

regarding selye and chronic response:
- what does acute stress that becomes chronic create?
- what can this lead to?
- what model is this related to?

A
  • it creates a situation that is not temporary
  • can lead to a decrease in growth, repair, brain function, organ function and immune function.
  • this is related to the biochemical response of the Faye subluxation model
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151
Q

regarding chronic stress:
- it can lead to increase blood ____
- the increase in insulin can decrease what?
- create difficulty producing and processing?

A
  • blood glucose, insulin and cholesterol
  • can decrease HGH
  • producing and processing Mg which helps with muscle relaxation
152
Q

regarding chronic stress:
- causes constriction of the blood vessels which increase? and produces ?
- is this pathology?

A
  • constriction of the blood vessels and increase blood pressure and weight gain
  • this in NOT pathology, it is physiology- reacting to stress in not a dz.
153
Q

what is JMPT?

A

proper motion and alignment of the spine is a genetic requirement for health. lack of motion is a stressor
- spinal joints that do not move will degenerate- sending stress to the brain- release of stress hormone- leads to a prolonged stress response- stress does not only come with pain.
- health is not how you feel- it is how you function
- function is the most important part of health- not pain

154
Q

what happens when chiro adjusts the spine?

A
  • decrease blood pressure
  • increase blood flow
  • decrease glucose
  • decrease cholesterol
  • decrease in catabolism
155
Q

following an adjustment what happens?

A
  • increase blood flow
  • improved immunity, memory, sleep, brain function- overall health
156
Q

what effect does movement have on the body overall?

A
  • movement stimulates brain activity and helps to block the stress response
  • to create health, decrease stress reaction and chronic stressors
157
Q

regarding cervical spine:
- define anterior head carriage
- explain alfre breig study
- what does dorr and davis study show

A
  • anterior head carriage- for every inch forward, you double the weight stress of the spine
  • alfre breig- loss of cervical curve can stretch the cord 5-7 cm and produce pathological tension putting the body in state of stress.
  • korr and davis- loss of cervical curve caused weakness in immune system, allowing for organ disease and increase sensitivity to pain.
158
Q

regarding upper motor neurons:
- where are they found?
- where do they carry information to?
- they are the main source of what?
- what cells are they made from

A
  • they are found in the cerebral cortex and brainstem
  • they carry information down to activate interneurons and lower motor neurons, which in turn directly signal muscles to contract and relax.
  • they are the main source of VOLUNTARY MOVEMENTS
  • made from pyramidal cells
159
Q

regarding upper motor neurons:
- describe the course that the upper motor neuron takes.
- what is the neurotransmitter involved- what does it do- what detects it?

A
  • UMN descends in the spinal cord to the level of the approp. spinal nerve root. there the UMN synapses with the LMN each of whose axons innervate a fiver of skeletal muscle
  • involved in glutamate which transmits the nerve impulses from the upper to lower motor neurons where it is detected by glutamatergic receptors
160
Q
  • what is another aka for UMN lesion?
  • where do these occur?
  • lesions can arise as a result of?
  • what are the results?
  • what ortho test would you conduct?
A
  • pyramidal insufficiency
  • neural pathway above the anterior horn of the spinal cord
  • result of a stroke, MS, spinal cord injury and brain injury-
  • results range from muscle weakness, decreased motor control, increased reaction of spinal reflexes causing spasticity, conus and excessive extensor plantar response
  • babinski to confirm
161
Q

regarding UMN pathways:
corticospinal tract:
- where does it travel from- to?
- what is the major function of this pathway?
- what does this control primary?
- where does the tract begin?
- injuries to the lateral cortical tract results in?
- ultimately corticospinal neurons synapse with ?

A
  • travels from the motor cortex to lower motor neurons in the ventral horn of the spinal cord.
  • fine boluntary motor control of the limbs- voluntary body posture adjustments
  • controls primary motor activity for the somatic motor system from the neck to the feet. involved in voluntary movements-
  • the tract begins in the primary motor cortex where the soma of pyramidal neurons are located within cortical layer V
  • ipsilateral paralysis- inability to move, paresis- decreased motor strength- and hypertonia- increased tone for muscles innervated caudal to the level of injury
  • they synapse directly onto alpha motor neurons for direct muscle control
162
Q
  • how many neurons in the CST become myelinated in the first 2 years?
  • connections to the somatosensory cortex suggest?
  • how is above possible?
  • after patients are lesioned they are paralyzed where?
A
  • more than 1 million neurons
  • the pyramidal tracts are also responsible for modulating sensory information from the body.
  • connections cross the midline at the level of the medulla and others at the level of the spinal cord, each side of the brain is responsible for controlling muscles on the opposite side of the body.
  • paralyzed on the corresponding side of the body- however they can re-learn some crude basic motion, just not find movements.
163
Q

regarding lateral corticospinal tract
- what is the largest portion of the CST?
- descending motor pathways carry motor signals from?
- they consist of ?
- the lateral coricospinal tract is a descending motor pathway that begins ? and decussates where? of what portion?
- then proceeds down the ______ side of the spinal cord.
- what is the largest part of the corticospinal tract?
- it extends throughout the entire length of the _____ and appears as an oval area in front of the _____ and medial to the _____ .

A
  • largest portion is lateral corticospinal tract
    90% of the CST fibers
  • from the brain down the spinal cord and to the target muscle or organ.
  • they consist of an upper motor neuron and a lower motor neuron.
  • begins in the cerebral cortex, decussates in the pyramids of the lower medulla- medulla oblongata or cervicomedullary junction- the most posterior division of the brain. -proceeds down the contralateral side of the spinal cord.
  • lateral corticospinal tract
  • length of the medulla spinalis
  • posterior column
  • posterior spinocerebellar tract
164
Q

regarding the lateral CST:
- where do the axons of the lateral CST weave into?
- control of more central axial and girdle muscles comes from the ?

A
  • into the anterior horns of the spinal cord.
  • anterior corticospinal tract.
165
Q

regarding the lateral CST:
- damage to different parts of the body depend on what?
- damage to above-
- example is
- damage to below
- example is
- what are mc complications?

A
  • whether the damage is above or below the pyramidal decussation.
  • above the pyramidal decussation cause contralateral motor deficits.
  • lesion at the pre-central gyrus in the right cerebral cortex then left side of the body will be affected.
  • the pyramidal decussation results in ipsilateral motor deficits.
  • example of this- spinal cord damage on the left side of the lateral corticospinal tract at the thoracic level can cause motor deficit to the left side of the body
  • mc complications are polio and ALS- causing muscle atrophy so must be at or below the brainstem
166
Q
  • what are the aka’f for anterior corticospinal tract?
  • this tract is usually (size)
  • it lies close to ??
  • it is present only in what portion of the spinal cord
  • what happens to its size as it descends?
  • where does it end?
A
  • aka’s include ventral corticopspinal tract/ bundle of turck/ medial corticospinal tract/ direct pyramidal tract/ anterior cerbrospinal fasciculus
  • usually small in size
  • lies close to the anterior median fissure
  • present only in the upper part of the spinal cord
  • it diminishes in size as it descends
  • it ends middle of thoracic region
167
Q

regarding anterior corticospinal tract:
- what makes these fibers different from lateral?
- where do they cross over?
- where do they synapse
- what movements does this tract control?

