Chapter 29 & 30: Musculoskeletal and Neuromuscular Dysfunction Flashcards

1
Q

What are some pediatric differences when compared to adults regarding musculoskeletal system?

4 differences

A
  • epiphyseal plate still elongating
  • ligaments and tendons are stronger than bones until puberty
  • bones are more porous and pliable – “bounce” rather than break
  • infant skull fontanels in process of closing
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2
Q

What are fractures?

4 aspects

A
  • break in a bone
  • common injury in children, but rare in infants except with MVA
  • most common broken bone in childhood (< 10 y.o.) = clavicle
  • may be caused by bicycle or sports injuries
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3
Q

What are the 4 types of fractures?

A
  • compound/open: fractured bone protrudes through skin
  • complicated: bone fragments have damaged other organs/tissues
  • comminuted: small fragments of bone are broken from fractured shaft and lie in surrounding tissue
  • greenstick: compressed side of bone bends but the tension side of the bone breaks, causing an incomplete fracture
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4
Q

What is a growth plate (epiphyseal/physeal) injury?

4 aspects

A
  • injury to the cartilage growth plate (epiphyseal plate), weakest point of long bones
  • frequent site of damage
  • may affect future bone growth
  • treatment:
    – ORIF – open reduction, internal fixation – prevents growth disturbances
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5
Q

What are some s/s of fractures?

7 s/s

A
  • swelling
  • pain or tenderness
  • deformity
  • diminished functional use
  • bruising
  • muscular rigidity
  • crepitus
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6
Q

What are general bone healing times for each stage of childhood?

A
  • neonatal = 2 - 3 weeks
  • early childhood = 4 weeks
  • later childhood = 6 - 8 weeks
  • adolescence = 8 - 12 weeks

– younger children have more osteoblasts –> faster healing times for fractures

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7
Q

How are fractures diagnosed? What are some interventions for fractures?

A
  • confirmed diagnosis with X-rays
  • treatments:
    – pain meds
    – PT & OT
    – skin assessments
    – neurovascular checks – CMS checks (circulation, movement, sensation)
    – pulses in extremities distal to fracture
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8
Q

What are the 5 P’s for fracture assessment?

A
  • pain
  • pulse – distal to fracture
  • pallor
  • paresthesia – sensation distal to fracture
  • paralysis – movement distal to fracture
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9
Q

What might be indicated by pain unrelieved by pain medication?

A

compartment syndrome

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10
Q

What are some education topics for families of casted pts?

5 education topics

A
  • don’t get it wet
    – use blow dryer on COOL setting if it gets wet
    – if too wet, will need to have it changed
  • don’t stick anything down cast
    – can cause skin breakdown or infection
  • supervise casted children to ensure nothing is stuck down cast
  • parents can perform home CMS checks
  • cast removal
    – child can touch saw to see that it won’t hurt
    – limb will look different when cast comes off (pale, smelly, smaller than other limb)
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11
Q

What is traction? What is it used for?

5 uses

A
  • an extended pulling force using weights for injured extremities
  • used to:
    – rest an extremity
    – position bones for healing
    – immobilize a fracture until healing is sufficient for casting
    – prevent contracture
    – reduce muscle spasms
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12
Q

What is developmental dysplasia of the hip (DDH)?

3 aspects

A
  • congenital hip dysplasia
  • abnormality in the development of the proximal femoral head and acetabulum where they are improperly aligned – femur head lies outside acetabulum
  • 2 types:
    – idiopathic – infant is neurologically intact
    – teratologic – neuromuscular defect
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13
Q

How is DDH diagnosed?

A

US or X-ray

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14
Q

What are some s/s of DDH?

5 s/s

A
  • asymmetrical gluteal and thigh folds – most common s/s
  • shortening of femur
  • Ortolani click
  • limited hip abduction
  • positive Trendelendburg sign with lordosis
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15
Q

What are some interventions for DDH?

2 interventions

A
  • swaddling
  • Pavlik harness – skin assessments
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16
Q

What is congenital clubfoot? What are the 4 types?

A
  • birth defect where one or both feet are twisted out of alignment
  • 4 types:
    talipes varus: inversion
    talipes valgus: eversion
    talipes equinovarus: plantarflexsion with toes lower than heel
    talipes calcaneus: dorsiflexion with toes higher than heel
17
Q

How is clubfoot treated?

A

serial casting

18
Q

What is osteomyelitis?

A
  • infection of bone and tissue around bone
  • AKA septic joint
19
Q

What are some s/s of osteomyelitis?

A
  • pain in affected bone
  • fever
  • irritability
  • guarding of affected limb
20
Q

How is osteomyelitis diagnosed?

5 diagnostics

A
  • based on s/s
  • radiography
  • bone scan
  • blood cultures
  • I&D, aspirate fluids – determine infection
21
Q

What are some interventions for osteomyelitis?

