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Aneesha CSI flashcards > Health check > Flashcards

Health check Flashcards

1
Q

The NHS Health Check can tell you whether you’re at higher risk of getting certain health problems such as what?

A

Heart Disease

Diabetes

Kidney Disease

Stroke

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2
Q

During the check-up you also discuss how to reduce the risk of these conditions and what other condition?

A

Dementia

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3
Q

If you are over what age will you be told the signs and symptoms of dementia to look out for?

A

65

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4
Q

Is everyone at risk of developing heart disease, stroke, type 2 diabetes, kidney disease and some types of dementia?

A

Yes

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5
Q

Is Cardiovascular risk the same for everyone?

A

No it varies from person to person

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6
Q

How long does the NHS Health Check take?

A

20-30 mins

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7
Q

What questions are asked by a health professional - often a nurse or healthcare assistant in an NHS Health Check?

A

Questions about your lifestyle

Questions about family history

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8
Q

What examinations are done by a health professional - often a nurse or healthcare assistant in an NHS Health Check?

A

Measurement of height and weight - calculate BMI

Take blood pressure

Do blood test - done either before or at the check

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9
Q

What can the personalised advice to improve your risk given in an NHS Health Check include?

A

How to improve your diet and the amount of physical activity you do

Taking medicines to lower your blood pressure or cholesterol

How to lose weight or stop smoking

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10
Q

Where do you have an NHS Health Check?

A

Usually at GP surgery or local pharmacy

Could happen at local library or leisure centre

Some areas they are offered from mobile units to passers-by and in workplace

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11
Q

Why might NHS Health Checks be paused in some areas?

A

At the moment may be paused due to COVID-19

Local council should be contacted to find out if programme is available in area

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12
Q

If you are between what age are you invited to have a free NHS Health Check every 5 years?

A

40-74 and do not already have a pre-existing condition

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13
Q

What should a patient do if they are eligible for a check but have not had one in the last 5 years or if they are not sure if they are eligible?

A

Ask at a GP surgery for an appointment

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14
Q

Do NHS Health Checks work?

A

Health conditions picked up by NHS Health Check, when added together are the biggest cause of preventable deaths in the UK with about 7 million people affected by them

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15
Q

In its first 5 years, how many heart attacks or strokes has the NHS Health Check been estimated to have prevented?

A

2,500 As a result of people receiving treatment after their Health Check

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16
Q

For every how many people having an NHS Health check is one person diagnosed with High Blood Pressure?

A

30-40

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17
Q

For every how many people having a Health check is 1 person diagnosed with Type 2 Diabetes Mellitus?

A

80-200

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18
Q

For every 6 in how many people having an NHS Health Check is 1 person identified as being at high risk for CVD?

A

10

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19
Q

Who runs the NHS Health Check Programme in a patient’s area?

A

Their local authority

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20
Q

What test on the NHS website assesses your risk of getting heart disease or stroke?

A

online Heart Age test

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21
Q

How do you find out if you are a healthy weight on the NHS website?

A

Health Weight Calculator

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22
Q

What does the How Are You quiz on the NHS website show?

A

How healthy you are

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23
Q

Equation to calculate cardiac output

A

Cardiac output= stroke volume*heart rate

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24
Q

Why does arterial pressure lead to vascular function and structural changes?

A

arterial pressure–>peripheral vascular resistance–>vascular function and structural changes

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25
Q

Why is it important to recognise hypertension?

A

Important to recognise hypertension as it is usually silent

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26
Q

What are 10 effects of hypertension?

A

Cardiac- coronary artery disease, left ventricular hypertrophy

Systemic vascular- TIA, stroke

Microvascular- Atherosclerosis (could increase risk of aneurysm), aneurysm

Renal- glomerulosclerosis (microvascular effects in kidney, will lead to kidney failure if left untreated), kidney failure

Visual- retinopathy, optic neuropathy (optic nerve damage)

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27
Q

Why is first BP measurement usually higher than normal and how do you avoid this?

A

Can be due to stress- ‘white coat’ hypertension when they see a healthcare professional

Best out of 3 measurements taken

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28
Q

If BP still high even after best of 3, what is done?

A

24 hour ambulatory blood pressure monitoring in the comfort of the patients home, can help avoid the white coat effect
Usually goes off 2x in a day and once in night which can be scary and so if this happens you can use BP monitor yourself at home 2x in day and once in night

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29
Q

If you still have high BP after 24 hour ambulatory blood pressure monitoring, what is done next?