A
  • they do no decussate at teh level of the medulla oblongata
  • they cross over in the spinal level they innervate
  • they synapse at the anterior horn with the lower motor neuron synapse at the target muscle
  • controls the movements of axial muscles of the trunk
168
Q

regarding corticobulbar tract:
- this tract is a two-neuron _____ motor pathway connecting the ____ in the _____ to the _____ pyramids, which are a part of the ______ also called bulbar region.
- what are these primarily involved in?
- the corticobulbar tract is one of the ____ tracts - what is the other…?

A
  • white matter/ motor cortex/ cerebral cortex/ medullary pyramids/ brainstems medulla oblongata (also called bulbar region)
  • involved in carrying the motor function of the non-oculomotor cranial nerves.
  • pyramidal tracts- the other being corticospinal tract.
169
Q

regarding CBT:
- what is the CBT composed of?
- what muscles are controlled?
- where do they terminate
- fibers that end in the sensory nuclei enhance ?

A
  • upper motor neurons of the cranial nerves
  • face, head and neck
  • motor neurons within brainstem motor nuclei.
  • or inhibit sensory transmission across various sensory nuclei- allows for the selective attention or inattention towards various stimuli
170
Q

regarding CBT:
- what does it innervate?
- also contributes?
- what % of fibers decussate?

A
  • cranial motor nuclei bilaterally- genioglossus muscles- CN V, VII , IX and XII
  • also contributes to the motor regions of cranial nerve X in the nucleus ambiguous
  • only 50% of fibers decussate
171
Q
  • what are Aka for coliculospinal tract?
  • what does this nerve tract coordinate
  • what “system” is this a part of>
  • the tract descends to the cervical spinal cord to terminate ?
  • what movements are coorinated?
A
  • tectospinal tract
  • head and eye movements
  • part of the extrapyramidal system- connect teh midbrain tectum and cervical regions of the spinal cord.
  • descends to the cervical spinal cord and terminates in Rexed Laminae-
  • VI, VII ad VIII to coordinate head, neck and eye movements- in response to visual stimuli
172
Q

regarding rubrospinal tract:
- where is this located?
- originates in?
- where does it cross over and descend?
- describe its travels in the spinal cord
- what is the tract responsible for?
- where does it terminate- - what does it function

A
  • located in the midbrain
  • originates in the magnocellular red nucleus
  • crosses to the other side in midbrain and descends in the lateral part of the brainstem tegmentum
  • travels through lateral funiculus then adjacent to the lateral corticospinal tract.
  • large muscle movemement regulation flexor and inhibiting extensor tone as well s fine motor control .
  • it terminates primarily in the cervical and thoracic portions of the spinal cord
  • it functions in upper limb but not in lower limb control.
173
Q

regarding vestibulospinal tract:
- this tract is a component of what system?
- they relay information from — to —- the nuclei receive info through ?
- what is the function of these motor commands ?

A
  • extrapyramidal system- classified as component of the medial pathway
  • from nuclei to motor neurons. The vestibular nuclei receive info through the vestibulocochlear nerve about changes in the orientation of the head.
  • function is to alter muscle tone, extend, and change the position of the limbs and head with the goal of supporting posture and maintaining balance of the body and head.
174
Q

regarding vestibulospinal tract:
- part of what system in the CNS
- what is primary role
- how is it able to respond correctly?
- when vestibular sensory neurons detect small movements of the body what does vestibulospinal tract command?

A
  • vestibular system
  • maintain head and eye coordination, upright posture and balance and conscious realization of spatial orientation and motion .
  • by recording sensory information from hair cells in the labyrinth of the inner ear. / then the nuclei receiving these signals project out to the extra ocular muscles, spinal cord and cerebral cortex to execute functions.
  • commands motor signals to specific muscles to counteract these movement and re-stabilize the body.
175
Q

regarding vestibulospinal tract:
- what are the 2 parts?
- which tract is found in the lateral funiculus?
- where does it originate?
- describe
-

A
  • lateral vestibulospinal tract and medial vestibulospinal tract
  • LVST is found in lateral funiculus, (bundle of nerve root in the spinal cord)
  • originates in the lateral vestibular nucleus or DEITERS NUCLEUS IN THE PONS- this nucleus extends fro pontomedullary junction to the level of abducens nerve nucleus in the pons
  • lateral vestibulospinal fibers descend uncrossed or ipsilateral in the anterior portion of the lateral funiculus of the spinal cord- fibers run down the total length of the spinal cord and terminate at the interneurons of laminae VII and VIII. additionally some neurons terminate directly on the dendrites of alpha motor neurons in the same laminae.
176
Q

regarding vestibulospinal tract 2 parts:
- what are the two parts?
- which tract is found in the anterior funiculus?
- where does it originate
- where does this nucleus extend
- describe.

A
  • lateral vestibulospinal and medial vestibulospinal
  • medial vestibulospinal tract is descending extrapyramidal motor neurons found in the anterior funiculus
  • originates in the medial vestibular nucleus or SCHWALBES NUCLEUS
  • this nucleus extends from the rostral end of the inferior olivary nucleus of the medulla oblongata to the caudal portion of the pons.
  • the medial vestibulospinal fibers joing with the ipsilateral and contralateral medial longitudinal fascicules- descend in the anterior funiculus of the spinal cord. terminate on neurons of laminae VII and VIII- innervates muscles that support the head. they fun down only to the cervical segments of the cord.
177
Q

VST reflexes:
- what organs are used?
- what is maintained?
- it can also be catagorized by?
- reflexes are important in the maintenance of?

A
  • vestibular organs- skeletal muscle
  • maintain balance, posture, and stability in environment with gravity
  • can be timing- dynamic reflex, static, reflex or tonic reflex. also sensory input can be canals, otolith or both. - below the neck
  • homeostasis
178
Q

tonic labyrintine reflex:
- define-
- what does it help babies master?
- what are the two forms it is found in
- what is forward
- what is backwards

A
  • TLR is a reflex that is present in newborn babies after birth and should be fully inhibited by 3.5 years.
  • master head and neck movements outside of the womb concept of gravity . - increase muscle tone, dev. of the proprioceptive and vestibular senses- and opportunities to practice with balance are consequences of this reflex.
  • when the head bends forward- the whole body arms, legs and torso curl together to form the fetal position
  • backwards- head is bent backward, the whole body arms legs and torso straighten and extend
179
Q

what is the righting reflex?

A
  • positions the head or body back into its normal position, in response to a change in head or body position. ex. is the cat righting reflex- reflex is initiated by sensory information from the vestibular visual and the somatosensory systems
180
Q

regarding reticulospinal tract:
- origins
- involved mainly in?
-

A
181
Q

what are the functions of the resticuloST?

A
  • integrates info from the motor systems to coordinate automatic movements of locomotion and posture
  • facilitates and inhibits voluntary movement- influences muscle tone
  • mediates autonomic functions
  • modulates pain impulses
  • influences blood flow to LATERAL GENICULATE NUCLEUS of the thalamus
182
Q

clinical sig. of the reticuloST

A
183
Q

regarding lower motor neurons:
- what are the 3 types
- a-MNS innervate?
- what are they directly responsible for?
- where are the cell bodies found? but also considered part of which system?
- what are the muscle fibers it innervates referred to?