5 interventions

A
  • blood cultures
  • broad-spectrum antibiotics, then targeted antibiotics
  • monitor labs
  • treat symptoms – antipyretics, pain meds, rest, fluids, nutrition
  • education on antibiotic compliance – takes a long time for this to heal; must remain compliant throughout treatment
22
Q

What is scoliosis?

2 aspects

A
  • complex spinal deformity in 3 planes:
    – lateral curvature
    – spinal rotation causing rib asymmetry
    – thoracic hypokyphosis
  • may be congenital or develop during childhood
23
Q

What are some s/s of scoliosis?

7 s/s

A
  • lateral curvature of spine
  • uneven waistline
  • unequal shoulders
  • unequal scapula
  • unequal hips
  • rib prominence
  • pain is not a normal finding – may indicate spinal mass
24
Q

What are some interventions for scoliosis?

2 interventions

A
  • bracing
  • spinal fusion if severe
25
Q

What is juvenile idiopathic arthritis (JIA)?

3 aspects

A
  • AKA juvenile rheumatoid arthritis
  • unknown cause
  • peak age = 1 - 3 y.o. & 8 - 10 y.o.
26
Q

What are some s/s of JIA?

9 s/s

A
  • stiffness
  • swelling
  • loss of mobility in affected joints
  • warm to touch, usually without erythema
  • tender to touch
  • symptoms increase with stress
  • growth retardation
  • iridocyclitis – inflammation of vascular layer of eye (iris and ciliary body)
  • uveitis – inflammation of uvea (middle layer of eye)
27
Q

What are the 4 diagnostic criteria for JIA?

A
  • age of onset = < 16 y.o.
  • 1+ affected joints
  • duration of arthritis longer than 6 weeks
  • exclusion of other forms of arthritis
28
Q

What are some interventions for JIA?

7 interventions

A
  • no specific cure
  • goals = preserve function, prevent deformities, relieve symptoms
  • ophthalmologist to treat iridocyclitis and uveitis
  • meds:
    – NSAIDs
    – SAARDs – slow-acting antirheumatic drugs
    – corticosteroids
    – cytotoxic agents
    – immunologic modulators
  • PT & OT
  • nutrition
  • exercise
29
Q

What is cerebral palsy?

3 aspects

A
  • group of permanent disorders of movement and posture
  • caused by intrauterine hypoxia or asphyxia
  • most common permanent physical disability in childhood
30
Q

How is cerebal palsy diagnosed?

5 diagnostics

A
  • assessment in first 2 years of life
  • neuro exams
  • history
  • neuro imaging – CT, MRI
  • metabolic and genetic testing
31
Q

What are some s/s of cerebal palsy?

7 motor s/s; 5 behavorial s/s

A

– motor signs:
* persistent primitive reflexes
* poor head control after 3 months
* stiff/rigid limbs
* arching back
* floppy tone – hypotonia
* unable to sit without support at 8 months
* clenched fists after 3 months

– behavioral signs:
* excessive irritability
* no smiling by 3 months
* feeding difficulties d/t frequent gagging or choking
* persistent tongue thrusting
* no reactions to sounds/talking

32
Q

What are some interventions for cerebal palsy?

5 interventions

A
  • surgery for spinal fusions – scoliosis is common
  • bracing
  • meds:
    – seizure meds – antispasmodics (Ativan), antiepileptics, Botox (relaxes muscles), Baclofen
    – analgesics
  • school and social support
  • PT
33
Q

What is muscular dystrophy (MD)?

4 aspects

A
  • largest group of muscular diseases in children
  • genetic origin
  • gradual muscule degeneration – progressive wasting and weakness
  • marked by increasing disability and deformity
34
Q

What is Duchenne muscular dystrophy (DMD)?

4 aspects

A
  • most severe and most common type of childhood muscular dystrophies
  • X-linked inheritance
  • onset = 3 - 7 y.o.
  • death usually occurs d/t respiratory or cardiac failure
35
Q

What are some s/s of DMD?

6 s/s

A
  • Gower sign – standing upright by first kneeling, then walking hands up straightened legs
  • frequent falls
  • lordosis – excessive inward curve of lower spine
  • enlarged muscles in thighs and upper arms
  • profound muscular atrophy
  • mental deficiency – developmental of cognitive delays (from walkie-talkie to nonverbal)
36
Q

How is DMD diagnosed?

5 diagnostics

A
  • prenatal testing if positive family hx
  • clinical appearance
  • blood demonstrating gene mutation
  • EMG (electromyography) and muscle biopsy
  • serum CPK and AST – elevated in first 2 years of life
37
Q

What are some interventions for DMD?

10 interventions

A
  • no effective treatment
  • goal = maintain function of unaffected muscles as long as possible
  • keep child as active as possible
  • ROM
  • bracing
  • ADLs
  • prevent contracture
  • promote mobility and independence
  • promote growth and development
  • prevent complications and isolation
38
Q

What is osteogenesis imperfecta?

A

genetic disease in which bones fracture easily with no obvious cause or minimal injury