Cells

Question 0

Increase in number of cells.

Answer 0

Hyperplasia

Question 1

Increase in size of cells resulting in increase of organ?

Answer 1

Hyper trophy

Question 2

Hypertrophy and hyperplasia of uterus during pregnancy?

Answer 2

Both Estrogen stimulated smooth muscle hypertrophy and hyperplasia.

Question 3

Wound healing, hypertrophy or hyperplasia?

Answer 3

Hyperplasia

Question 4

Type of cell death characterized by nuclear dissolution, without complete loss of membrane integrity.

Answer 4

Apoptosis

Question 5

Type of cell death which is energy-dependent, tightly regulated, and associated with normal cellular functions.

Answer 5

Apoptosis

Question 6

Type of cell death which results from a pathologic cell injury.

Answer 6

Necrosis

Question 7

Type of death associated with inflammation.

Answer 7

Necrosis

Question 8

It is the irreversible condensation of chromatin in the nucleus of a cell undergoing necrosis or apoptosis.

Answer 8

Pylons is

Question 9

It is the destructive fragmentation of the nucleus of a dying cell.

Answer 9

Karyorrhexis

Question 10

It is the complete dissolution of the chromatin of a dying cell.

Answer 10

Largo lysis

Question 11

This is the first manifestation of almost all forms of injury to cells.

Answer 11

Cellular swelling

Question 12

Small clear vacuoles within the cytoplasm, representing pinched-off segments of the endoplasmic reticulum.

Answer 12

Hydropic change or Vacuolar degeneration

Question 13

Appearance of lipid vacuoles in the cytoplasm.

Answer 13

Fatty change

Question 14

Surface blebs, increased eosinophilia of the cytoplasm, cellular swelling.

Answer 14

Reversible/Early Ischemic Injury

Question 15

Cell injury with loss of nuclei, cellular fragmentation and leakage of cellular contents.

Answer 15

Irreversible/Necrotic cellular injury

Question 16

These are chemical species with a single unpaired electron in the outer orbital.

Answer 16

Free radicals

Question 17

Most common cause of cell injury in clinical medicine.

Answer 17

Ischemic

Question 18

Composed of membrane-bound vesicles of cytosol and organelles seen in programmed-cell death.

Answer 18

Apoptosis bodies

Question 19

Restoration of blood flow to Ischemic but otherwise viable tissue paradoxically results in exacerbated and accelerated injury.

Answer 19

Ischemic-Reperfusion Injury

Question 20

Pathway of apoptosis triggered by loss of survival signals, DNA damage and accumulation of misfiled proteins. Inhibited by anti-apoptosis members of the Bcl family.

Answer 20

Mitochondrial/Intrinsic Pathway

Question 21

Pathway of apoptosis responsible for elimination of self-reactive lymphocytes and damage by cytotoxic T lymphocytes. Initiated by TNF receptors.

Answer 21

Death Receptors/Extrinsic Pathway