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Physio > oral physiology > Flashcards

oral physiology Flashcards

1
Q

genioglossus muscle

A

extrinsic tongue muscle. very fast with high amount of fast twitch fibers

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2
Q

what are the jaw opener muscles

A
  1. digastric2. lateral pterygoid (technically a PROTRACTOR)3. geniohyoid4. mylohyoid
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3
Q

digastric muscle is composed of mostly what type of fibers

A

fast

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4
Q

dysfunction of the superior head of the lateral pterygoid is associated with what

A

anterior displacement of the TM disc in TMJ dysfunction

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5
Q

there is a ___ amount of force generation needed for the jaw opening muscles

A

low amount of force generation….more important for movement instead of force production

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6
Q

what are the jaw closer muscles

A
  1. masseter2. temporalis3. medial pterygoid
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7
Q

the masseter muscle is composed of mostly what type of fibers

A

slow fibers (in both the deep and superficial parts)

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8
Q

what myosin isoforms predominate in the adult for the masseter muscle

A

slow fibers

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9
Q

what myosin isoforms predominate in the neonatal/embryonic masseter muscle

A

alpha cardiac

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10
Q

bruxing leads to ____ of the muscle

A

hypertrophy (gets larger)

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11
Q

bruxing hypertrophy leads to an increase in what

A

increase in the level of slow myosin…which could create less powerful contractions of the masseter which could affect chewing/speech/etc

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12
Q

the temporalis muscle is composed of mostly what type of fibers

A

faster type myosin than the masseter

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13
Q

the medial pterygoid muscle is composed of mostly what type of fibers

A

slower myosin

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14
Q

what is the pattern among jaw closer muscles (masseter, med pterygoid, temporalis)

A

compartmentalization….where there is greater amount of slow myosin fibers in the anterior and deep layers and faster myosin fibers in the posterior and superficial

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15
Q

what is kinesthesia

A

sense of movement and position

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16
Q

what provides feedback from the muscles to the CNS

A

rich sensory information is provided from the masticatory muscles to the CNS via sensory afferents. feedback can then be delivered from the CNS back to the masticatory muscles (to better control force generation and precision).

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17
Q

what are the two types of muscle fibers that make up a muscle spindle

A
  1. nuclear bag intrafusal fibers (clustered nuclei in the center of the fiber)2. nuclear chain intrafusal fibers (nuclei arranged in a chain/linearly within a fiber)
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18
Q

intrafusal refers to what

A

fibers that are WITHIN a muscle spindle rather than extrafusual fibers which are the muscle fibers that generate the “work” of a muscle during contraction (and are outside of the muscle spindle)

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19
Q

what are the 2 types of efferent nerve fibers associated with a muscle spindle

A

gamma and beta (rare)

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20
Q

what is the job of the efferent nerve fibers associated with a muscle spindle

A

to innervate the intrafusal fibers of the spindle

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21
Q

what are the 2 types of sensory/afferent nerve fibers associated with a muscle spindle

A
  1. 1a afferent/primary ending2. II afferent/secondary endings
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22
Q

what is the job of the sensory/afferent nerve fibers of the muscle spindle

A

they adjust the incoming signal to the muscles by providing info to the CNS about what is going on w/in the muscle. the CNS needs to know what to tell the extrafusal fibers to do.

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23
Q

what is the difference b/w Ia and IIa afferent fibers

A

-Ia provide MORE DYNAMIC signals (report to CNS earlier and give a more “robust” signal) to the CNS, but their signal DISSIPATES more during the length of a muscle stretch -IIa have a delayed response to the stimuli applied, but provide the signal more CONTINUOUSLY throughout the muscle stretch

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24
Q

what is the role of gamma efferent fibers within a muscle spindle

A

to maintain a high level of spindle sensitivity in SHORTENED muscles. when a muscle shortens, the gamma efferents signal to the intrafusal fibers (that they innervate) to shorten in their POLAR regions (outside of the muscle fiber). shortening of the polar region of intrafusal fiber causes lengthening of the equatorial (central) region of the muscle spindle. lengthening of the equatorial region of the spindle RESTORES THE SENSITIVITY of the spindle to future stretch stimuli

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25
Q

what would happen if the spindle didn’t “reset” its sensitivity after muscle shortening?

