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Nursing 111 & Pharmacology > NUS 111 Test #2 - Oxygenation and Perfusion > Flashcards

NUS 111 Test #2 - Oxygenation and Perfusion Flashcards

1
Q

this is mainly lung disease cause by bacterium called Mycobacteria

A

tuberculosis

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2
Q

this respiratory disease can spread to lymphatic and circulatory system which can take it to the brain and bones

A

TB

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3
Q

how is TB spread

A

airborne transmission

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4
Q

What precautions do we put in place for active TB patients?

A

Negative air pressure room
N95 respirator
Single room with closed door
Gown

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5
Q

how long does it take TB to show after exposure

A

2-10 weeks

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6
Q

once inhaled how does TB travel

A

down to alveoli and body ingests bacteria

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7
Q

a potentially fatal form of disseminated disease due to the hematogenous spread of tubercle bacilli to the lungs, and other organs

A

miliary TB

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8
Q

dry mask and they calcify and make disease dormant

A

ghon tubercle

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9
Q

you can get tb again from these particles

A

ghon tubercle

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10
Q

since it’s airborne transmission - how exactly is TB spread. what does a person do?

A

coughing, sneezing, laughing, singing, talking

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11
Q

waht are the risk factors for TB

A

foreign-born
suppressed immune system
homelessness, poverty level
minorities
advanced age
poor access to healthcare
multi drug resistant strains
subst abuse
infants and children exposed to high risk pts
traveling abroad

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12
Q

Comorbities

A

Malnutrition
Diabetes
Silicosis
Chronic Kidney disease
Gastric or Intestinal bypass Surgery

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13
Q

what puts healthcare workers at risk for TB

*** know this

A

Administration of aerosolized medications
Sputum-induction procedures, including suctioning and coughing procedures
Bronchoscopy
Intubations

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14
Q

what immunocomprmised individuals are at high risk for TB

A

Human immunodeficiency virus (HIV) infections
Malignancies: Head, neck, lung, hematologic
Long-term corticosteroid use
Immunosuppressive drug therapies
Organ transplantation

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15
Q

what are the comorbidities of TB

A

Malnutrition
Diabetes
Silicosis
Chronic kidney disease
Gastric or intestinal bypass surgery

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16
Q

gerontologic considerations for TB

A

have atypical manifestations in elderly
alter mental status
create unusual behavior - anorexia, weight loss
fever

the tuberculin skin test produces no reaction (loss of immunologic memory) or delayed reactivity for up to 1 week

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17
Q

clinical manifestations of TB

A

signs and symptoms of pulmonary TB are insidious
-low-grade fever, cough, night sweats, fatigue, weight loss.
- cough is nonproductive but progresses to be mucopurulent.

-Dyspnea, chest pain, and hemoptysis (bloody sputum) occur as the disease progresses.

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18
Q

how is TB diagnosed

A

complete history, physical examination, tuberculin skin test, chest x-ray, AFB smear, and sputum culture are used to diagnose TB

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19
Q

RN assessments for TB

A
  1. assess for asymptomatic vs symptomatic disease
  2. perform physical assessment: auscultating lungs, night sweats, sputum, living conditions, risk factors, assess for presentation : is it active or not
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20
Q

Most ppl exposed to TB are….

A

asymptomatic and exposing ppl bc they are unaware

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21
Q

how many ways is TB classified

A

6 classes

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22
Q

class 0 of TB

A

Class 0: No exposure; no infection

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23
Q

class 1 of TB

A

Class 1: Exposure; no evidence of infection

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24
Q

class 2 of TB

A

Class 2: Latent infection; no disease (e.g., positive PPD reaction but no clinical evidence of active TB)

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25
Q

class 3 of TB

A

Class 3: Disease; clinically active

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26
Q

class 4 of TB

A

Class 4: Disease; not clinically active

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27
Q

class 5 of TB

A

Class 5: Suspected disease; diagnosis pending

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28
Q

nursing interventions for TB re patient teaching (5 ways)

A

patient teaching:
1. how not to spread disease
2. set up airborne precautions
3. ensure adequate nutrition intake
4. have exposed family members get tested
5. monitor vitals, assess effective cough, may need suctioning

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29
Q

what are the meds will active TB be treated with –at least 4 medications

A

INH, rifampin, pyrazinamide, and ethambutol

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30
Q

Primary drug resistance re TB

A

Resistance to one of the first-line antituberculosis agents in people who have not had previous treatment

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31
Q

Secondary or acquired drug resistance re TB

A

Resistance to one or more antituberculosis agents in patients undergoing therapy

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32
Q

MDR re TB

A

Resistance to two agents, isoniazid (INH) and rifampin. The populations at greatest risk for MDR are those who are HIV positive, institutionalized, or homeless.

