38. Embolism Flashcards

(54 cards)

1
Q

VAE requirements

A

open vein
pressure in vein > subatmospheric pressure

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2
Q

VAE surgical position

A

can occur in any position

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3
Q

higher incident rate of VAE

A

sitting craniotomies

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4
Q

VAE presentation is dependent on

A

volume entrained
rate of air entrainment
presence of R-to-L shunt

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5
Q

as rate of air entrainment increases, what happens?

A

incr PAP
decr CO
RV overload
decr LV preload

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6
Q

nitrous and VAE

A

rapidly increases severity of VAE

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7
Q

VAE definitive diagnosis

A

Echo

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8
Q

VAE signs/symptoms

A

decr EtCO2 (stair step)
decr O2
tachycardia
hypotension

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9
Q

monitors to detect VAE

A

TEE
precordial doppler over RA

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10
Q

precordial doppler placement for VAE

A

over RA
between 3rd and 6th ribs

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11
Q

VAE treatment

A

prevent further entrainment
stop N2O
100% FiO2
aspirate air bubbles from CVC
give crystalloid to incr CVP
give vasopressor to correct htn
incr PEEP
head down/left tilt (durant’s)

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12
Q

polymethymethacrylate

A

exothermic
expansion
intramedullary pressures > 500mmHg

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13
Q

bone cement can cause embolism of

A

fat
bone marrow
cement
air

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14
Q

bone cement signs/symptoms

A

hypoxia
hypotension
arrythmias
PHTN
decr CO

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15
Q

bone cement embolism treatment

A

incr FiO2
euvolemia
adequate BP

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16
Q

rhabdo is associated with tourniquet inflation over _____ hrs

A

> 2hrs

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17
Q

pt experiences tourniquet pain after

A

60 mins

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18
Q

inflate tourniquet to _____

A

100mmHg above SBP

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19
Q

when are fat embolisms common

A

with long bone fractures

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20
Q

fat embolism signs/symptoms

A

dyspnea
confusion/coma
petechia
clotting dysregulation
hypoxia
resp failure
ST changes
decr EtCO2

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21
Q

signs/symptoms of RV strain

A

acute incr PHTN
pulm vasodilators
milrinone

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22
Q

fat embolism treatment

A

stabilization of frature
O2 therapy
hypotension correction

23
Q

ARDS

A

disruption of cap-alveolar membrane
albumin leak into alveolar space resulting in fluid in the lungs causing inflammation

24
Q

Acute lung injury

A

PaO2/Fio2 ration < 300

25
ALI causes
trauma sepsis aspiration
26
ARDS
PaO2/FiO2 ratio < 200 more severe than ALI
27
ARDS treatment
fix the cause
28
ARDS mgmt: Plat P
< 27 cmH2O
29
ARDS: TV
4-6 mL/kg
30
ARDS: FiO2
< 50%
31
ARDS: RR
< 30 breaths per min
32
ARDS: PEEP
<15 cmH2O
33
ARDS: drugs
NO prostacyclin PGE1
34
DVT
blood clot in vein that becomes thrombotic more common in lower extremities
35
DVT risk factors
obesity Age > 60 sx > 30 mins tourniquet use LE fracture immobilization > 4 days blood clotting disorders (Factor 5)
36
LE orthopedic risk of DVT
40-80% without prophylaxis
37
DVT pharmacological prophylaxis
LUFH warfarin LMWH (lovenox 40 mg)
38
DVT mechanical prophylaxis
IPC SCD compression stockings
39
what might lower risk of DVT
neuraxial anesthesia
40
DVT treatment
anticoags thrombotics IVC filter thrombectomy/embolectomy
41
AFE incidence rate
1:20,000
42
AFE mortality
86%
43
AF composition
fetal debri prostaglandins leukotrines
44
____ indicates severity of AFE
volume of entrainment
45
AFE is a _____ response
immune
46
AFE signs/symptoms
tachycardia cyanosis shock generalized bleeding
47
AFE phase 1
pulmonary vasospasm hypoxia resp acidosis
48
AFE phase 2
LV failure pulm edema uterine atony DIC
49
AFE phase 3
seizure coma
50
AFE treatment: pulm embolism
aspirate max PaO2 CPR
51
AFE treatment: hemodynamics
aggressive fluid resuscitation direct vasopressor (phenylephrine) milrinone after adequate resuscitation
52
AFE treatment: uterine atony
oxytocin methergine prostaglandin hemabate misoprostol
53
AFE treatment: DIC
transfusion
54
AFE treatment: seizures
benzos