A

bc the spindle could not restore its sensitivity, the muscle (extrafusal fibers) would keep working over a range of short lengths where the spindles would remain inactive and not be able to send modulating info to the CNS (….assuming this could result in injury of some kind?)

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26
Q

what are golgi tendon organs

A

receptors located in jn b/w the ends of muscle fibers and tendon (located w/in the tendon in series with muscle fibers). they generate signals that are proportional to the amount of force generated by the extrafusal (working) muscle fibers

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27
Q

how does a GTO respond to increasing forces

A

the more force applied/done by the extrafusal fibers, the more tension applied to the GTO and theremore the more a.p produced by the GTO

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28
Q

what are free nerve endings in joints activated by….what doe they respond to

A

mostly activated by PAIN, but some activated by mechanical stimuli (to sense joint position)

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29
Q

what is an EMG

A

non invasive gathering of info that records and analyzes muscle activation patterns. they don’t reflect forces generated across a joint (bc antagonist muscles can create a net 0 force).

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30
Q

how can an EMG be clinically relevant?

A

see what muscles are activated or no longer activated after unilateral damage/surgery has been perfomed (i.e a unilater marginal madiblectomy…aka mandible removed on one side)

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31
Q

depending of food consistency, mandibular movements during mastication are both ____ and _____

A

highly rhythmic and specific (controlled by the CNS!)

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32
Q

what are the 4 phases of CHEWING

A
  1. slow opening2. fast opening3. fast closing4. slow closing (to protect teeth/soft tissues)**this is highly CONSISTENT across species (even if they have different feeding behaviors)
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33
Q

the type of ____ differs b/w species with different feeding behaviors

A

myosin!carnivores–> masticatory herbivores–> alpha cardiac (rhythmic) omnivores–> unspecialized

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34
Q

what are the 3 phases of MASTICATION

A
  1. preparatory2. reduction3. pre-swallowing**variable activities of the openers and closers in ALL phases
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35
Q

characteristics of the preparatory phase

A

NAME?

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36
Q

characteristics of the reduction phase

A

NAME?

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37
Q

characteristics of the pre-swallowing phase

A

NAME?

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38
Q

what are the primary sites of control for mastication

A
  1. brainstem2. cerebral cortex3. and a little bit of the cerebellar cortex**these both include: nuclei, afferent fibers, and efferent fibers
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39
Q

the sensory nuclei that control mastication include

A
  1. trigeminal sensory nucleus2. trigeminal mesencephalic nucleus
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40
Q

what is the role of the trigeminal sensory nucleus

A

fibers extending from here INNERVATE the face and oral cavity (sensory) and then PROJECT to the cerebellar and cerebral cortexs

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41
Q

what is the role of the trigeminal mesencephalic nucleus

A

fibers extending from here are coming from the:1. spindle afferents of jaw closers2. mechanoreceptors in the PDL/ gingiva/palate

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42
Q

the motor nuclei involved in the control of mastication include

A
  1. trigeminal motor nucleus2. hypoglossal motor nucleus3. facial motor nucleusCN V, XII, and VII
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43
Q

what is the role of the trigeminal motor nucleus

A

includes alpha and gamma MOTOR fibers that innervate the jaw muscles. TOPOGRAPHIC organization

44
Q

what is the role of the hypoglossal motor nucleus

A

includes the MOTOR fibers that innervate the tongue muscles

45
Q

what is the role of the facial motor nucleus

A

includes the MOTOR fibers that innervate the facial muscles. TOPOGRAPHIC organization

46
Q

the motor nuclei and sensory nuclei that control mastication are located where

A

brainstem

47
Q

what is the role of the brainstem in mastication

A

fn autonomously. may contain a pattern generator. doesn’t require (although normally does) input from higher centers.

48
Q

describe the jaw closing reflex

A
  1. afferent fibers from muscle spindles send a signal to…2. trigeminal mesencephalic sensory nucleus which sends fibers to the…. 3. trigeminal motor nucleus to synapse here on…4. alpha motor neurons which innervate…5. extrafusal fibers of the jaw muscles (to close the mouth!)
49
Q

the jaw closing reflex is

A

monosynaptic…making it very fast (w/ very little modulation)

50
Q

describe the jaw opening reflex

A
  1. a stimulus in the oral cavity activates afferents that send a signal to….2. spinal trigemnal tract nucleus which sends fibers to the…3. interneurons that are located within the CNS which then send fibers to the…4. trigeminal motor nucleus to synapse here on…5. alpha motor neurons which innervate6. extrafusal fibers of the jaw muscles (to open the mouth)
51
Q

the jaw opening reflex is

A

polysynaptic…bc it is highly modulated for specific stimuli (depending on the stimuli size/consistency/etc)…causing it to take longer to execute