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33
Q

how many weeks is the TB initial phase treatment last

A

8 weeks

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34
Q

continuation therapy is what weeks - TB

A

18 or 31 weeks

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35
Q

what medications are used during the continuation phase of TB

A

INH and rifampin or INH and rifapentine,

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36
Q

how do you determine if initial phase treatment for TB was effective

A

assess sputum smears

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37
Q

what is prescribed with INH to prevent peripheral neuropathy

A

vitamin B6 (pyridoxine)

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38
Q

how many drugs can be used for multi drug resistent TB

A

up to 6 drugs

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39
Q

how long will the treatment be for multi drug resistent TB

A

6-12 weeks

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40
Q

who will receive prophylaxis treatment for TB

A

-Household family members of patients with active disease
-Patients with HIV infection who have a PPD test reaction with 5 mm+
-Patients with fibrotic lesions suggestive of old TB detected on a chest x-ray and a PPD reaction with 5 mm+
-Patients whose current PPD test results show a change from former test results, suggesting recent exposure to TB and possible infection (skin test converters)
-Users of IV/injection drugs who have PPD test results with 10 mm+
-Patients with high-risk comorbid conditions and a PPD result with 10 mm +

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41
Q

what foods should pt avoid if taking INH

A

foods that contain tyramine and histamine (tuna, aged cheese, red wine, soy sauce, yeast extracts)

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42
Q

what does INH prophylaxis treatment consist of

A

daily doses for 6 to 12 months

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43
Q

what lab work needs to be checked if pt is on INH prophylaxis

A

Liver enzymes, blood urea nitrogen (BUN), and creatinine levels are monitored monthly to detect changes in liver and kidney function

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44
Q

what is the TB skin test called

A

Mantoux test - screening test

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45
Q

how is the mantoux test red

A

-pt exposure not currently active TB
-pt needs to go back after 2 days

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46
Q

priority labs/diagnostics for TB

A
  1. smears of sputum
  2. chest xray
  3. mantoux test
  4. bacteriologic studies
  5. screening tools - interferon - gamma release assays (IGRAs)
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47
Q

waht are some collaborative goals for pts with TB

A
  1. stopping spread
  2. meals on empty stomach
  3. return normal pulmonary function
  4. complete resolution of disease
  5. absence of complications to food access
  6. conditions can reactive disease immune suppression malignancy, quit smoking
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48
Q

what does croup cause

A

swelling of larynx, trachea and large bronchi due to infiltration of WBC

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49
Q

what does the inflammation of croup lead to

A

mucus and noisy breathing

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50
Q

waht are risk factors for croup - 2

A

recent upper respiratory tract infection
younger than 6 years old

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51
Q

acute LTB (laryngotracheobronchitis)
signs/symptoms - 5

A
  1. caused by viral illness
  2. symptoms come on at night, usually middle of night
  3. hoarse voice
  4. is reaction to cold or something similar
  5. barking seal like cough
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52
Q

acute spasmodic croup
signs and symptoms - 6

A
  1. caused by allergens
  2. hereditary
  3. reflux/allergy
  4. like an allergic reaction only higher in respiratory system
  5. reaction to something within body
  6. responds well to allergy/reflux meds
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53
Q

clinical manifestations for croup

A
  1. upper airway obstruction due to swelling of larynx, trachea and bronchi
  2. stridor - inspiratory breathing
  3. seal like cough
  4. chest wall retractions
  5. any changes in mental status
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54
Q

know croup sounds——

https://www.youtube.com/watch?v=C1q6ATkMtm0

A
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55
Q

what are the RN assessments for croup - 8

A
  1. assess for inspiratory stridor, barking cough, hoarseness, tachycardia and tachypnea
  2. using assessory muscles
  3. fever
  4. irritable
  5. recent URTI
  6. auscultate lung sounds
  7. hydration
  8. diminished breath sounds at bottom of lungs or the bottom of lungs sound normal
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56
Q

waht are the nursing interventions for croup - 8

A
  1. facilitate airway clearance
  2. go outside in cold air to stop cough
  3. run hot shower and sit in room
  4. run humidifier
  5. maintain fluid balance
  6. decrease fear to stop fight or flight mode in child
  7. anti inflammatory meds
  8. give educ to parents about croup so they know what to look for
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57
Q

what are the labs/diagnostics for croup

A

observe clinical symptoms

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58
Q

meds for croup

A

dexamethasone: 0.15mg-0.6gm/kg orally

racemic epinephrine through neubulizer

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59
Q

safety measures for croup

A
  1. dont leave child alone
  2. maintain pt airway
  3. flu vaccine - type B for epiglottis
  4. Need to stay in hospital 4 hours after being administered racemic epinephrine
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60
Q

influenza virus affects respiratory tract how - 2 ways

A
  1. direct viral infections
  2. by damage from immune system response
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61
Q

this virus transmission occurs through a susceptible individuals contact with aerosols or inanimate objects can carry and spread disease and infectious agents from infected individual

