Glaucoma Flashcards

1
Q

The apposition of iris to the trabecular meshwork, which results in increased intraocular pressure:

A

Angle-Closure Glaucoma

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2
Q

Suddenly symptoms of blurred vision, seeing haloes around lights, red eye, pain, headache, nausea and vomiting:

A

Acute Angle-Closure Glaucoma presentation

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3
Q

The sudden and severe IOP elevation can quickly damage the optic nerve, resulting in:

A

Acute Angle-Closure Glaucoma

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4
Q

Is a true ophthalmic emergency, and a delay in treatment can result in blindness:

A

Acute Angle-Closure Glaucoma

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5
Q

When the pupil is mid-dilated, the distance between the iris and the lens is the shortest and can come into contact in individuals at risk for angle closure. When this occurs, aqueous humor cannot flow through the pupil into the anterior chamber, pushing the iris against the trabecular meshwork, so the aqueous humor cannot flow out of the eye, increasing IOP:

A

Pathophysiology

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6
Q

The normal IOP is:

A

10-21mmHg

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7
Q

In AACG, the IOP typically exceeds:

A

40mmHg

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8
Q

Pupillary block, Plateau iris, Use of medications, Increased iris thickness, Increased iris volume with dilation, Hyperopia, and Increased lens thickness in phacomorphic angle closure:

A

Mechanisms that can contribute to primary angle closure

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9
Q

AACG is more common in:

A

+40 years, Women, Asian persons, Persons with hyperopia, +Family history and Who have had AACG in one eye

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10
Q

Is a medical emergency that needs to be treated immediately:

A

AACG

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11
Q

Even with immediate treatment, AAC may result in:

A

Vision loss

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12
Q

Haloes and blurry vision result from

A

Corneal edema

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13
Q

The attack may have been precipitated by pupillary dilation, which may result from activities such as:

A

Movie theater, Taking medications that contain antihistamine or Using dilating eye drops

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14
Q

occluded anterior chamber angle in the affected eye and predisposing angle configuration (narrow occludable angle) in the contralateral unaffected eye (ophthalmic exam):

A

Gonioscopic visualization

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15
Q

Tonometry demonstrates:

A

An elevated IOP

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16
Q

Conjunctival injection, Fixed or sluggish and mid-dilated pupil, Shallow anterior chamber, Corneal epithelial edema and Bullae, and Flare (ophthalmic exam):

A

Slit-lamp exam

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17
Q

May reveal a swollen optic disc in an acute attack:

A

Ophthalmoscopy

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18
Q

May reveal a excavation if episodes have been chronic or repetitive:

A

Ophthalmoscopy

19
Q

Vesicles on the anterior subcapsular lens

A

Glaucoma flecks

20
Q

the most common cause of AACG:

A

Pupillary block

21
Q

Persons with susceptible anatomy have a narrow occludable angle, usually of less than:

A

20°

22
Q

HCTZ, Sulfamethoxazole and Topiramate:

A

Medications that can cause AACG

23
Q

“Individuals with hyperopia have ______ eyes, with shorter diameter, so the intraocular structures are spaced closer together”:

A

Smaller

24
Q

“Lens volume _________ with age and can decrease the amount of space in the anterior chamber”:

A

Increases

25
Q

Ultrasound biomicroscopy and Anterior segment optical coherence tomography:

A

Imaging studies

26
Q

The definitive treatment of AACG is:

A

Surgical

27
Q

Lowering the IOP minimizes damage to the:

A

Optic nerve

28
Q

If the IOP cannot be lowered sufficiently with meds:

A

Anterior Chamber Paracentesis

29
Q

Are used to help clear up the cornea, to reduce intraocular inflammation and to decrease iris edema:

A

Mediations for eye pressure and inflammation

30
Q

“In some cases, a _____ agent is used to prevent AAC recurrence”:

A

Miotic

31
Q

The treatment of choice for pupillary-block:

A

Laser iridotomy

32
Q

“If the cornea is extremely cloudy or the iris is too thick and an opening cannot be created using laser, _____________________ can be performed”:

A

Incisional peripheral iridectomy

33
Q

“If the patient has both a narrow occludable angle and a visually significant cataract (blurry vision due to cataract), ____________ is the treatment of choice”

A

Cataract surgery

34
Q

Meds:

A

Alpha-adrenergic agonists, Beta-blockers, Miotic agents, Carbonic anhydrase inhibitors and Prostaglandin

35
Q

Brimonidine and Apraclonidine

A

Alpha-adrenergic agonists

36
Q

Levobunolol, Betaxolol and Timolol:

A

Beta-blockers

37
Q

Pilocarpine:

A

Miotic agents

38
Q

Acetazolamide, Methazolamide, Dorzolamide and Brinzolamide:

A

Carbonic anhydrase inhibitors

39
Q

Latanoprost, Travoprost and Bimatoprost:

A

Prostaglandin

40
Q

Acute Glaucoma Treatment (First 60min):

A

Pilocarpina 2%, Timolol, Brimonidina, Acetazolamida oral, Corticoide topico ou Indentacao Corneana

41
Q

Se após tratamento a PIO diminui, pupila se contrai e a córnea fica clara:

A

Crise Bloqueada -> Iridotomia com Laser

42
Q

Após Iridotomia com Laser:

A

Timolol, Brimonidina e Corticóide tópico

43
Q

Se após tratamento permanecer com a crise não bloqueada:

A

Manitol Venoso —> Cirurgia