4 Etiologies of CVA/Stroke Flashcards

1
Q

What are the 4 types?

A

Ischemic - thrombotic
Ischemic - embolic

Hemorrhage - intracerebral
Hemorrhage - subarachnoid

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2
Q

Key question/observation for
Hemorrhage - intracerebral

A

Upon straining

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3
Q

Key question/observation for
Hemorrhage - subarachnoid

A

THUNDER CLAP headache

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4
Q

Ischemic Thrombotic CVA/Stroke

What is atherosclerosis?

What are the types?

Complications of the late stage?

A

“Athero” meaning soft, fatty, glue-like

“atheroma” meaning a fomration of “athero” within vessel that results in luminal narrowing

Thick - Hard - Less Elastic

Present with low distal perfusion, may develop aneurysms, prone to rupture with hypertension

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5
Q

Ischemic Thrombotic CVA/Stroke

What are the vessels at risk?

A

Internal carotid (carotid endarterectomy)

Upper vertebral (angioplasty)

Lower basilar (Rx & prevention)

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6
Q

Ischemic Thrombotic CVA/Stroke

What are the risks?

8

A

High cholesterol, triglycerides, lipids

Hypertension

Stress increases cortisol (lipids) & catecholamines

Hereditary

Sedentary lifestyle (lack of exercise)

Birth control pill, smoking & weight gain

Cardiac disease, dysrythmia

Diabetes Mellitus

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7
Q

Ischemic Thrombotic CVA/Stroke

What’s the prevention?

EMS care/Acute management?

A

Antiplatelet agents to prevent atherothrombosis

ASA & ticlopine are used most often

Carotid endarterectomy for internal carotid TIA

Angioplasty

Position the patient on their affected (body) side with the head elevated @ 30 degrees

High concentration oxygen & assisted ventilation with support of spontaneous breathing & augmentation of rate to no greater than 20 min.

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8
Q

Ischemic Embolic CVA/Stroke

What are the causes/risks for HYPERTENSION & ATHERTHROMBOTIC PLAQUE?

What are the causes/risks for IMPAIRED BLOOD FLOW LEADING TO STASIS?

What are the causes/risks for DAMAGED VALVE LEADING TO CLOT/FIBRIN EMBOLI?

A

It is an Embolus from atheromatous plaque in the carotid sinus or internal carotid artery.

Diabetes, hyperlipidemia, smoking, b/c pill, prior, overweight or + weight gain, sedentary lifestyle

CSM or Chiropractic neck manipulation.

Hyper viscosity syndromes & Glue sniffing

Recent AMI with dyskinetic left ventrical

Atrial Fibrillation

Rheumatic Heart disease

Prosthetic valves

Bacterial Endocarditis

Mitral Valve Prolapse

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9
Q

Ischemic Embolic CVA/Stroke

What area of the brain is USUALLY affected (the vessel)?

The # of emboli determines?

A

The left middle cerebral artery, especially the upper division, is the most frequent site of cerebral emboli b/c it is straighter which means less resistance to emboli

the extent of damage

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10
Q

Ischemic Embolic CVA/Stroke

What’s the prevention?

What’s the EMS care/Acute management for an ischemic embolic stroke?

A

Decrease other risk factors which cause hypertension & atherothrombotic plaque.

Avoid CSM & neck manipulation

Antiplatelet agents – warfarin more effective than ASA for preventing ischemic stroke
associated with AF

Thrombolytic therapy for acute care

Position the patient on their affected (body) side with the head elevated @ 30 degrees

High concentration oxygen & assisted ventilation with support of spontaneous breathing & augmentation of rate to no greater than 20 min

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11
Q

Intracerebral hemorrhage (ICH) CVA/Stroke

What does it mean?

How does it happen?

