Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

Core > Pediatrics: Chest > Flashcards

Pediatrics: Chest Flashcards

1
Q

How can you tell if a neonatal chest is hyper inflated or not?

A

Count the ribs:

More than 6 anterior or 8 posterior ribs above the diaphragm is too many (hyperinflated)

Note: Also look for flattening of the diaphragm or increased lucency under the heart.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is the best way to identify a pleural effusion on a neonatal chest radiograph?

A

Look for thickening of the minor fissure, then confirm by scrutinizing the costophrenic angle

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Hyperinflation and perihilar streaks on a neonatal chest radiograph…

A

Think MNoP

  • Meconium
  • Non-group B strep neonatal pneumonia
  • asPiration
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Diffuse granular opacities with normal/decreased lung volumes on a neonatal chest radiograph…

A
  • Surfactant deficiency disease
  • Group B strep neonatal pneumonia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Hyperinflation with ropy appearance of asymmetric lung densities in a term infant…

A

Think meconium aspiration

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Meconium aspiration is more common in…

A

Term or post-term infants

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What should you look for if you’re suspecting meconium aspiration…

A

Pneumothorax (present in 20-40% of cases)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Classic findings of meconium aspiration

A
  • Asymmetric rope-like pulmonary hyper densities
  • Hyperinflation
  • Pneumothorax (20-40% of cases)

Note: Usually in a term or post-term infant.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Meconium aspiration is usually associated with under inflation/hyperinflation

A

Hyperinflation (due to ball-valve effect from obstructing aspirated meconium)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Newborn with history of cesarean deliver with respiratory distress and bilateral coarse interstitial markings and fluid in the fissures that resolve by day 3 of life…

A

Transient tachypnea of the newborn

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Risk factors for transient tachypnea of the newborn

A
  • Cesarean section
  • Maternal sedation
  • Maternal diabetes
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What is the time course of transient tachypnea of the newborn?

A

Peaks at day 1 of life and resolved by day 3

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What is the most common cause of death in premature infants?

A

Surfactant deficiency disease (AKA respiratory distress syndrome)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Premature infant with respiratory distress and low lung volumes with bilateral granular opacities on radiography…

A

Think surfactant-deficiency disease

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

How can you differentiate surfactant-deficiency disease from group-B hemolytic neonatal pneumonia…

A

Surfactant-deficiency disease will not cause pleural effusions (which are common in group-B hemolytic neonatal pneumonia)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

True or false: A normal plain film at 6 hours of life excludes surfactant-deficiency disease

A

True

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Central bleb-like lucencies in an infant being treated for surfactant-deficiency disease…

A

Normal treatment response

Note: Do not confuse this with pulmonary interstitial emphysema.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Infants being treated for surfactant-deficiency disease are at an increased risk for…

A
  • Pulmonary hemorrhage
  • Patent ductus arteriosus
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

What is the most common type of pneumonia in newborns?

A

Group-B beta-hemolytic strep pneumonia (acquired during vaginal birth)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Bilateral granular opacities with low lung volumes in a newborn…

A
  • Surfactant-deficiency disease (if premature)
  • Group-B strep pneumonia (if pleural effusions are also present)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Does neonatal pneumonia usually have increased or decreased lung volumes?

A
  • Decreased lung volumes (group-B beta-hemolytic strep pneumonia)
  • Increased lung volumes (non group-B strep pneumonia)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Bilateral granular opacities, low lung volumes, and pleural effusions in a newborn…

A

Think group-B beta-hemolytic neonatal pneumonia

Note: If there were increased lung volumes, then you would think non group-B pneumonia.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Is group-B or non group-B strep pneumonia more associated with pleural effusions?

A

Non group-B strep neonatal pneumonia is more highly associated with pleural effusions (75% of cases)

Note: Group-B strep neonatal pneumonia is also highly associated with pleural effusions (25%) which can help distinguish it from surfactant-deficiency disease.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Patchy, asymmetric perihilar opacities with pleural effusions and hyperinflation…

A

Think non Group-B strep neonatal pneumonia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

Persistent pulmonary hypertension of the newborn

A

In utero, the pulmonary blood pressures are high (allowing blood to bypass the lungs). This high pulmonary pressure usually normalizes after the infant takes their first breath, but can persist.

Note: This can be primary (idiopathic) or secondary (due to hypoxia in the setting of meconium aspiration, pneumonia, etc.).

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

Linear lucencies on chest radiograph in a 6 day old infant being ventilated for surfactant-deficiency disease…

A

Think pulmonary interstitial emphysema (air escaping the alveoli and entering the interstitial/lymphatics due to high PEEP)

Note: This pt is likely to develop a pneumothorax if not corrected.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

How can you differentiate pulmonary interstitial emphysema from bronchopulmonary dysplasia?

