renal 5

Question 1

tf most of k is in interceullular fluid

Answer 1

T

Question 2

t/f 2/3 of fluid in body is ecf

Answer 2

f it is icf(28l to 14 l)

Question 3

plasma k when lower than 3.5 meq/l

Answer 3

hypokalemia

Question 4

normal plasma conc

Answer 4

3.5-5 meq/l

Question 5

what is the effect on the right

Answer 5

low k outside

hyperpolarized and hard to get to threshold

Question 6

symptoms of the curve on right

Answer 6

increased K on outside

very excitable

Question 7

det resting potential

Answer 7

k out

Question 8

after a meal plsma k increases stim k uptake thru

Answer 8

na/ k atpase

Question 9

insuline and epeniephrine

Answer 9

inc k uptake by inc. na k atpase turnover rate

Question 10

hyperkalemia

Answer 10

caused by insulin deficiency and beta antagonist

Question 11

epinephrine bind to – to inc na/k atpase turnover rate and k is brought into the cell

Answer 11

beta receptor

Question 12

treatment for hyperkalema

Answer 12

b agonist and insulin

Question 13

2 factors inc na/k atpase pump

so that k can be absorbed in tissue cells

Answer 13

plasma k

insulin and epinephrine

Question 14

tf after plasma K inc after a meal it immediately get translocated in a tissue cell and then immediately the plasma k level goes down to normal

Answer 14

F the plasma k level stays elvated to signal kidney to get rid of K by having more pumps activated

Question 15

tf after k accumulates in tisses cells after intestinal absorption there is fast renal excretion

Answer 15

F

renal excretion is very slow (little by little goes ijnto urine)

Question 16

plasma k

and aldosterone

Answer 16

signal excretion k in urine

Question 17

tf after K is absorbed in the intestines it immediately stays in the ECF

Answer 17

F

ECF doesnt want to saturate and it get transferred to Tissues