Cardiac 3 Flashcards

1
Q

Nitroglycerin (NTG, a nitrate)

A

is a drug of choice for acute anginal attacks. It dilates coronary arteries to increase oxygen to the myocardium, and dilate peripheral vessels so the heart does not have to work so hard to pump blood into them.

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2
Q

calcium channel blockers?

A

required for electrical excitability of cardiac cells and contraction of the myocardium and vascular smooth muscle. this relaxes vascular smooth muscle, which leads to decreased peripheral vascular resistance (after load) and decreased myocardial oxygen demand. {also used to decrease systolic and diastole blood pressures and to slow the heart rate.

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3
Q

what do anti platelets do?

A

inhibit platelet activation, adhesion, or procoagulant activity. (aspirin)

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4
Q

what are side effects and nursing implications of antiplatelets?

A

SE=increased risk of bleeding. NI= enteric coated may be given for daily dosing.

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5
Q

what are statins?

A
first- line drugs to reduce low- density lipoprotein by reducing cholesterol synthesis.        
atorvastatin (lipitor)
fluvastatin(LescolXL)  
lovastatin(Mevacor)
pravastatin(Pravachol)
simvastatin(Zocor)
rosuvastatin(Crestor)
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6
Q

what are side effects and nursing implications of statins?

A

SE: impaired liver functions, rhabdomyolysis (lethal breakdown or skeletal muscle) NI: tell patient to take in the evening when cholesterol synthesis is highest. Teach patient to report any muscle pain. monitor liver function studies.

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7
Q

anti-ischemic agent?

A

antianginal agent used as combination therapy for those not responding to other antianginal.
ranolazine (Ranexa)

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8
Q

what are side effects and nursing implications of anti-ischemic agent?

A

SE:prolongs QT interval on ECG, dizziness, headache, nausea
NI: may not be effective in women

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9
Q

what are beta blockers?

A

decrease pulse, BP, and cardiac output and suppress renin activity, decrease the risk of sudden death.
metoprolol (Lopressor, Troprol XL)
atenolol (Tenormin)

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10
Q

what are side effects and nursing implications of beta blockers?

A

SE: cold extremities, constipation, diarrhea, diaphoresis, dizziness, fatigue, and nausea ,abrupt withdrawal may result in diaphoresis, palpitations, headache and tremors
NI: if pulse less than 60 beats per minute or systolic BP less than 90 mm Hg. notify physician

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11
Q

what are calcium channel blockers?

A

dilate peripheral arteries, decrease myocardial contractility, depress conduction system, and decrease workload of the heart. in variant angina, reduce coronary artery spasms

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12
Q

what are side effects of calcium channel blockers?

A

SE: headache, peripheral edema, flushing, dizziness, atrioventricular blocks, nausea
NI:if pulse less than 60 beats per minute or systolic BP less than 90 mm Hg. notify physician. Administer before meals and at bedtime

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13
Q

what are nitrates?

A

vasodilate to reduce preload and afterload. reduce oxygen consumption of myocardium.

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14
Q

what are side effects and nursing implication of nitrates?

A

SE: hypotension, headache, orthostatic hypotension, dizziness
NI: document onset, type, radiation, location, and duration of the chest pain. Take apical pulse and BP pre- and post administration

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15
Q

what is unstable angina?

A

occurs when patient with worsening CAD and is notes by its changing or unpredictable pattern. rest does not decrease the chest pain of unstable angina. this pain may even occur when the patient is at rest. the eoisodes of of chest pain with unstable angina increase in frequency and severity. placing the patient at risk for myocardial infarction or sudden death. symptoms of angina are usually occur when an artery is narrowed by atleast 60% to 70%

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16
Q

what is a myocardial infarction or a heart attack?

A

the affected myocardial cells in the heart are permanently destroyed, An MI occurs from a partial or a complete blockage of a coronary artery, which decreases the blood supply to the cells of the heart supplied by the blocked artery. the extent of the cardiac damage depends on the location and amount of blockage in the coronary artery.

