2.2 Cardiovascular, Acute Coronary Heart Disease Flashcards

(55 cards)

1
Q

Acute Myocardial Infarction

Discuss coronary artery disease and angina pectoris

A

Coronary Artery Disease:
Impaired blood flow to the myocardium, usually caused by atherosclerotic plaque in the coronary arteries. Can be asymptomatic or may lead to angina pectoris, acute coronary syndrome, myocardial infarction, dysrhythmias, heart failure and death.

Angina pectoris:
Chest pain resulting from reduced coronary blood flow which causes imbalance myocardial blood supply and demand
May be due to coronary heart disease, atherosclerosis or vessel constriction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Acute Myocardial Infarction

Discuss pathophysiology of AMI

A

Patho AMI:
Blood flow to a portion of cardiac muscle is completely blocked, resulting in prolonged tissue ischemia and irreversible cell damage. Coronary occlusion is usually caused by ulceration or rupture of a complicated atherosclerotic lesion. When atherosclerotic lesion ruptures or ulcerates, substances are released to stimulate platelet aggregation, thrombin generation. Vessel constriction and a thrombus (clot) forms, occluding the vessel and interrupting blood flow to the myocardium

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Acute Myocardial Infarction

Identify assessment findings related to AMI and ACS

A

Pain:
heaviness/pressure/burning/tightness
substernal or precordial; may radiate to neck, arm, jaw
indigestion, upper back pain, atypical

Skin:
ashen, cool, clammy

Cardiovascular:
Initial elevation of BP and HR but BP can drop with decrease cardiac output
Shock= reduced perfusion of organs (brain, liver, kidneys, etc)
Abnormal heart sound S3 and S4, murmurs

Nausea and vomiting

Fever:
inflammatory process caused by cell death, increased WBC

Anxiety:
impending doom or denial

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Acute Myocardial Infarction

Describe the diagnostic process of ECG

A

ECG changes evolve over course of MI
ST depression= ischemia (inadequate blood supply)
ST elevation= injury
Q wave= infarction (obstruction of the blood supply)
T wave inversion= ischemia or injury

Ischemia: an inadequate blood supply to an organ or part of the body, especially the heart muscles

Infarction: obstruction of the blood supply to an organ or region of tissue, typically by a thrombus or embolus, causing local death of the tissue

ECG leads determine location of MI
ECG within 10 min of arrival to ER
**infarcted tissue doesn’t conduct normally

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Acute Myocardial Infarction

Describe the diagnostic process of cardiac enzymes

A
Cardiac enzymes (markers):
Proteins released when myocardial cells die, they determine the timing and severity of MI. Tests are repeated q6-8 hours for 24 hours

Myoglobin: first detectable but nonspecific with limited use (not often used)

Troponin: Elevates in 2-4 hours, peaks in 10-12 hours, return to baseline within 10 days. MOST specific indicator of MI

CKMB/CK ratio (heart specific/total body): Elevates in 4-8 hours, peaks 18-24 hours, return to baseline within 3 days.
Increased CK= muscle injury
Increased CK-MB= cardiac muscle injury

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Acute Myocardial Infarction

Describe the diagnostic process of stress testing

A

Stress testing
When cardiac enzymes are negative a stress test is ordered
Many stable outpatients have stress tests for initial screening
Exercise testing on treadmill and monitored
Medication induced stress when a patient can not walk on treadmill (meds: Persantine, dobutamine)
Myoview: nuclear isotope tracer (thallium) injected to evaluate UPTAKE into heart muscle
-assess coronary blood flow, perfusion to the myocardium
-scanned with exercise, then repeated at rest (2-3 hrs later)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Acute Myocardial Infarction

Describe the diagnostic process of echocardiogram

A

Echocardiogram:
Sonographic imaging of the heart
Transthoracic (more common) or Transesophageal, requires sedation
Evaluation of cardiac anatomy and function:
Muscle function/wall motion
Muscle thickness
Valve function
Chamber size and shape
Pericardial fluid
Thrombus
**does not evaluate vessels or conduction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Acute Myocardial Infarction