A

influenza

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62
Q

this creates inability of lung to perform its primary function of gas exchange which can result from multiple mechanisms, including obstruction of airways, loss of alveolar structure

A

influenza

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63
Q

risk factors for influenza - 6

A
  1. unvaccinated individuals
  2. compromised immune systems
  3. young/elderly
  4. chronic medical conditions
  5. pregnant women
  6. residetns of long term care facility
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64
Q

what needs to b e considered when old person gets flu

A
  1. immune system declines as adults age
  2. prevent secondary infection
  3. flu increases risk of heart attack 3-5x and stroke 2-3x in first two weeks of infection in 65+
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65
Q

clinical manifestations for influenza

A
  1. fever
  2. chills
  3. hypoxia
  4. severe headaches
  5. D & V
  6. cough
  7. muscle aches
  8. fatigue
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66
Q

RN assessments for the flu

A

vital signs, normal assessment of chest auscultation, look for signs of dehydration

subjective/objective data of pt: around anyone that’s sick, feeling sob, dyspnea on exertion, lung sounds, RR, BP, pulse ox, accessory msucle use, etc

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67
Q

labs/diagnostics for flu

A

health history - clinical findings
flu A & B test
RSV test
COVID test

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68
Q

how long do flu cultures take to come back

how long do rapids take

A

3-10 days

15-60 mins

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69
Q

nursing interventions for flu

A
  1. droplet precautions
  2. lots of fluids
  3. relief of symptoms
  4. keep track of I & O if severe
  5. antipyretics - Tyle & Motr
  6. analgesics - tyle & motr
  7. rest
  8. gargle warm salt water
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70
Q

safety considerations for pts with flu

A

droplet precautions
infants, elderly, hc workers
guillain-barre
anaphylaxis hypersensitivity to eggs

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71
Q

most effective strategy for managing influenza is preventative admin which is….

A

influenza vaccine yearly

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72
Q

” the ability of blood to transport oxygen-containing hemoglobin to cells and return carbon dioxide-containing hemoglobin to the alveoli”

A

perfusion

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73
Q

priority meds for the flu - 3

need to look up more details in ATI 17, 20, 23

A
  1. zanamivir (inhaler)
  2. oseltamivir (tablet)
  3. peramivir (IV)
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74
Q

infection of the lower respiratory tract caused by a variety of microorganisms, including bacteria, viruses, fungi, protozoa, and parasites

A

pneumonia

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75
Q

what are classifications of pneumonia - 5

A

community-acquired pneumonia (CAP),
hospital-acquired (nosocomial) pneumonia (HAP),
ventilator-associated pneumonia (VAP),
health care–associated pneumonia (HCAP), and
pneumonia in an immunocompromised patient.

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76
Q

waht are the vectors pneumonia can arise from - 5

A
  1. from normal flora present in patients whose resistance has been altered;
  2. aspiration of flora present in the nasopharynx or oropharynx;
  3. the inhalation of airborne microorganisms from other persons (sneezing, coughing, or talking);
  4. contaminated water sources or respiratory equipment;
  5. blood-borne organisms that enter the pulmonary circulation (hematogenous spread) and become trapped in the pulmonary capillary beds
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77
Q

most common microbe in CAP

***know this

A

streptococcus pneumoniae

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78
Q

risk factors for pneumonia – all types

A

smoking
age
malnutrition
post surgery
large family/lots of kids
pulmonary edema (fluid in lungs)
altered level of consciousness
toxic inhalation
chronic condition/pre existing hypoxemia

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79
Q

risk factors for HAP/VAP

A

Debilitation
Malnutrition
Altered mental status
Previous exposure to antibiotics (within the last 90 days)
Hospital stays of 5 days or longer
High rates of antibiotic resistance (hospital or unit-specific)
Immunosuppressive therapies or diseases
Prolonged (greater than 48 hours) intubation or a tracheostomy
Male gender

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80
Q

risk factors for HCAP

A

Hospitalization for 2 or more days in the last 3 months
Chronic dialysis within the last month
Home wound care within 30 days
Recent home IV therapy (antibiotic, chemotherapy)
Resident of a skilled nursing or extended-care facility
Family member with drug-resistant microorganism

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81
Q

risk factors for immunocompomised pneumonia

A

chemo treatmetns
autoimmune disorders
corticosteroids
malnourished
broad spectrum antibiotics

