Arrhythmias Flashcards

1
Q

Right atrial enlargement

A

large P wave amplitudes

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2
Q

Left atrial enlargement

A

large P wave durations, notching of P wave often seen due to out-of-sync conduction of right and left atria

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3
Q

2 Main causes of QRS Issues

A
  1. excessive generation of electrical potential in a particular direction, 2. more time required for depolarization wave to travel through muscle (ventricular enlargement)
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4
Q

Right ventricular enlargement

A

large S waves in I, II, aVF

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5
Q

Left ventricular enlargement

A

not always observed because left ventricle is already larger than right ventricle

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6
Q

Intraventricular conduction defects

A

impulses aren’t as fast, QRS duration will be longer

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7
Q

Left bundle branch block

A

wide and positive deflections in I, II, III, aVF, and inverted aVR and aVL, large R waves

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8
Q

Right bundle branch block

A

right axis deviation usually present, large S waves in I, II, III, aVF

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9
Q

Causes of arrhythmias

A
  1. abnormal rhythmicity of the pacemaker
  2. shift of pacemaker activity
  3. conduction blocks
  4. abnormal pathways of impulse transmission
  5. spontaneous generation of abnormal impulses
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10
Q

Sinus arrhythmia

A

impulses originate at the SA node at a varying rate, faster then slower, primary cause is respiration

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11
Q

Wandering atrial pacemaker

A

impulses originate from varying points in atria, P wave changes configuration, originate from various parts of atria, QRS still normal

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12
Q

Sinus arrest

A

SA node stops firing, escape beat

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13
Q

First degree AV block

A

not dangerous but will progress, PR interval is very long

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14
Q

Second degree AV block

A

Mobitz Type I- progressive lengthening of PR interval before dropped beat, block gets thicker, so PR interval length increases
Mobitz Type II- sudden QRS dropped without prior PR lengthening, atrial rate is greater than the ventricular rate (more P waves)

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15
Q

Third degree AV block

A

QRS rate is greater than the P rate (slower), SA node paces atria, Purkinje fibesr pace ventricular (separate rates), P and T waves are biphasic, ** no relationship between the two rates

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16
Q

Atrial flutter

A

atria contracting quickly

17
Q

Atrial fibrillation

A

no distinct P waves, irregularly spaced QRS complexes, no coordinated direction, AV node drives, no the SA node

18
Q

Ventricular paroxysmal tachycardia

A

runs of 3 or more VPCs in sequence

19
Q

Ventricular bigeminy

A

normal—->premature—->normal—->premature

20
Q

Ventricular fibrillation

A

cardiac output is 0, no QRS waves, decrease in velocity, shortening refractory period, most vulnerable during ventricular repolarization

21
Q

Supraventricular premature contraction

A

premature beats, P occurs before T wave

22
Q

Supraventricular paroxysmal tachycardia

A

clusters of premature contractions

23
Q

Ventricular premature contraction

A

usually no P wave, QRS wide and bizarre, morphology of QRS will vary with each focus