Describe and give rationale for nursing and medical interventions including medications

A

Nursing interventions for AMI

Anticipated orders:
telemetry, serial ECG
serial cardiac enzymes
oxygen
IV access (2 IV's, 18 g)
bedrest to reduce cardiac workload; BSC
light diet, small frequent meals but NPO initially
diagnostic testing
cath lab ASAP with STEMI

Medications:
Nitro- vasodilation, reduced cardiac workload, reduced anginal pain, improves perfusion. First line!

Morphine- drug of choice is nitro is not relieving pain, reduces anxiety, reduces cardiac workload

Aspirin- prevents platelet aggregation, chew for rapid buccal absorption

Heparin- does not lyse clots, prevents further buildup of thrombus

Beta Blocker (“olol”)- reduced O2 demand by reducing HR, BP and contractility, limit size of infarct, reduce risk of reinfarction and arrhythmias. Not used in shock state

Ace Inhibitors (“pril”)- prevents ventricular remodeling and heart failure, reduces mortality

Antidysrhythmic (amiodarone)- if needed, dysrhythmias are common

Cholesterol meds- underlying atherosclerosis

Laxatives- to avoid straining with BM

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Acute Myocardial Infarction

Discuss inter-professional collaboration, including providers and cardiac rehab

A

.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Acute Myocardial Infarction
Complications of AMI
DYSRHYTHMIAS

A

AMI Dysrhythmias:
Most frequent complication

Infarcted tissue is arrhythmogenic, alters impulse generation and conduction

PVC’s very common d/t ventricular irritability

Vtach and Vfib, frequent cause of sudden cardiac death, less common. Shockable rhythms

AV Blocks, common with anterior wall infarction, brady, right sided

Bradycardias, common with inferior wall infarction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Acute Myocardial Infarction
Complications of AMI
HEART FAILURE AND PULMONARY EDEMA

A

.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Acute Myocardial Infarction
Complications of AMI
PERICARDITIS

A

Pericarditis
Inflammation of pericardium: dual membrane sac, holds 30-50ml serous lubricant fluid. Inflammation may cause scarring and become chronic

Infectious causes: vial most common, heals with time

Noninfectious causes: many, including myocardial injury, autoimmune disease, cancer

Pericardial effusion: more than 50ml of fluid

Cardiac Tamponade: pressure around heart and unable to pump= decreased BP, narrow pulse pressure, muffled heart tones, JVD, anxiety, signs of shock

Assessment:
Chest pain: acute and sharp, worse on inspiration and relieved with leaning forward. Pericardial friction rub
Mild fever
Tachycardia
Increased WBC and SED rate
Slight increase in cardiac enzymes

Medica management:
Tylenol for fever
NSAIDS for inflammation, steroids if needed
Treat underlying cause

Surgical management:
Pericardiocentesis: needle aspiration
Pericardial window: removal of small rectangle of the pericardium
Pericardiectomy: removal of entire pericardial sac, typically requires a sternotomy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Acute Myocardial Infarction
Complications of AMI
MITRAL VALVE DYSFUNCTION and other structural defects

A

Mitral Valve:
Papillary muscle rupture
-Ischemia= papillary muscle dysfunction or rupture
-Sudden valve failure, typically mitral regurgitation, loud murmur, surgery immediately

Necrotic muscle= scar tissue, stiff, noncompliant

Ventricular aneurysm= scar tissue displaces outward

Myocardial rupture

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Acute Myocardial Infarction

Discuss rationale for hypothermia and nursing interventions

A

Hypothermia therapy:
Protects brain from cellular injury
Initiated after Vtach or Vfib arrest (when pulse is recovered but patient still unresponsive)

Goal:
Reduce metabolic rate and O2 demand
Stabilize cell membrane
Reduce ischemic damage and reperfusion injury caused by inflammation