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82
Q

old ppl considerations for pneumonia

get more detials from honan 303

A

can get missed due to not showing all signs of pneumonia

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83
Q

clinical manifestation for pneumonia

A

SOB
hypoxic
crackles and wheezes in lungs
fever
chest pain
productive cough
leukocytosis elevated (5000-10000 normal range)
fatigue
anorexia
tachypnea
SpO2 low
use secondary assessory muscles

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84
Q

RN assessments for pneumonia

A

-auscultate breath sounds
-monitor for complications
1. rate, depth, efforts of breath
2. sign of septic shock - low BP, tachycardia
3. use of accessory muscles
4. super infection
5. respiratory failure
6. thoracentesis
7. confusion
8. empyema
9. atelectasis

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85
Q

waht is a super infection and give example

A

overgrowth of good bacteria which then becomes bad

ex. c diff

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86
Q

what can septic shock lead to

A

multisystem organ failure

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87
Q

what is the leading cause of death for severe pneomina

A

respiratory failure

88
Q

fever, dehydration, hypoxemia, sleep deprivation, or developing sepsis … can lead to this

A

confusion

89
Q

collapsed alveoli.

it can improve with coughing and deep breathing

A

atelectasis

90
Q

nursing interventions for pneumonia

A

-incentive spirometer
-vaccines
-who’s at risk for pneumonia - ID and prevent them from getting it
-physical assessment
-oxygen therapy, hydration, antibiotics, neubulizer
-teaching how to cough and deep breathing techniques
-re-evaluate lung sounds, pulse ox, RR
-ambulating and staying active
-positioning
chest percussions to help loosen sputum

91
Q

pt teaching for pneumonia

A

-decrease the spread - stay home
-when to follow up with doctor
-take every dose of antibiotics
-know drug to drug interactions
-repeat chest xrays
-get vaccines
-cough can last for several weeks

92
Q

labs/diagnostics for pneumonia

A

chest x ray
CBC - check WBC
arterial blood gas

93
Q

meds that treat pneumonia - early onset (less than 5 days) with no risk of multi drug resistant pathogens

A

Ampicillin-sulbactam
OR
Ceftriaxone
OR
Levofloxacin, moxifloxacin, or ciprofloxacin
OR
ertapenem

94
Q

meds that treat pneumonia - late onset (5+ days) with risk of multi drug resistant pathogens

A

Cefepime
OR
Imipenem or meropenem
OR
Piperacillin-tazobactam AND Ciprofloxacin or levofloxacin
OR
amikacin, gentamicin or tobramycin AND vancomycin (MRSA)

95
Q

3 safety measures for pneumonia

A
  1. decreased perfusion/oxygenation
  2. any changes in cognition
  3. safety considerations based on meds they are given
96
Q

this is the balance between clot formation and clot dissolution

A

hemostasis

97
Q

balancing throughout the whole body

A

homeostasis

98
Q

why does the body need clotting mechanisms

A

to prevent bleeding

99
Q

what is fibrinolysis

A

clot dissolution

100
Q

what is thrombus

A

clot formation

101
Q

mini clots in atria which can become emboli and travel to the brain (stroke). top chambers of heart not opening properly

A

atrial fibrillation

102
Q

caused by a thrombus that impairs blood flow. blood clots off artery causing heart not to work bc blood isnt getting to body properly

A

myocardial infarction

103
Q

increases heart workload and contributes to atherosclerosis. heart is working harder, blood has smaller areas to go through

A

hypertension

104
Q

what 3 things affects perfusion by alterations in hemostasis

A
  1. atrial fibrillation
  2. myocardial infarction
  3. hypertension
105
Q

keeping a good balance is important btwn which two mechanisms

A

thrombus and fibrinolysis

106
Q

what is normal BP

A

120/80

107
Q

what is pre-htn bp

A

120-139/80-89

108
Q

stage 1 htn bp:

A

140-159/90-99

109
Q

stage 2 htn bp

A

160+/100+

110
Q

HTN crisis bp

A

180/120

111
Q

pathophysiology of HTN

A

thicker blood (viscosity) or small vessel radius =increase pressure
increase pressure (resistance) = higher BP
atherosclerosis (plaque) leads to narrowing of arteries
narrowing of the arteries leads to increase in BP

GOAL = manage resistance against what heart must pump

112
Q

HTN is known as

A

the silent killer - you dont know you have it

113
Q

high BP from an unidentifiable cause

A

primary HTN

114
Q

higher BP from an identifiable cause

A

secondary HTN

115
Q

if you stop medications abruptly, can have sudden increase of BP

A

rebound HTN

116
Q

what’s it called when BP higher than 180/120, true emergency - will lead to organ damage