52
Q

what is the role of “higher centers” in mastication

A

modulation of mastication

53
Q

what is the role of afferents in mastication

A

modulation of mastication (depending on stimuli/food consistency). variable receptors involved with the input/food stimuli type. afferents give input to brainstem and higher centers (cortex)

54
Q

what are the phases of swallowing (4)

A
  1. prepartory2. oral 3. pharyngeal 4. esophageal
55
Q

preparatory phase of swallowing is the same as the…

A

pre-swallowing phase of mastication

56
Q

the prep phase of swallowing is a fn of the

A

glossopalatal sphincter

57
Q

what is the glossopalatal sphincter and what is it composed of

A

a temporary sphincter composed of the posterior tongue and the soft palate (when they contact). it prevents food bolus from entering the pharynx.

58
Q

describe the prep phase of swallowing

A
  1. bolus formed and resting on tongue2. tip of tongue presses against max incision/ant hard palate3. region of tongue where food positioned rises laterally against pos teeth and palate4. palatoglossal sphincter forms
59
Q

what is the oral phase of swallowing

A

movement of the bolus from the oral cavity to the pharynx….but food still in the oral cavity

60
Q

describe the oral phase of swallowing

A
  1. formation of oral seal (as incisors and lips move close together)2. bolus is pushed toward the pharynx (ant tongue touches max alveolar ridge and ant hard palate)**3-5 occur TOGETHER!3. expansion of the hypopharynx to open “chute” to pharynx (tongue base moves down and forward)4. glossopalatal sphincter opens (palate moves up)5. food is prevented from going into nasal cavity (side walls of nasopharynx are close as palate contacts the pos pharyngeal wall)
61
Q

what are the muscles involved in the prep and oral phases of swallowing

A
  1. jaw closers2. facial muscles (formation of oral seal and stabilizes mandible)** FACULTATIVE GROUP of muscles (variable bc depends on food consistency)
62
Q

what is the pharyngeal phase of swallowing

A

movement of bolus from oropharynx to esophagus

63
Q

describe the pharyngeal phase of swallowing

A
  1. food is propelled by the posterior tongue from the oropharynx to the hypopharynx 2. food moves through pharynx by contraction of pharyngeal constrictors3. bolus enters the esophagus as upper esophageal sphincter relaxes4. opening to larynx closes as the epiglottis moves to a horizontal position
64
Q

is the epiglottis required to prevent aspiration

A

no

65
Q

what components allow for the movement of the epiglottis

A

hyoid bone (up), larynx (up), thryohyoid (contraction)

66
Q

what muscles are involved in the pharyngeal phase of swallowing

A

OBLIGATE group of muscles ….sequential in order of contraction

67
Q

what is the esophageal phase of swallowing

A

movement of food along entire esophagus

68
Q

describe the esophageal phase of swallowing

A
  1. peristaltic waves of contraction to move bolus2. lower esophageal sphincter opens3. food enters stomach
69
Q

the esophageal sphincters are ____ active

A

tonically active! they are are “inactivated” by BASAL ACTIVITY normally…but the basal activity stops before food enters the esophagus

70
Q

describe the contraction/relaxation of the upper esophageal sphincter

A

the upper is relaxed/opened before the pressure waves pass through the pharynx (to allow for food to pass through). then the upper contracts/closes after the bolus enters the esophagus. here, its pressure rises well above resting for a few seconds (to force food down)

71
Q

describe the contraction/relaxation of the lower esophageal sphincter

A

the lower is relaxed/open before the peristaltic waves reach it and remain relazed until the last series of swallows is complete. the lower then contracts/closes after last swallow. here, its pressure rises well above resting for a few seconds.

72
Q

what are the mechanisms the prevent aspiration

A

NAME?

