Week Five Flashcards

0
Q

What side of the brain needs to be working for perception

A

The right side which controls visual-spatial perception and constructional deficits

Sensory integratory area of the parietal lobe

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1
Q

What is perception and what does it involve

A

Creating of meaning out of a somatosensory inputs

  • vision
  • touch
  • taste
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2
Q

Damage to the left hemisphere vs damage to the right hemisphere

A

Left: Dominant
Results in language deficits and temporal (sequencing) deficits (including problems with initiation)

Right: Non-dominant
Results in disordered visual-spatial perception and constructional deficits

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3
Q

Left brain function vs right brain function

A
Left:
Logic
Detail oriented 
Facts rule
Words and language 
Maths and science
Order/pattern perception 
Reality based
Forms strategies 
Safe
Right:
Uses feelings
Big picture orientated 
Symbols and images present 
Can 'get it' - the meaning 
Can appreciate spatial perception 
Knows object function
Fantasy based 
Risk taking
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4
Q

What is our role in perceptual rehabilitation

A

Testing done by OT’s
Recognise presence of perceptual and motor planning deficits
Apply simple tests to establish severity and monitor progress
Understand the implications of these deficits for movement retraining
Know how to modify PT management in the presence of these deficits in order to optimise motor retraining and functional capacity

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5
Q

What are the different types of perceptual deficits

A
Body schema
Agnosia - including unilateral neglect 
Right/left discrimination 
Spatial relations 
Praxia
Tactile perception 

People with perception issues can’t work out speed or depth

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6
Q

What is body schema

A

Ask patient to point to named body parts

Draw a man

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7
Q

What is agnosia and what are the different types

A

May present as a complete denial of disability (anasognoisa)
Or as organ specific impairments (e.g. Visual agnosia - failure to recognise familiar objects)
Prosopagnosia - inability to recognise faces
Finger agnosia - failure to recognise which finger is touched (Gerstmann’s syndrome)

Important to note that the sensation in the organ is intact but the information is not being processed normally
May be associated with a cognitive impairment - e.g. Poor insight

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8
Q

What is unilateral neglect and what side is it most commonly on

A

Ask them to draw a clock, cross out all the lines, draw a face etc they will only do one side
Frequently on left side, due to right brain injury

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9
Q

What is left/right discrimination

A

Get them to show you their right hand, left ear, hold something in their right hand etc
Laterality
Sometimes need to tell them that their watch is on their left side - this may help them with L vs R

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10
Q

Spatial relations

A

Figure/ground (identify overlapping 2D or 3D objects)
Position one in front of the other
Verticality - rotate a ruler/walking stick in front of the patient and ask them to tell you when it is straight up and down
- associated with Pusher

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11
Q

What is tactile sensation and a test for it

A

Astereognosis - ask patient to recognise objects placed in their hand when their eyes are closed

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12
Q

What is a sensory inattention test

A

Tactile inattention or extinction

Able to identify and localised light touch when each limb is tested in isolation but not during bilateral simultaneous stimulation - will only say they can feel unaffected side

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13
Q

Perseveration

A

Observation of pen paper tasks, may be evident in speech and motor tasks

Patient becomes stuck at one component of the task and continues to repeat that component over and over again

An indication of failure to self monitor behaviour

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14
Q

What is praxia

A

Inability to perform a task, even though there’s nothing wrong with their motor path
Make sure when testing them that you use common objects and make common actions

Three types

  • ideational
  • ideomotor
  • constructional
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15
Q

What is dyspraxia

A

Difficulty in performing a willed purposeful movement or imitating a movement
Disorder affecting the purposeful execution of learned and meaningful activities

Inability to perform the activity is not a result of primary motor or sensory impairments, or deficits in comprehension, memory or motivation

Due to damage in the somesthetic association cortex of the parietal lobe
More commonly found in left sided lesions
May also be associated with damage in the premotor cortex of the frontal lobe causing problems with the execution and sequencing of purposeful movements

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16
Q

Ideomotor dyspraxia

A

Inability to execute ideas or previously learned movements
Difficulty orientating items in the correct plane
Poor initiation, timing and direction of movements

Difficultly performing the movement on command but can do it voluntarily/automatic
- can’t roll on command but can do it to get out of bed

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17
Q

Ideational dyspraxia

A

Inability to understand purpose of action/task, incorrect object use
- unable to put a coat on
Difficulty sequencing tasks
Parts of task omitted/wrong order
Difficulty conceptualising the task
Difficulty initiating the task
Know it’s a coat but don’t know to put it on when it’s cold

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18
Q

Constructional dyspraxia

A

Difficulty coping with visual representation, even simple pictures
Writing may appear crowded and poorly spaced on page
Difficulty creating 3D objects
May have difficulty moving in space
Inability to do more with spacing a timing

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19
Q

How do you manage perceptual deficits

A

Reassure family they’re not crazy or wilful
Ensure you educate the patient
Encourage patient to compensate in terms of scanning
Advise patient and family that they will most improve

Avoid fatigued noisy/busy environments and stress as these will reduce the processing of information/emphasis deficits

