1F RESPIRATORY Flashcards

(72 cards)

1
Q

Insert the medication canister into the plastic holder.

If a spacer is used, insert the MDI into the end of the spacer.

Shake the inhaler vigorously five or six times before using. Remove the cap from the mouthpiece.

Have the patient breathe in through the mouth and exhale.

With the inhaler properly positioned, have the patient hold the inhaler with the thumb at the mouthpiece and the index finger and middle finger at the top

Instruct the patient to take a slow, deep breath through the mouth and during inspiration, to push the top of the medication canister once.

Have patient hold the breath for 10 seconds and then exhale slowly through pursed lips

If a second dose is required, wait 1 to 2 minutes, and repeat the procedure by first shaking the canister in the plastic holder with the cap on.

When it is first used or if it has not been used recently, test the inhaler by spraying it into the air before administering the metered dose.

If a glucocorticoid inhalant is to be used with a bronchodilator, wait 5 minutes before using an inhaler that contains a steroid

Teach patients to self-monitor their pulse rate.

Caution against overuse because side effects and tolerance may result.

Teach patient to monitor the amount of medication remaining in the can-ister. Advise patient to ask a health care provider or pharmacist to estimate when a new inhaler will be needed based on the dosing schedule.

Teach patient to rinse their mouth after using an MDI. This is especially important when using a steroid drug. Rinsing the mouth helps prevent irritation and secondary infection to oral mucosa.

Avoid Smoking

Teach patient to do daily cleaning of equipment; this should include (1) washing the hands; (2) taking apart all the washable parts of the equipment and washing them with warm water; (3) rinsing: (4) placing the parts on a clean towel and covering them with another clean towel to air dry; and (5) storing the parts in a clean plastic bag once completely dry.
Alternate two sets of washable equipment to make this process easier.

A

Correct use of a Metered-Dose Inhaler

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2
Q

H1 Blockers of H1 Antagonists

A

Antihistamines

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3
Q

competes with histamine for receptor sites and prevent histamine response

A

Antihistamines

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4
Q

Act by blocking H1 receptors

Decreases nasopharyngeal secretions by blocking H1 receptor

A

Antihistamines

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5
Q

when stimulated, extravascular smooth muscles constrict

A

H1

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6
Q

when stimulated, gastric secretions occur

A

H2

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7
Q

Cold

Allergic rhinitis

Urticaria

Not used for anaphylaxis

A

Indications for Antihistamines

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8
Q

T/F: Most antihistamines are rapidly absorbed in 15 minutes, but they are not potent enough to combat anaphylaxis

A

TRUE

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9
Q

Mostly can cause drowsiness, dry mouth, decreased secretions and other anticholinergic symptoms

contained in many OTC cold remedies

A

1st Generation Antihistamines

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10
Q

Drowsiness

Dizziness

Fatigue

Disturbed coordination

Skin rashes

Anticholinergic symptoms

A

Side Effects of Antihistamines

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11
Q

Most prevalent type of upper respiratory infection

A

Common Cold

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12
Q

Caused by rhinovirus

A

Common Cold

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13
Q

Acute inflammation of the mucus membrane of the nose

A

Acute Rhinitis

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14
Q

Hay Fever; Caused by pollen or a foreign substance such as animal dander

A

Allergic Rhinitis

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15
Q

blocks the effects of histamine by competing for and occupying H1 receptor sites

A

Diphenhydramine

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16
Q

Primarily used to treat rhinitis

A

Diphenhydramine

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17
Q

closed angle glaucoma

urinary retention

peptic ulcer

small bowel obstruction

A

Contraindications of Diphenhydramine

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18
Q

can cause central nervous system depression if taken with alcohol, narcotics, hypnotics, or barbiturates

A

Diphenhydramine

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19
Q

Advise patients to avoid alcohol and other CNS depressants

A

Nursing Interventions of Diphenhydramine

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20
Q

Have fewer anticholinergic effects and a lower incidence of drowsiness

A

2nd Generation Antihistamines

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21
Q

Nonsedating Antihistamines

A

2nd Generation Antihistamines

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22
Q

Results from dilation of the nasal blood vessels caused by infection, inflammation, or allergy

A

Nasal Congestion

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23
Q

medications used to treat nasal congestion

A

Nasal Decongestant

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24
Q

stimulate the alpha-adrenergic receptors leading to vascular constriction of the capillaries within the nasal mucosa