A

hypertensive crisis

117
Q

modifiable risk factors HTN

A

-stress
-excessive dietary sodium
-sedentary lifestyle
-smoking
-alcohol
-drugs
-oral contraceptives
-diabetes type II

118
Q

this creates water retention means more water in CV system so increasing pressure

A

excessive dietary sodium

119
Q

nonmodifiable risk factors HTN

A

age
african american
family history
gender

120
Q

this to consider for old ppl and HTN

A

HTN increases with age
age causes functional & structural changes in body
risk of polypharmacy
Na+ restriction
lifestyle modifications
include family in teaching

121
Q

what are the symptoms of hypertension

A

HTN does not present with symptoms until its too late

122
Q

hypertension can speed up what disease

A

coronary artery diease

123
Q

what issues does hypertension create

A

organ damage, blurry vision, loss of vision, ischemia, pooling of blood

124
Q

pts with HTN have a higher chance of getting what

A

stroke

125
Q

what are the RN assessments for HTN

A

-monitor vital signs
-assess and reassess before and after giving BP medications
-CV and respiratory assessments

126
Q

what does a nurse look for during CV and respiratory assessments when pt has hypertension

A

looking for chest pain quality and length
edema
lung sounds - may hear crackles

127
Q

during HTN assessment - what does a nurse assess for signs of complications of (5)

A
  1. coronary artery disease (CAD)
  2. cerebrovascular disease
  3. peripheral vascular disease (VAD)
  4. nephrosclerosis
  5. retinal damage
128
Q

labs and diagnostics for HTN

A

CBC - complete blood count
BMP - basic metabolic panel
Serum lipid profile
serum liver function studies
serum thryroid tests
12 lead EKG
history & physical exam findings

129
Q

during a CBC for HTN - what do you specifically look at

A

platelets, RBC, WBC, hemoglobin, hematocrit

130
Q

during BMP for HTN - what do you specifically look at

A

BUN, creatine, electrolytes (Na, K, Mg, Cl, glucose, CO2)

131
Q

patient teaching for HTN

A

-low or normal fat - good v bad fats
-exercise
-no smoking/alcohol or other drugs
-decreasing stress
-high BP screenings
-screened for diabetes
-look for comorbid conditions - elevated lipids, diabetes, use alcohol or drugs
-teach signs and symptoms of distress so pt knows when to go to provider

132
Q

meds for hypertension

A
  1. beta blockers
  2. ACE inhibitors
  3. diuretics
  4. anticoagulants
133
Q

these are blood thinners

examples

A

anticoagulants

ex: heparin, warfarin, enoxaparin

134
Q

lower BP by pulling off fluid resulting in diuresis

list 2 examples

A

diuretics

ex: furosemide, bumetanide

135
Q

lower blood pressure. all drugs end in “pril”

examples

A

ACE inhibitors

ex: lisinopril, enalapril

136
Q

lower heart rate and blood pressure. all drugs end in “lol”

examples

A

beta blockers

ex: metoprolol, atenolol

137
Q

collaborative goals for pts with HTN

A

-lifestyle modifications
-avoid tobacco
-psychosocial
-diet change
-exercise

138
Q

pathophysiology of PVD - peripheral vascular disease

A

-characterized by reduced blood flow through vessels
-peripheral arterial disease (PAD) comes under the umbrella of PVD.
-lumens narrow —> blood flow decreases —> tissue ischemia

139
Q

unresolved tissue ischemia = ?

A

infarction

140
Q

risk factors for PAD - peripheral arterial disease

A

atherosclerosis (CAD)
smoking
obesity
family history
age
race

Pre-existing health conditions:
Coronary artery disease
Cerebral artery disease
Diabetes
Hypertension
Dyslipidemia
Clotting disorders
Hyperhomocysteinemia

141
Q

clinical manifestations for PAD

A

some pts are asymptomatic

unequal lower extremity pulses (or absence of pulses)
loss of hair on leg area
shiny appearance on area of the LE

142
Q

arterial assessment for pts with PAD

A

Be alert for the following signs and symptoms, known as the “Six Ps”:

Pain (severe, shooting, stabbing, or burning sensation)
Pallor (lighter color than the rest of the skin)
Pulselessness (no palpable pulse)
Poikilothermia (cool temperature to palpation)
Paresthesia (numbness, tingling, hot/cold sensations)
Paralysis (immobility [late sign], indicates severe tissue damage)

143
Q

what is intermittent claudication

A

pain may be described as aching or cramping in a muscle that occurs with the same degree of exercise or activity and is relieved with rest.