A

Pulmonary interstitial emphysema occurs in the first week of life (usually while ventilated)

Bronchopulmonary dysplasia occurs in pts older than 2-3 weeks of life

Note: Both can present with linear lucencies on chest radiograph.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

Treatment for pulmonary interstitial emphysema

A
  • Switch ventilation methods (e.g. decrease PEEP)
  • Place the infant with the affected side down (if PIE on the left, then place them left side down)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

Linear lucencies on the chest radiograph of a 20 day old infant being ventilated for surfactant-deficiency disease

A

Think bronchopulmonary dysplasia (AKA chronic lung disease)

Note: If this infant were less than 1 week old, then you would think pulmonary interstitial emphysema.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

Alternating regions of fibrosis (band-like opacities) and hyper aeration in an ex-premature infant who had prolonged ventilation…

A

Think bronchopulmonary dysplasia (AKA chronic lung disease)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

Post-term infant with respiratory distress…

A

Think meconium aspiration

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
32
Q

Term infant delivered by cesarean section with respiratory distress…

A

Think transient tachypnea of the newborn

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
33
Q

Term infant born to a mother who received sedation during delivery with respiratory distress…

A

Think transient tachypnea of the newborn

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
34
Q

Preterm infant with respiratory distress…

A

Think surfactant-deficiency disease (respiratory distress syndrome)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
35
Q

What are the major causes of pulmonary hypoplasia?

A
  • Primary (idiopathic)
  • Secondary (e.g. congenital diaphragmatic hernia, etc.)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
36
Q

What are the major causes of secondary pulmonary hypoplasia?

A
  • Things taking up too much space within the chest (e.g. congenital diaphragmatic hernia, neuroblastoma, giant congenital heart, sequestration, etc.)
  • Thinks outside the chest pushing on the chest (e.g. messed up rib cage such as in Jeune syndrome, large intraabdominal mass, etc.)
  • Things outside the chest that mess up normal lung development (e.g. oligohydramnios such as in premature rupture of membranes, Potter sequence, renal problems, etc.)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
37
Q

Reduced fetal thoracic circumference during an OB ultrasound…

A

Think pulmonary hypoplasia (possibly secondary to oligohydramnios)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
38
Q

Fetal lung to head ratio < 1 on an OB ultrasound…

A

Think pulmonary hypoplasia (possibly secondary to congenital diaphragmatic hernia, skeletal issues, or oligohydramnios)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
39
Q

Potter sequence pathophysiology

A

Dysfunctional kidneys results in no urine output (no amniotic fluid), which prevents normal lungs from maturing (the fetus must breath the amniotic fluid for normal pulmonary development)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
40
Q

What is the most common congenital diaphragmatic hernia?

A

Bochdalek hernia (“B” in the Back of the diaphragm)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
41
Q

Bockdalek hernias are usually on the left, but Bochdalek hernias on the right are associated with…

A

Group-B strep pneumonia

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
42
Q

Solitary, unilocular cyst abutting the trachea…

A

Think bronchogenic cyst

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
43
Q

Bronchogenic cyst with an air-fluid level

A

Think infected bronchogenic cyst

Note: These usually do not communicate with the airway, so an air-fluid level should make you think infection.

44
Q

Bronchopulmonary sequestration

A

Can be thought of as ectopic lung tissue that does not connect to the airways or pulmonary vasculature normally

Note: This can be intralobar or extralobar.

45
Q

Bronchopulmonary sequestration tissue usually gets it blood supply from…

A

The aorta via a feeding artery

46
Q

How can you differentiate intralobar from extralobar pulmonary sequestration on imaging?

A

You can’t, the difference is extralobar sequestration is covered by a pleural lining and drains to systemic veins whereas intralobar has no pleural covering and drains to pulmonary veins (but you can’t see either of these on imaging)

Note: The way to differentiate is by age and clinical presentation.

47
Q

How can you differentiate intralobar from extralobar pulmonary sequestration?

A

Age and clinical presentation:

Recurrent infections in an adolescent (intralobar)

Respiratory distress/cyanosis in an infant (extrtalobar)

48
Q

Which type of pulmonary sequestration is more often associated with other congenital anomalies?

A

Extralobar sequestration

Note: EXTRAlobar sequestration is associated with EXTRA things.

49
Q

Extralobar sequestration is associated with…

A
  • CCAMs
  • Congenital diaphragmatic hernias
  • Vertebral anomalies
  • Congenital heart disease
  • Pulmonary hypoplasia

Note: EXTRAlobar sequestration is associated with EXTRA things.

50
Q

Where is intralobar pulmonary sequestration most often located?

A
  • Left lower lobe (60%)
  • Right lower lobe (40%)
51
Q

Where is extralobar pulmonary sequestration most often located?