73
Q

which phases of swallowing are involuntary

A

pharyngeal (3) and esophageal (4)

74
Q

which phases of swallowing are voluntary

A

preparatory (1) and oral (2)….but normally subconcious

75
Q

what are the 3 components of the brainstem swallowing center

A
  1. sensory nuclei2. interneuronal network nuclei3. motor nuclei
76
Q

what are the 2 sensory nuclei of the brainstem involved in swallowing

A
  1. nucleus of the solitary tract2. trigeminal sensory nucleus**this is where the info comes in that says what/where/and what consistency the food is
77
Q

what are the 2 motor nuclei of the brainstem involved in swallowing

A
  1. nucleus ambiguous2. facial/trigeminal/hypoglossal nuclei (same as mastication)
78
Q

what is the interneuronal network of swallowing for

A

to mediate the interactions b/w the sensory and motor components of swallowing.

79
Q

where is the interneuronal network for swallowing located

A

distributed in the ventral and dorsal regions of the medulla

80
Q

what are the dorsal interneurons of the interneuronal network for swallowing for

A

initiation and programming/time of swallowing….can occur in the absence of sensory input. normally receives input from the nucleus of the solitary tract.

81
Q

what are the ventral interneurons of the interneuronal network for swallowing for

A

distribution of the excitation to the swallowing motor nuclei (nucleus ambiguous and CN V, VII, XII nuclei)

82
Q

cervical motor neurons are involved in what

A

control of swallowing…although not part of the brainstem swallowing center

83
Q

where does sensory info originate from during swallowing

A

pharynx, larynx, esophagus….afferents are sent to:-nucleus of solitary tract

84
Q

stimuli during swallowing can

A

activate afferent fibers to activate motor neurons or activate afferent fibers to INACTIVATE motor neurons (i.e the esophageal motor neurons are inactivated during oral and pharyngeal phases of swallowing)

85
Q

role of cortex in swallowing

A

can initiate swallowing if stimulated…but, not required for smooth, efficient swallowing

86
Q

describe the programming of infantile swallowing

A

NAME?

87
Q

describe the programming pattern of facultative muscles of infantile swallowing BEFORE tooth eruption

A

NAME?

88
Q

describe the programming pattern of facultative muscles of infantile swallowing AFTER tooth eruption

A

NAME?

89
Q

what is the consequence if infantile swallowing before tooth eruption is maintained even after tooth eruption

A

malocclusion…anterior open bite (from tongue thrust). controversial

90
Q

can infants swallow without interrupting respiration

A

no

91
Q

describe infant suckling mechanism

A
  • lips around nipple-mandible lowered-negative pressure in infants mouth-elevation of tongue and mandible =milk**so here, jaw opener and closer muscles are IMPORTANT
92
Q

is respiration maintained during infant suckling

A

yes!

93
Q

when does infant suckling programming begin

A

in utero

94
Q

describe the adult sucking mechanism

A

-driven by inspiratory muscles (think of sucking on a water bottle and get your heads out of the gutter)-lips form seal, but jaw opener/closer mm DON’T have a role in generating forces

95
Q

what is emesis

A

vomiting

96
Q

stimuli for vomiting

A
  1. physiological stimuli (**wine at psi thanksgiving dinner)2. disease-related stimuli
97
Q

what are the phases of vomiting (3)

A
  1. pre-ejection2. ejection3. post-ejection
98
Q

pre-ejection phase of vomiting

A

relaxation of proximal stomach (along with licking, salivation, tachycardia). duration can be min–>days

99
Q

ejection phase

A

2 phases (retching and ejection)

100
Q

describe the retching phase of the ejection phase of vomiting

A

glottis closed and inspiratory mm contract to…1. decrease thoracic pressure and 2. increase abdominal pressure3. upper esophageal sphincter relaxes during a “retch” but contracts b/w each one (1-2 sec apart)

101
Q

describe the expulsion phase of the ejection phase of vomiting

A

antrum of stomach relaxes, ab mm. contract, upper esophageal sphincter relaxes 1. thoracic pressure rises2. abdominal pressure rises 3. lower esophageal sphincter pressure is LOWvomit can travel from high pressure to low pressure…lovely.

102
Q

post-ejection phase of vomiting

A

cycle terminates of repeats

103
Q

what controls vomiting

A

medulla nuclei….info is brought in from (physiological or disease-related) stimuli via afferents to the sensory nuclei in the medulla

104
Q

bruxism causes

A

sleep phases….aka has a CNS origin… correlated with stress levels…excitation of jaw-closer motor neurons

105
Q

why is bruxism NOT due to occlusion interferences

A

tooth stimulation (caused by malocclusion) causes inhibition of jaw-closers

Physio (9 decks)

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