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17
Q

how is a myocardial infarction identified?

A

it is identified by type. Non-ST-segment elevation myocardial infarction (NSTEMI) is also known as a non-Q–wave MI. An ST-segment elevation myocardial infarction (STEMI) is also known as a Q-wave MI and is the deadliest type because it is usually caused by a complete blockage of the artery.

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18
Q

what is silent ischemia?

A

silent ischemia ocurs without pain and carrys many risks. the older adults and people with hypertension or diabetes are most often notes to have silent ischemia.

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19
Q

what is sudden cardiac death?

A

is a cardiac arrest triggered by lethal ventricular dysrythmias or asystole from the abrupt occlusion of a coronary artery. prompt treatment is required to prevent death.

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20
Q

when does a myocardial infarction happen?

A

it does not happen immediately. Ischemic injury evolves over several hours before complete necrosis and infarction take place. the ischemia proccess affects the subendocardial layer, which is the most sensitive to hypoxia.

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21
Q

what can prolonged ischemia produce?

A

it can produce severe cellular damage and necrosis of cardiac muscle. once necrosis takes place. the contractile function of the muscle is permenantly lost. if treatment is intiated within the first hour of symptoms of the MI, the area of damage can be minimized.

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22
Q

what wall does the left coronary artery feed?

A

feeds the anterior wall of the heart, which also includes most of the left ventricle. An occlusion in this area causes an anterior wall MI. when the left ventricle is affected, there can be severe loss of left ventricular function, leading to severe loss of left ventricular function, leading to severe changes in the hemodynamic status of the patient.

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23
Q

what does the right coronary artery feed?

A

feeds the inferior wall and parts of the atrioventricular node and the sinoatrial node. an occlusion of the RCA leads to an inferior MI abnormalities in impulse formation and conduction. Serious dysrhythmias can occur early in an inferior MI that may be life threatening.

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24
Q

what does the left circumflex coronary artery feed?

A

feeds the lateral wall of the heart and part of the posterior wall of the heart. A lesion in the circumflex leads to a lateral wall infarction of the left ventricle.

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25
Q

what are some signs and symptoms of MI?

A

chest pain is a classic symptom. the pain begins suddenly and continues without relief with rest or administration of NTG. the pain in the center of the chest is usually described as crushing, viselike, or as if an elephant is standing on the chest. the pain may radiate to the back, one or both arms and shoulders, neck, or jaw. the pain can imitate indigestion and a gallbladder attack with abdominal pain and vomiting. [shortness of breath, dizziness, nuas

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26
Q

what does time have to do with an MI?

A

“time is muscle” as time passes during the MI, more muscle is lost.

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27
Q

what is the leading cause of women death in the USA?

A

heart disease

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28
Q

what are some indicators of an MI?

A

patient history, ECG, and serum cardiac troponin I or T, myoglobin, and CK-MB levels. C-reactive protein is elevated in the presence of inflammation. Magnesium levels are also checked, especially for those in diuretic therapy. The ECg usually shows the area that has infarcted, as well as the ischemia areas of the heart.

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29
Q

what are some recommendations of onset chest pain?

A

the american heart association recommends chewing one uncoated adult aspirin at the onset of chest pain. delays i seeking care can limit treatment options and result in more cardiac damage.

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30
Q

how are patients repotting chest pain treated like?

A

they are treated as if they are having an MI until proven otherwise through testing.

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31
Q

when is oxygen administered?

A

it is administered immediately, usually at 2L/min via nasal cannula. oxygen therapy may be limited to the first 6 hours in stable patients. too much oxygen can lead to a systemic vasoconstriction,which may increase myocardial workload. Arterial blood gases (ABG’s)are drawn to determine the patients oxygen needs. oxygen saturation should be monitored and kept above 95%.

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32
Q

What is a percutaneous coronary interventions?

A

(PCI) is a variety of mechanical procedures used to increase blood flow and oxygen to the myocardium. Emergency PCI is used frequently in management of acute myocardial infarction with improved outcomes.