A

intraparenchymal means deep tissue (there’s a hematoma deep in the brain tissue)

  1. Bleeding occurs in the space surrounding the tissue.
  2. Distal brain cells become hypoxic immediately resulting in a focal presentation. (e.g., weakness in one hand).
  3. Blood is particularly irritating to local arteries which spasm in response to contact.
  4. The spasm may involve quite a large area resulting in an ever expanding / evolving presentation.
  5. Clot will form & brain tissue in the area becomes liquid.
  6. Clot eventually is removed by phagocytosis & replaced with blood vessels, astroglia fibers & fibrin to fill void.
    *Brain tissue does not replace dead cells.
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12
Q

Intracerebral hemorrhage (ICH) CVA/Stroke

What’s the symptom onset?

What are the complications?

What are the causes?

A

Slower to get worse
Initially focal

necrotic tissue & the edema causes herniation through the tentorium.

missile injury

tumors which bleed

hypertensive crisis (50% of cases)

arteriosclerosis

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13
Q

Intracerebral hemorrhage (ICH) CVA/Stroke

Where is it usually located?

Who does it affect?

A

anterior portion of the temporal lobes & the posterior portion of the frontal lobes

older men (68)

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14
Q

Intracerebral hemorrhage (ICH) CVA/Stroke

What’s the prevention?

What’s the EMS care/Acute management?

A

Stop the use of alcohol, tobacco, & use of cocaine & amphetamines

Mannitol & other osmotic agents reduce ICP caused by edema

Position the patient on their affected (body) side with the head elevated @ 30 degrees

High concentration oxygen & assisted ventilation with support of spontaneous breathing & augmentation of rate to no greater than 20 min

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15
Q

Intracerebral hemorrhage (ICH) CVA/Stroke

What’s the prognosis for a supratentorial hematoma GREATER than 5cm?

What about an infratentorial (pontine) hematoma GREATER than 3cm?

A

Poor

FATAL!!

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16
Q

Subarachnoid hemorrhage (SAH) CVA/Stroke

What’s the cause?

Who’s the target demographic?

What is the usual location, size, and apearance?

A

A rupture of blood vessels which merge with the sub-arachnoid space (the saccular aneurysms – circle of willis)
AND/OR
A laceration of arachnoid vessels caused by missiles or blunt trauma to brain (contusions).

45% younger than 55 y/o

Can affect children

the elderly & alcoholics

located at bifurcations

Small - pinpoint 2-3cm in diameter

Think walled blisters that gradually enlarge

17
Q

Subarachnoid hemorrhage (SAH) CVA/Stroke

What’s the symptom onset?

How long can it last?

What are the symptoms of meningeal irritation?

A

abrupt with the “worst headache” or “something snapped” CC

nuchal rigidity

sensorium deterioration

coma

convulsions

min-hours-days

Nuchal rigidity

Severe acute onset & intense headache

Photophobia

Projectile vomiting

Trauma may or may not be evident

18
Q

Subarachnoid hemorrhage (SAH) CVA/Stroke

What are the causes?

What are the complications?

A

Provoked by hypertension

Exacerbated by anticoagulation therapy or bleeding disorders

Occurs during periods of:
- stress
- exertion
- valsalva maneuvers
- coitus
- labour & delivery
- weight lifting - WITH or without the use of anaboic steroids

19
Q

Subarachnoid hemorrhage (SAH) CVA/Stroke

What are the complications?

What’s the prevention?

A

hemorrhage extending into the ventricle system

temporal lobe herniation

midbrain compression

Major therapeutic emphasis is on preventing predictable complications of re-rupture

20
Q

Subarachnoid hemorrhage (SAH) CVA/Stroke

What’s the EMS care/Acute management plan?

A

Re-bleeding management is central to the management of ruptured aneurysms.

Quiet dark room, given stool softener, analgesia

Position the patient on their affected (body) side with head elevated @ 30 degrees

High concentration oxygen & assisted ventilation with support of spontaneous breathing & augmentation of rate to no greater than 20 min.

21
Q

How many cc’s of blood can the brain tolerate WITHOUT clinical manifestations (symptoms)?

What’s considered lethal in the brainstem?

A

100cc

5ml clot