Goal temp: 32-34 C, cold saline, cooling blankets
24 hour treatment, then gradual rewarming

Complex ICU nursing care:
Ventilator- sedation and analgesia
Fluid and electrolyte management
Cardiac monitoring
Protect skin- necrotic, bleeding, pressure
Monitor for bleeding and kidney injury
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Cardiac catheterization

Identify indications for right and left catheterization

A

Coronary angiography:
Gold standard for looking at coronary anatomy
Dye and fluoroscopy visualization

Right heart cath:
Measures pressures in right side of heart and lungs
Venous puncture typically in groin

Left heart cath:
Determine location and severity of blockages
Arterial puncture, typically wrist or groin

Ventriculogram:
Measures left ventricular function
Requires additional dye

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Cardiac catheterization

Describe the procedure

A

.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Cardiac catheterization

Identify and give rationale for nursing interventions pre and post cath

A

Pre cath:
Baseline assessment

Post cath:
Neuro
Assess site:
-groin- bedrest 4-8 hrs w/ leg straight, HOB <30 degrees, worried about pseudoaneurysm (not true aneurysm but an injury to the lining of the artery where blood leaks out into the outer most layer, bulging, painful
-radial- pressure bands, no mobility restrictions
Distal circulation
Pain/symptoms
Cardiac rhythm
Restenosis (artery closes again after corrective surgery)
Reperfusion, may also cause angina (ex: foot falls asleep, tingling, pain)
Monitor CBC, BMP and I/O

Medications:
Nitrates
Calcium channel blockers (“dipine”, open arteries)
Antiplatelets (can be dual antiplatelet therapy)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Acute Coronary artery disease

Discuss the use of thrombolytics and nursing care

A

Thrombolytics (clot busters):
Used when there is no access to cath lab for PCI
Reperfusion is the goal
Treat and transfer to a PCI facility for angiography and PCI
Medications: Alteplase (tPA), Reteplase (rPA), Streptokinase within first 6 hours of symptoms onset

Nursing care:
HOB <15 degrees, bedrest
Cardiac monitoring
Monitor for bleeding
AVOID injections, catheter placement r/t bleeding after thrombolytic is administer (bleeding risk).
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Acute Coronary artery disease:
Revascularization: Percutaneous Coronary Intervention (PCI)
Discuss the use of angioplasty and complications

A

Angioplasty is a procedure used to open blocked coronary arteries (balloon inflation of artery) caused by coronary artery disease. It restores blood flow to the heart muscle without open-heart surgery

Complications:
Dissection- artery damage
Vasospasm- artery irritation
Dysrhythmias
Restenosis- artery closes again
CVA- stroke risk
CIN- contrast induced nephropathy
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Acute Coronary artery disease
Revascularization: Percutaneous Coronary Intervention (PCI)
Discuss the use of stents

A

Coronary stents are now used in nearly all angioplasty procedures. A stent is a tiny, expandable metal mesh coil. It is put into the newly opened area of the artery to help keep the artery from narrowing or closing again.

Complications:
Dissection- artery damage
Vasospasm- artery irritation
Dysrhythmias
Restenosis- artery closes again
CVA- stroke risk
CIN- contrast induced nephropathy
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Acute Coronary artery disease
Revascularization: Percutaneous Coronary Intervention (PCI)
Discuss the use of atherectomy

A

Atherectomy remove plaque buildup from an artery. Removing this plaque allows blood to flow more freely through the artery.

An atherectomy often treats artery blockages that contain plaque — fatty substances made up of cholesterol, fats, calcium, and other substances.

