213 Flashcards

(78 cards)

1
Q

What is ventilation?

A

Active breathing. Inspiration and expiration.

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2
Q

What is hyperventilation and what can it cause?

A

Breathing off CO2 and respiratory alkalosis.

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3
Q

What is hypoventilation and what can it cause?

A

Retain CO2 and respiratory acidosis.

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4
Q

What is hypoxemic respiratory failure?

A

Low O2 leading to low blood O2 levels.

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5
Q

What is hypercapnic respiratory failure?

A

Lungs can’t remove CO2 causing high CO2 levels in blood, causing an increase in rate and depth of respirations.

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6
Q

What should you look for in hypercapnic patients and how should they be treated?

A

Mental status change and give them a BIPAP.

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7
Q

High CO2 is a PaCO2 above?

A

45.

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8
Q

What is gas exchange? How does it affect your patient?

A

CO2 out and O2 in. Exchange of gas between alveolar air and blood.

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9
Q

Hypoxemia is an PaO2 of?

A

Less than 80 as normal range is 80-100.

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10
Q

What are early signs of hypoxemia?

A

AMS- restlessness, agitation, confusion. High vitals- tachypnea, tachycardia, hypertension.

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11
Q

Why might vitals be high early on with hypoxemia?

A

The body is desperately trying to push the little amounts of oxygen it does have around.

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12
Q

What are the late vital signs of hypoxemia?

A

Low vital signs like bradypnea, bradycardia, hypotension, cyanosis, dysrhythmias.

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13
Q

What are the expected ABG numbers of a COPD patient?

A

Low PaO2 which correlates to hypoxemia - low O2. High PaCO2 over 45 (acidosis) that correlates to hypercapnia - too much CO2. pH less than 7.35 acidosis.

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14
Q

Is anemia common in COPD patients? And how does the RBCs cause risk to the patient?

A

No as RBC are actually increased due to hypoxia and they are also thicker blood causing risk of CVA or stroke.

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15
Q

What is COPD?

A

Irreversible damage to the alveoli and bronchi of the lungs that causes decreased gas exchange.

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16
Q

How does decreased gas exchange in COPD patients affect the lungs?

A

The double C’s- Chronic air trapping causing decreased gas exchange and decreased inflammatory damage. CO2 high which limits airflow and prevents exhalation.

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17
Q

What are some possible causes of COPD?

A

Smoking and car mechanics, Alpha antitrypsin deficiency - genetic disorder affecting protein used to protect the lungs.

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18
Q

How does smoking affect the lungs in COPD patients?

A

It damages the alveoli and ciliary action.

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19
Q

What are some parts or subsections of COPD?

A

Chronic bronchitis, emphysema, hyperactivity airway disease.

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20
Q

What does emphysema cause?

A

PINK- pursed lips and pink skin, increased chest (barrel chest), no chronic cough, tripoding.

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21
Q

What is emphysema?

A

Loses lung elasticity and inflation due to loss of surfactant which helps the lungs stretch.

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22
Q

Advanced emphysema can cause?

A

Frequent infections and risk of pneumothorax.

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23
Q

Chronic Bronchitis Signs and symptoms?

A

BLUE- Bluish skin (cyanosis), long term (chronic) cough/sputum, unusual lung sounds (crackles and wheezes), edema peripherally (due to cor pulmonale). Last 3 months more, productive cough and mucous, more severe in the morning.

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24
Q

What is cor pulmonale?

A

Caused from COPD. Patient experiences right-sided heart failure and body is rocked with fluids which can increase pressure in the lungs due to pulmonary hypertension.