A
  • a-MNs, b-MNs and y-MNs
  • innervate extrafusal muscle fibers of skeletal muscle
  • responsible for initiating their contraction-
  • found in central nervous system- but also considered a part of somatic nervous system a branch of the PNS because their axons extend into the periphery to innervate skeletal muscles.
  • alpha motor neuron and the muscle fibers it innervates is a MOTOR UNIT.
184
Q

regarding a-MNs:
- where are they found?
- what do the remaining a-MNs innervated

A
  • a-MNs innervating the head and neck are found in the brainstem
  • the remaining innervate the rest of the body and are found in the spinal cord
  • a-MNs are directly proportional to the amount of fine motor control in that muscle
185
Q

a-MNs in the spinal cord
- where are they located?
- what do thes provide?

A
  • located in gray matter that forms the ventral horn
  • provide the motor comp. of the spinal nerves that innervate muscle of the body
186
Q

what are renshaw cells

A
  • inhibitory interneurons found in the gray matter of the spinal cord and are associated in two ways with an alpha motor neuron
187
Q

regarding renshaw cells:
- how do they operate?

A
  • they receive an excitatory collateral from the alpha neurons axon as they emerge from the motor root, and are kept informed of how vigorously that neuron is firing.
  • they send an inhibitory axon to synapse with the cell body of the initial alpha neuron and an alpha motor neuron of the same motor pool.
  • renshaw cell action represents a negative feedback mechansim-
  • renshaw cell may be supplied by more than one alpha motor neuron collateral and it may synapse on multiple motor neurons
188
Q

what is the clinical significance of renshaw cells?

A
  • they are target of the toxin of clostridium tetani- a gram positive spore forming anerobic bacterium that lives in the soil and causes tetanus.
  • when wounds are contaminated with tetani the toxin travel to the spinal cord where it inhibits the release of glycine, an inhibitory nt from renshaw cells. - as a result, alpha motor neurons become hyperactive and muscles constantly contract.
189
Q

what is the primary output of a-MNs?

A

extrafusal muscle fibers

190
Q
A
191
Q
A
192
Q
  • what a muscles general resting level of tension?
  • how is it maintained
  • what is the purpose
A
  • called muscle tone
  • maintained by the motor neurons innervating the muscle
  • purpose is to maintain posture and assist in quicker movements.
193
Q
A
194
Q

regarding sensory nerve fibers:
-what are the types of sensory fibers?

A
  • 1A-A alpha / responsible for proprioception
  • 1B-A alpha/ golgi tendon organ
  • II-A beta/ secondary receptors for muscle spindle, cutaneous mechanoreceptors and some nociception
  • III-A delta/ touch, pressure, fast pain and temp.
  • IV-c / nociceptors of paleospinothalamic tract, temp. and olfaction
195
Q

regarding sensory 1A fibers:
- this is a sensory fiber of a ? called the ? found in muscles
- what does the information from this fiber contribute?
- what are the special sensory receptors called
- what does the body need to keep moving properly?
- what are muscle spindles- where are they found- where do they lie
- which fibers are the largest and fastest fibers
- where are the cell bodies housed?

A
  • stretch receptor called muscle spindle- found in muscles- the information carried by type 1a fibers contribute to the sense of proprioception.
  • for the body to keep moving properly the NS has to have constant input of sensory data coming from areas such as the muscles and joints.
  • to receive a continuous stream of sensory data the body has developed special sensory receptors called proprioceptors.
  • muscle spindles are a type of proprioceptor and they are found inside the muscle itself. they lie parallel with the contractile fibers.
  • these 1A fibers are the largest and the fastest fibers
  • the cell bodies are housed in the DORSAL ROOT GANGLIA (IA FIBERS)
196
Q

type 1B sensory fibers
- in _____ responds to muscle tension changes
- the organ is innervated by ?
- what happens when force is applied to a muscle?

A
  • golgi tendon organ responds to muscle tension changes
  • innervated by primary afferents called group 1b fibers, which have specialized endings that weave in between the collagen fibers.
  • the golgi tendon organ is stretched, causing the collagen fibers to squeeze and distort the membranes of the primary afferent sensory endings.
197
Q

type II fibers:
- this is another type of sensory fiber.
- under pathological conditions they may become?
- are these fast adapters?

A
  • type II sensory fiber- group Aa is another type fo sensory fiber, which participate in the sensation of body position (proprioception) / type II sensory fiber group Ab is a type of sensory fiber, the second of the two main groups of touch receptors
  • under pathological conditions they may become HYPER-EXCITABLE leading to stimuli that would usually elicit sensations of tactile touch causing pain.
  • no these are slow adapters. - it is thought that the 1a fibers signal the degree of change in muscle movement and the type 11 fibers signal the length of the muscle which is later used for forming the perception of the body in space.
198
Q

class 1a, 1b and II are all responsible for the sensory portion of ___

A

proprioception

199
Q
A
200
Q

-this group of nerve fibers are unmyelinated and have a small diameter and low conduction velocity
- these nerve fibers are nociceptors of the?

A
  • group C nerve fibers.
  • anterior spinothalamic tract and warmth receptors
201
Q

what is circulatory hypothesis?

A
  • proposes sympathetic hyperactivity leads to vasoconstriction and a threat of relative ischemia in involved area.
  • chiropractic adjustment improves circulation by restoring joint function and removing the source of sympathetic irritation.
  • musculoskeletal mobility and strength also influence venous and lymphatic flow. alterations can have negative impacts on health
  • vascular response = result of stimulation to the ANS (lateral horn) - there is a need for ample blood supply for optimal function.
  • vasoconstriction resulting from sympathetic hyperactivity reduces blood volume - posing threat of ischemia.
  • signs of reduced circulation to the tissue may be altered texture, moisture or temp.
  • a healthy respiratory pump depends on a functioning diaphragm and flexible spine and rib cage.
  • conditions or injuries that lead to the loss of musculoskeletal mobility and strength result in loss of ability to move blood and lymph- leads to loss of vascularization in the affected tissues.
202
Q

neuroplasticity model

A
  • subluxation is a central segmental motor control problem. CNS is not controlling the movement pattern of spinal segments correctly- leading to altered input ot the brain which affects central processing
203
Q

define neuroplasticity

A
  • the ability of the brain to adapt to changes in an individuals environment by forming new neural connections over time-
  • explains how the human brain is able to adapt, master new skills, store memories and info and even recover after TBI
204
Q

neuroplasticity model

A
  • NP model is another type of dysafferentation. the brain is being provided inaccurate picture of the environment- challenging the body to adapt to changes as it should
  • the brain has ability to grow, change and modify itself as new things are learned and new processes are undergone.
  • ## the main way that the brain improves is by forming neural pathways. - this is communication that the brain has for recognizing information and when new neural pathway are formed- mental health, memory and overall brain functioning improves.
205
Q

what is somatic visceral disease simulation?

A
  • vert. subluxation can simulate the symptoms of visceral disease-
  • aka’s pseudovisceral disease, organ disease mimicry, somatic visceral disease mimicry syndrome and somatic simulation.
  • Nansel and Szlazak- state that patients with apparent visceral conditions who respond to spinal manipulation do not have true visceral disease. - challenge the theory..
206
Q

what is the mechanism for visceral mimicry syndrome?