Unlike an angioplasty, which moves plaque to the side of the artery, an atherectomy completely removes plaque from the artery

Complications:
Dissection- artery damage
Vasospasm- artery irritation
Dysrhythmias
Restenosis- artery closes again
CVA- stroke risk
CIN- contrast induced nephropathy
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Coronary Artery Bypass Grafting (CABG)
Revascularization
Identify indications for CABG

A

CABG indications:
Complete occlusion and wire cannot pass
Multivessel disease (3 or more vessels blocked)
Left main coronary artery disease
Failed PCI stent
Severe left ventricular dysfunction, low ejection fracture
DM (prone to stents blocking back up)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Coronary Artery Bypass Grafting (CABG)
Revascularization
Describe the procedure

A

CABG procedure:
Place new vessels (bypass) from aorta or internal mammary artery to distal coronary arteries

Grafts (conduit): internal mammary artery (IMA), saphenous vein graft (SVG), radial artery

Sternotomy: large incision, cutting through sternum to access heart

Thoracotomy: incision on side of chest, very painful.

Cardiopulmonary Bypass (CPB) machine: provides a bloodless field for cardiac surgery. Venous blood is drained to a reservoir, oxygenated and sent back to the body using a pump, skipping heart/lungs. Sometimes pt can be off pump

Hypothermia: metabolism drops = less O2 demand

Temporary pacing wires

Chest tubes: fluid and air drainage

Invasive hemodynamic monitoring by arterial lines or central line

24
Q

Coronary Artery Bypass Grafting (CABG)
Revascularization
Identify potential complications

