Brulures, Engelures, TEN/S Flashcards
What are the 5 guiding principles for burn management
Resuscitation
Resurfacing
Reconstruction
Rehabilitation
Recovery
What are the 3 major predictor of burn mortality
Age
TBSA
Inhalation
3 mécanismes de transfert d’énergie?
Convection, Conduction, Radiation
Décrire les Zones de Jackson
Nécrose (Coagulation) – doit être excisé
Stase – salvage possible avec ressuscitation
Hyperémie – viable
5 mechanism de brulure
- Chaleur
- Électrique
- Friction
- Chimique
- Radiation
Qu’est-ce que le burn shock? Nommez 3 mécanismes
Réaction systémique à la brulure lorsque extensive où la perfusion tissulaire est insuffisante pour maintenir le delivery de O2
Environ 10-15% TBSA chez une personne âgée, 20% chez un adulte et 30% chez un enfant
3 mécanismes :
1. Relaxation endothéliale de NO
2. Augmentation de la perméabilité capillaire
3. Translocation de fluide interstitiel
Phase of edema with burns
Immédiate – minutes, augmente dans les tissus brulés
Delayed – 12-24h, dans les tissus brulés et non brulés
Maximum : à 12h dans une petite brulure et 24h dans une grosse brulure
Indication for burn resucitation and duration
> 20% (>15% if geriatric or peds) (does not include 1st degree)
For the first 24-48h crystalloids firth 24h and colloids 2nd 24h
Endpoint of resuscitation
- Débit urinaire
- BP
- Base deficit
- Lactates
- Autres : Index cardiaque >3.5L/min/m2, TVC
Modified Brooke Formula
2xTBSAxkg
first half in first 8h and second have in next 16h
+ add maintenance for children
Target 0.5cc/kg/h or 30-50cc/h average
What is fluid creep
Surviens quand >6cc/kg/h repletion
Conséquences :
ARDS
Infections nosocomiales
Augmente la mortalité
Risque de compartiment
Score for mortality prediction in Burns
Baux score: Age + %TBSA + 17 if inhalation
50% mortality if score >110
Is inhalation injury present 50% mortality if score >100
Describe rule of 9’s
Palm without digits 1%
Head and neck: 9%
Anterior chest and abdomen: 18%
Back :18%
Each upper limb: 9%
Each lower limb: 18%
Geneitals: 1%
Difference between second degree deep and third degree burns
Both do not blanch
3rd degree burn is insensate
Blistering absent in third degree
Signs of inhalation injury
Carboxyhemoglobine >10%
Sooth in airway
Singed nasal hairs
Respiratory distress
Workup for electrical injury
CK
Tropo
ECG
Cardiac monitoring
Alkali versus acids that cause deeper injury
Alkali
Treatment of alkali and acids burns
Copious 15-20 minute irrigation
hydrofluoric acid: systemic irregularities and treatment
Systemic hypocalcemia
Treat with calcium gel, intradermal calcium gluconate or intraarterial calcium gluconate if severe
Cardiac monitoring
What is the end point for use of calcium gluconate
resolution of pain
What is the antidote to white phosphorous
Copper sulfate 0.5%
What are the metabolic changes seen with white phosphorous
HypOcalcemia, hypERphosphatemia, cardiac arrythmias
What is the mechanism of copper sulfate
Binds to phosphorous sulfate and prevents oxidization with contact to air. Turns particles black to remove them
What is the antidote for phenol burns
Polyethylene glycol (IV sodium bicarbonate can also be given to help with cardiac arrythmia)
In electrical burns what is the tissue with the most resistance
Bones
In electrical burns what is the tissu with the least resistance
Nerves
In increasing magnitude, list tissues resistance to electrical injuries
Trick: NeVer Mind PeTit GrOs
- nerves -> vessels ->muscles -> peau -> tendons -> gras -> os
Other than joule heating, name another mechanism of electrical burn that causes destruction of cells
Electroporation (formation of pores in cell lipid bilayer. This results in calcium influx and apoptosis)