A

Lifestyle advice
exercise (circulation to skin and muscles for vasodilation and increased Renal function)
Diet
stop smoking

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30
Q

Give an example of lifestyle advice to do with diet

A

Usually hypertensive individual has high salt and vulnerable to renin so salt intake should be controlled

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31
Q

If BP still high after lifestyle changes, what should be done?

A

Give medication

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32
Q

What 4 things should be offered to all patients with hypertension?

A

Test for presence of protein in urine by sending a urine sample for estimation of albumin: creatinine ratio and test for haematuria using a reagent strip

Take a blood sample to measure glycated haemoglobin (HbA1c), electrolytes, creatinine, estimated glomerular filtration rate, total cholesterol and HDL cholesterol

Examine the fundi for the presence of hypertensive retinopathy

Arrange for a 12 lead ECG to be performed

((all test for end organ damage))

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33
Q

Describe the order of the 5 electrical events in the cardiac cycle

A

SA node signal
Atrial contraction
AV node signal
Ventricular depolarisation
Ventricular repolarisation

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34
Q

What is the name of the cell types in the SA node?

A

Autorhythmic myocytes

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35
Q

What is the name of the wave created by the SA node?

A

P-wave

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36
Q

How is the AVN signal represented on ECG and is this a slow or fast signal?

A

Isoelectric line

Slow transduction to allow ventricular filling before contraction

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37
Q

What wave does the depolarisation of the Bundle of His show on ECG and is this slow or fast signal?

A

Isoelectric line

Fast as it is insulated

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38
Q

What occurs in the Q wave?

A

Shows septal depolarisation via bundle branches

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39
Q

What does the R wave show?

A

Ventricular depolarisation by Purkinje fibres

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40
Q

What does the S wave show?

A

Late ventricular depolarisation and this is in opposite direction to lead II

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41
Q

What does the T wave show and why is this positive deflection on ECG?

A

Ventricular repolarisation which occurs in opposite direction to lead II but since it it is repolarisation in the opposite direction, it has positive deflection (depolarisation in opposite direction would have had negative depolarisation)

The part that depolarises first is what repolarises last

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42
Q

Summary diagram for ECG tracing

A
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43
Q

Where do each of the chest electrodes go?

A

V1- right sternal border in the 4th intercostal space
V2- left sternal border in the 4th intercostal space
V3- Halfway between V2 and V4
V4- Mid-clavicular line in the 5th intercostal space
V5- Anterior axillary line at the level of V4
V6- Mid axillary line at the level of V4

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44
Q

Which direction does electrical conduction travel from and to?

A

From negative electrode to positive electrode as depolarisation leads to a lower intracellular charge compared to extracellular charge

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45
Q

What direction do each of leads I-III go from in relation to the limbs?

A
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46
Q

What is the correct way to read an ECG?

A
  1. Check right patient, DOB, date and time
  2. Look at calibration and running speed to see how big each square is
  3. Rate - R-R interval (300/number of big square)
  4. Rhythm - equidistant R-R interval
  5. Axis - normal is -30 to 90 and see whether it is positive or negative deflection
  6. P-Wave - are they present in V1, are they normal morphology
  7. P-R interval - Within 3-5 small squares and if longer it is a type of heart block, is it regular without the lead
  8. QRS complex - height, amplitude and the width otherwise there is abnormality in ventricular depolarisation
  9. ST-Segment - Flat on isoelectric wave
  10. T-Wave - Morphology
  11. Q-T interval- abnormal or not
  12. Go through each lead systematically and see how to interpret it
47
Q

What could large QRS complexes on the V leads suggest in the context of LV hypertrophy?

A

Large QRS complexes on the V leads - difference in ventricular depolarisation

Hypertrophied LV means more mass in LV so more muscle that needs to contract and so the QRS amplitude increases

48
Q

What could ST segment depression suggest?

A

Abnormally electrical impulse goes through the muscle

49
Q

What could t wave inversion mean?

A

Myocardial Ischaemia

50
Q

What is diastolic dysfunction?

A

Stiffening of the heart’s ventricles

51
Q

Explain the pathophysiology of diastolic dysfunction?

A

Increased peripheral vascular resistance so increased afterload

Adapts to this by pressure and volume related remodelling of the LV

Hypertrophy occurs and thicker muscle builds

LV remodels so much that it causes diastolic dysfunction

52
Q

What is systolic dysfunction

A

Increased circulating volume leads to remodelled LV and so this leads to eccentric hypertrophy to allow the large volume which leads to systolic dysfunction

53
Q

How does calcium channel blocker lead to reduction in blood pressure?