A
  • visceral afferent nerves transmit nociceptive information from internal organs
  • somatic afferrent nerves transmit nociceptive info from deep connective tissue - then transmitted to CNS pathways
207
Q

what is decreased axoplasmic transport theory?

A
  • the decreased axoplasmic transport theory is a nonimpulse mechanism based not on transmission of signals within the NS but on the intra-axonal transport and exchange of macromolecular materials.
  • this theory involves the neurotrophic relationship between neurons and end organs or target cells.
208
Q

what are trophic substances?

A
  • macromolecules produced in the neuron cell body, packaged by the golgi apparatus, transported within the axon and release at the synapse.
  • these substances have influence to maintain the proper vitality and fx of the target tissues and are considered essential for the maintenance of proper tissue function.
209
Q

what transport methods occur within the axon?

A
  • axoplasmic transport is faster form of transport and carries material in BOTH DIRECTIONS
  • axoplasmic flow is the slower (slow to flow) form of transport and carries materials ONLY TOWARD THE AXON.
210
Q

axon

A
  • every neuron in the brain has an axon.
  • they are also called nerve fiber- part of the neuron that carries nerve impulses away from the cell body.
  • cytoplasmic protrusions from the cell body of a neuron.
211
Q

anatomy of the axon

A
  • axons are covered by membrane known as an axolemma
  • the cytoplasm of an axon is called axoplasm
  • the end branches of an axon are called telodendria
  • the swollen end of a telodendron is axon terminal - forming a synaptic connection
212
Q

korr

A
  • proposes that any factor that causes derangement of transport mechanism in the axon could cause trophic influences to become detrimental
  • cause could be direct mechanical insult such as deformations of nerves and roots. including compression, stretching, angulation and torsion that occur commonly and disturb the intra axonal transports mechanism and intra neural microcirculation
  • this adverse affect on axoplasmic transport is explained in the context of the nerve compression hypothesis
213
Q

what is wallerian degeneration

A
  • consequences of interruption of axonal transport
  • lack of vital proteins and materials causes degeneration of the axon.
  • direct mechanical insult- compression stretching, angulation and torsion- compression hypothesis- -
214
Q

sustained hyperactivity

A
  • places increased energy demands on the cell, thus affecting cellular metabolism and axonal transport. this links the theory to Korr’s fav. theory of facilitation.
215
Q
  • how does the nervous system determine responses of the body?
  • the NS acts as ______ and ______ for the body
  • what are the primary components of the NS?
A
  • to changes in internal and external environments-
  • messenger and coordination
  • the CNS- brain and spinal cord AND peripheral nervous system- cranial and spinal nerves
216
Q

regarding neurons:
- what is neuron derived from?
- what is the cell body?
- what is a dendrite?
- what is the axon?
- what is the telodendria?
- what is the ganglia?

A
  • neuron is derived from ectoderm, from the neural tube, neural crest cells or ectodermal placodes
  • trophic: contains the nucleus and metabolic machinery of the cell
  • receptive: form extensions into tissues that may synapse to one or many other neurons
  • conductive: a long cytoplasmic process also called the nerve fiber- can transmit nerve impulses over a long distance without diminution of amplitude of the signal.
  • transmissive: the terminal branches of an axon- make contact with other neurons at synapse
  • groups of neuron cell bodies that lie peripheral to the CNS in vertebrates
217
Q

regarding types of neurons:
- this neuron has a cell body located near the middle of the axon- like a retinal neuron
- this neuron has a cell body that lies off to one side of the axon- like a sensory neuron
- this neuron has a cell body that lies very close to dendrites- like a motor neuron

A
  • biopolar
  • unipolar
  • multipolar
218
Q

describe the transmission of an action potential.

A

The nerve impulse is electrical that passes as a wave along the surface membrane of a nerve fiber. Normally the nerve cell is resting potential based on the concentration of sodium and potassium ions inside and outside

219
Q

what are the steps that the neuron goes through during action potential ?

A
  1. high concentration of sodium ions outside- negative charge inside cell compared to outside
  2. axon stimulated, ion channels open causing action potential, sodium ions rush into cell, membrane potential reversed and membrane is depolarized.
  3. nerve impulse travels down the axon as a wave of depolarization.
  4. sodium pumped outside of cell and resting potential restored.
220
Q

regarding neurotransmitters:
- where are they located?
- how are they released?

A
  • the telodendria contains synaptic vesicles that contain the neuro transmitters. which are acetylcholine, noradrenaline, serotonin and dopamine
  • they are released when the nerve impulse reaches the telodendria and then cross the synaptic cleft to reach the dendrites of the next neuron in line causing the impulse to be sent by the postsynaptic nerve cell.
221
Q

ss

A

aa

222
Q

ww

A

ss

223
Q

ss

A

ss

224
Q

nodes of ranvier

A

periodic gap in the insulating sheath myelin on the axon of certain neurons that serves to facilitate the rapid conduction of nerve impulses. Nodes of ranvier are approx. 1 um wide and expose the neuron membrane to the external environment.

225
Q

oligodendrocytes

A
  • specialized glial cells that wrap themselves around neurons present in the CNS-
  • primarily responsible for maintenance and generation of the myelin sheath that surround axons
  • they also participate in axonal regulation and the sculpting of higher order neuronal circuits.
226
Q

astrocytes

A

-

227
Q

microglial cells

A
  • specialized pop. of macrophages that are found in the central nervous system.
  • they remove damaged neurons and infections and are important for maintaining the health of the CNS
228
Q

satellite cells

A

small cuboidal cells that surround clusters of neuron cell bodies called ganglia

229
Q

ependymal cells

A
  • derived from primitive neural ectoderm- form the lining of the cavities of the brain and spinal cord.
  • classified with neuroglia
  • embryonic ependymal cells are ciliated and some retain celia permanently
230
Q

what is neuronal circuitry- what does it include?

A

the general parts of the neuronal circuitry include three basic types of neurons-
- primary sensory or afferent
- motor or efferent neurons
- interneurons

231
Q

what do primary sensory neurons carry?

A

they are afferent neurons - carry impulses from free nerve endings or receptor cells into the central nervous system

232
Q

what do motor- neurons carry?

A

they are efferent neurons (exit) carry impulses from the CNS to effectors, such as muscles or glands

233
Q

what do interneurons receive?

A

they receive signals from sensory neurons, integrate information and send signals to motor neurons

234
Q

what do somatic fibers relate to?

A

to the skin and its derivatives, and to voluntary muscles.

235
Q

what are visceral fibers related to?

A

involuntary muscles and glands of the organ systems

236
Q

describe the reflex arc

A
  • messages from the receptor organs are transferred within the spinal cord directly from afferent fibers to efferent fibers, which then send appropriate messages to effector organs
  • the function of the spinal cord is to receive incoming impulses, integrate and coordinate them, transmit them to wherever they should go within the CNS, and send responses to the PNS as appropriate
237
Q

what is the general structure of the spinal cord?

A
  • the grey matter lies on the interior of the cord- whit matter lies exterior
  • grey matter resembles the letter H- with the upper arms called the dorsal columns or horns and the lower arms called the ventral columns or horns
  • the grey commissure makes up the cross arm of the H and transmits fibers from on side of the cord ot the other
  • the external white matter is divided into right and left sides by the doromedian sulcus and the ventromedian fissure
  • the dorsal horn of the cord receives terminations of primary sensory neurons
  • the ventral horn contains the dendrites and cell bodies of motor neurons
238
Q

what is a trophic nerve?
what is trophic function of neuroglia?

