4.1.1 Communicable diseases, disease prevention and the immune system Flashcards

(48 cards)

1
Q

4 pathogens you need to know

A

-viruses
-bacteria
fungi
protoctists

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2
Q

What do bacteria do?

A

produce toxins that damage body cells

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3
Q

What do viruses do?

A

Use host cells to replicate before bursting out and destroying cells

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4
Q

What do protoctists do?

A

Take over the cells and break them open

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5
Q

What do fungi do?

A

Digest living cells to destroy them- some also produce toxins

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6
Q

How does climate increase the risk of communicable diseases?

A

-Warmer conditions allow mosquitoes to breed
- increased heat so more KE for chemical reactions + reproduction

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7
Q

How do social factors increase the risk of disease?

A

lack of healthcare etc

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8
Q

bacterial diseases to remember

A

meningitis, ring rot,, TB

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9
Q

Viral diseases to remember

A

HIV/AIDS, influenza, tobacco mosaic virus

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10
Q

Which protoctist diseases to remember

A

late tomato blight, malaria

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11
Q

which fungal diseases to remember

A

black sigatoka, athlete’s foot, ringworm

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12
Q

How is ring rot spread

A

direct contact

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13
Q

How is TB transmitted

A

airborne, contaminated food + drink

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14
Q

The effect of TB on the body

A

damage lungs + suppress the immune system

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15
Q

The effect of bacterial meningitis on the body

A

Damage the membranes of the brain and cause blood poisoning

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16
Q

The effect of HIV/AIDS on the body

A

gradually destroys the immune system

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17
Q

The effect of influenza on the body

A

Kills ciliated epithelial cells in the gas exchange system

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18
Q

The effect of athlete’s foot on the body

A

Causes cracking and scaling of skin between the toes

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19
Q

The effect of ringworm on the body

A

Causes a circular red rash

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20
Q

The effect of malaria on the body

A

Damages the red blood cells, liver, and brain

21
Q

Give 4 examples of vectors

A

wind, water, animals, humans

22
Q

Four factors that increase the risk of communicable disease in plants

A

Crop variety, Overcrowding, mineral nutrition, and Climate change

23
Q

Effect of ring rot on the plant

A

damages leaves, tuber and fruit

24
Q

The effect of TMV on plants

A

Damages the leaves, flowers and fruits

25
The effect of black sigatoka on plants
Attacks and destroys leaves, turning them black
26
Effect of late tomato blight on plants
Destroys leaves, fruit and tuber
27
3 physical plant defences
waxy cuticle, cell wall, production of callose
28
What are pathogens?
microbes that cause infections and disease
29
What is a bacteriophage?
Viruses that attack bacteria
30
What are parasites?
Organisms that benefit at the harm of the host
31
Explain waxy cuticles as a plant defense
Waxy cuticles - Leaves and stems are covered in a waxy cuticle which provides a physical barrier against pathogens.
32
Explain the cell wall as a plant defence
Cell walls - Plant cells are surrounded by cell walls that form a physical barrier against pathogens
33
Explain callose deposition as a plant defence
Production of callose - When pathogens attack plants, they produce a polysaccharide called callose. This is deposited between the cell wall and the cell-surface membrane, making it harder for pathogens to enter cells.
34
3 methods of direct transmission (animals)
- direct contact - innoculation (animal bites, sharing needles, cuts in skin) - ingestion (contaminated food/water)
35
2 mechanical responses to infection in plants
36
method of direct transmission (plants)
- direct contact
37
2 methods of indirect transmission
- contaminated soil - vectors
38
skin as a physical animal defence
contains skin flora which outcompete pathogens for space on skin
39
explain blood clotting as a skin defence in animals
form if the skin is cut
40
explain lysozymes as an animal defence
lysozymes are found in tears and saliva and contain hydrolytic enzymes which digest pathogens
41
explain inflammation as an animal defence (3)
- damaged cells trigger mast cells to release histamines and cytokines - histamines cause vasodilation and make capillaries more permeable - so more wbc's can be delivered to the site of damage
42
explain cytokines as an animal defence
attract phagocytes which can engulf and destroy pathogens
43
2 types of phagocytes
macrophages or neutrophils
44
phagocytosis (7)
- damaged cells/pathogens release cytokines which attract phagocytes to the site of infection - opsonin protein attaches to pathogens to mark them and make it easier for macrophages and neutrophils to engulf them - phagocytes have receptors which can attach onto chemicals on the surface of pathogens - pathogen engulfs the pathogen into a vesicle to create a phagosome - phagocytes have lysosomes in them with hydrolytic enzymes - lysosome fuses with phagosome 2 expose pathogen to lysozyme, lysozyme hydrolyses the pathogen+ and any soluble useful molecules are absorbed into cytoplasm of phagocyte - phagocytes present antigen of digested pathogen on their surface =apc's
45
where are the two types of lymphocytes produced and where do they mature
- bone marrow (produced) - b cells mature in bone marrow, T cells mature in thymus
46
cell mediated response (t-cells)
- receptors on t-cells bind 2 antigens on apc's - causes t-cells 2 divide rapidly by mitosis (clonal expansion) - examples of apc's = infected body cells, macrophage
47
4 physical plant defences
- cellulose cell walls - lignified layer - waxy upper cuticle - old vascular tissue blocked to prevent spread of pathogen
48
2 mechanical