psychopathology: ocd Flashcards

1
Q

Outline 2 behavioural Characteristics of OCD

A

compulsions

avoidance

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2
Q

Outline 2 emotional Characteristics of OCD

A

anxiety + distress

co-morbid depression

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3
Q

Outline 2 cognitive characteristics of OCD

A

obsessive thoughts

hyper-vigilance

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4
Q

What is the Concordance rate of OCD between MZ and DZ twins?

A

MZ= 68%
DZ= 31%

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5
Q

How does the comparison of concordance rates support the genetic explanation? A03

A

high concordance rates show evidence that OCD can be genetically predisposed to those related, especially twins

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6
Q

What is the evidence between other family members?

A

parents also with OCD= 37&
siblings also with OCD= 21%

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7
Q

How does the evidence between family members support the genetic explanation? A03

A

shows that even if not twins, likelihood of also getting OCD through a genetic predisposition is still possible- still a genetic link

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8
Q

What is an alternative explanation as to why OCD runs in families? A03

A

the diathesis stress model- genes increase the likelihood of OCD however there still has to be an environmental trigger

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9
Q

Name 2 neurotransmitters linked to OCD, and whether activity is increased or decreased in OCD

A

serotonin levels - low levels mean transmission of mood regulated information doesn’t happen, resulting in low moods and anxiety, (activity decreased)

excess dopamine- too much information being passed from neuron to neuron leading to compulsive behaviours (increased activity)

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10
Q

What evidence is there to support the brain chemical explanation? (hint: effectiveness of drug treatments - Soomro et al) A03

A

Soomro et al found that medications targeting serotonin, like SSRIs, were effective in reducing OCD symptoms when compared to placebo. This suggests that serotonin plays a role in OCD, supporting the idea that brain chemicals are involved in the disorder.

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11
Q

Name 2 different areas of the brain linked to OCD

A

orbito-frontal cortex
basal ganglia

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12
Q

What symptoms of OCD are the areas of the brain linked to?

A

orbito-frontal cortex = low mood, poor mood regulation

basal ganglia = compulsive behaviours

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13
Q

Why can’t we say changes in these brain regions cause OCD? A03

A

because there may be other variables such as genetics, also causing OCD

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14
Q

Describe how SSRIs work and how they reduce symptoms of OCD

A

selective serotonin re-uptake inhibitors, they prevent re-absorption and breakdown of serotonin which increases the levels of serotonin in the synapse

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15
Q

Explain research to support the effectiveness of SSRIs e.g Soomro et al. A03

A

Soomro et al. conducted a study where they looked at how well medications called SSRIs work for OCD. They gathered data from different studies and found that SSRIs are more effective at reducing OCD symptoms compared to placebo pills. This research suggests that SSRIs, which affect serotonin levels in the brain, are helpful in treating OCD. This supports the idea that serotonin plays a role in OCD.

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16
Q

What are the Side effects of SSRIs?

A

nausea, headaches, insomnia, dizziness, digestive problems, anxiety

17
Q

Why is Drug treatment more cost effective and less time consuming than СВТ? А03

A

medications are usually more accessible than CBT appointments are, they also act faster and do not take multiple sessions to work, they have a lower initial cost for the patient.

18
Q

Why is Drug treatment often combined with CBT? A03

A

to enhance the effectiveness and tackle cognitive and emotional symptoms, the treatment can also be tailored to each individual patient

19
Q

Why might drugs not work for some people with OCD? A03

A

the side effects may clash with other drugs that are being taken, some people may have addiction issues meaning they cannot take specific medical drugs, people may be forgetting and not take the drugs when needed to, and the drugs may not target the right areas of a persons OCD