A

-one concerned with regulation of nutrition.
- provision of nutrients for neurons. structural support for nerve cell. Release and absorption of ions and neurotransmitters for improved modulation of cell-to-cell communication between neurons.
- repair of the nervous system

239
Q

what is neurodystrophic hypothesis?

A
  • interaction between the fx of the central nervous system and the body’s immunity that lends support to the hypothesis that neural dysfunction is stressful to the body locally and globally.
  • this results in lowered tissue resistance, modifications to the nonspecific and specific immune responses occur, as well as altered tropic function of the involved nerves.
240
Q

which hypothesis demonstrated neuroendocrin-immune connections in animal experiments and clinical investigations?

A

Selye- General Adaptation Syndrome- GAS

241
Q

the stress response in GAS involves?

A

psychological, psychosomatic and sociological components

242
Q

what did selye observe as it relates to GAS?

A
  • selye observed non-specific changes that are labeled general adaptation syndrome
  • he also observed very specific responses that depended on the stressor and on the part of the subject, which he termed local adaptation syndrome.
243
Q

what did selye feel about long term stress?

A

long term stress would lead to diseases of adaptation, including cardiovascular disease, high blood pressure, connective tissue disease, stomach ulcers and headaches.
- stressors can lead to profound health consequences.

244
Q

what do theorists say about stressful events?

A
  • they trigger cognitive and affective responses that, in turn induce sympathetic nervous system and endocrine changes and ultimately impair immune function.
245
Q

how does stress affect immune functions?

A

through central nervous system control of hypothalamic pituitary adrenal HPA Axis and sympathetic adrenal medullary Axis

246
Q

stressors produce ?

A

reliable immune changes

247
Q

what did segerstrom and miller analyze?

A

analyzed different types of stressors separately and found that the immunologic effect of stressors depends on their duration.

248
Q

what if individuals do not develop the same syndrome with same stressors?

A

mason suggested that emotional stimuli under the influence of internal- genetics, past experiences, age, sex or external conditioning (drugs, diet, hormone use) are reflected in the responses of the-
- endocrine
- autonomic system
- musculoskeletal system

249
Q

what did stein, schiavi and camerino contribute to neuroimmunology?

A
  • they demonstrated psychosocial and neural influences on the immune system
  • they showed the hypothalmus has a direct effect on the numeral immune response, explaining that the psychosocial factors can modify host resistance to infection.
  • researchers were able to stimulate both sympathetic and parasympathetic responses by stimulating different parts of the hypothalamus.
250
Q

what is sympathetic response? what is another name>.

A
  • characteristic of fight or flight
  • also known as ergotropic
251
Q

what are some ergotropic responses?

A
  • primarily sympathic
  • excitement, arousal, action
  • movement of body or parts
  • increased heart rate, BP and resp. rate
  • increased blood sugar
  • increase muscle tension
  • increased dioxide consumption
  • increased carbon dioxide elimination
  • pupil dilation
252
Q

what are parasympathetic responses?
what is another name for it?

A
  • relaxation that promotes a restorative process
  • also known as trophotropic
253
Q

what are trophotropic responses?

A
  • primarily parasympathetic
  • energy conservation
  • decreased heart rate- BP and resp rate
  • increased GI function
  • decreased muscle tension
  • decreased dioxide consumption
  • decreased carbon dioxide elimination
  • pupil constriction
254
Q

what is neuroscience, endocrinology and immunology joined? what does this involve?

A
  • psychoneuroimmunology PNI
  • various organs including brain, glands- spleen, bone marrow and lymph nodes joined to one another in a network of communication, linked by information carriers called NEUROPEPTIDES
255
Q

what is PNI grounded in?

A

PNI is a scientifically solid study, grounded in well-designed experiments and in the resolute tenets of behaviorism

256
Q

what are nodal points?

A
  • the first components of the process of linking the systems of the body together, and ultimately the body and mind, are the receptors found on the surface of the cells in the body and brain.
257
Q
  • what type of receptors are found in the spinal cord? what does it filter?
A
  • almost every peptide receptor could be found in the spinal cord site- it filters all incoming bodily sensations.
258
Q

all locations with the information from any of the 5 senses enters the nervous system, there is a high concentration of neuropeptide receptors- what are these regions called?

A

Nodal Points

259
Q

-what is the skin, spinal cord and all organs points of ?
-what do health care providers that incorporate touching and movement in their treatment affect?

A
  • nodal points of entry into the psychosomatic network
  • they affect them all in their treatment of patients.
260
Q

spinal lesions (vertebral subluxation complex) are associated with exaggerated sympathetic activity.

A

Korr

261
Q

they employed the term “tone” in reference to ambient nervous system activity. High sympathetic tone may alter organ and tissue responses to hormones, infectious agents, and blood components.

A

Korr

262
Q

sympathetic stimulation was induced in human volunteers by exhaustive exercise. They found that acute sympathetic stimulation leads to selective release of immuno regulatory cells into the circulation, with subsequent alterations in cellular immune function.

A

Murrey et al.

263
Q

“growing evidence suggests that immune function is regulated in part by the sympathetic nervous system. Sympathetic nerve endings densely innervate lymphoid tissue such as the spleen, lymph nodes and thymus, and lymphoid cells have beta 2 andregenergic receptors.

A

murrey et al.

264
Q

in their experiments there was a sharp rise in T suppressor/cytotoxic cells and natural killer cells following sympathetic stimulation. Only modest rises were seen in T helper and B cells. The cells most affected, the T suppressor/cytotoxic cells and the natural killer cells, are those with the largest density of beta receptors

A

murrey et al.

265
Q

they reported that the neurotransmitter norepinephrine is present in post-ganglionic sympathetic fibers which innervate lymphoid organs and act on the spleen.

A

felten

266
Q

available receptors on cells in the white pulp and the localized neurotransmitter terminal which directly contact T lymphocytes in the periarticular lymphatic sheath.

A

felten

267
Q

they propose that norepinephrine in lymphoid organs fulfills the criteria for neurotransmission and plays a significant role in the modulation of immune responses

A

felten

268
Q

stressful conditions lead to altered measures of immune function and altered susceptibility to a variety of diseases. many stimuli which primarily act on the central nervous system can profoundly alter immune responses. the two routes available to the central nervous system for communication with peripheral organs are neuroendocrine channels and autonomic nerve channels

A

felton

269
Q

cells can learn to associate responsiveness to antigens and other immuno active agents, with responsiveness to signals originating in the CNS delivered via neuroendocrine or autonomic nervous channels. they propose storage memory of stimuli in the immune system rather than in the brain

A

Grossman et al.

270
Q
  • a small controlled study of HIV positive patients
  • the effects of specific upper cervical adjustments on the immune system CD 4 cell counts of HIV positive individuals was studied.
  • half the patients received atlas adjustments based upon grostic upper cervical analysis- other half received a placebo- instrument applied to the mastoid bone.
  • over 6 month period of the study- the control group experienced a 7.96 decrease in CD4 cell counts, while the adjusted group experienced a 48% increase in CD4 cell counts over the same period.
A

HIV study- Selano et al.