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25
What are COPD manifestations?
New dyspnea, weight loss, clubbing (chronic hypoxia - vasodilation), prolonged expiration, respiratory acidosis, compensatory polycythemia.
26
When assessing someone showing signs of polycythemia in COPD patients, they may need what drug and why?
Low dose heparin due to increased thickness in RBC that can cause stroke.
27
What must you report with a patient exhibiting polycythemia?
High hemoglobin levels.
28
When treating COPD medications, it is important to not use what kind of medications?
Opioids or benzos.
29
What are some examples of a short and long acting bronchodilator?
Albuterol and salmeterol.
30
What are bronchodilators?
Widen airway to breathe easier.
31
What is a common beta 2 agonist?
Albuterol.
32
What is albuterol used for? and why?
Brutal asthma attack because it is the only rescue inhaler.
33
What is a common anticholinergic? and what does it do? When should it be used in an asthma attack?
Dries out the body. Ipratropium. 2nd.
34
Why are methylxanthines so toxic especially theophylline?
They can cause seizures if given over 20 mcg, fast HR, and dysrhythmias.
35
What are methylxanthines examples?
Theophylline, aminophylline.
36
What should be avoided when taking methylxanthines?
Beta blockers as they lower heart rate, cimetidine and ciprofloxacin as they increase toxicity and caffeine.
37
What is important to let the doctor know of before giving the next dose of methylxanthine?
Tachycardia.
38
How do xanthine bronchodilators work?
Inhibit phosphodiesterase and increase cAMP causing bronchodilation.
39
What issues can beta agonists cause?
Tachycardia, hypertension, palpitations.
40
After using beta 2 agonist, a patient may feel?
Increased productive cough, decreased anxiety, mild hand tremors, and difficulty sleeping.
41
What should be avoided with beta 2 agonists?
Beta blockers as they can cause bronchospasm.
42
How do you know a beta 2 agonist is effective? And what should you monitor?
Decreased respirations, 90% or more oxygen saturation. Heart rhythm and B/P.
43
Routes for beta 2 agonists?
PO or inhalant but inhalant is preferred.
44
Examples of inhaled corticosteroids?
Beclomethasone, budesonide, fluticasone, and methylprednisolone.
45
When should inhaled corticosteroids be used in an asthma attack and which one is typically used?
3rd and methylprednisolone.
46
What are common side effects of inhaled corticosteroids?
Sugar increase and sores in mouth (infection).
47
After using inhaled corticosteroids, it is important to do what?
Rinse the mouth out to prevent thrush and candida and don't swallow the water or use spacers.
48
What is important with oxygen therapy in COPD patients?
O2 goal isn't as high as people without COPD. Avoid large amounts, remove apnea, CO2 poisoning, remove COPD respiratory drive.
49
What is a common antibiotic for COPD?
Ancef.
50
How should metered dose inhalants be used?
Patient sit upright, shake before you take, hold breath for 10 seconds so med can get deep in airway, exhale with pursed lips, 3 mins between puffs, rinse mouth.
51
What are examples of corticosteroids?
Prednisone (oral), inhaled corticosteroids, Advair.
52
What are some side effects of oral prednisone?
Hypernatremia, hyperglycemia, hypokalemia, GI bleed (black tarry stool).
53
What are the side effects of anticholinergics?
Can't see, pee, poop, or spit.
54
How should you treat the side effects from anticholinergics?
Gum or fluids.
55
When are anticholinergics contraindicated?
If already dry, glaucoma, urinary retention, bowel obstruction.
56
Examples of anticholinergics? Short and long acting.
Atrovent (short acting), Respimat (short), Spiriva (long).
57
Example of a mucolytic?
Acetylcysteine (Mucomyst).
58
What is Acetylcysteine used for overdose wise?
Tylenol poisoning.
59
Acetylcysteine is contraindicated in?
Asthma patients as it causes bronchospasms.
60
Nursing considerations for Acetylcysteine?
Resp insufficiency, inadequate coughing mechanism, gag reflex depression, secretions may occlude if not cleared.
61
What is asthma?
Chronic inflammatory disease in pathway of lungs bronchi, or bronchioles.
62
What occurs during an asthma attack?
Bronchoconstriction, inflammation causing tightness and increased mucous production from goblet cells in respiratory tract.
63
Why is it hard to exhale with asthma attacks?
Resp tract is constricted so O2 can't get in and CO2 can't get out causing air to be trapped preventing exhalation.
64
Etiology of asthma?
Genetic disorders and environmental factors triggered by allergens.
65
Signs and symptoms of asthma?
A-accessory muscle use, S-SOB, T-tight chest, H-high pitch wheezing, M-minimal diminished breath sounds, A-3 A's absent breath sounds (silent chest), acidosis (O2 retention), air trapping (prolonged exhalation).
66
Test for asthma?
Pulmonary function test - how well lungs work.
67
Peak Expiratory Flow Rate (PEFR)?
Measure of the fastest flow of exhaled air after a maximal inspiration.
68
Treatments for asthma?
Bronchodilators and corticosteroids, antinflammatories, epinephrine injections, inhaler, leukotriene inhibitors.
69
What are leukotriene inhibitor examples? And what are they used for?
"Lukast" Montelukast (Singulair). Used for long-term management.
70
Are leukotriene inhibitors used for asthma attacks?
Not for acute asthma attacks. They are for long onset and used to prevent inflammation that causes asthma attacks.
71
What is cromolyn?
Mast cell stabilizer used for asthma to block the massive swelling.
72
When is the best time to use Cromolyn?
10-15 minutes before activity.
73
Nasal Cannula?
1-6 lpm. Short- low O2 after surgery. Long- can dry out membranes need humidification.
74
Partial rebreather?
6-10 lpm.
75
NRB?
10-15 lpm for carbon monoxide poisoning. If bag deflates, increase O2.
76
Simple face mask?
In exchange for partial rebreather. 6-10 lpm.
77
Venturi mask?
A face mask and reservoir bag device that delivers the most accurate oxygen. Used for unstable COPD.
78
Face tent?
Facial trauma high humidification.