144
Q

what is the hallmark symptom of PAD

A

intermittent claudication

145
Q

what causes intermittent claudication

A

by the inability of the arterial system to provide adequate blood flow to the tissues in the face of increased demand for nutrients and oxygen during exercise

146
Q

how is intermittent claudication relieved

A

Relieved by stopping muscle use

147
Q

overall review of intermittent claudication

A
  1. Cramp-like pain in a muscle
  2. Consistently reproduced with the same degree of exercise or activity
  3. Relieved by stopping muscle use
  4. Caused by inability of the arterial system to provide blood flow that keeps up with increased demand
  5. Site of arterial disease can be determined by the location of claudication
  6. Pain occurs in muscle groups distal to the diseased vessel
  7. 70–80% of patients do not have worsening symptoms
  8. 10% of claudicants will progress to critical limb ischemia (CLI) (White, 2016)
  9. Dependent position reduces pain
148
Q

during a focused assessment - waht do you look for for PAD

A

Structural changes, resulting from chronic lack of oxygen and nutrient delivery to the tissues:
Hair loss distal to the occlusion
Thick, opaque nails; shiny, dry skin
Skeletal muscle atrophy

Skin color changes:
Elevational pallor
Dependent rubor (red color when limb dependent from dilated damaged vessels)

Pulse changes:
Pulses diminished or absent below area of stenosis/obstruction-pedal, posterior tibial, popliteal, femoral
Cool extremity distal to occlusion

Sensation changes:
Paresthesias
Numbness
Tingling of extremities

Ulcers or gangrene

Edema

149
Q

lab/diagnostics for PAD

A

-c-reactive protein (CRP) - inflammation marker
-homocysteine level
-serum lipid profile - triglycerides, HDL/LDL & Cholesterol
-doppler ultrasound - of the extremities
-ankle-brachial index
-angiography - locates extent of the PAD. Contrast dye injected which can confirm or refute arterial disease

150
Q

surgical revascularization to improve blood supply from aorta into femoral artery and the vessels below the formal artery. incision made down the medial part of leg with PAD

A

arterial bypass

151
Q

surgical procedure that may be performed with or without a stent. balloons inserted into vessel where might be plaque

A

angioplasty

152
Q

post op nursing interventions for PAD

A
  1. check distal pulses
  2. assess incision site
  3. perform NV checks to leg (6 Ps)
  4. assess for signs/symptoms of compartment syndrome
  5. monitor for bleeding/thrombosis
  6. assess mental status (any signs for decreased perfusion)
  7. health promotion - assess for PAD risk factors
  8. avoid heating pads/heating blankets
  9. use compression socks/devices
  10. moist dressings
  11. control blood sugars
  12. assess any wounds for s/s of infection
153
Q

what is the pt teaching that needs to happen post op regarding PAD

A
  1. wear socks, shoes
  2. keep feet dependent for maximum blood flow to LEs
  3. no bare feet
  4. diet modification - decrease high cholesterol foods, decrease saturated fats, less refined sugar, encourage vitamin c, proteins & zinc
154
Q

meds for PAD

A

-analgesics for pain
-cilostazol (pletal) for intermittent claudication
-clopidogrel (plavix) for protection from platelet aggregation
-aspirin or other anticoagulants to keep blood thin
-medications to treat atherosclerosis:
*ACE inhibitors
*diuretics

155
Q

collaborative goal for PAD - arterial

A

anticoagulant therapy (IV heparin then warfarin), surgical interventions - thrombectomy, fem/pop

156
Q

collaborative goal for PAD - venous

A
  1. compression devices, dressing care, nutrition/diet
157
Q

arterial ulcer:
location

A

location: Distal to arterial stenosis, heels, toes, over bony prominences, metatarsals, malleoli, between toes, trauma points

158
Q

arterial ulcer: ulcer-base

A

Dry, pale gray or yellow; may be necrotic

159
Q

arterial ulcer: shape

A

Border regular and well demarcated

160
Q

arterial ulcer: surrounding tissue

A

Pale; cooler than other skin areas. In longstanding insufficiency, skin is thin.

161
Q

arterial ulcer: edema

A

Minimal unless leg is dependent often

162
Q

arterial ulcer: pain

A

Claudication. Rest pain; continuous pain worsens with elevation and eases with dependency.