A
  • Left lower lobe (90%)
  • Subdiaphragmatic (10%)
52
Q

Which pulmonary sequestration type usually presents with recurrent infections?

A

Intralobar

Note: The pleural covering of extralobar sequestration protects it from infections.

53
Q

Where do pulmonary sequestrations drain veins to?

A
  • Pulmonary veins (intralobar)
  • Systemic veins (extralobar)
54
Q

Recurrent pneumonia in the left lower lung…

A

Think intralobar pulmonary sequestration

55
Q

Congenital lobar emphysema

A

When a bronchial pathology, such as bronchial atresia, leads to progressive air trapping resulting in a hyper lucent, hyper expanded lobe on radiographs that gets worse over time

56
Q

What is the most common location for congenital lobar emphysema?

A

Left upper lobe

57
Q

Treatment for congenital lobar emphysema?

A

Lobectomy

Note: The lobe with air trapping will not get better due to whatever bronchial pathology is causing the air trapping.

58
Q

Question has a series of chest radiographs that show a left upper lobe opacity that resolves at the next time point and then has additional time points showing that where the opacity was is not getting hyper lucent and hyperexpanded, eventually causing mass effect on the mediastinum…

A

Think congenital lobar emphysema

59
Q

CCAM

A

Congenital cystic adenomatoid malformation (AKA CPAM), a congenital malformation where lung tissue is replaced by dysfunctional adenomatoid tissue, usually only involving one lobe. The adenomatoid tissue does have a connection to the airway, which allows them to fill with air creating cystic spaces.

60
Q

Time course of CPAM

A

AKA CCAM (congenital cystic adenomatoid malformation)

Most spontaneously decrease in size by the third trimester

61
Q

Treatment for CCAM

A

Congenital cystic adenomatoid malformation (AKA CPAM)

Surgical removal of adenomatoid tissue (due to the small risk of malignant transformation to pleuropulmonary blastoma or rhabdomyosarcoma)

62
Q

You see what looks like a CCAM and then identify a feeding artery from the aorta to the “adenomatoid” tissue…

A

Not CCAM, if there is an arterial feeder then it is a pulmonary sequestration

63
Q

Cystic air spaces clustered in the left upper lobe of a kid…

A

Think congenital lobar emphysema (due to left upper lobe location)

Note: Could also be CCAM.

64
Q

Cystic air spaces clustered in the left lower lobe of a kid…

A

Think pulmonary sequestration (due to left lower lobe location)

Note: Also look for a diaphragmatic hernia as these also favor the left lower lobe.

65
Q

How can you differentiate a congenital pulmonary malformation from a primary lung tumor on MCQs?

A

Developmental/congenital pulmonary lesions in general are present on the second trimester screening ultrasound (if they tell you the 2nd trimester ultrasound was normal and now there is a pulmonary mass, think primary lung tumor

Note: This is a general rule, but there are exceptions.

66
Q

What is the most common primary lung malignancy in children?

A

Pleuropulmonary blastoma

67
Q

Child with a large right-sided, pleural-based pulmonary mass without chest wall invasion or calcifications (2nd trimester screening ultrasound was normal)…

A

Think pleuropulmomary blastoma (most common primary lung malignancy in children)

68
Q

How can you distinguish a pleuropulmonary blastoma from an Askin/Ewing sarcoma in a child with a lung mass?

A

Both can appear as masses centered at the pleura, but Askin/Ewing sarcoma should have chest wall invasion (and PPB does not)

69
Q

Pleuropulmonary blastomas are associated with…

A

Multilocular cystic nephrons (10%)

70
Q

What is the most common primary lung mass in children?

A

Inflammatory myofibroblastic tumor (benign)

71
Q

Child with a calcified, lobulated lung mass in the lower lobe…

A

Think inflammatory myofibroblastic tumor

72
Q

Characteristic appearance of inflammatory myofibroblastic tumor on MRI

A

T2 bright (looks like fluid because they are composed of myxoid stroma)

73
Q

What is the path of an umbilical venous catheter?

A

Umbilical vein -> left portal vein -> ductus venous -> hepatic vein -> IVC

74
Q

What is the proper position for an umbilical venous catheter?

A

IVC-right atrium junction

75
Q

Complications of umbilical venous catheter placement

A
  • Hepatic infarction or portal hypertension (if malpositioned in a portal vein)
  • Hepatic hematoma formation (if UVC erodes into the liver parenchyma)
76
Q

What is the path of an umbilical artery catheter?

A

Umbilical artery -> iliac artery -> aorta

77
Q

What is the proper position of an umbilical artery catheter?

A

T8-T10 (can also place lower at L3-L5)

Note: The important point is to not place the tip near the renal arteries, where it can cause thrombosis.