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33
Q

what is balloon angioplasty ?

A

is a catheterization laboratory, a catheter with a balloon tip is interested, usually via the femoral artery, and advanced into the heart to open the blocked coronary artery. once the blocked artery is entered, the balloon in the catheter is inflated and the atherosclerotic plaque is compressed. the dilated vessel is able to deliver more oxygen- rich blood to the myocardium. angioplasty can be done with or without the placement of stents.

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34
Q

what is a coronary artery stent?

A

a coronary artery stent is placed during angioplasty, is used to prevent closure of a coronary artery from an athersclerotic lesion. a stent is an expandable metal mesh tube that is implanted at the site of blockage in the coronary artery. a stent provides support to a coronary artery wall at the area of stenosis to keep blood flowing through the artery.

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35
Q

what are some complication associated with stent placements?

A

include thrombosis (formation of a blood clot inside a blood vessel), bleeding from anticoagulation, stent occlusion, or coronary artery dissection.

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36
Q

what is a drug eluting stent, and what is it coated with?

A

coated with immunosuppressants medication that can be released at the implantation site to reduce the risk of restenosis.

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37
Q

for those with unstable angina taking tha “fab four “ has shown beneficial to health care, what is the fab four?

A

fab four includes anitplatelets, statins, ACEI’s, and beta blockers.

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38
Q

what kind of effect is occurs when the “fab four” are given?

A

a synergistic effect in fighting plaque. which means that they have greater positive result.

39
Q

What is MONA a mnemonic for?

A

guidelines for remembering the medications to give in treating a suspected MI; Morphine,Oxygen, Nitroglycerin, and aspirin.

40
Q

what are some medications used to treat myocardial infarction?

A

analgesics, angiotensin- converting enzyme inhibitor, anticoagulants, antidysrthmics, antiplatelets.

41
Q

why are analgesics given for an MI?

A

given for relief of chest pain. morphine is the most common, patient should be monitored for hypotension, respiratory depression, over sedation, morphine sensitivity,. in addition to pain relief morphine also helps with reducing anxiety, opens bronchioles, and increases peripheral bloos pooling to decrease preload.

42
Q

what are vasodilators used for in an MI?

A

NTG can be administered for vasodilation to supply more blood to the myocardium to reduce pain and the workload of the heart.

43
Q

when would you not administer a nitrates in and MI?

A

if systolic pressure of less than 90 mm Hg, or 30 mm Hg or more below the baseline. severe bradycardia less than 50 beats /mn, or if pt has taken a phosphodiesterase inhibitor. Catastrophic hypotension may occur.

44
Q

what is thrombolytic therapy used for in MI?

A

used to dissolve a blood clot that is occluding a coronary artery. this therapy needs to be started within a specific time range from the onset of symptoms,usually 1 to 6 hours.

45
Q

what kind of activity can pt do after MI?

A

first patients are kept on bed rest to decrease myocardial oxygen demand. a bedside commode for bowel movements is usually ordered to reduce straining. then activity is advanced gradually.

46
Q

what is an intra- aortic ballon pump?

A

to support the ischemic heart, an intra-aortic balloon pump may be used to increase circulation to the coronary arteries and reduce the work of the heart.

47
Q

why is diet and weight loss important after and acute phase of MI?

A

during an acute phase of MI, small easily digest meals are served. Caffeine is restricted because it increases heart rate and causes vasoconstriction. fluids may be restricted if pt is in heart failure as well. initially a low- sodium and liquid diet is started. then, low- fat, low- cholesterol, and low - sodium diet may be ordered. if pt is obese, weight loss can reduce cardiac output.

48
Q

what is a bypass surgery?

A

when a blood vessel from the leg or chest is used to reroute blood around a segment of a coronary artery that is narrowed by atherosclerosis.