A

Inhibit L-type calcium channel, decreasing calcium influx in vascular smooth muscle leading to downstream inhibition of myosin-light chain and this prevents cross-bridge formation and smooth muscle contraction leading to dilation which reduces blood pressure

54
Q

What effect does decreasing peripheral resistance have on vascular pressure?

A

Decreased peripheral resistance → decreases vascular pressure

55
Q

What dose do you start with of CCBs?

A

Start with the lowest dose first and then you can move up

56
Q

What is the difference between dihydropiridenes and non-dihydropiridenes?

A

Non-dihydropiridenes - more negative inotropic effects

Dihydropiridenes - more potent vasodilators

57
Q

Choice of anti-hypertensive drug

A
58
Q

Why are hypertensive patients less receptive to ACEi or ARB?

A

Less responsive to Renin and so are less receptive to ACEi or ARB as they have high salt

May be of black African or African-Caribbean family origin

59
Q

Why is the blood pressure still high despite drug treatment?

A
  • Non-Adherence (Intentional - they do not want to take the medication and are not engaging with the prescriptionUnintentional - may forget and not feel they are getting any benefit from it)
  • Side effects
    Ankle oedema - swelling of the ankles (most common)
    Change in bowel habits - Calcium channels present in gut mucosa therefore affected gut motility and changes in these
    Palpitations - decreasing blood pressure which is detected by baroreceptors and so the heart is pumped harder to increase pressure which the patient feels the heartbeat for
    Headaches and flushings - Dilated blood vessels so more blood to head and this gives flushing and headaches
  • Multiple drugs required (70% patients fail to control hypertension on one medication
    CCB with ACEi, ARB or Thiazide-like diuretic)
  • Ethnicity (Can increase risk of resistance to anti-hypertensives)
  • White coat syndrome (Could still be stressed when coming in to see the doctor’s about the results of the blood pressure tests)
60
Q

What is the function of pacemaker cells?

A

Set rate of heartbeat by sending out action potentials across cells

61
Q

What percentage of cells are pacemaker cells?

A

1%

62
Q

What is meant by the auto-rhythmicity of the pacemaker cells?

A

They are continually able to generate action potentials to send out to the rest of the heart

Every pacemaker cells has the ability to generate a new AP given a certain time

63
Q

Where do skeletal muscle cells get their signal to send out APs from?

A

Neurons

64
Q

What cells receive the AP from pacemaker cells?

A

Myocytes making up the myocardium

65
Q

What is meant by polarisation?

A

More positive ions outside the cell than inside (negative membrane potential inside the cell compared to the outside)

66
Q

What is meant by depolarisation?

A

Membrane potential is smaller and so it is more positive than in polarisation as the amount of positive ions on the outside is less than inside during depolarisation when compared with polarised state

67
Q

What is a depolarisation wave?

A

When one cell after another is depolarised

68
Q

What is the SA node?

A

Group of pacemaker cells in the upper right quadrant of the right atrium

69
Q

What is the function of the SA node?

A

Generates signal for the atria to contract

70
Q

Does the depolarisation wave move faster in myocytes or in pacemaker cells?

A

Faster in pacemaker cells

71
Q

How does the depolarisation wave in the right atria reach the left atria?

A

Atrial internodal tracts - Bachmann’s bundle

72
Q

Other than the atrial myocytes, what other structure does the SA node send signals to?

A

AV node

73
Q

What causes the conduction to slow at the AV node?

A

Smaller diameter so higher resistance
Slower Ca2+ channels used rather than faster Na+ channels

74
Q

Why is slowing of conduction at AV node useful?

A

Allows time for ventricular filling

75
Q

Describe the path of the conduction after AV node?

A

AV node → Bundle of His → Bundle branches in interventricular septum → Purkinje Fibres

76
Q

What is the significance of the His-Purkinje system being fast?

A

Heart contracts in a coordinated way to give a forceful push

77
Q

If the SA node pacemaker cells fail to send out a signal to contract what happens?

A

Other atrial pacemaker cells send signal at 60-80 per minute
(Ectopic pacemakers - Not in SA node)
If these fail, AV node pacemaker cells send signal at 40-60 per minute

If these fail, ventricular pacemaker cells send signals at 20-40 per minute

78
Q

What is meant by cardiac conduction velocity?

A

Velocity of depolarisation through myocardium (m/s)

79
Q

Describe the action potential at a molecular level?