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20
Q

Management of left/right discrimination

A

Don’t confuse them by saying L and R

Use visual and tactile input or movement commands

21
Q

Management of neglect

A

Position patient in room so that attention to the affected side is maximised
Window on affected side, family to stand on affected side, approach them from their affected side
Minimise distractions
Encourage the to visually scan the environment into their affected side and tell you what they can see - get family to do this also

22
Q

Management of Pusher

A

Treat in safe, supported environment with upright cues
Provide plenty of tactile stimulation
A mirror can be effective but make sure you don’t confuse them
Explain what’s happened to them at that they need to listen to visual cues - their eyes are correct, but their brain is telling them something else

23
Q

Management of tactile perceptual deficits

A

Encourage them to consciously attend to sensory cues, initially used vision to supplement and then withdraw as they become more able to reply on somatosensory inputs
Use vision to compensate for deficits

24
Q

Management of perseveration

A

Don’t allow patient to perseveration in a task

Stop that particular task altogether and do something completely different then come back to it later

25
Q

Management of poor shared attention and concentration

A

Decreased distractions (don’t talk or challenge their attention during task performance, focus on one task at a time in a quiet environment)
Mention a consistent treatment routine
Don’t rush or overload them with complex tasks
Consistency and routine will help

26
Q

Management of ideomotor dyspraxia

A

Subcortical cues - use vision rather than verbal cues, physical guidance to initiate and follow through movement
Set up environment to encourage automatic movement patterns - external rather than internal practice - reaching for a cup rather than practising parts
Develop a routine/sequence of moments to encourage automaticity

Lay clothes out in a certain pattern to help them get used to a certain routine
Encourage sequence of movement

27
Q

Management of ideational dyspraxia

A

Assist with the first steps of the task
Simple to complex object use - brush hair before tying laces
Develop functional routines
Initially provide full prompts/assist throughout task, as they learn slowly withdraw these
Close supervision during practise to reduced mistakes - patient losing purpose of task

28
Q

Management of constructional dyspraxia

A

Break task down into sub-components
Start with simple tasks
Assist with components of tasks to prevent frustration

29
Q

What is conversion syndrome

A

Present with neurological deficits but there’s nothing actually wrong with them
Neurological psychological issues
don’t allow them to say that they can’t do it, as usually they can
Incongruencies as you assess them

30
Q

What are primary impairments following a CNS injury

A

Loss of voluntary motor control - reduced strength, reduced speed yo generate force, reduced dexterity, reduced selectivity, influence of mass patterns
Loss of sensation
Impaired perfection and cognition - including a reduced conscious state
Impaired coordination
Impaired vision
Impaired speech

Note: loss of sensation can lead to floppy gait, not just ataxia etc

31
Q

What are the 8 secondary changes that result from a CNS injury

A

Develop as a consequence of primary impairments

  1. Musculoskeletal changes - contractures
  2. Learned non-use
  3. Adaptive motor behaviours - compensatory behaviours
  4. Subluxation/joint malalignment - can cause pain
  5. Pain - peripheral and central syndromes
  6. Reduced cardiovascular fitness
  7. Increased muscle fatiguability
  8. Depression and insomnia
32
Q

What are some musculoskeletal changes

Immobility/disuse leading to:

A

Atrophy
Increased fat content - more fat/fibrous tissue than muscle
Adaptive change in muscle fibre profile
Deconditioning - muscular endurance and cardio wise
Adaptive soft tissue shortening (non-neural changes)

33
Q

Musculoskeletal changes

Adaptive soft tissue shortening can lead to

A

Rest/long duration in shortened position
Overactivity/hypertonia contributes to shortening
Muscle imbalance - shortening agonist, lengthing antagonist
- will change length tension ratio

34
Q

When does the greatest level of atrophy occur

A

In LMNL as there’s no messages going to the muscle telling it to contract
Will get atrophy very quickly
Lesion below L2 is below the spinal cord (in corda equina) so will be a LMNL

35
Q

What are the primary muscles affected by adaptive soft tissue shortening

A

UL

  • shoulder adductors
  • internal rotators
  • elbow flexors
  • wrist and finger flexors

LL

  • hamstrings
  • adductors
  • plantarflexors

Having let ankle get to such PF, need to get tilt table to get them upright

36
Q

What are adaptive motor behaviours

A

Attempts to perform purposeful movements using remaining muscles give a reflection of patients ‘best’ attempt given the state of their system and the environmental difficulties in which they function

Best attempt coupled with secondary changes can significantly contribute to the development of abnormal movement patterns

New compensatory strategies become established and reduce the potential for recovery - want to prevent these

37
Q

What is learned non-use

A

When using it gives them negative feedback so they don’t want to use it
Negative reinforcement as it doesn’t work
Get used to/learn not to use it - can’t maximise the potential of the limb after this stage and it becomes very difficult to learn to reuse these again

38
Q

What is constraint-induced movement therapy

A

Constrain the good arm so that they can’t use it
Need to have
- 30* wrist extension
- 20* finger extension
Only suitable for 10% of stroke patients
Need a behaviour contract
Need toy have good arm constrained for at least 8 hours a day