49
Q

what is minimally invasive direct visualization coronary artery bypass? (MIDCAB)

A

techniques done without a cardiopulmonary bypass. several small incisions are used to access to the coronary artery instead of a sternotomy. this technique uses a thorascope. (one or two- vessel disease pt’s may use this technique. )

50
Q

what a coronary artery bypass ? (CPB)

A

combines peripheral cardiopulmonary bypass with minimally invasive heart access. the advantage is that the heart is not beating during surgery. several vessel repairs can be done and other areas of the heart, such as valves, can also be repaired with this technique.

51
Q

what helps identify life threatening dysrhythmias and determine the degree of cardiac damage?

A

continous cardiac monitoring, serial ECG, and laboratory values.

52
Q

when does a cardiac rehabilitation and exercise start ?

A

begins when the patients acute symptoms are relieved. first phase of rehab occurs in the hospital. activities for each hospital day , such as types and amounts of self- care and activity, are specified in protocols.

53
Q

what is peripheral vascular disease? (PVD)

A

may either be arterial or venous in origin. PVD is very common in people who are older or diabetic. it is important for the nurse to understand whether the origin is arterial or venous to prevent serious complications from occurring.

54
Q

what are some causes of arterial thrombosis?

A

occurs to where there is injury to the arteral awl, sluggish flow, or plaque formation secondary to athersclerotic changes. other causes polycythemia, dehydration, and repeated needlesticks.

55
Q

what are some causes of an arterial embolism?

A

dysrhythmias, prosthetic heat valves, and rheumatic heart disease.

56
Q

what are the 6 clinical signs of acute arterial occlusion?

A

the sic P’s ( pain, pallor, pulselessness, paresthesia, paralysis, and poikilothermia.

57
Q

what is done to treat the effected limb (embolism)?

A

anticoagulants are administered immediately, IV heparin to prevent further clotting, warfarin is added. the heparin is continued until therapeutic warfarin level is reached.

58
Q

what dissolves a thrombus or embolus?

A

thrombolytic agent

59
Q

what is peripheral artery disease?

A

disorder of the arteries usually caused by a chronic, progressive narrowing of arterial vessels that leads to an obstruction or occlusion. Usually occurs more often in lower extremities.

60
Q

what is the purpose of the arterial system?

A

to deliver oxygen- rich blood to the vascular beds. anything that impedes this flow causes an imbalance of supply and demand for oxygen.

61
Q

what intermitted claudication?

A

pain in the calves of the lower extremities associated with activity or exercise.

62
Q

what is a percutaneous transluminal angioplasty?

A

used to dilate a narrow peripheral vessel, although it does not provide longterm results, is is similar to PCI.

63
Q

what is peripheral atherectomy?

A

another invasive procedure used to remove plaque from the atherosclerotic arteries.

64
Q

what is raynauds disease?

A

a vasoconstriction response causing ischemia from exposure to cold and stress.

65
Q

what are some therapeutic measure for raynauds disease?

A

keep hands warm, gloves should be worn when going outside, cleaning a refrigerator, or preparing cold foods,. Avoiding caffeine, alcohol. and smoking.

66
Q

what is thromboangiitis Obliterans? (burger’s disease)

A

recurring inflammation and thrombosis of small and medium arteries and veins of the hands and feet. cause is unknown but thought to be autoimmune.

67
Q

what are some signs and symptoms of Buergers disease?

A

intermittent claudication, numbness on extremities, or decreased sensation, cool extremities, lower extremities may be red or cyanotic when in dependent position, and pulse may be diminished.

68
Q

what is an aneurysm?

A

bulging, ballooning, or dilation at a weakened point of the artery.

69
Q

when is an aortic aneurysm silent?

A

when less than 4 cm. incidence increases with age.

70
Q

what are the types of aneurysm?

A

Fusiform: the entire circumference of the artery is dilated.
Saccular: one side of the artery is dilated
Dissecting: a tear in the inner layer causes of cavity to form between the layers of the artery and fill with blood.

71
Q

aneurysm symptoms?

A

back pain, flank pain, abdominal fullness, nausea, pulsating mass in abdomen, severe sudden back pain with rupture, shock from blood loss.