271
Q

concluded that concepts of Neuroimmunomodulation, somatosympathetic reflex, and spinal fixation provide a theoretic basis for using spinal manipulation in the management of certain disorders involving the immune system, including asthma, allergic rhinitis and the common cold.

A

Fidelibus

272
Q

MS dysfunction can result in immune dysfunction and that, by removing MS dysfunction, spinal manipulation can affect the immune function.

A

Fedelibus

273
Q

studies on infantile colic indicate that chiropractic treatment results in a reduction of the daily length of daily colic period. 94% of infants were helped by chiropractic within 14 days.

A

klougart and associates

274
Q

this study compared spinal manipulation with medication- infants in chiropractic group showed 67% reduction in colic hours- as opposed to 38% with medication

A

Wiberg, nordsteen and nilsson

275
Q

30 examined asymptomatic randomized to determine if HVLA C/S manipulation would effect salivary cortisol secretion- no response- concluded that CMT did not induce enough anxiety sufficient to disrupt the homeostatic mechanism and activate the HPA axis

A

Whelan- salivary cortisol

276
Q

reported a slight but statistically sig. increase in B-endorphin levels in asymptomatic males after cervical manipulation-

A

Vernon

277
Q

-this is the only extended line of investigation into the effect of chiropractic on immune function.- single manipulation in the thoracic and lumbar spine produced a short term priming of polymorphonuclear cell response to an in vitro particulate challenge. observed enhanced chemiluminescent respiratory burst in both asymptomatic and symptomatic study participants.
- enhanced polymorphonuclear cell activity was associated with slight, but statistically significant rise in plasma substance P.
- this increase may prime circulating phagocytic cells for enhanced resp. burst.
- still not enough evidence to determine if there is actual direct effect on immune system

A

brennan

278
Q

there is growing body of scientific research to support a connection between chiropractic and immune system.
- the nervous system senses damage, infectious agents and foreign bodies with help of chemical releasing immune cells and deals with problems by deploying different types of immune cells to carry out specific procedures.
- disturbances in the nervous system- diminish the ability of an individual to sense and repair damage and combat infection, cancer etc.

A

lit. review Ari Cohn

279
Q

what is this: nerves can become compressed through impingement from intersegmental spinal biomechanical derangements and can have biomechanical, functionaland clinical significance. this is one of the original theories related to subluxation

A

nerve compression hypothesis

280
Q

what is the most likely place for nerve root compression?

A

the IVF- an extended interpedicular zone- tunnel- transforaminal ligaments create functional compartments within the IVF that reduce the functional compartment or space available for the spinal nerve and roots.

281
Q

orthopedist states the close proximity of cervical nerve roots to the anterior and posterior walls of the cervical IVF made them vulnerable to compression- irritation from mechanical derangement or inflammation. they coined the term cervical syndrome to describe the group of symptoms and clinical findings resulting from irritation or compression of the cervical nerve roots in the IVF

A

ruth jackson

282
Q

a medical radiologist nots that arthritic and degenerative changes may become an important factor resulting in foraminal encroachment- he coined the term chronic cervical syndrome to describe a set of symptoms that patients with cervical IVF encroachment suffer.

A

hadley

283
Q

what are the symptoms that hadley states patients suffer from IVF encroachment- chronic cervical syndrome?

A
  • paroxysmal deep or superficial pain in head, face, ear, throat, or sinuses
  • sensory disturbances in the pharynx
  • vertigo
  • tinnitus with diminished hearing
    -vasomotor disturbances- sweating, flushing, lacrimation, salivation
284
Q

in cases of spinal degeneration- normal physiologic motion- may be enough to compromise the space around the nerve root to such a degree that very little safety margin is left. additionally with degeneration, the nerve loses its flexibility and develops adhesions with the IVF walls. This may create the conditions for a chronic threat of compression and mechanical irritation leading to inflammation of the nerve root.

A

panjabi

285
Q

90% of patients with low back pain have dysfunction indicating that changes exhibited are mainly those of abnormal function with slight anatomic changes to the three joint complex, the intervertebral disc and zygopophyseal joints. Of the remaining ten percent he claims that fifty percent of those have lateral nerve root entrapment. With dynamic recurrent lateral entrapment there is a laxity of the posterior joints and of the annulus causing abnormal movement of the vertebrae resulting in a narrowing of the lateral nerve canal and tapping on the main spinal nerve as this passes along the canal- indicates that manipulation is an effective method of treatment for both patients with dysfunction and lateral nerve root entrapment .

A

kirkaldy-willis

286
Q

pain in and from the neck results from the mechanical factor of encroachment of space and impairment of movement. decrease in the space in which pain-sensitive tissues lie or through which they pass compresses these tissues, resulting in possible pain and loss of functions. pain is more apt to-occur if pressure is acute and transient, whereas loss of function is more likely as pressure is prolonged and continuous. the most likely to compress are the intervertebral foramina and within the spinal canal. the tissues in these specific areas are the nerves and their coverings, blood vessels, ligaments, joint capsules and dura mater. encroachment of space resulting in pressure upon these tissues may result in pain or loss of function.

A

cailliet

287
Q

what effects does skeletal muscle have on nerve compression?

A
  • the pathway taken by nerves after leaving the IVF often traverses next to or through skeletal muscle. Neurons may be subject to compression and torsion, as well as chemical influences. adhesions, constrictions and angulations may occur within a nerve traveling through muscle. the nerve sheath surrounding the nerve roots is an extension of the meninges surrounding the spinal cord, leaving open the possibility that peripheral nerve compression could lead to central effects. peripheral nerve entrapments are also commonly seen in a chiropractic compression may be associated with the MYOLOGIC, INFLAMMATORY AND BIOCHEMICAL components of VSC
288
Q

what are the influences of vascular and sympathetic on nerve compression?

A

nerve compression may have the potential to lead to ischemia of nerve cells. Ischemia in the cells of the dorsal root ganglia may lead to progressive loss of sensory function, including propreoception. additionaly edematous pressure from congestion of the venous drainage in the area may affect nerve conduction. also potential for autonomic changes due to the position of the cervical sympathetic ganglia- positioned against the vertebral column and subject to stress imposed by abnormal motion. vascular and sympathetic effects of nerve compression may be associated with the vascular and neurologic components of VSC.

289
Q

what are neurophysiological effects of nerve compression?

A

spinal nerve roots may be susceptible to compression- indirect increases in pressure affecting the nerve root is more likely- Sharpless concluded that 10mm hg produced conduction block and rydevik determined venous blood flow to spinal roots was blocked with 5-10 mmHg pressure.

290
Q

how does mechanical tension and the muscle dural connection contribute to nerve compression?