A

Nasal Decongestant

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25
Frequent use results to tolerance and rebound nasal congestion
Nasal Decongestants
26
used primarily for allergic rhinitis including hay fever and acute coryza
Systemic Decongestants
27
May decrease beta blocker effect
Pseudoephedrine + Beta blockers
28
increased possibility of hypertension or cardiac dysrhythmia
Decongestant + MAOIs
29
Increased restlessness and palpitations
Decongestant + Caffeine
30
Effective for treating allergic rhinitis
Intranasal Glucocorticoid
31
with anti-inflammatory action — allergic rhinitis symptoms of rhinorrhea sneezing and congestion
Intranasal Glucocorticoid
32
Continuous use = dryness of the nasal mucosa may occur
Intranasal Glucocorticoid
33
Nervous Restless Rebound nasal congestion
Side Effects of Decongestant
34
Hypertension Cardiac Disease Hyperthyroidism Diabetes Mellitus
Contraindications of Decongestant
35
Headache Nasal Irritation Pharyngitis Fatigue Insomnia Candidiasis
Side Effects of Intranasal Glucocorticoid
36
Act on the cough control center in the medulla to suppress the cough reflex
Antitussives
37
for nonproductive and irritating cough
Antitussives
38
Nonopioid Opioid Combination Preparations
Types of Antitussives
39
Benzonatate
Nonopioid
40
Codeine Dextromenthorphan Guaifenesin Homatropine 1.5mg & Hydrocodone 5mg
Opioid
41
Guaifenesin Dextromethorphan
Combination Preparations (with expectorant)
42
provides temporary cough relief due to non-productive cough
Dextromethorphan
43
Acts by decreasing the excitability of the cough center in the medulla
Dextromethorphan
44
Loosen bronchial secretions so they can be eliminated by coughing Used for productive cough with or without nonpharmacologic agents
Expectorants
45
inflammation of the mucous membrane of one or more of the maxillary, frontal, ethmoid or sphenoid sinuses
Sinusitis
46
Antibiotic may be prescribed for acute or severe sinusitis acetaminophen fluids and rest may be helpful
Sinusitis
47
Inflammation of the throat or sore throat
Acute Pharyngitis
48
can be caused by a virus, beta-hemolytic streptococci or other bacteria
Acute Pharyngitis
49
can occur alone or with common cold and rhinitis or acute sinusitis Elevated temperature cough
Acute Pharyngitis
50
Throat culture first (should be obtained to rule out beta-hemolytic streptococci infection)
Acute Pharyngitis
51
Chemoreceptors are sensors that are stimulated by changes in these gases and ions. Chemoreceptors are located centrally and peripherally The central chemoreceptors, which are located in the medulla near the respiratory center and the cerebrospinal fluid, respond to an increase in carbon dioxide and a decrease in pH by increasing ventilation. However, if the carbon dioxide level remains elevated, the stimulus to increase ventilation is lost. Peripheral chemoreceptors are in the carotid and aortic bodies. It responds to changes in oxygen levels. A low blood oxygen level stimulates the peripheral chemoreceptors which in turn stimulate the respiratory center in the medulla, and ventilation is increased. If oxygen therapy increases the oxygen level in the blood, the oxygen may be too high to stimulate the peripheral chemoreceptors, and the ventilation will be depressed. Take Note: the tracheo-bronchial tube consists of smooth muscles whose fibers spiral around the trachea-bronchial tube. Contraction of these muscles constrict the airway. The sympathetic and the parasympathetic nervous system affects the bronchial smooth muscles in opposite ways.
Respiration
52
The vagus nerve releases acetylcholine, which causes Bronchoconstriction The sympathetic nervous system releases epinephrine which stimulates beta 2 receptors in the bronchial smooth muscle, resulting in Bronchodilation These two nervous systems counterbalance each other to maintain homeostasis.
Respiration
53
Increase bronchodilation by bronchial smooth muscles. Phosphodiesterase enzyme can inactivate cAMP
Cyclic Adenosine Monophosphate (cAMP)
54
Chronic Obstructive Pulmonary Disease Restrictive Pulmonary Disease
Lower Respiratory Infections
55
Caused by airway obstruction with increased airway resistance of airflow to lung tissues Chronic bronchitis Bronchiectasis Emphysema Asthma
Chronic Obstructive Pulmonary Disease
56
Results in irreversible lung tissue damage
Chronic Obstructive Pulmonary Disease
57
Results to the decrease in total lung capacity as a result of fluid accumulation or the loss of elasticity of the lungs
Restrictive Pulmonary Disease
58
Pulmonary edema Pulmonary fibrosis Pneumonitis Lung tumors Thoracic deformities (scoliosis) Disorders affecting thoracic muscular wall
Restrictive Pulmonary Disease
59
Characterized by periods of bronchospasm
Bronchial Asthma
60
results when the lung tissue is exposed to extrinsic or intrinsic factors that stimulate broncho constrictive response
Bronchospasm
61
Progressive lung disease Caused by smoking or chronic lung infections Bronchial inflammation and excessive mucous secretion > airway obstruction
Chronic Bronchitis
62
Hypercapnia and Hypoxemia > respiratory acidosis
Chronic Bronchitis
63
Abnormal dilation of the bronchi and bronchioles due to frequent infection and inflammation
Bronchiectasis
64
Bronchioles become obstructed by the breakdown of the epithelium of the bronchial mucosa and tissue fibrosis may result
Bronchiectasis
65
Progressive lung disease caused by smoking, atmospheric contaminants, or lack of alpha-antitrypsin protein that inhibits proteolytic enzymes that destroy alveoli
Emphysema
66
Terminal bronchioles become plugged with mucous, causing a loss in the fiber and elastin network in the alveoli Alveoli enlargement as many of the alveolar walls are destroyed
Emphysema
67
Airway obstruction Dyspnea Decreased gas exchange Fatigue
Patient Problems on Chronic Pulmonary Disease
68
Bronchodilators (sympathomimetics, parasympatholytic, and methylxanthines to assist in opening narrow airways) Glucocorticoids (decreases inflammation) Leukotriene Modifiers (used to reduce inflammation in lung tissue) Expectorants (assist in loosening the mucus from the airways) Antibiotics (to prevent serious complications from bacterial infections)
Medications for Chronic Obstructive Pulmonary Disease
69
used to manage COPD.
Sympathomimetics or Alpha and Beta 2 Adrenergic Agonists
70
Acts on Alpha 1, Beta 1, Beta 2 Adrenergic Receptor Sites Promotes bronchodilation and elevates blood pressure Used during anaphylaxis reaction through Subcutaneous route
Sympathomimetics: Epinephrine
71
Beta 2 adrenergic agonist Stimulates beta 2-adrenergic receptor in the lungs which relaxes the bronchial smooth muscle > bronchodilation High dose or overuse may cause some degree of beta 1 response > nervousness, tremor, and increased PR
Albuterol
72
Asthma Bronchospasm
Indications of Albuterol