Clinical Assessment of the Spine and Spinal Cord 2 Flashcards

1
Q

Additional Sensory Testing

A

two-point discrimination
Double simultaneous stimulation (looking for extinction)

Testing higher order (cortical) sensory processing:
Graphesthesia
Stereognosis- ability to recognize based on texture, size, temperature

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2
Q

Clinical Assessment of Motor Function

A
Inspection
Observation
Palpation
Individual muscle group testing
pronator drift
fine motor movements
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3
Q

The abdominal reflex (special)

A

elicited by drawing a line away from the umbilicus along the diagonals of the 4 abdominal quadrants. A normal reflex draws the umbilicus toward the direction of the line that is drawn.

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4
Q

cremasteric reflex (special)

A

elicited by drawing a line along the medial thigh and watching the movement of the scrotum in the male. A normal reflex results in elevation of the ipsilateral testis.

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5
Q

anal wink reflex (special)

A

elicited by gently stroking the perianal skin with a safety pin. It results in puckering of the rectal orifice owing to contraction of the corrugator-cutis-ani muscle.

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6
Q
  1. Complete Cord Transection
A

Tracts: All ascending sensory & descending motor/autonomic tracts.

Deficit: Sensory + motor levels below lesion; may also have root signs at site.

Note: Spinal shock followed by UMN signs.

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7
Q
  1. Central Lesions:
A

Tracts:Initially involve crossing ST

E.g.s: Syringomyelia (fluid-filled cavity in cord), ependymomas, cord contusion.

Deficit: PP/Temp loss at level of lesion, with
sparing of position sensation.

Note: Cape-like distribution if in C-spine.

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8
Q
  1. Posterior Column Syndrome:
A

Tracts: PC

E.g.s.Tabes dorsalis (form of neurosyphilis)

Deficit: Bilateral loss of position & vibration
sensation

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9
Q
  1. Combined Anterior Horn Cell-Pyramidal Tract Syndrome:
A

Tracts:CS and LMN cells in cord.

E.g.s:Amyotrophic lateral sclerosis (Lou Gehrig’s disease)

Deficit: Loss of bilateral strength.

Note: Fasciculations, atrophy, ↑ or ↓DTR,
normal sensation

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10
Q
  1. Brown-Sequard (Hemi-Section):
A

Tracts: Crossed ST + uncrossed PC + crossed CS

E.g.: Compression by herniated discs, tumor extramedullary abscess, etc.

Deficit: Below lesion, loss of
CL PP/Temp
IL Position
IL strength.

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11
Q
  1. Posterolateral Column Syndrome:
A

Tracts: PC + CS

E.g.: B12 deficiency (aka subacute combined degeneration)

Deficit: Bilateral loss of position & vibration, and strength.

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12
Q
  1. Anterior Horn Cell Syndrome:
A

Tracts: None - lower motor neuron (cell).
E.g.: Spinal muscular atrophy, polio virus

Deficit: Bilateral loss of strength.

Note: Fasciculations, ↓ tone + ↓ DTRs
with sparing of all sensory tracts and bladder functions.

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13
Q
  1. Anterior Spinal Artery Occlusion:
A

Tracts: ST + CS

E.g.: Anterior spinal artery occlusion.

Deficit: Bilateral loss of strength + PP/Temp,
with sparing position sense.

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14
Q
  1. Pyramidal Tract Syndrome:
A

Tracts: CS
E.g.: Primary lateral sclerosis.
Deficit: Bilateral UMN weakness with spastic gait, ↑ DTRs, but complete sparing of all sensory tracts and bladder function.

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15
Q
  1. Myelopathy with Radiculopathy:
A

Tracts: Any or all 3 tracts (esp. CS)

E.g.s:Cervical spinal stenosis, may be congenital or degenerative.

Deficit: Bilateral UMN syndrome with spastic gait, ↑ DTRs + IL or CL root signs + possible bladder dysfunction.

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16
Q

CAUDA EQUINA Syndrome

A
-EARLY root pain radiating to legs
Leg weakness & ↓ DTRs (LMN)
-Patchy, asymmetric "saddle"
Late bladder dysfunction
Late bowel & sexual dysfunction
17
Q

CONUS MEDULLARIS

A
Late pain in thighs & buttocks
Pelvic floor muscle weakness
SYMMETRIC "saddle" anesthesia, numb 
-EARLY bladder dysfunction
-EARLY bowel & sexual dysfunction
18
Q

Lhermitte’s Sign

A

Neck flexion results in “electric shock” sensation down the back and/or into arms.
Attributed to posterior column disease (MS, disc, B12 def, mass).

19
Q

Cervical Stenosis

A

Congenital or acquired narrowing of central cervical spinal canal.
Can result in UMN signs in legs +/- bladder dysfunction.

20
Q

Immediate muscle weakness and hypotonia, hyporeflexia or areflexia (“Spinal Shock”)
Followed by spasticity and HYPERreflexia in days to weeks (including extensor plantar response: Babinski’s sign)
SPASTIC PARESIS

A

UMN:

21
Q

Muscle weakness, hypotonia, hyporeflexia, areflexia are all immediate and long-lasting
FLACCID PARESIS
FASCICULATIONS
ATROPHY

A

LMN

22
Q

Detrusor (smooth) muscle, activated by the preganglionic parasympathetic outflow from the

A

S2-S4 segments.

23
Q

The involuntary (smooth) sphincter, controlled by sympathetic outflow from the ________ of the spinal cord

A

T10-L2 segments

24
Q

Skeletal muscle of the pelvic floor, innervated by alpha motor neurons from the ________

A

S2-S4 segments

25
Q

Late bladder/ sex problem

Early Pain

A

Cauda Equina

26
Q

Early Bladder

Late pain

A

Conus Medullaris

27
Q

Traumatic Spinal Cord Injury (SCI)

A

Spinal Shock following spinal cord injury is a specific term that relates to the loss of all neurological activity below the level of injury, including loss of motor, sensory, reflex, and autonomic function. Physiologic disruption of cord

28
Q

Neurogenic Shock:

A

low blood pressure, slowed heart rate that results from the disruption of the autonomic pathways within the spinal cord.

Hypotension results from decreased systemic vascular resistance from loss of sympathetic tone.

Bradycardia results from unopposed vagal tone.

29
Q

Spinal Shock

A

48- 72 hours transient disruption
return bulbo-cavernouses reflex shows ending
(anal contraction)

30
Q

Primary vs Secondary injury

A

primary: cannot repair

31
Q

Sequelae of SCI

A

bladder, DVT, pressure ulcer, pneumonia, gi hemorrhage, autonomic dysreg

32
Q

Lhermitte’s Sign:

A

Neck flexion results in “electric shock” sensation down the back and/or into arms. Attributed to posterior column disease (MS, disc, B12 def, mass).

33
Q

Radiculopathy

A

Sensory and/or motor dysfunction due to injury to a nerve root. Can be caused by herniated disk

s/s:
Lhermitets
nerve pain
reliefe: rest, nsaids