A
  • mechanical tension on the pain dura mater may contribute to things such as headache- rectus cap. posterior minor extends from the occiput to the posterior arch of the atlas and connects via a bridge of tissue to the dura. this connection may resist inward folding of the dura which may compromise cs fluid flow when the neck is extended. the dura is sensitive and tension applied during surgery caused headache. the muscle dura connection may transmit forces from the neck muscles to the pain dura. this muscle-dura connection may represent an anatomic basis for the effectiveness of spinal adjustment- this may decrease muscle tension and reduce pain by reducing the forces between c1 and C2.
291
Q

absolute contraindication

A

manipulation should not be performed on the patient

292
Q

relative contraindication

A

caution should be used in applying manipulation and consideration given to modifying the procedure

293
Q

informed consent

A

required by insurance carriers= some level of protection- list “possible” problems can create concerns in patients mind

294
Q

complication

A

problem that occurs after the application of a procedure

295
Q

contraindication

A

a problem identified before a procedure is applied that makes application of the procedure inadvisable

296
Q

most common adverse reactions to adjustment

A
  • localized discomfort
  • headache
  • tiredness
  • radiating discomfort
    typically self resolve within 24 hours
297
Q

there is no difference in incidence of stroke in patients visiting DC’s vs. those visiting MD/s

A

cassidy, boyle and cote

298
Q

what are common presenting complaints when possible stroke

A

severe neck pain
sever head pain- different than anything I have experienced before
dizziness
unsteady
vertigo
under age 45

299
Q

what are some associated activities with dissection?

A
  • calisthenics, stretching, yoga
  • wrestling
  • stargazing
  • hair stylist
  • receiving CPR
  • painting ceiling
300
Q

what are the five D’s

A

dizziness- vertigo lightheadedness
drop attacks- loss of consciousncess
diplopia- other visual disturbances
dysarthria
dysphagia

301
Q

what are the 3 N’s?

A

nausea, vomitting
numbness on one side of face or body
nystagmus

302
Q

what do you do if patient has a dissection?

A
  • do not adjust
  • keep patient still
  • record vital signs
  • evaluate pupils
  • nystagmus, tremor, extremity dysfunction
  • can patient swallow, speak, facial numbness
  • call 911
  • follow up with patient daily
  • make detailed records
  • DO NOT ABANDON PATIENT
303
Q

the idea that a long term problem with movement leads to (this)?

A

degeneration

304
Q
A

sandoz 4 phase model of degeneration

305
Q
A

kirkaldy-willis 3 phase model of degeneration

306
Q

certain regions of the central nervous system will become facilitated, or more likely want to “send the message” this is _____ of segmental dysfunction

A

korr model of segmental dysfunction - non inflammatory model

307
Q
A

korr

308
Q

this model is based on the concept of segmental facilitation, having been caused by either a poorly coordinated motion, muscle tension, or visceral spasm-

A

patterson-steinmetz model- non inflammatory model

309
Q

what is the difference between patterson steinmetz model and korr model?

A
  • segmental facilitation may last even after the instigating stimulus is removed. the term used to describe this is the Neural scar- this abnormal segmental reflex circuit itself participates in maintaining- the symptom. a self maintaining subluxation- extended care plan- spinal learning
310
Q

-this model proposes that segmental dysfunction creates both the mechanical and chemical stimulation necessary to lead to activation of both nociceptors and the spino-thalamic tract
- the spinothalamic tract rns from the spine to the thalamis- this pathway takes information from the spine to the brain that can lead to sensations- like pain, temp, and crude touch. This may lead to articular pain- with (P- part) associated reflex muscular changes. (T-part)

A

dvorak model

311
Q

postural muscles shorten (A in part) and fast twitch muscles fatigue. all of this causes muscle spindle activity to increase, resulting in post contraction sensory discharge triggering further contraction. those postural muscles shortening will likely be associated with histochemical changes- potassium concentration- that further maintain the post-contraction sensory discharges. the muscles will soon experience relative hypoxemia and altered joint movement

A

dvorak

312
Q

-spinal dysfunction leads to articular pain and stimulates reflex effects on the muscle
- reflex effects lead to mscle spindle discharge
- muscle spindle discharge lead to 1a fibers post sensory fiber discharge
- this leads to muscle contraction and shortening of the postural slow twitch fibers creating histochemical change- this may include changes in potassium concentrations within the muscles

A

dvorak model

313
Q

those histochemical changes and shortening of the postural slow twitch muscles lead to relative hypoxemia
this hypoxemia leads to damage of the muscle tissue and pain
this muscular damage and pain causes muscular imbalance eventually
disturbed joint movement

A

dvorak

314
Q

trauma or postural strain og skeletal muscles first generates a myofascial trigger point. segmental facilitation is initiated, disruption of small blood vessels leads to the release of platelets and serotonin which sensitizes nerve ending. connective tissue damage results in the breakage of mast cells and the release of histamine, also sensitizing nerve endings. sustained local contraction creates a region of uncontrolled metabolism- uncontrolled metabolism causes additional histamine release depletion of local ATP stores, and the progressive failure of relaxation

A

gatterman - goe

315
Q

by reducing local blood flow, the sustained contractions may stimulate the autonomic nerves and a somato somatic response. the result is local accumulation of metabolites, such as prostaglandins and histamines that further sensitize the nerve endings. they predicted that this self perpetuating cycle is painful, resists stretching and decreases range of motion of the adjacent joints

A

gatterman and goe

316
Q
A

Mense model

317
Q
A

evans minimal energy theory

318
Q

what is cord compression?

A
  • compressive myelopathy- BJ hole in one
  • caused by severe subluxation and ligament laxity at C1-C2
  • thought to cause dentate traction and direct mechanical irritation to the cord
  • lateral translation and distraction can also irritate the cord
319
Q

what is cord traction?

A
  • part of the theory behind SOT- tech by Dr. major dejarnette
  • cranio-sacral meningeal tension
  • craniopelvic manipulation to restore CSF flow
  • CSF pump- top sphenobasilar
  • bottom sacral
320
Q

what is vertebral artery insufficiency - cervical rotation causing symptoms of-

A

nausea
nystagmus
numbness

321
Q

what is dural torque-dentate cord distortion

A
  • dentate ligament- aka denticulate- cross bridges stabilizing the cord, rotational stress torque attachments from pia to dura mater
  • dr.john grostic
322
Q

explain dural torque

A

dr. grostics theory - becuase of C1-2 area controls 50% of the flexion/extension and rotation of the C-spine, it does not have the restrictive control of the joints, as the lower cervical spine does.
- it can then shift and cause compromise ot the spino-cerebellar and spino thalamic tracts. These tracts lay closest to the dentate ligament.

323
Q

dentate ligaments-

A
  • triangular shaped ligaments that anchor the spinal cord along its length down each side to the dura mater. the ligaments arise from the pia mater and attach firmly to the arachnoid and dura
  • the purpose of the dentate ligaments is to stabilize the spinal cord during movement
324
Q

CSF component of dural torque.

A
  • improper circulation of the CSF due to irritation of the dentate ligament has been suggested as a mechanism that is improved with manipulation
  • this rational also lends itself to the stasis of CSF causing aberrant flow and lack of nutrition to the tissues bathed in CSF
325
Q

what does Dr.Grostics paper 1988 suggest?

A
  • 2 mechanisms/
  • direct mechanical irritation
  • venous occlusion causing stasis and ischemia to related tissues- dr. palmer and dr. dejarnet
326
Q

can cervical distraction due to movement of the spine cause conduction block?

A

jarzem reported that there was decreased spinal blood flow and somatosensory interruption with experimental cord distraction.