A
CABG complications:
Bleeding
Pneumothorax
Cardiac Tamponade
Stroke
Dysrhythmias
Delirium (acute)
Ileus: temporary lack of muscle contract in intestines
Renal failure
Infection
Pneumonia
25
Coronary Artery Bypass Grafting (CABG) Revascularization Prioritize post op nursing interventions
Postop CABG: Know what the patient had done CBP (cardiopulmonary bipass) affects every system- inflammatory response Monitor cardiac output Oxygenation: vent, suction, airway, extubation, O2 Lungs not inflated during procedure (atelectasis Acute pain Dysrhythmias common Temporary pacer (right atrial, left ventricular) Strict I/O- report <30ml/hr x 2hr Chest tube patency, output, air leaks Labs: ABG's, lytes, blood count, coags, renal function Manage IV fluids VS Hemodynamic monitoring Heart sounds: S3/S4, respiratory compromise ``` First several hours is recovery Once stable, recovery is aggressive -early mobilization -pulmonary rehab (inspirex, cough and deep breathing, flutter, ambulation, nebs) -bowel regimen -multimodal pain management -oral care TID with tooth brush -gum chewing -IPC's, SCD's -assess and manage delirium ```
26
Discuss heart transplant surgery
Heart transplant surgery: When mechanical support is not enough Only definitive treatment for end-stage HF Postop care similar to CABG Dysrhythmias common Myocardial stunning common (initial post op HF) Donor heart is denervated- lacks autonomic regulation, HR runs higher 90-110 Infection and rejection are major risks- antirejection meds are critical but increase infection risk
27
Hemodynamic monitoring | Explain the concepts of cardiac output (CO) and cardiac index (CI)
Cardiac output: Amount of blood the heart pumps in 1 minute Cardiac output= heart rate X stroke volume Cardiac index: Index for size, better indicator of cardiac performance Cardiac output adjusted for body size of patient Provides more meaningful data about the heart's ability to perfuse the tissues, therefore is more accurate indicator of the effectiveness of the circulation than CO
28
Hemodynamic monitoring | Describe how preload, afterload and contractility influence stroke volume (SV) and cardiac output (CO)
Stroke volume includes: preload, afterload and contractility Heart rate X stroke volume = cardiac output Preload: amount of blood remaining in the ventricle. Preload is the end-diastolic stretch on the muscle fibers. Pressure inside the heart just before contraction. Usually volume dependent Afterload: PRESSURE that the ventricular muscles must generate to overcome the pressure in the aorta. Afterload = resistance. Components of afterload: BP, valve compliance (stiff valves, arterial wall compliance (tight arteries) Contractility: inherent ability of the myocardium to contract independent of preload or afterload, velocity and extent of myocardial fiber shortening; quality of the pump. Cannot be directly measured. Influences myocardial oxygen consumption: increased contractility= increased myocardial work= increase O2 consumption. Hear rate: affected by the SNS. Autonomic reflexes trigger heart rate to compensate for changes in cardiac output. Multiple meds are chronotropic, affecting the heart rate. Increasing the heart rate increases cardiac output to the point where it interferes with ventricular filling
29
Hemodynamic monitoring | Discuss the hemodynamic effects of common vasoactive medications
.
30
Hemodynamic monitoring | Explain the rationale for invasive and noninvasive hemodynamic monitoring
Invasive Hemodynamic Monitoring (ART line): Assess cardiovascular function in patients who are critically ill or unstable. Main goal: to evaluate cardiac and circulatory function and the response to interventions. Noninvasive: Pulses, HR, BP, mentation, skin, breath sounds, neck veins. Labs: BNP, ABG, BMP, LFT, lactic acid Invasive: Measure pressures, flow and oxygenation in cardiovascular system Pressure in vessel converted to electrical waveform Parameters include: HR, arterial BP, central venous or right atrial pressure, pulmonary pressures and cardiac output **trends are more significant than individual readings
31
Arterial Pressure Monitoring | Explain the significance of mean arterial pressure (MAP)
Mean arterial pressure (MAP): Average pressure in the arterial system during systole and diastole REFLECTS THE PERFUSION PRESSURE Calculate MAP: Systolic pressure + 2x diastole pressure/3 Ex: 120/80 BP 120 + 160 = 280/3 = 93 ``` Normal: Systolic 100-119 Diastolic 60-79 MAP 70-90 <60 is hypoperfusion, not supplying O2 <40 is circulatory collapse ``` **DIASTOLE IS TWICE AS LONG AS SYSTOLE
32
Arterial Pressure Monitoring | Identify priority nursing responsibilities and rationale for Art-Lines
