neuromuscular system and PNF 1.3 Flashcards

1
Q

What is the other name for type 1 (slow twitch) muscle fibres?

A

slow oxidative muscle fibres

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2
Q

characteristics of type 1 muscle fibres

A
  • have a slower contraction speed
  • have a high aerobic capacity due to high mitochondrial density, high myoglobin content, and high capillary density
  • low fatiguability
  • low anaerobic capacity
  • low force produced due to small motor neurone size
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3
Q

what is another name for type 2a muscle fibres

A

Fast oxidative glycolytic muscle fibres

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4
Q

characteristics of type 2a muscle fibres

A
  • large motor neurone six=ze causing a large force produced from contraction
  • high anaerobic capacity
  • medium aerobic capacity (medium fatiguability) due to medium myoglobin content, medium mitochondrial density, medium capillary density
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5
Q

what is another name for type 2b muscle fibres?

A

fast glycolytic muscle fibres

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6
Q

characteristics of type 2b muscle fibres

A
  • large motor neurone size = large force production
  • very high anaerobic capacity
  • low aerobic capacity: low mitochondrial density, low myoglobin content, low capillary density
  • high fatiguability
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7
Q

what is the effect of training on fibre type

A
  • fibre type tends to be genetically determined
  • training can increase size of muscle fibres (hypertrophy) resulting in greater strength
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8
Q

what is a motor unit?

A

consists of a motor neurone and it’s muscle fibres
- only one type of muscle fibre can be found in one particular motor unit

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9
Q

function of motor neurone

A

transmits the nerve impulse to the muscle fibre so that a contraction can occur

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10
Q

what is the neuromuscular junction

A

where the motor neurone and muscle fibre meet

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11
Q

what is the all or none law?

A
  • once the nerve impulse stimulates the muscle fibres, either all of them contract or none of them do (it isn’t possible for a motor unit to partially contract0
  • minimum amount of stimulation is required to start a contraction. If sequence of impulses is less than the threshold, no muscle contraction will occur.
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12
Q

what is wave summation and how does it control the strength of a contraction?

A

the greater the frequency of the stimuli, the greater the tension developed by the muscle (greater force of contraction)

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13
Q

how does spatial summation control the duration of a contraction?

A

It involves recruiting motor units whilst others rest and then swapping allowing a longer contraction. e.g. helpful for a climber holding onto the wall

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14
Q

how does spatial summation control the strength of a contraction?

A
  • the correct number and size of motor units are selected.
  • e.g. a strong contraction would involve recruiting large numbers of large motor neurones (type 2b)
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15
Q

what does PNF stand for?

A

Proprioceptive neuromuscular facilitation

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16
Q

what is PNF?

A

an advanced stretching technique used to increase the range of motion at a joint

17
Q

function of muscle spindles

A
  • they detect how far and how fast the antagonist is being stretched by sending impulses to the CNS. They will initiate a stretch reflex/contraction by the CNS sending impulses back to the muscles when they detect overstretching. This prevents overstretching which can cause injury
  • found between muscle fibres
18
Q

function of Golgi tendon organs

A
  • found between the muscle fibre and tendon
  • detect increased tension in the muscle when the antagonist isometrically contracts and will autogenic inhibition. This is a sudden relaxation of the antagonist.
19
Q

advantages of PNF

A
  • increased range of motion at a joint
  • increased muscle strength
  • increased muscle flexibility
20
Q

disadvantages of PNF

A
  • high risk of injury
  • requires a partner and good communication
  • not suitable for younger athletes
21
Q

explain the CRAC method of PNF

A
  • a passive stretch occurs and muscle spindles detect how far and fast the muscle is being stretched by sending impulses to the CNS
  • the stretch is done until tension is felt, which will occur if the muscle is overstretched due to CNS sending impulses back to muscle causing a contraction to prevent overstretching
  • antagonist then isometrically contracts in response to impulse
  • Golgi tendon organs detect this contraction and initiate autogenic inhibition. This is a sudden relaxation of the antagonist in response to high amounts of tension. Finally the agonist contracts and the joint is able to move further