327
Q

intervertebral encroachment theory IEF

A
  • pressure on the contents of the IVF either cause increase or decrease neural activity
  • increased activity causes paresthesia, pain, hypertonicity, vasoconstriction and sweating
  • decreased activity causes numbness, muscle weakness/paralysis, dry skin and vasodilation
  • the dorsal root ganglia, not the nerve root is susceptible to encroacnment and may be stimulated
  • the DRG is the most sensitive neurological tissue to compressive forces
328
Q

regarding IET:
- stimulation of the DRG causes?

A

release of substance P and vasoactive intestinal peptides VIP at their peripheral terminal. this suggest that the peripheral origins of the sensory fibers are important sites for neurological mediated effects.

329
Q

IET cont:
-irritation to the DRG due to encroachment cause the release of?

A
  • peptides in the intervertebral discs and zygapophyseal joints that cause a breakdown of the structural proteins. this leads to disc degeneration and destruction of the zygapophyseal joints
330
Q

facts about the dorsal root ganglia:

A
  • located in the middle of the IVF in the cervical spine
  • it migrates medially in the thoracic and lumbar spine
  • substance P normally inhibits pain in the CNS
  • reminder- substance P is a neurotransmitter
331
Q

what is the function of the DRG?

A
  • nociception
  • stimulated by temperature- nociception, chemical and mechanical stimuli
  • consists of high and low threshold receptors
332
Q

what is substance P?

A
  • located in many cells including- glia,neurons, endothelial cells of the capillaries and lymphatics, stem cells, fibroblasts, WBC’s and many organs
  • SP is a first responder to noxious pain stimuli
  • regarded as a defender of stress- repair and survival
333
Q

substance P functions

A
  • potent vasodilator and broncho constriction through the vagus system
  • responsible for a portion of local inflammatory response to infection and injury
  • pain perception
  • other include mood- emotions, cell growth = vomiting etc.
334
Q

proprioceptive insult hypothesis-

A
  • a sub component of the somatosomatic relfex model
  • undue irritation to the mechanreceptors of the articular structures, possibly from anomaly or injury, bombard the internuncial- (connections between neurons) pool that result in spasms
  • think assymetry
335
Q

fixation theory- segmental facilitation or impulse theory)

A
  • self perpetuating viscious cycle
  • lessened mobility lead to
  • aberrant relfexes lead to
  • segmental fascilitation leads to
  • soft tissue aberration- spasm, edema
336
Q

axonal transport vs. nerve compression

A
  • nerve compression is more associated with tone
  • results in altered nerve transmission and energy by a structural cause that leads to body pathology
  • impingement includes pressure, traction, torsion, stretch, angulation
337
Q

nerve compression theory

A
  • tend to be more global or systemic
  • increased transmission- increased tone
    -decreased transmission - decreased tone
338
Q

axonal transport - aberrations:

A
  • generally one nerve or plexus local with decreased protein supply in micro tubular network
  • loss of tropic function, caused by nerve compression or injury
  • results in Wallerian degeneration and local tissue death - Hiltons law
339
Q

reviewing wallerian degeneration

A
  • assoc. with the axonal aberration theory
  • an active process of degeneration of a nerve that is cut or crushed
  • a related retrograde degeneration- called wallerian-like degeneration is associated with diseases like ALS
340
Q

wallerian degeneration

A

severed or crushed axons actively self destruct through a gradual cascade in 3 stages

341
Q

regarding wallerian degeneration:
- what is step 1

A
  • axon degeneration-
  • immediately following injury within 30 minutes, there is a separation of the proximal and distal ends of the nerve. after a short latency period, the transacted membranes are sealed, which initiates the formation of axonal sprouts. this occurs in less than a day and allows for nerve regeneration in certain instances.
342
Q

regarding wallerian degeneration:
what is step 2

A

myelin clearance-
- this occurs around the 7th day when macrophages are signaled by schwann cells to do their clean up work. rate of clearance is slower in the CNS than the PNS due to the clearance of the myelin.
- in addition- it is slower in the CNS because of oligodendrocytes being in the way. also the blood tissue barrier will slow the clearing of the CNS
- slower in the CNS vs. PNS

343
Q

regarding wallerian degeneration:
- what is step 3?

A

regeneration -
-if the soma/cell body is damaged, a neuron can NOT regenerate
- if the injury is towards the end of the axon, it can regenerate at a rate of up to 1 mm/day

344
Q

what is microtubular transmission associated with?

A

axoplasmic transport- if you see microtubular the answer is going to be AXT- one nerve or plexus and involves trophic process

345
Q

what is decreased immunity and compromised neuromusclular tone assoc. with?

A

neurodystrophic- think stress and immuntiy

346
Q

what is irritated joint receptors assoc with?

A

proprioceptive insult- when you hear mechanoreceptors- think PI- due to the body reacting to injury- overuse altered biomechanics due to assymetry

347
Q

what is nerve transmission altered by inhibited retrograde flow associated with?

A

axoplasmic transport- RETROGRADE is the key word

348
Q

what is wallerian degeneration assoc with?

A

AXTransport- effecting one nerve or plexus

349
Q

what is hypomobility of segment impinges nerve root?

A
  • nerve compression
350
Q

what is tone of tissue associated with

A

nerve compression - tone is generally associated with nerve compression

351
Q

what is perceived chronic stress associated with?

A

neurodystrophic- think selye

352
Q

what is protein supply associated with?

A

AXT- trophic substances always related to AXT

353
Q

what is antibody production associated with?

A

neurodystrophic effecting the immune system

354
Q

what is weekend warrior associated with

A
  • proprioceptive insult injury and altered biomechanics
355
Q

what is fixations associated with

A

neurodystrophic-

356
Q

what is mechanoreceptors associated with?

A

proprioceptive insult- if you see mechanoreceptors, it is the body responding to mechanical stress and assymetry

357
Q

what is microtubular assoc with

A

AXT

358
Q

what is altered transmission assoc with

A

Nerve compression

359
Q

what is Selye assoc with?

A

neurodystrophic

360
Q

wht is IVF encroachment assoc with

A

nerve compression

361
Q

hat is influenza assoc with

A

neurodystrophic - immune system

362
Q

what is systemic response assoc with

A

nerve compression

363
Q

what is trophic assoc with

A

AXT

364
Q

what percentage of the flexion extension and rotation of the cervical spine is at o-c1-c2?

A

50%

365
Q

who originated SOT tech

A

dejarnette

366
Q

what is the name coined to describe the close proximity of the cervical nerve roots to the IvF causing compression

A

cervical syndrome

367
Q

what keep assists in keeping the muscle spindle taut

A

Gamma MN

368
Q

alpha motor neurons are considered part of the CNS and PNS

A

true

369
Q

____ motor neurons are the most numerous

A

alpha

370
Q

what tract integrates information from the motor system to coordinate autonomic movements of locomotion and posture

A

reticulospinal

371
Q

the tectospinal tract____

A

mediates reflex postural movements of the head

372
Q

the vestibulospinal systme is able to respond correctly by recording sensory information from ___

A

hair cells in the labyrinth of the inner ear

373
Q

the corticospinal tract controls

A

primary motor activity from the neck to the feet

374
Q

hat neurotransmitter transmits nerve impulses to UMN and LMN

A

glutamate

375
Q

a reversible pathophsiologic state consisting of unnotced, misdirected neurophysiological reactions is?

A

dysponesis