39
Q

Subluxation and joint malalignment

A
Where 
- shoulder, wrist and fingers 
Results in 
- pain
- impingement 
- adoptive soft tissue shortening 
- joint restriction
- joint dysfunction 
- hygiene issues 

Common with muscle overactivity
Can be painful or can have it without pain
Discourage pulley systems and self assisted range in shoulders as pushing them past 100-110* IR and flexion can cause a lot of impingement
Encourage them to take care of their arm and don’t pull things or twist them

40
Q

When does subluxation/joint malalignment most often occur

A

Frequently occurs early in the flaccid upper limb due to:

  • muscle paralysis around GHJ
  • effect of gravity
  • poor UL positioning
  • poor UL self care
  • poor handling by health professionals

Measure of subluxation - length between acromion and humeral head, or take photograph and measure from there

41
Q

What are Decorticate syngeries

A

Contractures of biceps and wrist and finger flexors
Contracture in LL due to shortening of the calf muscles
Unlikely to get quads contracture as they’re sitting a lot, may get hip flexor contractures though due to sitting

42
Q

Pain as a secondary impairment

A

Most commonly reported in the shoulder and wrist
84% of stroke patients will experience shoulder pain which may be caused by:
- subluxation
- subacromial bursitis
- adhesive capsulitis
- biceps/supraspinatus tendonitis

Shoulder pain is correlated with:

  • reduced strength of external rotators, elevators, abductors, especially rotator cuff control
  • loss of external rotation, passive and active ROM

Pain may also be associated with sensation of central pain syndrome - CRPS
Abnormal processing of sensory input - thalamic pain
Pre-existing joint pain may be exacerbated by immobility
Incorrect passive movement - such as using pulleys
Joint trauma

43
Q

Physical deconditioning - reduced CV fitness, muscle fatiguability

A

Immobility leads to deconditioning very quickly
Particularly important in older patients
Younger patients may experience significant weight loss and reused CV fitness

De conditioning can severely limit:

  • participation in rehabilitation
  • being active in the community following discharge

The more immobile you are the quicker you decondition
Need to encourage them to exercise
Get them upright so that they’re not lying around in bed all day

44
Q

Depression and insomnia

A

Overwhelming loss of social and physical capacity may lead to depression and insomnia
This can compound cognitive dysfunction and affect participation in rehabilitation and outcomes

The biggest predictor of elderly exercising is dog ownership
Recovery increases dramatically with family and pet input

45
Q

What is the key to prevention

A

Anticipation
Anticipate what might happen and implement strategies to prevent these
Get in there early with good management skills

Start rehab ASAP, even if it’s just in their rooms

46
Q

What can we do to manage and prevent contratcures and what are the ‘at risk’ muscles

A

Need to be in stretched position for at least 8 hours a day
Supportive splinting where there are muscle imbalances
Avoid restrictive splinting as this can contribute to learned non-use
But them in a hoist or tilt table
Rapid loss of range may require medication
Frequent movement
Stretching doesn’t help to lengthen muscle but does help with joint nutrition

At risk muscles

  • long head of biceps
  • hamstrings
  • gastrocnemius

Note: unless severe muscle overactivity is preset, active rehab with sufficient practice of functional tasks will prevent loss of joint range

47
Q

How can we facilitate/support existing muscle activity

A

Modify environment to allow intact muscles to work - eliminate gravity, friction, utilise active/assisted strategies, modify environment
Exercise over appropriate ranges
Facilitate correct movement - hands on, support
Incorporate assistive devices - FES

48
Q

How can we encourage the use of the affected side

A

UL

  • constraint induced movement therapy
  • intensive practice

LL

  • use for bed mobility
  • weight bearing tasks
  • PWSTT
  • encourage symmetry

Get window on affected side and get them to look out it
Encourage family to touch and stimulate the affected side

49
Q

How can we utilise motor learning principles

A

Client centred goal setting
Provide facilitation/alter environment
Appropriate feedback to achieve desired movement pattern
Use functional tasks
Pre-empt maladaptive behaviour or learned non-use
Use metal practise
Provide opportunities for extended practice to allow multiple repetitions
Don’t say too much in cognitive phase etc
Appropriate feedback

50
Q

How can we assist in maintaining and improving physicals fitness

A

Commence rehab ASAP
Ensure therapy opportunities are fully used
Proved extra opportunities for practise
Tailor program to individual needs
Consider reps, load, speed, work capacity
Use appropriate clinical measures
What to push them to fatigue
Brian interprets pain - how can we manage this
In early rehab BW is enough

51
Q

How can we ensure early obstacles to recovery are addressed

A

Early referral to medical staff/clinic,a psychologist if depression or insomnia appear to be interfering with rehabilitation
Collaborative client centred goal setting - essential for otibatkon
Provide opportunities to utilise therapy assistance and particularly family and friends as a supportive family is the best indicator of favourable rehabilitation outcome

Anticipate potentilla changes and prevent these