emergencies Flashcards

1
Q

What is Subdural hematoma (SDH)

A

caused by bleeding in the potential space between the dura and the arachnoid membranes.
-r/t tearing of the bridging veins that drain from the surface of the brain to the dural sinuses, but arterial rupture accounts for approximately 20 to 30 percent of cases.

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2
Q

What is the most common cause of SDH in middle aged men

A

Head trauma in motor vehicle accidents, falls, and assaults

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3
Q

high risk factors for SDH

A
  • old pt with history of chronic alcohol abuse, and those with previous traumatic brain injury
  • **overall incidence of SDH is highest among older adults
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4
Q

common mental status is 50% of cases:

A

coma

-38% = transient “lucid interval” that is followed by a progressive neurologic decline to coma.

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5
Q

s/s of chronic SDH

A

insidious onset of headaches, light-headedness, cognitive impairment, apathy, somnolence, and occasionally seizures.

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6
Q

radiology for suspected SDH

A

CT of the head

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7
Q

pt c/o sudden, severe headache & described it as “worst headache of my life”

A
  • aneurysmal subarachnoid hemorrhage (SAH)

- r/t hemorrhagic stroke

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8
Q

associated s/s with SAH

A

brief loss of consciousness, seizures, nausea or vomiting or meningismus

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9
Q

if SAH suspected, np should order ____to dx

A

Noncontrast head CT

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10
Q

if non contract CT is negative for SAH, what needs to be done is there is a high suspicion

A

Lumbar puncture is mandatory if there is a strong suspicion of SAH despite a normal head CT.

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11
Q

what are the Complications of SAH

A

rebleeding, vasospasm and delayed cerebral ischemia, hydrocephalus, increased intracranial pressure, seizures, hyponatremia, cardiac abnormalities, and hypothalamic dysfunction.

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12
Q

what is Anaphylaxis

A

an acute, potentially lethal, multisystem syndrome resulting from the sudden release of mast cell- and basophil-derived mediators into the circulation

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13
Q

how is anaphylaxis classified?

A

“immunologic” or “nonimmunologic.”

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14
Q

what is Immunologic anaphylaxis

A

it includes both immunoglobulin E (IgE)-mediated and immunoglobulin G (IgG)-mediated reactions (which have not been identified in humans), as well as immune complex/complement-mediated mechanisms.

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15
Q

what is Nonimmunologic anaphylaxis

A

caused by agents or events that induce sudden, massive mast cell or basophil degranulation, without the involvement of antibodies.

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16
Q

How to confirm Anaphylaxis and determining the cause(s)

A

detailed hx!!! - r/o asthma, COPD. Meds may exacerbation- i.e. Ethanol, NSAIDs, and opiates; beta blockers, ace inhibitors, Beta-adrenergic blockers administered orally or topically
-refer to allergy/immunology specialist

17
Q

major causes of community-acquired bacterial meningitis in adults in developed countries

A

Streptococcus pneumoniae, Neisseria meningitidis, and, primarily in patients over age 50 to 60 years or those who have deficiencies in cell-mediated immunity= Listeria monocytogenes.

18
Q

major causes of healthcare-associated bacterial meningitis

A

staphylococci and aerobic gram-negative bacilli

19
Q

how to differentiate bacterial from viral meningitis

A

conduct CSF analysis including Gram stain and culture

20
Q

what is the Kernig sign

A

used to r/o meningitis
-Place patient supine with hip flexed at 90 degrees. Attempt to extend the leg at the knee. (+) = resistance to extension at the knee to >135 degrees or pain in the lower back or posterior thigh

21
Q

what is the Brudzinski sign

A

used to r/o meningitis
-Place patient in the supine position and passively flex the head toward the chest. (+) = when there is flexion of the knees and hips of the patient.

22
Q

what is Jolt accentuation of headache

A

Patient rotates his/her head horizontally two to three times per second. (+) = exacerbation of his/her headache with this maneuver.

23
Q

what is a hypertensive crisis?

A
  • systolic pressure ≥180 and/or diastolic pressure ≥120 mmHg

- acute rise in blood pressure over a previously normal baseline, even if the presenting pressure is <180/120 mmHg

24
Q

tx of hypertensive crisis

A

DONT lower the blood pressure too quickly or too much, as ischemic damage can occur in vascular beds that have grown accustomed to the higher level of blood pressure (ie, autoregulation).
*****For most hypertensive emergencies, mean arterial pressure should be reduced by about 10 to 20 percent in the first hour and then gradually during the next 23 hours so that the final pressure is reduced by approximately 25 percent compared with baseline.

25
Q

What are the major exceptions to modest and gradual blood pressure lowering over the first 24 hours?

A
  • The acute phase of an ischemic stroke – The blood pressure is usually not lowered unless it is ≥185/110 mmHg in patients who are candidates for reperfusion therapy (table 1) or ≥220/120 mmHg in patients who are not candidates for reperfusion therapy.
  • Acute aortic dissection – The systolic blood pressure is rapidly lowered to a target of 100 to 120 mmHg (to be attained in 20 minutes).
  • Spontaneous hemorrhagic stroke – The systolic blood pressure can be rapidly reduced if no contraindications exist.
26
Q

T/F: The most important aspect of care for the patient with a hypertensive emergency is assuring that high-quality outpatient follow-up is available.

A

TRUE