A

Calcium and sodium slip through gap junctions and trigger voltage gated sodium channels in adjacent cells to open leading to AP which causes next cell to allow sodium through its gap junction triggering another AP

80
Q

Describe the conduction velocity at the atria, AV node, Bundle of His and Purkinje fibres and the Ventricles

A
81
Q

In the t wave is there negative or positive deflection?

A

T wave shows repolarisation, however there is positive deflection as the last cells to depolarise (ventricular) are the first to repolarise and so the repolarisation wave which goes from negative to positive will travel in the opposite direction to the lead which then will show itself as positive deflection

82
Q

What are the limb electrodes?

A

The limb electrodes are placed on the right arm, left arm, left leg and right leg

The right leg is used as a neutral and so the left leg is used to show the signal directions on ECG

Together they make leads I, II, III, aVF, aVL, aVR

83
Q

What are the precordial electrodes?

A

They are the chest electrodes of V1-V6 placed across the chest

84
Q

In which direction do each of the chest leads show deflection in?

A
85
Q

Which are the inferior leads?

A

Leads II, III, AVF are inferior leads

86
Q

What are the inferior leads supplied by?

A

Right coronary artery

87
Q

Which are the lateral leads?

A

Leads I, aVL, V5, V6

88
Q

What are the lateral leads supplied by?

A

Circumflex artery

89
Q

Which are the septal leads?

A

V1, V2

90
Q

What are the septal leads supplied by

A

Left anterior descending artery

91
Q

Which are the anterior leads?

A

V3, V4

92
Q

What are the anterior leads supplied by?

A

Right coronary artery

93
Q

How many seconds and mV is one small box on an ECG?

A

0.04s in width

0.1mV in height

94
Q

What does the P wave signify?

A

Atrial depolarisation via SA node

95
Q

What is the isoelectric line after the P wave a show of?

A

AV node depolarisation and this shows the slow signal transduction which is protective of the ventricular filling

96
Q

How is the PR interval measured and what does it show?

A

From beginning of P wave and end of isoelectric line

Time between start of atrial contraction and ventricular contraction

97
Q

What is a normal PR interval?

A

0.12-0.20s

3-5 small boxes

98
Q

What does the Q wave signify?

A

Signal passing through the bundle branches in which there is septal depolarisation

This is shown as negative deflection as the signal goes through all the myocytes in the thick intraventricular septum which shows it to be in opposite direction of lead II

99
Q

What causes the R wave?

A

Signal conduction through large Purkinje fibre going through left ventricle

100
Q

What causes the S wave?

A

Negative deflection caused by late ventricular depolarisation in the right ventricle via Purkinje fibres

101
Q

What is the ST segment?

A

No change in electrical activity after hitting the J point (isoelectric line)

102
Q

What causes the T wave?

A

Wave of negative charge due to repolarisation in the opposite direction to lead II shows positive deflection as opposed to S wave which was positive charge in opposite direction and so showed as a negative deflection

103
Q

Why is the T wave more spread out?

A

Repolarisation is a slower process that takes place at slightly different times for each cardiomyocyte instead

104
Q

Why does atrial repolarisation not show itself on ECG?

A

The small vectors this creates is lost by the larger vectors created by ventricular depolarisation

105
Q

If there is an irritable atrial cell in left atrium which becomes an atrial extopic focus, is the PR interval going to be longer or shorter?

A

Longer as the atrial ectopic focus is farther away from the AV node

106
Q

In first degree heart block is the PR interval longer or shorter than normal?

A

Longer as the conduction through AV node is slower

107
Q

How does a ventricular ectopic focus alter the QRS complex?

A

Becomes wider as it takes longer to depolarise all the myocytes

108
Q

How long is a normal QRS Complex?

A

0.10s

109
Q

How do you define an intermediate and prolonged QRS Complex?

A

Intermediate - 0.10-0.12s

Prolonged - >0.12s

110
Q

What does the QT interval show?

A

Ventricular systole from depolarisation through to repolarisation

111
Q

How long is an abnormally long QT interval in men and women respectively?

A

Men - 440ms

Women - 460ms

At 60bpm

112
Q

If a male has a QT interval of 400 at 90bpm is this normal?

A

Not necessarily as QT interval is supposed to change with rate

As Rate increases, QT interval should reduce

113
Q

How do you calculate the adjusted QT interval for a ceratin rate?

A

:)

Aneesha CSI flashcards (4 decks)

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