163
Q

arterial ulcer: pulses

A

May be absent or diminished; often disappears with exercise

164
Q

venous ulcer: location

A

Around ankle, lower third of leg, more often on medial side

165
Q

venous ulcer: ulcer-bas

A

Generally shallow but may be deep. Pink, but may be beefy red with granulation tissue. Ulcer bed usually moist. May have copious drainage

166
Q

venous ulcer: shape

A

Irregular border

167
Q

venous ulcer: surrounding tissue

A

Darkened color in gaiter area. Temperature higher than other skin areas. Brawny edema. Skin may be thick and fibrotic (woody). May be oozing and crusted

168
Q

venous ulcer: edema

A

may be severe

169
Q

venous ulcer: pain

A

Aching, throbbing, heaviness. Superficial stinging when open to air during dressing changes

170
Q

venous ulcer: pulses

A

Usually present with only venous etiology but may be difficult to palpate with edema

171
Q

what is the pathophysiology of VTE/PE

A

it’s unknown

172
Q

VTE =

A

blood clot that forms in the “deep veins”

173
Q

PE =

A

blood clot in the arteries of the lungs

174
Q

Virchow’s tirad evidence

A

stasis of blood, vessel wall injury, and altered coagulation

175
Q

platelet aggregation can lead to ___ ___

A

pulmonary embolism

176
Q

risk factors for vte/pe

A
  1. blood pooling AFIB blood pools in top chambers of heart
  2. surgery - immobility; distrupting hemostasis
  3. cardiovascular disease
  4. cancer
  5. sickle cell disease
  6. trauma to vein
  7. recent childbirth; pain
  8. oral contraceptives; prolonged mechanical ventilation (PE); IV drug use; tobacco use
  9. hormone therapy; corticosteroids; recent bone fracture; travel/been on a plane
  10. Virchow’s triad
177
Q

3 parts of virchow’s tirard

A
  1. venous stasis
  2. endothelial damage
  3. blood hypercoagulability
178
Q

what is venous stasis

A

immobility; dysfunctional vein valves; changes in blood flow

179
Q

what is endothelial damage

A

releases clotting factors

180
Q

what is blood hypercoagulability

A

increase in fibrin production; oral contraception, chemo

181
Q

what are the manifestations for VTE/PE

A
  1. unilateral leg edema
  2. pain
  3. hemoptysis
  4. low grade fever 100.4
  5. brown color with venous ulcer - stasis dermatitis
  6. redness/swelling/warmth
  7. pulmonary embolism - sudden or new onset of SOB
  8. hurts when breathe
  9. cramping in legs, tender, dull achy - typically in calf. warm to touch
182
Q

what is Homan’s sign

A

pain in calf during dorsiflexion

183
Q

labs for vte/pe

A
  1. prothrombin time (PT)/international normalized ration (INR)
  2. aPTT
  3. platelets
  4. h/h: hemoglobin & hematocrit
  5. D-dimer
184
Q

what is prothrombin time and normal range

A

time measured for blood to clot
11-13.5 sec is normal range

185
Q

this is used for herapin, this is sensitive test to make it more accurate

A

aPTT

186
Q

waht is the normal range for aPTT

A

20-45 seconds

187
Q

this is important for clotting. if not clotting puts pt at risk for bleeding

A

platelets

188
Q

measuring function of capable of carrying oxygen to tissues

A

hemoglobin & hematocrit

189
Q

do we want hemostasis in the body

A

yes

190
Q

this can indicate a clot is there and is breaking down.

A

d-dimer

191
Q

this can be an indication of PE, pregnancy.

less than ____ can rule out PE

A

d-dimer

less than 500

192
Q

diagnositcs for vte/pe

A

duplex ultrasound
sprial ct scan - ct angio
v-q scan (ventaliation/perfusion scan)

193
Q

this examines the blood flow in major arteries and veins in the arm and legs

A

duplex ultrasound

194
Q

this is uses a dye to see blow flow. with dye you can see structures, see if th re is tissue damage

A

spiral ct scan - ct angio

195
Q

nursing considerations for spiral ct scan

A

-if pt is aware of any allergies to dye
-any renal deficiencies
-pt given IV fluids

196
Q

administers radioactive subustance via inhale & IV to see how much pts getting to see if theres a mismatch and where issue lies

A

v-q scan

197
Q

if performing diagnostic test for vte/pe and pt is pregnant which test do you do

A

v-q scan

198
Q

RN interventions for vte/pe

A
  1. prevent thrombus from start
  2. monitor lab values
  3. early and frequent ambulation
  4. compression socks
  5. cardiac enzymes
  6. elevate legs
  7. check for bleeding risk
  8. pain medicaton
  9. check cap refill
199
Q

if pt has VTE what RN intervention do you NOT do

A

dont’ use socks or SCDs bc it will break it off and send it to the heart or lungs

200
Q

elevating legs does what for vte/pe

A

raising legs decrease swelling and help with venous returns.

