Burn Flashcards

1
Q

What is the mechanism of burn healing?

A

by keratinocyte proliferation and migration

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2
Q

Epitheliazation of superficial burn

A

cell migration from would PERIPHERY

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3
Q

Epithelizalization from PTB

A

cell migration from wound periphery and skin appendages ( sweat glands and hair follicles )

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4
Q

Epithelizalization of FTB

A

Wound contraction and fibroplasia
Excision and closure with grafting
FTB greater than 1cm diameter will not heal without surgery

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5
Q

tx of superficial 1st degree burn?

A

Healing takes 10 days

Emollient and palliation

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6
Q

tx of 2nd degree superficial PTB

A

healing takes 10-21 days
semi-occlusive dressing for moisture control
Palliation ( to reduce the pain without treating the underline cause like taking pain meds )
Topical

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7
Q

tx of 2nd degree deep PTB

A

takes a longer time to heal ( 6wks)
loss of dermal structure and appendage slows healing
debridement and skin grafting appropriate especially with large BSA injury

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8
Q

3rd degree FTB tx

A

had to be debrided
skin grafting
reginal skin plasty
Flaps: sin flap, muscle flap, fasciocutaneous flap, free tissue transfet

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9
Q

4th degree burn tx

A

Excision/ amputation

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10
Q

What are some appropriate graft recipient sites?

A
Will take :
granualtion 
peritenon
periosteum 
muscle
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11
Q

graft recipeient sites that wont take?

A

cartilage
necrotic tissue
nerve
infected sites

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12
Q

Infection prevention

A

Topical antibiotics

Systemic antibiotics prophylaxis

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13
Q

Topical antibiotics

A

silver sulfdiazine
Bactroban
Sulfamylon
Anticoat

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14
Q

Systemic antibiotics prophylaxis

A

not found to improve outcomes
may cause secondary infections
bacterial resistance
study in children demosnrate higher infection rates
Appropriate peri-operatively with wound excision

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15
Q

what group of people have increased risk of infection with burns ?

A

Diabetics

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16
Q

T/F ? Diabetes is associated with poor outcomes in burn pts

A

False!!!! Hyperglycemia is associated with poor outcomes

17
Q

what is osteochisis?

A

fracture of bones from direct splitting of bone as current passes through the bone - do x-ray

18
Q

complications of electrical burns

A
  • compartment syndrome
  • myonecrosis and myoglobulinemia leading to renal failure
  • microvascular thrombosis resulting in prolonged tissue necrosis, progressive gangrene
19
Q

Progressive gangrene and necrosis requires

A
  • staged sequential exploration
  • avoid early, aggressive definitive treatment
  • Best with conservative meticulous debridement than early definitive amputation
  • Amputations should be left open until its clear that deep tissue necrosis is non progressive
20
Q

Partial thickness skin grafting

A

higher success of take than full thickness graft (FTSG)
renewable donor site
greater surface of coverage with fenestration
donor site healing more painful than FTSG
Graft contracture
Pigment and skin texture changes

21
Q

Full thickness skin grafting

A
more cosmetic than PTB
less graft contraction
less painful with donor site healing
-TAKE LESS READILY THAN STSG
-LESS SURFACE AREA COVERAGE 
-DONOR SITE NOT RENEWABLE
22
Q

types of split thickness skin grafting?

A

Thin
Intermediate
Thick

23
Q

Which of the split thickness skin grafting method is the best?

A

Thicker STSG preserve more reticular collagen , vascular plexus, dermal appendeges to create more cosmetic appearance

24
Q

What are some late complications burns

A
malignant degeneration ( Squamous cell carcinoma)
Dystrophic calcification
Chronic pain 
Shoe gear accomodation 
contracture and deformity
25
Q

Late challenges of burns

A

Preventing Contracture

Treating Contracture

26
Q

treating contracture

A

Scar revision
Tendon lengthening
External fixation

27
Q

How to develop a treatment plan?

A

Think in layers :)