72
Q

what are diagnostic tests of aneurysm?

A

ultrasound, chest x-ray, MRI, CT scan

73
Q

what are varicose veins?

A

elongated, tortuous, dilated veins. the exact cause is unknown. the condition tends to be familial, varicose veins are divided into primary and secondary varicosities.

74
Q

what are the lead factors that may lead to varicose veins?

A

wall defects have been identified as a familiar tendency and may be inherited, any factor that may contribute to increasing hydrostatic pressure within the legs, such as prolonged standing, pregnancy, obesity, may promote venous dilation.

75
Q

what are therapeutic measures for varicose veins?

A

primary goals is to improve circulation, relieve pain, and avoid complications. treatment is not usually indicated if the problem is only cosmetic. elastic compression socks should be used as ordered. injection sclerotherapy or lasers treat superficial varicosities. minimally invasive ablation procedures include radio - frequency ablation ( closure procedure) or endogenous laser therapy.

76
Q

what are signs and symptoms of varicose veins?

A

disfigurements of lower extremities with primary varicosities, may be dull pain, especially after prolong standing. this can be relieved by walking or elevating the extremity. with secondary varicosities, the pain and disfigurements may be more severe. edema or ulceration can develop if circulation is severely compromised.

77
Q

what is venous insufficiency?

A

a chronic condition, damaged or aging valves within the veins interfere with blood return to the heart, causing pooling of blood in the lower extremities.

78
Q

what can Chronic venous insufficient lead to?

A

can lead to venous stasis ulcers.

79
Q

what are venous stasis ulcers?

A

are the end result of chronic venous insufficiency. dysfunctional valves in the venous system prevent or reduce venous blood return. (develop from increased pressure and rupture of small veins)

80
Q

what are therapeutic measures for venous stasis ulcers?

A

decrease edea, heal skin ulcerations, compression socks are necessary to decrease edema. bed rest and elevation of legs above the heart.

81
Q

what is vascular surgery?

A

vascular impairments requiring surgery may be acute or chronic and involve arteries, veins, or lymphatic vessels. when limb is at risk for amputation, then surgical vascular grafting may be done.

82
Q

An endarectomy is?

A

arteriosclerotic plaques are dissected from the lining of the arterial awl and removed.

83
Q

Angioplasty is?

A

minimally invasive techniques may be used to open plaque - blocked arteries. these techniques include ballon or laser angioplasty. A flexible laser tipped catheter is inserted into an artery and advanced at site of the blockage.

84
Q

where are stents placed?

A

inside an artery to provide support to the artery walls and keep them open

85
Q

lymphangitis is what?

A

bacterial infection of the lymphatic channels. it can occur in the arms or legs and commonly caused by staphylococcus or streptococcus bacteria. serious infection that can causes sepsis and be fatal.

86
Q

what does the lymphatic system do?

A

return fluid from the tissues in the body to the bloodstream. it is a pump less system with oneway valves that return the fluid to the heart.

87
Q

what results if any interruption in the flow of lymphs?

A

results in edema

88
Q

signs and symptoms of lymphangitis?

A

painful red streaks in the extremity. fever and chills may be present. lymph nodes in the area of infection can be enlarged or painful.

89
Q

what is a classical symptom of peripheral arterial occlusive disease?

A

intermittent claudication.

90
Q

what is the purpose of CABG surgery?

A

increase blood flow to the myocardium

91
Q

what are atypical symptoms of an MI?

A

absence of chest pain, fatigue, cramping, anxiety, falling, feeling of impeding doom

92
Q

symptoms for women with MI?

A

epigastri or abdominal pain, chest discomfort, pressure, burning, arm, should, neck, jaw , or back pain. discomfort pain between shoulder blades, SOB, fatigue, indigestion or gas pain, nausea vomiting.

93
Q

to whom does a silent MI usually occur to?

A

most often occur to older adults or those with diabetes regardless of age.

94
Q

treatment should be sought after how long of unrelieved pain?

A

5 minutes