201
Q

what medications do you use to treat vte/pe

A

heparin , warfarin, enoxaparin

202
Q

what lab do you use to monitor heparin

what is the antidote

how would it be administered

A

PTT or aPTT

protimin sulfate

sub q or IV - two inches below umbilicus

203
Q

warfarin - what is the lab used to monitor it

what is the antidote

how administered

A

PTINR

Vitamin K

Is pill only

204
Q

enoxaparin - what lab is used to monitor med

A

none - look at CBC or platelets

205
Q

what are the nursing interventions for heparin, warfarin, enoxaparin

A
  1. look for bleeding
  2. swallowing capability
  3. check diet to make sure they aren’t ingesting too much vitamin K
206
Q

collaborative goals for vte/pe

A

pain relief
medication compliance
decreased edema for pts with VTE
no skin breakdown

207
Q

safety considerations for VTE/PE

A
  1. bleeding
  2. BP - watch for low if pt is bleeding, HR will go up
  3. avoid IM injections
  4. check for delayed cap refill, confusion, mental status changes
  5. notify dentist/provider if having surgery/procedure
  6. dont stop taking meds
  7. decrease edema, no skin breakdown - can cause venous ulcers if have skin breakdown
  8. teach about prevention
  9. nutrition therapy
  10. ambulation, ROM exercise, reposition pt every 2 hours if can’t get out of bed
208
Q

A 69-year-old man has been identified as having peripheral artery disease (PAD) and is motivated to slow the progression of the disease. Which of the following measures addresses the most common underlying cause of PAD?

A

Making lifestyle changes that address atherosclerosis

Atherosclerosis is implicated in the etiology of PAD. Hypertension and low activity levels may also exacerbate or contribute to the disease, but these are less significant than arteriosclerosis. Increased intake of antioxidants has not been shown to affect the course of PAD.

209
Q

An obese patient with a history of smoking and hypertension underwent a coronary artery bypass graft for the treatment of coronary artery disease. During the patient’s postoperative recovery, he developed a DVT. The development of this event was a result of the interaction between which of the following pathophysiological phenomena? Select all that apply.

  1. Stasis of blood resulting from immobility and surgery
  2. Injury to one or more of the patient’s vessel walls
  3. Cardiac stress resulting from surgery
  4. Alterations in the normal process of coagulation
  5. The presence of an inflammatory process
A

1, 2, 5

-Stasis of blood resulting from immobility and surgery
-Injury to one or more of the patient’s vessel walls
-The presence of an inflammatory process

210
Q

Which observation regarding ulcer formation on the client’s lower extremity indicates to the nurse that the ulcer is a result of venous insufficiency?

A

Large and superficial

Ulcerations are in the area of the medial or lateral malleolus (gaiter area) and are typically large, superficial, and highly exudative. Superficial venous insufficiency ulcers cause minimal pain. The base of a venous insufficiency ulcer shows a beefy red to yellow fibrinous color.

211
Q

A woman has sought care from her primary care provider, stating, “The front of my foot aches constantly these days, and it’s gotten so bad that it keeps me up at night.” The nurse should recognize that this patient may be experiencing the symptoms of:

A

Severe arterial insufficiency

212
Q

A client is being discharged following pelvic surgery. What would be included in the patient care instructions to prevent the development of a pulmonary embolus?

A

Tense and relax muscles in the lower extremities.

213
Q

A client is being discharged home with a venous stasis ulcer on the right lower leg. Which topic will the nurse include in client teaching before discharge?

A

Application of graduated compression stockings

Graduated compression stockings usually are prescribed for clients with venous insufficiency. The required pressure gradient is determined by the amount and severity of venous disease. Graduated compression stockings are designed to apply 100% of the prescribed pressure gradient at the ankle and pressure that decreases as the stocking approaches the thigh, reducing the caliber of the superficial veins in the leg and increasing flow in the deep veins. These stockings may be knee high, thigh high, or pantyhose.

214
Q

The nurse is caring for a patient with peripheral arterial insufficiency. What can the nurse suggest to help relieve leg pain during rest?

A

Lowering the limb so that it is dependent

Persistent pain in the forefoot (i.e., the anterior portion of the foot) when the patient is resting indicates a severe degree of arterial insufficiency and a critical state of ischemia. Known as rest pain, this discomfort is often worse at night and may interfere with sleep. This pain frequently requires that the extremity be lowered to a dependent position to improve perfusion to the distal tissues.

215
Q

Which risk factor is related to venous stasis for deep vein thrombosis (DVT) and pulmonary embolism (PE)?

A

Obesity

Obesity is a risk factor for DVT and PE related to venous stasis. Trauma, pacing wires, and surgery are related to endothelial damage as a risk factor for DCAT and PE.

Nursing 111 & Pharmacology (12 decks)

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