Duplex/Color flow venous imaging Flashcards

1
Q

Sources for false positive?

A
  • extrinsic compression: tumors, ascites, and pregnancy
  • peripheral arterial disease: decreased venous filling
  • chronic obstructive pulmonary disease: elevated central venous P
  • improper Doppler angle or probe pressure
  • superior vena cava syndrome
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2
Q

Sources of false negative studies?

A

prox obstruction

technically limited studies

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3
Q

May be difficult to thoroughly evaluate what peripheral veins in the lower extremity secondary to vessel size, depth, and course?

A

infra-popliteal

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4
Q

Difficult to thoroughly evaluate what peripheral upper extremity veins secondary to bony structures?

A

subclavian and brachiocephalic/innominate

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5
Q

What is the patient positioning for upper extremity peripheral vein?

A

supine or low fowlers position

arm in pledge position

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6
Q

What settings can be adjusted to maximize color filling and flow patterns?

A
  • adjust color scale to detect slower velocities
  • change wall filters
  • increase color gains
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7
Q

What is another word for compressibility?

A

coaptation

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8
Q

What are the possible venous flow patterns?

A

spontaneous, phasic, augment with distal compression, augment with proximal release

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9
Q

Signal immediately heard at all sites except what vein?

A

posterior tibial

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10
Q

what is the lower extremity phasicity?

A

increase with expiration and decreases with inspiration

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11
Q

what is the upper extremity phasicity?

A

decreases with expiration and increases with inspiration

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12
Q

pulsatile venous flow pattern evident with fluid overload or what?

A

congestive heart failure

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13
Q

What is a technique to collapse the subclavian and innominate?

A

a quick breath through pursed lips should collapse vein

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14
Q

Flow reversal usually in response to a valsalva maneuver or during prox manual compression indicates what?

A

venous refulx

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15
Q

Reflux is identified when reversed flow last more than how long?

A

1 sec

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16
Q

Deep inspiration causes the abdominal and pelvic veins what?

A

dilate

17
Q

Phasic, bi-directional/pulsatile Doppler signals are in what veins?

A

IVC, renal, and hepatic

18
Q

minimally phasic, continuous Doppler signals are in what veins?

A

portal, splenic, and mesenteric

19
Q

There is minimal flow fluctuation in the portal vein and flow is variable in the hepatic veins when?

A

during inspiration

20
Q

If flow is not spontaneous at the CFV, FV, and/or Pop V what could be the cause?

A

obstruction distal to or at the site

21
Q

If flow is not phasic, but rather continuous, what could be the result?

A

a proximal obstruction

22
Q

Where might an obstruction be if the is no augmentation with distal compression seen?

A

obstruction may be between where you are compressing and where you are listening, or slightly more proximal

23
Q

If there is no augmentation with proximal release, where might an obstruction be?

A

proximal

24
Q

If flow increases during proximal compression, what does that signify?

A

venous reflux

25
Q

A compressible vessel with evidence of rauleau formation on B-mode could be what?

A

normal or suggest proximal obstruction

26
Q

What is the appearance of chronic clot?

A
  • highly echogenic
  • visible collateralization or recanalization may be evident
  • vessel not dilated; may retract over time
27
Q

Flow characteristics with chronic clot?

A
  • abnormal Doppler venous signals may be evident, such as continuous, decreased phasicity, or no augmentation
  • venous reflux lasting >1 sec
28
Q

Where is an IVC interruption device usually placed?

A

below renal veins and may appear as bright echogenic lines

29
Q

With systemic venous hypertension what is evident?

A

persistent dilated vessels

30
Q

Increased portal venous pressure can result in what flow alterations?

A
  • reversed flow in portal vein (hepato-fugal)

- collateral development

31
Q

What is Budd-Chiari Syndrome?

A
  • results from hepatic occlusion

- primary site of obstruction may be hepatic vein, sinusoids, or IVC

32
Q

What are the clinical findings of Budd-Chiari Syndrome?

A

hepatomegaly, abdominal pain, sudden onset of ascites