78
Q

Complications of umbilical artery placement

A

Renal arterial thrombosis (which is why positioning is important)

79
Q

How can you differentiate an umbilical venous catheter from an umbilical artery catheter?

A

The umbilical artery catheter courses inferiorly before turning around to go superior

80
Q

Contraindication for an umbilical artery catheter

A

Omphalocele

81
Q

What should you look for when evaluating umbilical artery catheter placement

A
  • Catheter should remain left of midline on radiographs (in aorta)
  • Tip should be at T8-T10 (or L3-L5)
82
Q

Where should the catheters be for venoarterial ECMO?

A
  • Right atrium (take deoxygenated blood)
  • Aortic arch (give oxygenated blood)

Note: This can help rest the left ventricle, which doesn’t have to pump as much blood in venoarterial ECMO.

83
Q

Where should the catheters be for venovenous ECMO?

A

Both catheters in the right atrium (usually a dual lumen catheter)

Note: This only provides lung support, not the extra cardiac support that venoarterial ECMO can provide.

84
Q

Chest radiographs demonstrate near complete white out of the bilateral lungs after initiating ECMO…

A

This is a normal finding (ECMO causes atelectasis and other physiological changes that looks like severe edema)

85
Q

Complications of ECMO

A
  • Hemorrhage (due to anticoagulation needed for ECMO)
  • Brain bleeds (high risk due to anticoagulation and sick state of the pt)
86
Q

What additional complication is possible for venoarterial ECMO that doesn’t exist for venovenous ECMO?

A

Neurologic ischemic complications

Note: For venoarterial ECMO a carotid artery is usually sacrificed while placing the aorta catheter, leading to increased risk for neurogenic ischemic complications.

87
Q

Classic imaging findings in viral pneumonia

A
  • Peribronchial edema
  • Air trapping
  • Subsegmental atelectasis
88
Q

What is the classic imaging finding seen in RSV infections?

A

Segmental or lobar atelectasis (particularly in the right upper lobe)

Note: In addition to the peribronchial edema seen in non-specific viral pneumonia.

89
Q

5 y/o with a circular mass in the posterior lower lobes…

A

Think round pneumonia and recommend a follow up chest radiograph to confirm resolution

90
Q

Round pneumonia is classically caused by…

A

Strep pneumoniae

Note: Only children (usually <8 y/o) get round pneumonia because you don’t have good collateral ventilation pathways.

91
Q

5 y/o with a rounded, pleural-based mass in the apex of the lung…

A

Think neonatal atypical peripheral atelectasis (similar to round pneumonia, but peripherally-based)

92
Q

Centrilobular airspace opacities with fat attenuation in a child…

A

Think lipoid pneumonia and try to figure out what might have been aspirated

93
Q

Expiratory radiographs demonstrate hyperlucency of the left lung that persists on left lateral decubitus views…

A

Air trapping in the left lung, suspicious for a bronchial foreign body in a child

94
Q

Chest radiographs demonstrate hyperlucency of the left lung and the left lung appears smaller than the right lung (history of a prior viral lung infection)…

A

Think Swyer James (post-infectious obliterative bronchiolitis)

95
Q

Multiple tracheal and lung nodules with some cavitation in a child…

A

Think papillomatosis

96
Q

Papillomatosis is associated with a 2% risk of…

A

Squamous cell carcinoma

97
Q

If you manipulate papillomatosis nodules…

A

You may cause dissemination of the HPV and cause more nodules

98
Q

3 y/o with history of sickle cell disease and new opacities on chest radiograph…

A

Think acute chest due to sickle cell disease

99
Q

What are imaging signs a kid might have sickle cell disease?

A
  • Big heart
  • Bone infarcts (look at the humeral heads)
  • H-shaped vertebrae on lateral films
  • Cholecystectomy clips
100
Q

What is the pulmonary distribution of cystic fibrosis findings?

A

Apical predominance (lower lobes are less involved)

101
Q

Classic chest imaging findings of cystic fibrosis

A
  • Apical-predominant bronchiectasis
  • Hyperinflation
  • Pulmonary arterial hypertension
  • Mucous plugging (lots of finger-in-gloving)

Note: Look for fatty replaced pancreas on CT and biliary cirrhosis.

102
Q

Upper lobe predominant bronchiectasis in a child…

A

Think cystic fibrosis

103
Q

Lower lobe predominant bronchiectasis in a child…

A

Think primary ciliary dyskinesia

104
Q

Classic imaging findings of primary ciliary dyskinesia

A
  • Lower lobe predominant bronchiectasis
  • Situs inversus (50%; AKA Kartageners)
105
Q

Why do cystic fibrosis and primary ciliary dyskinesia cause male infertility?

A

Males with cystic fibrosis are missing their Vas Deferens

Males with primary ciliary dyskinesia make sperm that can’t swim

Core (98 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions