Trauma of Young Permanent Teeth Flashcards

1
Q

What are the 3 main principles of luxation injury management?

A
  1. Reduction
    - Reposition teeth and tissues to their original position
  2. Fixation – via splinting
  3. Endodontic monitoring
    - Pulpal status
    - Periodontal healing/non-healing
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2
Q

What are the aims of fixation?

A

Aims to stabilise and maintain teeth position to
- Optimise healing outcomes for pulp and PDL
- Improve function and provide comfort

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3
Q

What must be done before splinting and after repositioning?

A

Post-positioning x-ray to verify position

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4
Q

What must be taken note of when suturing gingiva as part of the reduction step?

A

Gingiva must be sutured back in correct position if not there will be tissue healing by secondary intention => ugly

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5
Q

How long are permanent teeth splinted for and what are the recommended follow up timingss?

A

Splint for 4 weeks

Follow up intervals (post-trauma)
- 2 weeks
- 4 weeks (remove splint)
- 8 weeks
- 12 weeks
- 6 months
- 1 year
- yearly for at least 5 years

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6
Q

Describe the process of splinting

A

After post-positioning x-ray
- Bend wire to sit passively on teeth
- Attach flexible wire splint to teeth with CR

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7
Q

For lateral luxation injury, what is the recommended definitive treatment?

A
  • For teeth with complete root formation, pulp will likely become necrotic.
  • At 2 week post injury, should commence RCT.
  • Remove pulp and place corticosteroid-AB or Ca(OH)2 as intracanal medicament – prevent inflammatory external resorption
  • Complete obturation at 4 week post injury recall (remove splint after obturation completed)
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8
Q

What are possible sequelae of trauma in permanent dentition?

A
  1. Loss of vitality
  2. Internal/external root resorption
  3. Pulpal calcification/obliteration
  4. Ankylosis/replacement resorption
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9
Q

Rank the types of traumatic injuries by their chance of pulp necrosis

A
  1. Intrusion
  2. Lateral luxation/Extrusion
  3. Concussion/Subluxation
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10
Q

What is the pathogenesis of inflammatory resorption?

A

Occurs due to pulp necrosis:
- Stimulus from infected pulp space will transverse the root and sustain inflammation around the root
- Inflammatory resorption will continue til the whole root is resorbed

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11
Q

Why is there a need for endodontic monitoring post-trauma?

A
  • Protective layer of pre-cementum is damaged during trauma
  • Inflammation due to injury occurs which can cause 3 kinds of resorption
    1. Inflammatory resorption
    2. Surface resorption
    3. Replacement resorption
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12
Q

What is the pathogenesis of surface resorption?

A

Occurs when PDL cells are still viable
- Cementum healing can occur – only surface resorption

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13
Q

What is the pathogenesis of replacement resorption?

A

Occurs when large surface of PDL has been damaged
- Replacement resorption will take over inflammatory resorption (bone fills space) => ankylosis
- Occurs in severe injuries: intrusion, avulsion, lateral luxation

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14
Q

What may be a presentation of ankylosis?

A

Ankylosed tooth is infraoccluded – adjacent teeth have grown with alveolus

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15
Q

What should be noted about teeth with pulp canal obliteration?

A

pulp canal obliteration => slender neurovascular bundle => ortho movement will ↑ risk of tooth becoming non-vital

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16
Q

What should be included in a diagnosis of root fracture?

A
  • Single/multiple
  • Horizontal/vertical
  • Level: apical/middle/coronal ⅓
  • Degree of separation between fragments (radiographically)
17
Q

What is the treatment for root fracture?

A
  1. Reduce fracture & splint
  2. Pulp protection: Cvek pulpotomy (1-2mm) then
    - Ca(OH)2
    - MTA (causes discolouration)
    - Biodentine
  3. Seal off access cavity with CR
  4. 4 week review: splint removal &
    restore aesthetics
    - If fracture is located cervically, stabilisation for a longer period of time (up to 4 months) may be needed
18
Q

What are the possible healing outcomes for root fractures?

A
  • Hard tissue union
  • Interposition of CT
  • Interposition of bone & CT
  • Granulation tissue (coronal pulp necrosis)
    => worst outcome – appears as RL at fracture radiographically
19
Q

Describe transient apical breakdown

A
  • Occurs after luxation injuries
  • Injured tissues undergo spontaneous repair in pulp & periapical area
  • Followed by surface resorption &/or obliteration of pulp canal
  • No permanent damage to pulp
20
Q

How do teeth with transient apical breakdown present?

A
  • PARL
  • No response to vitality tests
  • Grey discoloration
  • No other symptoms
21
Q

What is the treatment for intrusion injuries for permanent teeth with complete root formation?

A

If tooth intruded <3mm: allow re-eruption without intervention
If no re-eruption within 8 weeks
- Reposition surgically and splint for 4 weeks
- Reposition orthodontically before ankylosis develops

If tooth intruded 3mm or more: reposition surgically

Pulp will almost always become necrotic – pulpect should be done at 2 weeks