Dressing: tubing secure/ transparent dressing over insertion site Immobilize: padded splint to immobilize in neutral position (wrist slightly hyper extended) Assess: distal extremity for neurovascular assessments/changes ``` Complication: distal ischemia Thrombus Infection Nerve injury ``` Nursing care: Assess site and distal perfusion Flush lines Maintain dressing and lines **NEVER INFUSE ANYTHING THROUGH ART LINE
33
Arterial Pressure Monitoring | Describe Allen's test
Allen's test is used to evaluate ulnar circulation Patient clench fist, apply pressure to both radial and ulnar arteries. Without releasing pressure have patient to unclench fist to relaxed position. Palm should be blanched due to lack of blood flow Release pressure from ulnar artery to observe return of color within 7 seconds If blood flow returns in 7-15 seconds= blood flow impaired If blood flow returns in >15 seconds blood flow is considered inadequate and the radial artery should not be used in this hand
34
Invasive Lines (PAC= Pulmonary artery catheterization, CVC= Central venous pressure): complications and nursing care
``` Complications: CVC (central line pressure, access by jugular, clavicle) Infection (CLABSI): central line associated bloodstream infection Air emboli Insertion site: -bleeding -hematoma -pneumothorax PA (Pulmonary artery catheterization, access in mid arm) lines: -ventricular dysrhythmias -pulmonary infarction -pulmonary artery rupture -balloon rupture- emboli ``` Nursing care: Verify placement by x-ray prior to infusion Flush line to keep patent Assess distal circulation with limb sites Assess sites for infection, infiltration, phlebitis Strict aseptic technique, scrub the hub Follow protocols for dressing and tubing changes Hold pressure when pulling
35
Describe the effect of heart disease of reproductive health
Oral contraception | Hormone replacement therapy
36
Summarize essential aspects of discharge teaching
``` Risk factor modifications: Smoking cessation Cardiac diet Glycemic control Weight loss ``` Activity guidelines: restrictions and goals ``` Medications ASA/Antiplatelets Beta blockers Nitrates ACE/ARB Statin Diuretics Anticoagulants Antiarrhythmics ``` Multidisciplinary: RN, provider, PT/OT/ST, case manager, social work, cardiac rehab, palliative care Assess capacity for ADL's, mobility , medication management, wound care
37
Angina aggravating factors
``` O2 Supply: Atherosclerosis Anemia Lung disease Dysrhythmias Hypovolemia Valve disorders ``` ``` O2 Demand: Exertion Cardiomyopathy Tachycardia Hypertension Hyperthermia Stress ``` **Myocardial ischemia: insufficient O2 supply= cardiac chest pain
38
Impaired Coronary Perfusion
Coronary artery occlusion with stable plaque Platelet aggregation forms a thrombus Coronary vasospasm (Printzmetal's angina) Drop in BP= reduced coronary perfusion pressure Autoregulatory systems fail
39
Acute Coronary Syndrome (ACS)
Spectrum of myocardial ischemia- injury- infarction- necrosis Not getting enough O2 to heart muscle, supply and demand is off balance Most often due to atherosclerosis Unstable angina Acute myocardial infarction (AMI) -NSTEMI (non ST elevation myocardial infarction) -STEMI (ST elevation myocardial infarction)
40
Cardiac Diagnostics Additional Labs
CBC- increased WBC r/t inflammation and damaged muscle PT/PTT- baselines before intervention BMP- electrolytes and renal function Liver Function Test ABG's- oxygenation and acid/base balance Assess risk factors: lipid profile, Hgb A1C
41
``` Define: Unstable angina NSTEMI STEMI Necrosis ```
Time is muscle. Damage begins within 30 minutes Unstable angina- ischemia: partially occluding thrombus NSTEMI- injury (not full thickness): T wave inverted or ST depressed, enzymes positive, platelet aggregation, early thrombus STEMI- infarction (full thickness): Elevated ST segment, may have Q wave, thrombin rich thrombus, conduction problems can cause life threatening dysrhythmias Necrosis- permanent damage **GOAL: restore blood flow, treat dysrhythmias, stabilize
42
MONA
Morphine: relieves pain, decreases workload O2: keep stats >92% Nitroglycerin: relieves pain, dilates arteries and veins, improves coronary perfusion, reduces workload (usually given first) Aspirin: platelet inhibitor
43
Quality: Core Measures for ACS
On arrival: Aspirin ECG Time to PCI or fibrinolytics: 90 min CMS mandate for STEMI Smoking cessation counseling ``` At discharge: Statin Beta Blockers Aspirin ACE/ARB with MI, DM, CHF ```
44
AMI Complication: Infarct Extensions
``` Extension of the original infarct Presents with same s/s as original MI Typically seen 10-14 days after MI Risks is minimized by medication: Aspirin Beta blockers ("olol") ACE ("pril") inhibitors or ARB ("sartan") Revascularization procedures ```
45
AMI complication: encephalopathy
Encephalopathy: Neurologic injury following cardiac arrest Arrest survivors- high risk for hypoxic brain injury and permanent neuro deficits Statistics- up to 80% are comatose, 10-30% have "meaningful neurologic recovery Oxygen and ATP stores that "feed" the brain are used up in about 5 minutes Prevention: Excellent CPR- push hard, push fast Early defibrillation Targeted temperature management or therapeutic hypothermia
46
AMI complications: Pump failure
Pump Failure: Acute heart failure from muscle wall injury Severity dep on location and size of MI Cardiogenic shock- cardiac output cannot meet O2 demands and organs begin to fail Left HF: Large anterior MI (LV) S/S: crackles, dyspnea, frothy cough, S3 gallop, mentation changes, cough Right HF: Large inferior MI (RV) S/S: JVD, ascites, peripheral edema, hypotension
47
Heart Failure classifications
HF classifications: Acute vs Chronic: Refers to onset and current ability to compensate Left vs Right: One always leads to the other over time Systolic vs Diastolic: Systolic- disease of pumping Diastolic- disease of filling Low output vs High output: Low- cardiac disorder with increased cardiac output High- increased metabolism, cardiac output can't meet demands: hyperthyroidism, sepsis
48
Heart Failure: nursing care
Monitoring: Labs- BNP, renal, lytes, weights, I/O, BP, HR, breath sounds Education: Meds, diet, daily weights, activity/rests balance, when to call, follow up appointments Medication: Beta blockers ("olol")- reduce O2 demand, workload, HR and BP ACE ("pril")/ARB ("sartan")- vasodilation, reduce O2 demand, workload, remodeling, cardiomyopathy Entresto- combo of ARB and sacubitril, increase natriuretic peptides Diuretics
49
What are hemodynamics?
Blood movement Study of blood flow or circulation Management of volumes and pressures to optimize the delivery of oxygen to all the cells of the body ``` REMEMBER THE CELL The end goal is NOT blood pressure Goal is cellular respiration Cellular respiration is the foundation for life Focus on oxygen delivery to the cell ```
50
Preload Assessment | RIGHT AND LEFT
Right: Central Venous Pressure (CVP) Noninvasive- JVD, peripheral edema, weight Left: Pulmonary Capillary Wedge Pressure (PCWP) Noninvasive- lung sounds, BP, urine output (low), weight
51
Afterload Assessment | RIGHT AND LEFT
Right: Pulmonary vascular resistance (PVR) Elevated PVR = pulmonary hypertension Left: Systemic vascular resistance (SVR) Noninvasive- diastolic blood pressure
52
Arterial Pressure Monitoring with ART LINE?
Direct, continuous monitoring of systolic, diastolic and mean arterial pressure Easy access for arterial blood samples and ABG's Assess blood volume and response to vasoactive meds Most common placement is radial (femoral and brachial also used) Before placement, check Allen's test
53
What is Central Venous Catheter (CVC) and Central Venous Pressure (CVP)
Central Venous Catheter (CVC): Venous access and monitoring central venous pressure Multiple types of CVC access, not all monitor CVP Most common is triple lumen, can read CVP -most common, subclavian, internal jugular, femoral -femoral CVC are not accurate but are useful for trending Purple Power PICCS can be used for CVP monitoring Ports are NEVER used for CVP monitoring ``` Central Venous Pressure (CVP): Pressure in right atrium- ASSESS preload Assesses venous return to the heart- think volume Normal CVP value- 2-6 mmHg (higher in sick people) Increased CVP: -right sided heart failure -volume overload -pulmonary hypertension -cardiac tamponade Decreased CVP: -hypovolemia -shock ```
54
What is Pulmonary Artery Catheter (PAC) and Pulmonary Artery Pressure (PAP)
Pulmonary Artery Catheter (PAC): Placed for detailed assessment for stroke volume and management of shock status High risk- requires meticulous care and monitoring Venous access (right internal jugular most common) -balloon tip "floats" through right ventricle and into pulmonary artery -wedges in small pulmonary artery branch -balloon is then deflated and catheter tip rest there Calculated assessments not directly measured -Cardiac output/index: overall pump function -Systemic Vascular Resistance: left and right, afterload assessment, think constriction (afterload) -Stroke Work Index: left and right, contractility assessment ``` Pulmonary Artery Pressure (PAP): Measured continuously by pulmonary artery catheter Normal PAP 15-30/8-15 Increased PAP -left sided heart failure -increased pulmonary arterial resistance (pulmonary HT, mitral valve disease, hypoxia) Decreased PAP -low circulating blood volume ``` Arterial waveform
55
Pulmonary Capillary Wedge Pressure (PCWP)
PCWP: Reflects left atrial and ventricular pressures Measured when the inflated balloon wedges in the pulmonary vasculature Also called pulmonary artery occlusive pressure and pulmonary artery wedge pressure High risk procedure: Effectively creates a PE Balloon can rupture Can injure the artery Increased PCWP: Left sided heart failure Cardiac tamponade Decreased PCWP: Low circulating blood volume